ライフサイエンスコーパス: molecular layer

1 ell dendrites invading a given volume in the molecular layer.

2 nation in the inner one-third of the dentate molecular layer.

3 pines in different dendritic segments in the molecular layer.

4 in revealed immunostaining of the cerebellar molecular layer.

5 elrhodopsin or electrical stimulation in the molecular layer.

6 fibers and boutons extended into the dentate molecular layer.

7 ctrode was directly evaporated on top of the molecular layer.

8 a network of inhibitory interneurons in the molecular layer.

9 ayer between the top metal electrode and the molecular layer.

10 at least one recurrent mossy fiber into the molecular layer.

11 Cs extended to the outer edge of the dentate molecular layer.

12 in the Purkinje cells and other cells of the molecular layer.

13 the metal atoms penetrating or damaging the molecular layer.

14 e cell and Purkinje cell layers, but not the molecular layer.

15 layer and overelaboration in the superficial molecular layer.

16 cted following electrical stimulation in the molecular layer.

17 mossy fibers) in the granule cell layer and molecular layer.

18 hat terminate in the outer two-thirds of the molecular layer.

19 lized foci on the superficial surface of the molecular layer.

20 er, terminates in the inner one-third of the molecular layer.

21 ure granule cells and formed synapses in the molecular layer.

22 By 48 h the dendrites first appeared in the molecular layer.

23 le cell layer but only Kv4.3 subunits in the molecular layer.

24 ausing their dendrites to extend through the molecular layer.

25 d branching dendrites that extended into the molecular layer.

26 rkinje cells are misplaced in the cerebellar molecular layer.

27 they had developing synapses on them in the molecular layer.

28 nanoscale films of dodecane down to a single molecular layer.

29 rmation to spatially distinct regions of the molecular layer.

30 directly affected by orbital energies of the molecular layer.

31 hibitory HIPPs distribute axon fibers in the molecular layer.

32 f the electron energy after it traverses the molecular layer.

33 local synapses and vasculature in the inner molecular layer.

34 ound along dendrites in the granule cell and molecular layers.

35 eep layers and parallel fiber input in their molecular layers.

36 und along the direction perpendicular to the molecular layers.

37 e within a single molecular layer or between molecular layers.

38 ar to very distinct alterations on different molecular layers.

39 terfaces between metallic thin films and C60 molecular layers.

40 ell nanoparticles with two components in the molecular layer, a framework molecule to form the shell,

41 At photon energies where the molecular layer absorbs, a secondary phenomenon is opera

42 , involving fiber sprouting into the dentate molecular layer and a parallel fiber retraction from the

43 spinous terminals in the dentate gyrus inner molecular layer and CA1 stratum radiatum and in (ii) pos

44 s was present in the inner one-fourth of the molecular layer and extended bilaterally throughout the

45 mma-7 is enriched in Purkinje neurons in the molecular layer and glomerular synapses in the granule c

46 ation of Purkinje cell dendrites in the deep molecular layer and overelaboration in the superficial m

47 CD200/CD200R interaction seem to include the molecular layer and pial surface in neonates and blood v

48 other types of neurons sprout into the inner molecular layer and synapse with granule cell dendrites

49 -Mott limit, and the interaction between the molecular layer and the substrate acts to influence the

50 ing from partial charge transfer between the molecular layer and the substrate.

51 rmed dense axonal arborizations in the inner molecular layer and throughout the hilus.

52 they progressively expanded into the middle molecular layer and, most ventrally, formed a distinct b

53 icantly larger dendritic arborization in the molecular layer, and a more triangular cell body than gr

54 nd dendrites, sprout axon collaterals in the molecular layer, and form new synapses with granule cell

55 pyramidale neuropil and dentate gyrus inner molecular layer, and increases in the strata oriens and

56 ice also had larger somata, more axon in the molecular layer, and longer dendrites than controls.

57 ry glomeruli, hippocampal CA3 and cerebellar molecular layer, and moderate binding in the cerebral co

58 fibres extending to the outer portion of the molecular layer, and more climbing fibre-Purkinje cell s

59 tiple potassium sites within initially dense molecular layers, and thus interact with the PAH anion p

60 l connectivity of the GABAergic cells in the molecular layer are not well understood.

