ライフサイエンスコーパス: mobility shift assay

1 atio was 50 as determined by electrophoretic mobility shift assay.

2 e-hybrid assays in yeast and electrophoretic mobility shift assay.

3 h RvE1 treatment as shown in electrophoresis mobility shift assay.

4 ter regions was confirmed by electrophoretic mobility shift assay.

5 ding SF1, as measured by the electrophoresis mobility shift assay.

6 higher than for dsDNA in an electrophoretic mobility shift assay.

7 e-specific RNA binding in an electrophoretic mobility shift assay.

8 ExsA was demonstrated via an electrophoretic mobility shift assay.

9 munoprecipitation (ChIP) and electrophoretic mobility shift assay.

10 er region was observed in an electrophoretic mobility shift assay.

11 ed elongation complexes as measured in a gel mobility shift assay.

12 bility to bind to the tcpA promoter in a gel mobility shift assay.

13 nd protein was determined by electrophoretic mobility shift assay.

14 ion of GATA3 was analysed by electrophoretic mobility shift assay.

15 ated G or A alleles in a gel electrophoretic mobility shift assay.

16 ble complex with Bdnf RNA in electrophoretic mobility shift assays.

17 moter DNA was analyzed using electrophoretic mobility shift assays.

18 binding was investigated by electrophoretic mobility shift assays.

19 multiple shifted species in electrophoretic mobility shift assays.

20 and PntR were identified by electrophoretic mobility shift assays.

21 transcriptase (qRT)-PCR, and electrophoretic mobility shift assays.

22 orated by immunoblotting and electrophoretic mobility shift assays.

23 r the biotin operator DNA in electrophoretic mobility shift assays.

24 his location by ChIP-seq and electrophoretic mobility shift assays.

25 the CbbR-cbbLS promoter interactions in gel mobility shift assays.

26 d using light scattering and electrophoretic mobility shift assays.

27 nd to each of these sites in electrophoretic mobility shift assays.

28 the virB promoter region in electrophoretic mobility shift assays.

29 promoters in vitro by use of electrophoretic mobility shift assays.

30 egration sequences (attB) in electrophoretic mobility shift assays.

31 tometry, transactivation and electrophoretic mobility shift assays.

32 in vitro as demonstrated by electrophoretic mobility shift assays.

33 n polymerase chain reaction, immunoblot, and mobility shift assays.

34 Electrophoretic mobility shift assay analysis indicates that the SNP dif

35 Electrophoretic mobility shift assay analysis with antibodies against c-

36 ugh DNase I footprinting and electrophoretic mobility shift assay analysis.

37 F65, as determined by an RNA electrophoretic mobility shift assay and a chromatin immunoprecipitation

38 during di-snRNP assembly by electrophoretic mobility shift assay and accompanying conformational cha

39 Electrophoretic mobility shift assay and chromatin immunoprecipitation a

40 Electrophoretic mobility shift assay and chromatin immunoprecipitation a

41 We then used an electrophoretic mobility shift assay and chromatin immunoprecipitation a

42 vation studies combined with electrophoretic mobility shift assay and chromatin immunoprecipitation a

43 Promoter, electrophoretic mobility shift assay and chromatin immunoprecipitation a

44 Electrophoretic mobility shift assay and chromatin immunoprecipitation a

45 By electrophoretic mobility shift assay and chromatin immunoprecipitation c

46 Using mobility shift assay and chromatin immunoprecipitation,

47 C1 promoter was confirmed by electrophoretic mobility shift assay and chromatin immunoprecipitation.

48 promoter were identified by electrophoretic mobility shift assay and DNase I footprinting.

49 h DNA-binding, which we confirmed by electro-mobility shift assay and isothermal titration calorimetr

50 Electrophoretic mobility shift assay and NMR revealed that the KH domain

51 NP function was evaluated by electrophoretic mobility shift assay and promoter luciferase assay.

52 The implementation of electrophorethic mobility shift assay and pull-down experiments coupled w

53 ctivate ODO1, as revealed by electrophoretic mobility shift assay and yeast one-hybrid analysis, plac

54 Here we demonstrate through mobility shift assays and calorimetric measurements that

55 Electrophoretic mobility shift assays and chromatin immunoprecipitation

56 ntified it using competitive electrophoretic mobility shift assays and chromatin immunoprecipitation.

