ライフサイエンスコーパス: molecular bases

1 gely apoE-dependent processes with different molecular bases.

2 kinje cell death in these mice have distinct molecular bases.

3 s, COU phenotypes are likely to share common molecular bases.

4 resonance spectroscopy to link them to their molecular bases.

5 dysmotility, and investigate the underlying molecular bases.

6 The cellular and molecular bases allowing tissue regeneration are not wel

7 been discovered in several cancer types, the molecular bases and consequences of spliceosome aberrati

8 enetic contribution, but the relationship of molecular bases and environmental exposures appears intr

9 mans, and motivates future research into the molecular bases and functional consequences of sex chrom

10 ere, recent advances in understanding of the molecular bases and physiologic roles of the mechanisms

11 mas spurred many efforts to understand their molecular bases and physiological consequences(5-14).

12 l role in diabetic vascular disease, but its molecular bases are not completely defined.

13 plant recipients, for which the cellular and molecular bases are not well understood.

14 the biological importance of UV vision, its molecular bases are not well understood.

15 disease development; however, the underlying molecular bases are not well understood.

16 disorder in humans, although the underlying molecular bases are poorly understood.

17 get genes of Snail1 and Snail2, although the molecular bases are presently unknown.

18 Yet the genetic and molecular bases are unclear, as very few iron uptake gen

19 ed in its export during germination, but the molecular bases are unclear.

20 ial fraction of idiopathic cases, the actual molecular bases are usually undetermined.

21 k, of disease and has diverse biological and molecular bases as revealed by cloning of QRLs and ident

22 Our results clarify the cellular and molecular bases behind the first genetically engineered

23 forward.IMPORTANCE Our understanding of the molecular bases behind the interplays between human noro

24 To understand the molecular bases behind these evolutionary changes, we so

25 cClelland et al. have begun to elucidate the molecular bases by which antibodies alone can impact pat

26 e underlying causative genes, as well as the molecular bases by which they mediate susceptibility, ar

27 plementing available reviews by highlighting molecular bases for a selective immune response.

28 vances that have begun to reveal the diverse molecular bases for adapter-mediated regulation of p97 f

29 The molecular bases for AICAR toxicity at the cellular level

30 ory hair cells have highlighted the possible molecular bases for amplification and the components of

31 Collectively, these findings constitute molecular bases for assembly and inhibition of transfera

32 The molecular bases for both primary photoreceptor and RPE d

33 Despite decades of studies, the molecular bases for both remain elusive.

34 The molecular bases for differences in lipid transfer activi

35 and provides insights into the cellular and molecular bases for enhanced HIV transmission in syphili

36 ll RNA sequencing (scRNAseq) to identify the molecular bases for functional distinctions between moto

37 These findings elucidate the molecular bases for hCLDN-9 selective ion permeability a

38 significantly increased understanding of the molecular bases for high-affinity peptide/MHC binding by

39 itive impact on brain function; however, the molecular bases for how exercise affects the structure a

40 Studies characterizing the molecular bases for individual differences in AT in the

41 To understand the molecular bases for its cell death regulatory activity,

42 The molecular bases for its unique clinical presentation are

43 ening disease, but little is known about the molecular bases for its unique clinical presentation.

44 objective of this study was to elucidate the molecular bases for K(+) current diversity in porcine gr

45 idates the ping-pong mechanism, explains the molecular bases for Lnt's substrate promiscuity, and sho

46 come, and recent evidence suggests different molecular bases for male and female MDD.

47 The pathologic and molecular bases for myocardial ischemia-reperfusion inju

48 SC graft survival, jeopardizing cellular and molecular bases for neural repair in vivo.

49 r complex axonal arbors but the cellular and molecular bases for neurite length-dependent vulnerabili

50 de novel means for examining the genetic and molecular bases for neurodevelopmental impairment in mod

51 toward a more complete understanding of the molecular bases for neurofibromatosis 1 and neurofibroma

52 ality in ligand binding orientations suggest molecular bases for partial agonism at full-length recep

53 tem human brain studies providing additional molecular bases for parvalbumin-positive interneuron alt

54 The molecular bases for phosphodiesterase 5 (PDE5) catalytic

55 As such, they are valuable to understand the molecular bases for PM lateral organization in plants.

56 These findings provide insight into the molecular bases for Salmonella Typhi's host specificity

57 of action of these agents is available, the molecular bases for selective tumor cell killing by chem

58 Unfortunately, the molecular bases for self-renewal of vertebrate cells rem

59 Here the molecular bases for SMX high-affinity block are investig

60 The molecular bases for species barriers to lentiviral repli

61 channels (TRPC) have been implicated as the molecular bases for store-operated channels (SOC).