61 Such molecular layers are at the quantum limit of device mini

62 Molecular layers are prepared by allowing sessile drops

63 Analysis of the molecular layer as a function of coverage reveals an unp

64 Granule cell axons enter the molecular layer as parallel fibers without bifurcating.

65 f the Ca(2+) indicator Fluo-4 throughout the molecular layer as well as the granule cells.

66 luation of thickness changes in the adsorbed molecular layers as a result of chaperone binding to rec

67 Changes in the molecular layer, as a result of enzymatic reactions, wer

68 l layer potentiated responses throughout the molecular layer, as well as in the CA3 region.

69 This study confirms that thinning of the molecular layer associated with disease pathogenesis is

70 Films are constructed one molecular layer at a time by using the layer-by-layer, d

71 sistive switching are strongly affected by a molecular layer at the graphene/BaTiO3 interface.

72 ances have enabled the profiling of multiple molecular layers at single-cell resolution, assaying cel

73 radial responses did not activate measurable molecular layer beams in transverse slices.

74 In the molecular layer, both isoforms were at highest levels wi

75 rials that consist of one or a few atomic or molecular layers, bottom-up assembly of 2D crystalline m

76 and large central cells are confined to the molecular layer but are not planar.

77 rth, with a primary dendrite pointing to the molecular layer, but at this stage, with several neurite

78 in the inner and outer zones of the dentate molecular layer, but it was reduced in the dentate hilus

79 nt path potentiated responses throughout the molecular layer, but left responses in the CA3 region un

80 dendrites of Golgi cells are confined to the molecular layer, but their axon arbors are either confin

81 ansfection, fibers were present in the inner molecular layer, but they progressively expanded into th

82 inous GABAergic synapse numbers in the outer molecular layer changed most.

83 ices were restricted to sublayers within the molecular layer, conducting slowly away from the stimulu

84 irection of rectification is reversed if the molecular layer consists of naphtalene diimides having l

85 ils, based mainly on solid-state NMR data, a molecular layer consists of two beta-sheets (residues 12

86 colloidal synthesis, in which self-assembled molecular layers control the alignment between materials

87 MeHg-induced increase in fluorescence in the molecular layer correlated with that of increased sEPSC

88 Herein, molecular layer deposition is used to form a nanoscale "

89 artificial SEI layer for Li-metal anodes via molecular-layer deposition (MLD) is reported.

90 The molecular-layer deposition of a flexible coating onto Si

91 ells that were ectopically positioned in the molecular layer displayed a more rapid functional matura

92 pattern, with dense innervation in the inner molecular layer, dorsal MCs have a more diffuse distribu

93 h marked uniaxial characteristics within the molecular layer due to the presence of one-dimensional p

94 nd an enlargement of the CF territory in the molecular layer during development.

95 Shear fluidization of a film of five to six molecular layers during an individual slip event should

96 asing lattice resistance to shear sliding of molecular layers during plastic deformation.

97 Molecular layer ectopia, clusters of misplaced cells in

98 lacunosum-moleculare of CA1 and the dentate molecular layer elicited GABAergic inhibitory responses

99 s increased dark Purkinje cells (PC), a thin molecular layer, fewer synapses, a loss of dendritic spi

100 eased Kv1.2, Kv1.3, and Kv1.6 in the dentate molecular layer following chronic Ro25,6981 treatment.

101 h allowed for the clear visualization of the molecular layer, granular layer, and white matter in chi

102 Although previous studies focused on the molecular layer, here, we shift attention onto the mossy

103 ochemical analyses on confined electroactive molecular layers, herein exemplified with electroactive

104 cribe the prevalence and cytoarchitecture of molecular-layer heterotopia in C57BL/6J mice and related

105 particular, we found that the prevalence of molecular-layer heterotopia vaired according to the sex

106 nt protein were abnormally positioned in the molecular layer, hilus, and granule cell layer.

