ライフサイエンスコーパス: monocyte progenitor

1 population arising from a common granulocyte/monocyte progenitor.

2 risk cases revealed expansion of granulocyte-monocyte progenitors.

3 Cs, multipotent progenitors, and granulocyte-monocyte progenitors.

4 ting with a greater expansion of granulocyte-monocyte progenitors.

5 t-shifted with selective loss of granulocyte-monocyte progenitors.

6 (SCF)-dependent EML cell line to granulocyte/monocyte progenitors.

7 used to document the distribution pattern of monocyte progenitors.

8 nal differentiation of committed granulocyte/monocyte progenitors.

9 xtracellular vesicle transfer to granulocyte-monocyte progenitors.

10 l-forming potential is a particular trait of monocyte progenitors.

11 In addition, common monocyte progenitors accumulate cholesterol and lipids,

12 karyocytic-erythroid progenitor, granulocyte-monocyte progenitor and hematopoietic stem cell bone mar

13 cells, which correlated with an increase in monocyte progenitors and a decrease in granulocyte proge

14 rated an increase in the growth potential of monocyte progenitors and a significant decrease in granu

15 layed transcriptional profiles distinct from monocyte progenitors and closely resembling primary micr

16 on and differentiation of murine granulocyte-monocyte progenitors and human CD34(+) progenitors.

17 sma gondii, there was enhanced generation of monocyte progenitors and increased numbers of inflammato

18 absence of dramatic increases in bone marrow monocyte progenitors and was independent of chemokine (C

19 viral transcripts could be detected only in monocyte progenitors and were also associated with reduc

20 pool (common myeloid progenitor, granulocyte-monocyte progenitor, and megakaryocyte-erythroid progeni

21 ors (common myeloid progenitors, granulocyte monocyte progenitors, and megakaryocyte erythroid progen

22 Monocyte progenitors are induced in mycobacterial infect

23 Inducible monocyte progenitors are so far undescribed circulating

24 appaB activity causes changes in granulocyte/monocyte progenitor behavior.

25 n from the common myeloid to the granulocyte/monocyte progenitor but is not required beyond this stag

26 ntained; we subsequently identified not only monocyte progenitors but also diverse HSPCs within the g

27 ha is required for generation of granulocyte-monocyte progenitors, but the subsequent role of C/EBPal

28 proinflammatory cytokines, overt granulocyte/monocyte progenitor cell apoptosis, and failure to recon

29 rrow progenitors and demonstrate that common monocyte progenitor cells express high levels of APOBEC1

30 tic stem cells, but not immediate myeloid or monocyte progenitor cells, contained full microglia repl

31 med progenitor cells and cycling granulocyte-monocyte progenitor cells, providing evidence for NUP98-

32 nd osteoclasts are derived from monocytes or monocyte progenitor cells, the ways in which they are fo

33 ion of Lin-Sca1+c-Kit+ (LSK) and granulocyte-monocyte progenitor cells.

34 that Ly6C(hi) monocytes develop from common monocyte progenitors (cMoPs) and reside in the bone marr

35 nitor (MDP) stage and remains high in common monocyte progenitors (cMoPs).

36 lineage, the common myeloid and granulocyte-monocyte progenitors (CMP/GMP), have been shown to prote

37 ghly purified common myeloid and granulocyte-monocyte progenitors (CMPs/GMPs) accelerated myeloid rec

38 resulted in an expansion of the granulocyte/monocyte progenitor compartment and was associated with

39 rs are so far undescribed circulating common monocyte progenitor descendants with high giant cell-for

40 d IFN-gamma promoted a regulatory program in monocyte progenitors during development.

41 ) was determined in both total nucleated and monocyte progenitor enriched bone marrow cells.

42 ause in addition to lymphoid and granulocyte-monocyte progenitors, FLT3(-) Mk- and E-restricted downs

43 lls (HSCs), short-term HSCs, and granulocyte-monocyte progenitors from individuals with high-risk (-7

44 ate mapping revealed that yolk-sac and fetal monocyte progenitors gave rise to the majority of cardia

45 em cell dislocation, and CD41(-) granulocyte-monocyte progenitor (GMP) aggregation.

46 proteins preferentially activate granulocyte-monocyte progenitor (GMP) genes.