62 Elucidation of the molecular bases for strain-specific host ranges and cros

63 ence will be the potential to understand the molecular bases for structural features of chromosomes t

64 , and oligodendrocytes, and they provide the molecular bases for subsequent neurovascular dysfunction

65 Some of the genetic and molecular bases for the aggressive behavior of pancreati

66 and molecular modeling studies disclosed the molecular bases for the binding of 19 to CXCR4 and for i

67 Herein, we report on some of the molecular bases for the cellular toxicity of homocystein

68 Moreover, the cellular and/or molecular bases for the developmental defects observed i

69 To address the molecular bases for the different viral lifestyles, we c

70 ong-chain carboxychromanol provide potential molecular bases for the differential anti-inflammatory e

71 are interested in learning the cellular and molecular bases for the existence of UVB susceptibility

72 Here we identify the molecular bases for the initiation of primer synthesis b

73 t 5 years, investigators have discovered the molecular bases for the major inherited blistering disor

74 Here, we describe the molecular bases for the md- and mg-dependent rescue of t

75 The biochemical and molecular bases for the most common forms of EDS (types

76 The molecular bases for the parcellation of the visual corte

77 Molecular bases for the period length and sustainability

78 The molecular bases for the recruitment to double-strand bre

79 ent in mammalian development, the underlying molecular bases for the resulting phenotypes remain to b

80 To understand the molecular bases for the roles played by the periskeletal

81 To characterize the molecular bases for the unique properties of the AVE, we

82 es, and structural studies have revealed the molecular bases for their activities.

83 udies lay the groundwork for elucidating the molecular bases for their distinct differentiation pathw

84 Consequently, the sequence requirements and molecular bases for their functions, as well as their ro

85 Understanding the genetic, developmental and molecular bases for these changes will provide insights

86 To identify possible molecular bases for these clinical manifestations, we fi

87 The molecular bases for these differences are presently unkn

88 The molecular bases for these differences in inheritance pat

89 The cellular and molecular bases for these early defects remain enigmatic

90 n decision-making but the neural circuit and molecular bases for these functions are little known.

91 have diverse effects on physiology, yet the molecular bases for these impacts remain mostly unknown(

92 The global genetic and molecular bases for these interspecies divergences were

93 To elucidate the molecular bases for these observed clinical differences

94 The molecular bases for these relationships remain largely u

95 In order to identify cellular and molecular bases for this difference, we performed compar

96 r historical ATRA/chemotherapy regimens, the molecular bases for this effect remain unknown.

97 The structural and molecular bases for this mechanism are not well understo

98 is widespread in plants and animals, but the molecular bases for this phenomenon remain elusive.

99 The molecular bases for this restriction are unknown.

100 cloning-candidate gene approach to identify molecular bases for two historic axolotl pigment phenoty

101 er structure and function should clarify the molecular bases for variations in CBS gene expression in

102 ng the interplays between the mechanical and molecular bases for vein graft restenosis.

103 To decipher the molecular bases governing seed germination, this study p

104 ans, especially among the elderly [2], their molecular bases have not been defined.

105 ght into the diagnosis, pathophysiology, and molecular bases, it is now possible to diagnose the diso

106 The molecular bases linking PrP sequence, infection suscepti

107 We report the molecular bases of 5 Er blood group antigens: the recogn

108 In contrast, the molecular bases of acinar dysplasia and congenital alveo

109 insight into the development, evolution and molecular bases of acquired and congenital melanocytic n

110 mography, current genetic diversity, and the molecular bases of adaptations in the bush dog and maned

111 Then, we review the cellular and molecular bases of affinity-dependent selection within t

112 cently, two breakthroughs in the long-sought molecular bases of alkane formation and polyester synthe

113 The biochemical and molecular bases of all autosomal dominant periodic fever

114 Over the past 20 years the molecular bases of almost all the major blood group anti

115 a critical function of this receptor in the molecular bases of antisocial personality.

116 1L as well as novel probes to understand the molecular bases of apoptosome regulation and turnover.