107 The inner molecular layer (IML) displayed a corresponding volumetr

108 ic reorganization of the dentate gyrus inner molecular layer (IML) is a pathophysiological process th

109 row time window and stack their axons in the molecular layer in chronological order from deep to supe

110 noreactivity was more prominent in the inner molecular layer in prenatally choline-deficient rats com

111 tput (I/O) relationship in the dentate gyrus molecular layer in response to lateral perforant path st

112 rains of electrical stimuli delivered to the molecular layer in vitro, release adenosine via a proces

113 eficits, as measured by the thickness of the molecular layer, in all mice in which Klhl1 was deleted

114 limited to fibers located in the granule and molecular layer, in an orientation consistent with granu

115 The structures and energy levels of the molecular layers included donor molecules with relativel

116 ion of parallel fibers, migration across the molecular layer, incorporation into the internal granule

117 Gene-ontology analysis across different molecular layers indicates that although chromatin recon

118 ted staining was detected at the inner third molecular layer indicating glutamatergic sprouting.

119 immature granule cell neurons in the dentate molecular layer induced by ECS.

120 ity into a beam-like response, implying that molecular layer inhibition does not prevent beam-like re

121 Therefore, molecular layer inhibition is compartmentalized into zeb

122 Molecular layer inhibitory interneurons generate on-beam

123 a low-affinity antagonist at parallel fiber-molecular layer interneuron (PF-MLI) synapses.

124 Furthermore, CFs systematically targeted molecular layer interneuron cell bodies, especially at t

125 idence indicates electrical coupling between molecular layer interneurons (basket and stellate cells)

126 postnatal survival of both granule cells and molecular layer interneurons (basket and stellate cells)

127 increased by release of endogenous GABA from molecular layer interneurons (MLIs) and spillover activa

128 A receptors (NMDARs) expressed by cerebellar molecular layer interneurons (MLIs) are not activated by

129 munication between climbing fibers (CFs) and molecular layer interneurons (MLIs) in the cerebellum is

130 Inhibition from molecular layer interneurons (MLIs) is thought to play a

131 known that, during movement, the activity of molecular layer interneurons (MLIs) of the cerebellar co

132 aired recordings from synaptically connected molecular layer interneurons (MLIs) of the rat cerebellu

133 Information processing by cerebellar molecular layer interneurons (MLIs) plays a crucial role

134 Molecular layer interneurons (MLIs) provide powerful inh

135 eir spatiotemporal activity is controlled by molecular layer interneurons (MLIs) through GABA(A) rece

136 However, the contribution of molecular layer interneurons (MLIs) to these processes i

137 elieved to receive GABAergic inhibition from molecular layer interneurons (MLIs).

138 (TARPs) on presynaptic AMPARs in cerebellar molecular layer interneurons (MLIs).

139 Molecular layer interneurons (MLIs, stellate and basket

140 GABA release from molecular layer interneurons activates parallel fiber GA

141 cal discoveries concerning the importance of molecular layer interneurons and their plasticity, the s

142 Because they receive input from many CFs, molecular layer interneurons are well positioned to dete

143 Molecular layer interneurons exhibit bidirectional firin

144 or (NMDAR) expression on axons of cerebellar molecular layer interneurons have produced conflicting r

145 Here we perform cell-attached recording of molecular layer interneurons in cerebellar slices from r

146 Here, we address the roles of inhibitory molecular layer interneurons in establishing Purkinje ce

147 Our data uncover complementary roles for molecular layer interneurons in shaping the rate and pat

148 By recording from multiple molecular layer interneurons in the cerebellar cortex, w

149 racellular recordings from granule cells and molecular layer interneurons in vivo, selective pharmaco

150 ortex has led to suggestions that cerebellar molecular layer interneurons laterally inhibit Purkinje

151 ive currents obtained under voltage clamp in molecular layer interneurons of juvenile rats or mice re

152 simple synapses between parallel fibers and molecular layer interneurons of rat cerebellar slices.

153 ry GABA(A) receptor-mediated transmission in molecular layer interneurons of the mouse cerebellum.