47 these TdT(+) cells coincide with granulocyte-monocyte progenitor (GMP) immunophenotype, their accessi

48 trast, SRSF2 mutant cases show a granulocyte-monocyte progenitor (GMP) pattern and high levels of sen

49 e the emergency expansion of the granulocyte-monocyte progenitor (GMP) population and upregulate the

50 sP3KB null mice, the bone marrow granulocyte monocyte progenitor (GMP) population was expanded, and G

51 expansion and differentiation of granulocyte/monocyte progenitor (GMP) populations, which is due in p

52 , there was a strong skew toward granulocyte-monocyte progenitor (GMP) production at the expense of e

53 lpha then directs the LMP to the granulocyte-monocyte progenitor (GMP) stage, while inhibiting lympho

54 nd differentiation skewed toward granulocyte-monocyte progenitors (GMP) during joint and intestinal i

55 on myeloid progenitors (CMP) and granulocyte-monocyte progenitors (GMP) protects against death follow

56 hGM-CSFR-expressing (hGM-CSFR+) granulocyte/monocyte progenitors (GMPs) and megakaryocyte/erythrocyt

57 Granulocyte-monocyte progenitors (GMPs) and monocyte-dendritic cell

58 llows us to separate oligopotent granulocyte-monocyte progenitors (GMPs) and their lineage-committed

59 Purified granulocyte/monocyte progenitors (GMPs) gave rise to eosinophils as

60 Granulocyte-monocyte progenitors (GMPs) have been previously defined

61 e show that RUNX1 is required in granulocyte-monocyte progenitors (GMPs) to epigenetically repress tw

62 n myeloid progenitors (CMPs) and granulocyte/monocyte progenitors (GMPs).

63 n myeloid progenitors (CMPs) and granulocyte/monocyte progenitors (GMPs)], which have a distinct gene

64 s of the myelomonocytic lineage (granulocyte-monocyte progenitors) harbouring previously acquired dri

65 a greatly increased multipotent granulocyte-monocyte progenitor in the spleen.

66 ny-stimulating factor receptor expression in monocyte progenitors in burn sepsis.

67 ignificantly altered distribution profile of monocyte progenitors in norepinephrine-depleted mice com

68 C/EBPalpha increases granulocyte relative to monocyte progenitors in Runx1-deleted marrow cells.

69 of mature monocytes and myeloid granulocyte-monocyte progenitors in the bone marrow and spleen of hy

70 vealed a skewing of HSPCs toward granulocyte-monocyte progenitors in the CAC group, consistent with a

71 esicles and, when transferred to granulocyte-monocyte progenitors, increased protein translation, cel

72 d progeny: granulocyte progenitors (GPs) and monocyte progenitors (MPs).

73 ating cell (LIC) population has a neutrophil-monocyte progenitor (NMP) phenotype.

74 ges differentiated from bone marrow or blood monocyte progenitors of HDM-allergic mice or asthma pati

75 ges differentiated from bone marrow or blood monocyte progenitors of HDM-allergic mice or asthma pati

76 age-negative marrow cells and in granulocyte-monocyte progenitors or common myeloid progenitors.

77 EoPs and Eos, but not granulocyte-monocyte progenitors or neutrophils, expressed Helios an

78 +)/c-Kit(+), common myeloid, and granulocyte-monocyte progenitor populations in the BM.

79 Common monocyte progenitors potently produce cytokines in respo

80 g common myeloid progenitors and granulocyte-monocyte progenitors, resulting in lower rates of myeloi

81 Flow cytometric analysis of early and late monocyte progenitors showed a significantly altered dist

82 alysis of early (ER-MP12) and late (ER-MP20) monocyte progenitors showed an increase in monocyte line

83 the bone marrow starting at the granulocyte-monocyte progenitor stage and reduced systemic expansion

84 a +37(-/-) mice rapidly progress through the monocyte progenitor stage to develop directly into Ly6C(

85 d each of these fates or whether specialized monocyte progenitor subsets exist before inflammation.

86 sophils and eosinophils acted on granulocyte-monocyte progenitors to transcriptionally programme the

87 actor receptor (M-CSFR) on bipotent GMPs and monocyte progenitors via the signal transducer and activ

88 olony-stimulating factor receptor density in monocyte progenitors was assessed by 125I macrophage col

89 Monocyte progenitors were 5-bromo-2'-deoxyuridine (BrdU)

90 cription factor expression, these neutrophil/monocyte progenitors were reprogrammed to take on erythr

91 the commitment of these cells to granulocyte/monocyte progenitors, whereas dominant-negative Stat5 co