57 biquitin conjugation include electrophoretic mobility shift assays and detection of epitope-tagged or

58 Results from electrophoretic mobility shift assays and DNA pulldown assays with ChIP-

59 te for BldD, as was shown by electrophoretic mobility shift assays and DNase I footprinting analysis.

60 Moreover, electrophoretic mobility shift assays and DNase I footprinting revealed

61 t of this competition model, electrophoretic mobility shift assays and DNase I footprinting showed th

62 within the cbbLS promoter by the use of gel mobility shift assays and DNase I footprinting.

63 l nuclear protein binding in electrophoretic mobility shift assays and drives increased expression of

64 Using electrophoretic mobility shift assays and fluorescence anisotropy, we re

65 The results of electrophoretic mobility shift assays and quantitative analysis of prgQ

66 y VqsM has been confirmed by electrophoretic mobility shift assays and quantitative real-time polymer

67 ognition, using quantitative electrophoretic mobility shift assays and reporter gene activation assay

68 Based on electrophoretic mobility shift assays and RNA footprinting, the H. pylor

69 sequence elements on dimer formation via gel mobility shift assays and size exclusion chromatography.

70 Gel mobility shift assays and surface plasmon resonance anal

71 We validated regulatory DNA sequences by mobility shift assays and with luciferase reporters usin

72 Using gel-mobility-shift assays and surface plasmon resonance spec

73 logs in terms of DNA binding (as revealed by mobility shift assays) and multimerization (as revealed

74 romatin immunoprecipitation, electrophoretic mobility shift assay, and both knockdown and overexpress

75 Through promoter mapping, electrophoretic mobility shift assay, and chromatin immunoprecipitation

76 Bioinformatic, electrophoretic mobility shift assay, and gene expression analysis found

77 on using UV melting studies, electrophoretic mobility shift assay, and RNase A footprinting.

78 promoter was demonstrated by electrophoretic mobility shift assay, and the MisR binding sequences wer

79 g/overexpression approaches, electrophoretic mobility shift assays, and ChIP revealed that DDR2 acts

80 luciferase reporter assays, electrophoretic mobility shift assays, and chromatin immunoprecipitation

81 terial one-hybrid screening, electrophoretic mobility shift assays, and coimmunoprecipitation experim

82 d by using a promoter truncation series, gel mobility shift assays, and DNase I footprinting.

83 h in vitro, as determined by electrophoretic mobility shift assays, and in cells, as determined by Ch

84 validated, both in vitro, by electrophoretic mobility shift assays, and in vivo, by chromatin immunop

85 nding site was defined using electrophoretic mobility shift assays, and its importance was investigat

86 romatin immunoprecipitation, electrophoretic mobility shift assays, and luciferase reporter assays we

87 fluorescence anisotropy and electrophoretic mobility shift assays, and our NMR structure of phosphom

88 sing saturation mutagenesis, electrophoretic mobility shift assays, and RNA-sequencing profiling of c

89 romatin immunoprecipitation, electrophoretic mobility shift assays, and VE-cadherin-luciferase report

90 nalyzed by western blotting, electrophoretic mobility-shift assay, and immunohistochemistry in liver

91 sensitive acetyl transferase electrophoretic mobility shift assay applicable both for kinetic analysi

92 Electrophoretic mobility shift assays are widely used in gel electrophor

93 Electrophoretic mobility shift assay as well as chromatin immunoprecipit

94 Moreover, as determined by electrophoretic mobility shift assays, BioR binds the predicted operator

95 Electrokinetic preconcentration coupled with mobility shift assays can give rise to very high detecti

96 Using EMSA (electrophoretic mobility shift assay), ChIP (chromatin immunoprecipitati

97 e reporter luciferase assay, electrophoretic mobility shift assay, chromatin immunoprecipitation assa

98 Co-transfection analyses, electrophoretic mobility shift assays, chromatin immunoprecipitation, an

99 atin immunoprecipitation and electrophoretic mobility shift assays confirm are bound by Hand2 and Pho

100 Electrophoretic mobility shift assay confirmed that STAT3 bound to the m

101 Furthermore, electrophoretic mobility shift assays confirmed specific binding of Fur

102 Gel mobility shift assays confirmed that CcrR directly binds

103 atin immunoprecipitation and electrophoretic mobility shift assay data revealed that FOXO 3a regulate