154 A(A) antagonists and reflect the activity of molecular layer interneurons on their targets.

155 Feed-forward inhibition from molecular layer interneurons onto granule cells (GCs) in

156 inoid release from PCs and GABA release from molecular layer interneurons provide the primary means o

157 ithin the 480 mum lateral distance examined, molecular layer interneurons reduced the strength of gra

158 we examined how feedforward inhibition from molecular layer interneurons regulates adaptation of the

159 upting this balance by selectively silencing molecular layer interneurons results in unidirectional f

160 pal slices and identified a subpopulation of molecular layer interneurons that expressed immunocytoch

161 In the cerebellar cortex, molecular layer interneurons use chemical and electrical

162 ion from granule cells to Purkinje cells and molecular layer interneurons will be strengthened during

163 okes inward GABAergic currents in cerebellar molecular layer interneurons with rise times of 2 ms, co

164 made up of Purkinje cells, climbing fibers, molecular layer interneurons, and cerebellar efferent ce

165 nd adults, PCs make synapses onto other PCs, molecular layer interneurons, and Lugaro cells, but not

166 nctional connectivity between granule cells, molecular layer interneurons, and Purkinje cells in rats

167 ical synapses and gap junctions with various molecular layer interneurons, including other NGFCs.

168 h prior studies of spillover transmission to molecular layer interneurons, these results reveal that

169 xcitatory granule cells (GCs) and inhibitory molecular layer interneurons-in processing of whisking s

170 inhibitory GABAergic synapses of cerebellar molecular layer interneurons.

171 parallel fiber synaptic input to downstream molecular layer interneurons.

172 lity at synapses between parallel fibers and molecular layer interneurons.

173 rites of Purkinje cells, efferent cells, and molecular layer interneurons.

174 targets, the Purkinje cells, Golgi cells and molecular layer interneurons.

175 The molecular layers investigated included donor molecules w

176 When the molecular layer is based on a molecule with a high HOMO-

177 In the cerebellum, the molecular layer is regarded as the main NVC determinant.

178 lved, and the coordination between different molecular layers is unclear.

179 Discrete molecular layer lesions demonstrate that PFs contribute

180 characterized by progressive thinning of the molecular layer, loss of Purkinje cells and increasing m

181 The partly charged end-groups of the grafted molecular layer may act as a top gate.

182 In the molecular layer, mGluR2/3 labeling slightly declined in

183 hat NGFCs form an integral part of the local molecular layer microcircuitry generating feed-forward i

184 1(nmf164) mouse cerebellum, microglia in the molecular layer (ML) are activated and contacting dendri

185 reas dentate gyrus granule cells (DGGCs) and molecular layer (ML) interneurons predominantly express

186 e from the outer to the inner regions of the molecular layer (ML) of the DG.

187 ) including a granule cell layer (GCL) and a molecular layer (ML) that continuously crosses adjacent

188 ward migration of GABA interneurons into the molecular layer (ML) were arrested, disrupting layer and

189 al cells (BGs), astrocytes of the cerebellar molecular layer (ML), express various receptors that can

190 fibers (pfs) and apical GoC dendrites in the molecular layer (ML).

191 lar colocalization numbers were found in the molecular layer of cerebellar sections.

192 piny branchlets of the Purkinje cells in the molecular layer of cerebellum and in the olfactory bulb.

193 naptic terminals of GABAergic neurons in the molecular layer of cerebellum.

194 hinal cortex, CA1/subiculum border and outer molecular layer of dentate) were initially affected, wit

195 Formula: see text] are observed for a single molecular layer of dodecane trapped between crystallogra

196 e coating is a covalently tethered, flexible molecular layer of perfluorocarbon, which holds a thin l

197 alterations to the general morphology of the molecular layer of the cerebellar cortex that are the re

198 ressed in punctate structures throughout the molecular layer of the cerebellar cortex.

199 ween granule cells and Purkinje cells in the molecular layer of the cerebellar cortex.

200 pic neurons and progenitors collected in the molecular layer of the cerebellum and adjacent tectum.

201 rpendicular deposits of prion protein in the molecular layer of the cerebellum.

202 layers I and II of the cerebral cortex, and molecular layer of the cerebellum.