117 The cellular and molecular bases of asymmetric morphogenesis remain large

118 sts to more rapidly uncover the cellular and molecular bases of behavior.

119 in the ocean, but little is known about the molecular bases of biological pressure tolerance.

120 The molecular bases of brain development and CNS malignancie

121 in vivo to enhance our understanding of the molecular bases of brain function and dysfunctions.

122 eran insects is crucial to fully explain the molecular bases of Bt specificity and insecticidal actio

123 cells can play a vital role in revealing the molecular bases of cancer development and progression.

124 s approaches to uncover novel mechanisms and molecular bases of cardiovascular pathophysiological man

125 To investigate the molecular bases of cephalopod brain and body innovations

126 To examine the molecular bases of chronic tuberculosis and the role of

127 ese data provide the first insights into the molecular bases of cis -acting flanking sequences modify

128 ovide a set of candidates for dissecting the molecular bases of climate adaptations, as well as insig

129 Conceivably, the molecular bases of cocaine reward might display sufficie

130 As the molecular bases of colonic motor and sensory function ar

131 a powerful tool in elucidating not only the molecular bases of color vision, but the processes of ad

132 werful approach to further understanding the molecular bases of complex diseases.

133 predictions provide an understanding of the molecular bases of complications and means of identifyin

134 ovide an opportunity to discover unsuspected molecular bases of DCM, enabling pre-clinical risk detec

135 To investigate the molecular bases of dental fluorosis, we analyzed the eff

136 sed to formulate testable hypotheses for the molecular bases of disease phenotypes associated with th

137 ful tool to provide further insight into the molecular bases of disorders associated with abnormal mo

138 e as a well-defined model for studies on the molecular bases of drug-protein interactions in ABC tran

139 Improved understanding of the molecular bases of dystroglycanopathies will lead to mor

140 olving this paradox requires elucidating the molecular bases of efficient nutrient distribution and r

141 Unraveling the molecular bases of exocrine regeneration may identify ne

142 nd molecular framework for understanding the molecular bases of FLNA-MVD, which is crucial for the de

143 To clarify the molecular bases of flowering time evolution in crop dome

144 rogress toward understanding the genetic and molecular bases of FTD, no class of selectively vulnerab

145 suvanine as a template, we shed light on the molecular bases of FXR antagonism, identifying the essen

146 Understanding the molecular bases of galectin-ligand interactions, particu

147 Understanding the genetic and molecular bases of gene function is of increasing import

148 tein mutagenesis, we gained insight into the molecular bases of glycolipid recognition by Mincle.

149 , further enhancing our understanding of the molecular bases of GPCR-mediated signaling.

150 ystem, which permits study of the neural and molecular bases of habit learning, has important implica

151 The molecular bases of heteromeric assembly and link between

152 We investigated the molecular bases of high FVIII levels in 2 Italian famili

153 Despite much study, the molecular bases of HIV-1 latency and reactivation are in

154 Furthermore, we provide the molecular bases of how delta/alphabetaTCRs bind to their

155 llectively, our study provides insights into molecular bases of how environments have shaped peach ge

156 The molecular bases of how host genetic variation impacts th

157 However, the structural and molecular bases of how the lipophilic drugs reach the bi

158 and will accelerate our understanding of the molecular bases of human disease and cancer.

159 technologies are critical to understand the molecular bases of human diseases.

160 the development of the human fundus and the molecular bases of human gastric physiology and pathophy

161 calcium imaging, and mutagenesis to explore molecular bases of human TRPV6 inhibition by the antifun

162 To understand better the molecular bases of humidity sensing, we investigated sev

163 related hydrazone derivatives to define the molecular bases of immune-stimulation activity.

164 Little is known about the molecular bases of individual differences in EFs.

165 acetylcholinesterase species, ascertain the molecular bases of inhibition by Elec410, and document t

166 The molecular bases of inwardly rectifying K(+) (Kir) curren

167 L pathogenesis remain unclear, including the molecular bases of latency and reactivation, the site(s)

168 tion are central issues in understanding the molecular bases of liver regeneration.

169 e to human malignancies, but the genetic and molecular bases of many rare cancers remains unclear.

170 ry responses are ubiquitous and diverse, the molecular bases of mechanosensation in most cases remain

171 vide a useful model for investigation of the molecular bases of medulloblastoma and for evaluation of

172 metalloamyloids are needed to understand the molecular bases of metal-amyloid interactions.