104 Electrophoretic mobility shift assays demonstrate that cFos distinctly i

105 ents, mutation analyses, and electrophoretic mobility shift assays demonstrate that the sequence CGAC

106 Electrophoretic mobility shift assay demonstrated that Foxo1 suppressed

107 ent with these observations, electrophoretic mobility shift assay demonstrated that phenylmethimazole

108 Transactivation analysis and electrophoretic mobility shift assay demonstrated that PtrWNDs and EgWND

109 RNA electrophoresis mobility shift assays demonstrated a direct interaction

110 Electrophoretic mobility shift assays demonstrated decreased binding of

111 Electrophoretic mobility shift assays demonstrated increased NFAT-DNA bi

112 Electrophoretic mobility shift assays demonstrated that CcpE binds to th

113 Electrophoretic mobility shift assays demonstrated that FadR binds to th

114 Electrophoretic mobility shift assays demonstrated that hFXR-K210R and -

115 Electrophoretic mobility shift assays demonstrated that IolR recognized

116 Electrophoretic mobility shift assays demonstrated that nuclear extracts

117 Electrophoretic mobility shift assays demonstrated that the CcpA DNA bin

118 Electrophoretic mobility shift assays demonstrated that the four predict

119 Electrophoretic mobility shift assays demonstrated that VtlR binds direc

120 In vitro electrophoretic mobility shift assays demonstrated the potential functio

121 Gel mobility-shift assays demonstrated that assembly of the

122 Employing electrophoretic mobility-shift assays, DNA footprinting, and in silico a

123 Electrophoretic mobility shift assay documented the activation of NF-kap

124 Electrophoretic mobility shift assay (EMSA) analysis disclosed that SarR

125 ese values were confirmed by electrophoretic mobility shift assay (EMSA) analysis, which also suggest

126 ciferase reporter assays and electrophoretic mobility shift assay (EMSA) analysis.

127 Here, we use electrophoretic mobility shift assay (EMSA) and atomic force microscopy

128 Using electrophoretic mobility shift assay (EMSA) and chromatin immunoprecipit

129 ing site as determined by an electrophoretic mobility shift assay (EMSA) and DNase I protection.

130 Using electrophoretic mobility shift assay (EMSA) and isothermal titration cal

131 Electrophoretic mobility shift assay (EMSA) experiments using an IE62 fr

132 for our in vivo studies and electrophoretic mobility shift assay (EMSA) for our in vitro studies, we

133 We performed electrophoretic mobility shift assay (EMSA) using wild-type sequence der

134 Second, electrophoretic mobility shift assay (EMSA) was used to demonstrate the

135 Electrophoretic mobility shift assay (EMSA) was used to identify SNPs th

136 g enzyme-1 (BACE1) genes for electrophoretic mobility shift assay (EMSA) with different fragments of

137 gion were demonstrated in an electrophoretic mobility shift assay (EMSA), and a Mur binding site was

138 immunoprecipitation (ChIP), electrophoretic mobility shift assay (EMSA), and luciferase assays revea

139 the Bdnf gene, we performed electrophoretic mobility shift assay (EMSA), chromatin immunoprecipitati

140 Using electrophoretic mobility shift assay (EMSA), purified LsrR and CRP prote

141 ing, as assessed in vitro by electrophoretic mobility shift assay (EMSA), revealed that a 14-bp seque

142 al repeat DNA as assessed by electrophoretic mobility shift assay (EMSA), the mutations did not disru

143 nd ICB2 was studied using an electrophoretic mobility shift assay (EMSA).

144 promoter was demonstrated by electrophoretic mobility shift assay (EMSA).

145 le Thermophoresis (MST), and Electrophoretic Mobility Shift Assay (EMSA).

146 Electrophoretic mobility shift assays (EMSA) and chromatin immunoprecipi

147 munoprecipitation (ChIP) and electrophoretic mobility shift assays (EMSA) experiments showed that ER8

148 Luciferase reporter and electrophoretic mobility shift assays (EMSA) showed that POU3F2 induces

149 FINDINGS: Luciferase assays, electrophoretic mobility shift assays (EMSA), and RNA expression by RT-P

150 Using recombinant ArsR in electrophoretic mobility shift assays (EMSA), we localized binding to a

151 bind TR DNA, as assessed by electrophoretic mobility shift assays (EMSA).