203 VGLUT1 was highly expressed in VCNm and the molecular layer of the DCN, whereas VGLUT2 was expressed

204 ted a decrease in spine density in the outer molecular layer of the dentate gyrus (DG) beginning as e

205 gates through the perforant path (PP) to the molecular layer of the dentate gyrus (DG) where informat

206 of compact beta-amyloid plaques in the outer molecular layer of the dentate gyrus and Layers II and I

207 ll decreases in synapse density in the outer molecular layer of the dentate gyrus at both 6-9 and 15-

208 ults indicate that MOPP cells located in the molecular layer of the dentate gyrus contribute to feed-

209 nificantly increased synaptic density in the molecular layer of the dentate gyrus in Tg2576 mice.

210 ifically, spines of new neurons in the outer molecular layer of the dentate gyrus were more readily i

211 it labeling at perisynaptic locations in the molecular layer of the dentate gyrus where these subunit

212 which innervates the outer two-thirds of the molecular layer of the dentate gyrus, was one of the fir

213 and amyloid deposits are often found in the molecular layer of the dentate gyrus, which is the termi

214 stimulation of the perforant pathway in the molecular layer of the dentate gyrus.

215 er of the Caspr2 extracellular domain in the molecular layer of the developing cerebellum.

216 s. radiatum, CA2 and CA3 s. pyramidale, and molecular layer of the DG, was increased by 34.0%, 8.9%,

217 similar patterns of termination in the inner molecular layer of the DG, we discovered that the axonal

218 sicular glutamate transporter 1 in the outer molecular layer of the hippocampal dentate gyrus on the

219 n the activated dendritic lamina, the middle molecular layer of the hippocampal dentate gyrus.

220 density of GABAergic synapse markers in the molecular layer of the hippocampus of TS mice.

221 and ectopic neurons in the dentate hilus and molecular layer of the hippocampus.

222 extracellular space (ECS) of the anisotropic molecular layer of the isolated turtle cerebellum.

223 ssoon in the active zones of synapses in the molecular layer of the mouse cerebellum.

224 evealed a number of immature features in the molecular layer of the mutant cerebellar cortex, includi

225 in in dendritic spines down to 40 mum in the molecular layer of the visual cortex of an anesthetized

226 Similar beams were observed in the molecular layer of thick transverse slices but not sagit

227 A single molecular layer of titanium diselenide (TiSe2) is a prom

228 3 GPa are found using Raman spectrometry for molecular layers of 1-nm in thickness.

229 intracellular distribution of Fabp7 mRNA in molecular layers of hippocampus.

230 successive hopping diffusion into successive molecular layers of molecular spintronics devices.

231 scopy reveals that these ribbons, only a few molecular layers of protein thin, rival the mechanical p

232 through the addition or removal of discrete molecular layers of water alters cation exchange selecti

233 synapse densities in the dentate gyrus outer molecular layer (OML) correlated with DNMS recognition a

234 ume of the somatostatin-immunopositive outer molecular layer (OML), innervated by projections from th

235 ce intensity as well as the thickness of the molecular layer on the observed photocurrent.

236 cterization of structure and interactions of molecular layers on an optical waveguide surface for a w

237 h charge injection barrier caused by organic molecular layers on its basal plane, thus the best perfo

238 mperature and then regenerated with the same molecular layer or a different one by "renewing" the int

239 epending on whether they are within a single molecular layer or between molecular layers.

240 the number of GABAergic interneurons in the molecular layer or hilus.

241 It is organized in ridges of ganglionic and molecular layers, oriented perpendicular to the granular

242 numbers of excitatory synapses in the inner molecular layer per granule cell could be calculated.

243 ge of 2,280 asymmetric synapses in the inner molecular layer per granule cell, which was reduced to 6

244 numbers of excitatory synapses in the inner molecular layer per hippocampus in pilocarpine-treated c

245 modified rabies virus, we demonstrated that molecular layer perforant pathway (MOPP) cells innervate

246 g that CB1R downregulation in the cerebellar molecular layer plays a key role in THC-induced cerebell

247 her density of dendritic spines in the inner molecular layer postlesion accompanied by an increase in

248 e signals conveyed by parallel fibers in the molecular layer predict the patterns of sensory input to

249 Increase in the molecular layer roughness in response to the sialylation

250 ture the electrochemical transformation of a molecular layer self-assembled on a tapered gold microel

251 zed excitatory neuronal class in the dentate molecular layer, semilunar granule cell (SGC), has been

252 ranule cells but had axon collaterals in the molecular layer, significantly larger dendritic arboriza

253 e they require a Burgers vector spanning two molecular-layer spacings, distorting the crystal lattice

254 In the rat cerebellar molecular layer, spillover of glutamate between parallel

255 Molecular layer stimulation of the intact cerebellum evo

256 ons reliably phase-lock to regular trains of molecular layer stimulation.