173 itro experiments were performed to study the molecular bases of NAC thrombolytic effect, including pl

174 In this review we consider the cellular and molecular bases of neuronal migration into the postnatal

175 insight into these processes elucidates the molecular bases of numerous acquired and inherited hemat

176 tagenesis assays were employed to unveil the molecular bases of OPR1 regulation by senescence and JA.

177 l ultimately herald the understanding of the molecular bases of ovatoxins bioactivity.

178 An understanding of the molecular bases of pathogenicity in Blastomyces dermatit

179 Nevertheless, little is known about the molecular bases of pathogenicity in these algae-like org

180 s is of great interest for understanding the molecular bases of phenotypic diversification and pollin

181 have revealed many 'unusual suspects' in the molecular bases of phenotypic evolution, including upstr

182 Although the molecular bases of PKCdelta activation are being unfolde

183 To elucidate the genetic and molecular bases of plant-Verticillium interactions, we h

184 The cellular and molecular bases of platelet release by terminally differ

185 In Drosophila melanogaster, the genetic and molecular bases of post-mating changes in the female's b

186 the evolutionary, biochemical, genetic, and molecular bases of primary metabolic diversity could pro

187 Our results provide insights into the molecular bases of primate brain organization and demons

188 n, antigen identification, understanding the molecular bases of protective immune responses, and adju

189 However, the molecular bases of proteotoxicity by soluble LCs have no

190 ntal studies are redefining the cellular and molecular bases of pulmonary arterial hypertension (PAH)

191 Here, we investigated the molecular bases of rcd-1-dependent cell death.

192 The molecular bases of reactogenicity are unknown, but it ha

193 t and that are valuable in understanding the molecular bases of replicational accuracy.

194 the focus of many development studies of the molecular bases of retinotopic map formation, the role o

195 f MeCP2 has implications for research on the molecular bases of Rett syndrome, autism, and genomic im

196 etabolomics, knowledge about the genetic and molecular bases of rice seed metabolomes at different de

197 The molecular bases of ROS production and the primary cellul

198 d metabolome of sALS cells to understand the molecular bases of sALS metabotypes and the impact of EH

199 However, the anatomical, cellular and molecular bases of schizophrenia remain unclear.

200 Transcript profiling has shown the molecular bases of several biological processes in plant

201 However, the genetic and molecular bases of sociability are poorly understood.

202 At present, molecular bases of spectral tuning in rhodopsin-like (RH

203 The molecular bases of spectral tuning in the UV-, violet-,

204 We investigated the molecular bases of spondyloepiphyseal dysplasia (SED) as

205 The molecular bases of such cell behaviors (e.g. apical cons

206 However, the molecular bases of such conflicts remain poorly understo

207 The structural and molecular bases of such effects are unknown.

208 We understand the molecular bases of T cell receptor (TCR) recognition of

209 outcomes with a quantitative basis, and the molecular bases of that evolution have been explored.

210 Deciphering the molecular bases of the CMs is required for therapy and i

211 erefore identified in RIF1 dual function the molecular bases of the co-dependency of the replication-

212 Here, we analyze the genetic and molecular bases of the divergence of body pigmentation p

213 We also define the molecular bases of the energetic coupling between the po

214 The molecular bases of the evolved phenotypes were then eluc

215 h an in silico receptor-driven approach, the molecular bases of the hA1- and hA3AR recognition and ac

216 The molecular bases of the high degree of stereospecificity

217 .17/13/21 genetic lineage, we elucidated the molecular bases of the human norovirus-glycan interactio

218 An understanding of the molecular bases of the morphogenesis, organization, and

219 ained binding data were rationalized and the molecular bases of the observed hA3 AR affinity and hA3

220 al DNA translocation may shed light into the molecular bases of the packaging mechanism of bacterioph

221 Comparisons of the molecular bases of the phenotypic differences in SADDAN

222 However, the cellular and molecular bases of the skeletal abnormalities associated

223 To investigate the molecular bases of the SMS phenotype, we constructed BAC

224 ng 2 associated with aHUS) and unraveled the molecular bases of the so-called deregulation activity o

225 agents of acute respiratory disease, but the molecular bases of their distinct pathobiology are still

226 les of such dual traits, and the genetic and molecular bases of their evolution have not been identif

227 ute toward malignant behaviors; however, the molecular bases of their oncogenic properties are largel

228 To identify the molecular bases of their partnership, we generated recom

229 The cellular and molecular bases of these actions are unknown.

230 To understand the molecular bases of these activities, we identified 298 g

231 anscripts were carried out to understand the molecular bases of these altered P deprivation responses

232 persist into later life stages; however, the molecular bases of these changes remain unknown.