152 leotide library by employing electrophoretic mobility shift assays (EMSA).

153 o bound TR DNA as assayed by electrophoretic mobility shift assays (EMSA).

154 A binding with the use of an electrophoretic mobility-shift assay (EMSA) and confocal microscopy.

155 Additionally, electrophoretic mobility shift assays (EMSAs) and DNase I footprinting w

156 Electrophoretic mobility shift assays (EMSAs) confirmed binding of Rep t

157 ility and efflux assays, and electrophoretic mobility shift assays (EMSAs) confirmed compromised affi

158 Importantly, electrophoretic mobility shift assays (EMSAs) determined that a recombin

159 platform for high-throughput electrophoretic mobility shift assays (EMSAs) for identification and cha

160 Electrophoretic mobility shift assays (EMSAs) identified positive DNA-pr

161 tigen, we introduce affinity electrophoretic mobility shift assays (EMSAs) in a high-throughput forma

162 Like the PTE, electrophoretic mobility shift assays (EMSAs) indicated that eukaryotic

163 Electrophoretic mobility shift assays (EMSAs) revealed that the overexpr

164 Western blot analysis and electrophoretic mobility shift assays (EMSAs) showed that TC-EC contact

165 formed EA-binding assays and electrophoretic mobility shift assays (EMSAs) to elucidate a mechanism f

166 Electrophoretic mobility shift assays (EMSAs) were performed to investig

167 Electrophoretic mobility shift assays (EMSAs) were used to prove that H.

168 ctor binding was analyzed by electrophoretic mobility shift assays (EMSAs) with Jurkat T-cell nuclear

169 nmoR, which was confirmed by electrophoretic mobility shift assays (EMSAs) with the purified NmoR pro

170 protein purification steps, electrophoretic mobility shift assays (EMSAs), and mass spectrometry ana

171 e fusion proteins, and using electrophoretic mobility shift assays (EMSAs), the IHFalpha-IHFbeta prot

172 s, including SW blotting and electrophoretic mobility shift assays (EMSAs).

173 The microfluidic mobility shift assay establishes a scalable format for t

174 A combination of electrophoretic mobility shift assay experiments and bioinformatic analy

175 Electrophoretic mobility shift assay experiments demonstrated RTCS bindi

176 th infected cell extracts in electrophoretic mobility shift assay experiments, (iv) supershift assays

177 n the absence of PCNA, using electrophoretic mobility shift assays, fluorescence intensity changes an

178 Luciferase reporter and electrophoretic-mobility shift assay for the FUT6 variant rs78060698 usi

179 Gel mobility shift assays further demonstrated that DDX1 for

180 Electrophoretic mobility shift assay gel shift patterns suggested that a

181 binant Exo1 and nuclease and electrophoretic mobility shift assays, here we determined that DNA hairp

182 es, mutagenesis studies, and electrophoretic mobility shift assays identified a PPARalpha response el

183 dow for functional impact on electrophoretic mobility shift assay identifies rs806371 as a novel regu

184 8378; multiplexed competitor electrophoretic mobility shift assays implicated FOXA as the protein.

185 Using electrophoretic mobility shift assays in HeLa cell extracts, we show tha

186 regulation of Shp by Vdr using reporter and mobility shift assays in transfected human embryonic kid

187 Electrophoretic mobility shift assay indicated that P-RhpR has a higher

188 Electrophoretic mobility shift assays indicated that BpaB also binds wit

189 However, in contrast to E. coli, gel mobility shift assays indicated that neither E. coli nor

190 Importantly, electrophoretic mobility shift assays indicated that purified CspE, but

191 Electrophoretic mobility shift assays indicated that specific nuclear pr

192 Furthermore, electrophoretic mobility shift assays indicated the presence of an activ

193 ssays, bind KLF16 in vivo In electrophoretic mobility shift assays, KLF16 binds specifically to a sin

194 etion assay of IL-17, and by electrophoretic mobility shift assay of activating protein-1 (AP-1).

195 reporter into cell lines or electrophoretic mobility shift assay of lysate.