257 an energy loss that is strongly dependent on molecular layer structure and thickness.

258 ity, and allow examination of the effects of molecular-layer structure, thickness and contact work fu

259 In the cerebellar molecular layer, synaptic input reduces [Ca](o) by up to

260 l layer that contains all somata and (2) the molecular layer that contains the dendritic forest.

261 s (SGCs), glutamatergic neurons in the inner molecular layer that generate long-duration plateau pote

262 These results reveal UNG as a new molecular layer that shapes the specificity of AID-induc

263 f local field potential power in the dentate molecular layer that were accompanied by transient incre

264 ome shell proteins, which self-assemble into molecular layers that most likely constitute the facets

265 thickness of a material approaches a single molecular layer, the large surface energy can cause piez

266 ness of approximately 8 nm consisting of 7-8 molecular layers, the deposition of FgAalpha251 was term

267 ith the transverse-oriented, slow-conducting molecular layer, thereby permitting complex temporal pro

268 xponential dependence of junction current on molecular layer thickness (d) similar to that observed f

269 isease progression as assessed by histology (molecular layer thickness and an overall severity score)

270 deletions) and an intermediate reduction in molecular layer thickness in mice with reduced levels of

271 id not result in full recovery of cerebellar molecular layer thickness or prevent Bergmann glia degen

272 unctions, the energy loss is linear with the molecular layer thickness, with a slope of 0.31 eV/nm.

273 type, but did not prevent BG process loss or molecular layer thinning, while excision of ataxin-7 fro

274 d on electronically addressing a molecule or molecular layer through the formation of a metallic cont

275 d by direct electron-beam evaporation on the molecular layers through masks defined by electron-beam

276 ethod of forming 'soft' metallic contacts on molecular layers through surface-diffusion-mediated depo

277 m a horizontal bipolar shape situated in the molecular layer, through a transitional triangular and t

278 tic light absorption in the sun-facing front molecular layer, through which sun light must propagate

279 deposited remotely and then diffuse onto the molecular layer, thus eliminating the problems of penetr

280 The grafting of molecular layers to carbon-based materials provides a wa

281 napses in the inner one-third of the dentate molecular layer was Glu-CB1 -RS, 53.19% (glutamatergic t

282 dendritic segments in different areas of the molecular layer were differentially regulated.

283 leptic rats excitatory synapses in the inner molecular layer were larger than in controls.

284 Those in the middle molecular layer were more likely to be influenced by the

285 of HEGC apical dendrites that penetrated the molecular layer were significantly reduced compared with

286 rection of rectification was reversed if the molecular layers were also reversed.

287 ut Ag present, and the spectra of oligomeric molecular layers were completely consistent with those o

288 The structure and orientation of the molecular layers were probed with ATR-FT-IR and Raman sp

289 When the energy levels of the two molecular layers were similar, the device had electronic

290 xclusively in the ganglionic layer below the molecular layer where parallel fibers terminate.

291 fuse distribution and expand into the middle molecular layer where they overlap and interact with inn

292 d delta were highly expressed in the dentate molecular layer, whereas alpha1, alpha2, alpha3, alpha5,

293 e cell dendrites in the inner portion of the molecular layer, whereas parallel fibres form synapses o

294 mitochondrial staining, particularly in the molecular layer, which was independent of stress-induced

295 at integrates perturbations across different molecular layers, which identifies a sub-network consist

296 staining revealed the three sublamina of the molecular layer, while immunostaining for vesicular glut

297 om the interaction of excitons in an organic molecular layer with surface plasmons in a metallic film

298 n of diazonium reagents enables formation of molecular layers with sufficient integrity for use in mo

299 f deep stellate cells are distributed in the molecular layer, with fine axon arbors in the ganglionic

300 ns of the outer and middle one-thirds of the molecular layer, with frequent axonal projections across