233 quite disparate phenotypes, deciphering the molecular bases of these conditions has served to highli

234 Until recently, the molecular bases of these disease subtypes have been uncl

235 To understand the molecular bases of these diseases, we generated construc

236 Molecular bases of these divergences are poorly understo

237 The molecular bases of these interactions, however, are unkn

238 However, the molecular bases of these properties are only partially e

239 The molecular bases of these sensory processes are largely u

240 tudies have begun to yield insights into the molecular bases of these unique features of the hairpin

241 Little is known about the molecular bases of this calcium-independent activation.

242 nal approaches, we set out to understand the molecular bases of this crucial interdependence through

243 s do not proliferate in these cells, yet the molecular bases of this exclusion remain only partially

244 The molecular bases of this interaction are revealed here by

245 The molecular bases of this intimate association appear to b

246 To clarify the molecular bases of this metabolic feature, we investigat

247 To establish the structural and molecular bases of this observation, we performed X-ray

248 able cytogenetic strategies to elucidate the molecular bases of this organization using Caenorhabditi

249 The physiological and molecular bases of this pervasive abnormality are unknow

250 esponsible for cancer invasion; however, the molecular bases of this phenomenon are poorly understood

251 The molecular bases of this process are poorly understood.

252 The molecular bases of this remarkable adaptation are, howev

253 This study determines the molecular bases of three of these S-haplotypes that conf

254 ndamental resource to reveal the genetic and molecular bases of traits in forward genetic studies.

255 Our understanding of the cellular and molecular bases of transduction of painful stimuli has b

256 provides new insights into the cellular and molecular bases of tumorigenesis and points to the poten

257 r human biology and disease and uncovers the molecular bases of valuable traits.

258 cy and clinical phenotypes, to elucidate the molecular bases of variability in manifestations of MAT1

259 provide unique resources to investigate the molecular bases of viral virulence and fitness and also

260 he world, numerous discoveries regarding the molecular bases of vision have been made through genetic

261 r loss, known collectively as alopecias, the molecular bases of which are entirely unknown.

262 ysiologic implications, but their underlying molecular bases remain incompletely defined.

263 ite this success, the underlying genetic and molecular bases remain largely unknown, limiting the bro

264 ex and its inputs in higher mammals, but the molecular bases remain largely unknown.

265 depend on vesicular trafficking events whose molecular bases remain poorly known.

266 s sense gravity for spatial orientation, the molecular bases remain uncertain.

267 s to congenital heart defects, their precise molecular bases remain unknown in the majority of patien

268 protein interactions, which are the dominant molecular bases responsible for mediating circuit develo

269 This study uncovers the molecular bases responsible for the differential activat

270 To begin to study the molecular bases that determine the selective interaction

271 The molecular bases that govern NT-3 expression within the c

272 e of the microbiota in human physiology, the molecular bases that govern the interactions between the

273 The molecular bases underlying inflammatory responses are no

274 stinent smokers, yet the neuroanatomical and molecular bases underlying it are unclear.

275 al alterations in brain organization and the molecular bases underlying neuronal clinical features re

276 However, the exact molecular bases underlying peptide selection and binding

277 siology of kidney disease and aging, but the molecular bases underlying the biologic outcomes on the

278 mbined with an improved understanding of the molecular bases underlying the dysregulation of connexin

279 The molecular bases underlying the pathogenesis of neurodege

280 However, the molecular bases underlying the phenotypic and functional

281 tinent to this concept, discuss the possible molecular bases underlying the selective targeting of th

282 The molecular bases underlying the subsistence of diatoms in

283 eine modification and help understanding the molecular bases underlying the surprisingly large role o

284 which were used to decipher the genetic and molecular bases underlying the variation of this respons

285 Here, we attempt to determine the molecular bases underlying the wide range of binding pro

286 However, the molecular bases underlying these associations remain unr

287 However, the molecular bases underlying these evolutionary changes ha

288 To better understand the molecular bases underpinning these processes, we analyze

289 While there is a growing appreciation of the molecular bases underpinning variable (V)d1(+) and Vd2(+