196 Electrophoretic mobility shift assays on the shortlist detected allele-s

197 with mutational analysis and electrophoretic mobility shift assays, our results provide insights into

198 In mobility shift assays, PHR1 and its close homologue PHL1

199 dies utilizing a pulse-chase electrophoretic mobility shift assay protocol revealed that mutating eit

200 Electrophoretic mobility shift assays provide further evidence that A. p

201 es upon modulation of HOTAIR Electrophoretic mobility shift assays provided further evidence that HOT

202 In the electrophoretic mobility shift assay, purified recombinant Reb1p was sho

203 Electrophoretic mobility shift assays, quantitative reverse transcriptio

204 by coimmunoprecipitation and electrophoretic mobility shift assays, respectively.

205 Electrophoretic mobility shift assay results demonstrate Myc-Max heterod

206 Electrophoretic mobility shift assays reveal two discrete complexes with

207 ated in resistant plants and electrophoretic mobility shift assay revealed sequence-specific binding

208 An electrophoretic mobility shift assay revealed similarity between human a

209 Electrophoretic mobility shift assay revealed that PMA stimulated DNA bi

210 Electrophoretic mobility shift assays revealed a direct binding of KLF9

211 Electrophoretic mobility shift assays revealed increased binding of 8S m

212 Eletrophoretic mobility shift assays revealed increased binding of unme

213 Electrophoretic mobility shift assays revealed that ChrA specifically bi

214 Electrophoretic mobility shift assays revealed that FKPB51 overexpressio

215 Moreover, mobility shift assays revealed that Msn4 binds beta-oxid

216 Electrophoretic mobility shift assays revealed that MtrB enhances the bi

217 Electrophoretic mobility shift assays revealed that NFATc2 and CSL bind

218 Electrophoretic mobility shift assays revealed that RegX3 binds directly

219 Electrophoretic mobility shift assays revealed that SarX protein bound t

220 east one-hybrid analysis and electrophoretic mobility shift assays revealed that the transmembrane do

221 Electrophoretic mobility shift assays revealed that Tim enhances DDX11 b

222 RNA electrophoretic mobility shift assays (RNA-EMSA) were used to confirm th

223 Electrophoretic mobility shift assays show that Mn(II) restores DNA bind

224 Electrophoretic mobility shift assays show that RTV1 binds to DNA in vit

225 Electrophoretic mobility shift assays show that the K protein binds to a

226 An electrophoretic mobility shift assay showed that Arn prevents H-NS from

227 Electrophoretic mobility shift assay showed that OsbZIP48 binds directly

228 A gel mobility shift assay showed that the regulator proteins

229 evealed VDR-dependent inhibition of SHP, and mobility shift assays showed direct binding of VDR to en

230 However, electrophoresis mobility shift assays showed less binding of HNF-3beta t

231 Electrophoretic mobility shift assays showed that AaNAC2, AaNAC3, and Aa

232 -hybrid system technique and electrophoretic mobility shift assays showed that AioR interacts with th

233 ) one-hybrid experiments and electrophoresis mobility shift assays showed that AtNAP could physically

234 In this study, electrophoretic mobility shift assays showed that AtWRKY30 binds with hi

235 Electrophoretic mobility shift assays showed that Fur binds upstream of

236 Immunoprecipitation-qPCR and electrophoretic mobility shift assays showed that MdMYB88/MdMYB124 act a

237 ophyll cell protoplasts, and electrophoretic mobility shift assays showed that NAP can bind directly

238 Gene expression and electrophoretic mobility shift assays showed that the 5.3-kDa antimicrob

239 Electrophoretic mobility shift assays showed that the CRR1 SBP domain bi

240 Mobility shift assays showed that the transcription fact

241 rase reporter assays and RNA electrophoretic mobility shift assays showed that wild-type, but not zin

242 and electrophoretic mobility shift assay shows that RBM24 directly binds to

243 otein interaction studies by electrophoretic mobility shift assay suggested hypoxia response and an a

244 Electrophoretic mobility shift assay suggested that the G allele interac

245 Mobility shift assays suggested that E2F interacts with

246 these interact with ABI4 in electrophoretic mobility shift assays, suggesting that sequence recognit

247 We introduce a microfluidic mobility shift assay that enables precise and rapid quan

248 We showed by electrophoretic mobility shift assay that the C terminus of KNL2 binds D

249 , we have recapitulated our findings using a mobility shift assay that was developed and employed by

250 We also show through electrophoretic mobility shift assays that OsARID3 specifically binds to

251 t fluorescence quenching and electrophoretic mobility shift assays that probe siRNA binding by the di

252 We demonstrate using electrophoretic mobility shift assays that Rv0678 binds to the mmpS5-mmp

253 vitro dimethyl sulfate footprinting and gel mobility shift assays, that DnaA(L366K) in either nucleo

254 When examined by electrophoretic mobility shift assay, the triterpenoid suppressed nuclea

255 However, based on electrophoretic mobility shift assays, the divergent CTRs do appear to p

256 In electrophoretic mobility shift assays, the purified rPG2212 protein did

257 In addition to electrophoretic mobility shift assays, this model was corroborated by fu

258 In this study, we used electrophoretic mobility shift assay to analyze 46 arylstibonic acids fo

259 riophage lambda Cro and used electrophoretic mobility shift assays to compare binding of each variant

260 We used deep sequencing and electrophoretic mobility shift assays to derive in vitro GR binding affi

261 ions, in situ hybridization, electrophoretic mobility shift assays to determine binding sites in targ

262 In a previous study, we used gel mobility shift assays to determine that BreR binds at tw

263 n the current study, we used electrophoretic mobility shift assays to examine the binding of OLE RNA

264 DNase hypersensitivity, and electrophoretic mobility shift assays to study protein-DNA binding, we i

265 We used electrophoretic mobility shift assay, transient transcriptional activati

266 tly reduced ability to bind 3' and 5' RNA in mobility shift assays, use the DNA target to prime rever

267 etry of a band excised after electrophoretic mobility shift assay using a ZTRE probe.

268 ear transcription factors by electrophoretic mobility shift assay using digoxigenin (DIG)-labeled pro

269 Electrophoretic mobility shift assays using mouse retinal nuclear extrac

270 Electrophoretic mobility shift assays using the ilvE promoter and a puri

271 Mobility-shift assays using a control RNA detected an RN

272 Gel mobility shift assays validated the identity of the APUM

273 Electrophoretic mobility shift assays verified formation of a sandwich c

274 A gel mobility shift assay was used to examine the effect of p

275 Use of the methods for electrophoretic mobility shift assays was demonstrated for binding of th

276 , NMR, microcalorimetry, and electrophoretic mobility shift assay), we have characterized the structu

277 Using a phospho-protein mobility shift assay, we demonstrate that WRKY33 is phos

278 atin immunoprecipitation and electrophoretic mobility shift assay, we show that TH has a direct recep

279 By electrophoretic mobility shift assays, we confirmed binding of FOXA prot

280 Using electrophoretic mobility shift assays, we defined a RegX3 binding site i

281 of in vitro translation and electrophoretic mobility shift assays, we demonstrate that a PCBP/nsp1be

282 Using RNase digestion, DNAzyme, and RNA mobility shift assays, we demonstrate the absence of nak

283 luorescence polarization and electrophoretic mobility shift assays, we demonstrate the recognition of

284 te-directed mutagenesis, and electrophoretic mobility shift assays, we identified a GLI2 binding site

285 Using electrophoretic mobility shift assays, we identified CREB as the regulat

286 By electrophoretic mobility shift assays, we identified four additional Sma

287 Using reporter gene and electrophoretic mobility shift assays, we identify an 11-bp fragment of

288 Using electrophoretic mobility shift assays, we observed differential CEBPB bi

289 By electrophoretic mobility shift assays, we show weaker binding of protein

290 on resonance diffraction and electrophoretic mobility shift assays were consistent with PARTICLE trip

291 In silico analysis and electrophoretic mobility shift assays were used to assess SNP function.

292 Electrophoretic mobility shift assays were used to assess the binding of

293 Gel mobility shift assays were used to measure the binding a

294 ption were also reflected in electrophoretic mobility shift assays where CcpE bound to the citB promo

295 investigated by competitive electrophoretic mobility shift assay, which revealed that the two AC-ric

296 gradient composition and the development of mobility-shift assays, which rely on discrimination of m

297 ion factor-binding sites and electrophoretic mobility shift assays with MCF-7 nuclear protein demonst

298 Electrophoretic mobility shift assays with purified His(10)-MrpC2 and Fr

299 by DNase I footprinting and electrophoretic mobility shift assays, with some DNA-binding capacity be

300 ng yeast 3 hybrid assays and electrophoretic mobility shift assays, Zar2 was shown to bind specifical