ライフサイエンスコーパス: monocyte-derived macrophage

1 ytoplasmic and nuclear HIV-1 DNA in infected monocyte-derived macrophages.

2 ered by phenotypic overlap of microglia with monocyte-derived macrophages.

3 (PXR), in M. tuberculosis infection in human monocyte-derived macrophages.

4 endent maturation of IL-1beta in human THP-1 monocyte-derived macrophages.

5 complexin monocytic THP-1 cells and primary monocyte-derived macrophages.

6 of BBI on HIV infection of peripheral blood monocyte-derived macrophages.

7 n of IFN-beta in human primary monocytes and monocyte-derived macrophages.

8 er cells control the initial accumulation of monocyte-derived macrophages.

9 vation is inhibited by PGE2 in human primary monocyte-derived macrophages.

10 receptor for bacterial RNA in primary human monocyte-derived macrophages.

11 of cells that comprises conventional DCs and monocyte-derived macrophages.

12 49 human alveolar epithelial cells and THP-1 monocyte-derived macrophages.

13 hough the cells can be complemented by adult monocyte-derived macrophages.

14 c-derived Kupffer cells and peripheral blood monocyte-derived macrophages.

15 in both THP-1-derived macrophages and human monocyte-derived macrophages.

16 noted in postefferocytotic peripheral blood monocyte-derived macrophages.

17 an dermal CD14(+) cells are CD11b(+) CD64(+) monocyte-derived macrophages.

18 population of experimentally-infected human monocyte-derived macrophages.

19 ty of the peptide on in vitro cultured human monocyte-derived macrophages.

20 rn recognition and defense response genes in monocyte-derived macrophages.

21 acrophages of embryonic origin distinct from monocyte-derived macrophages.

22 d primarily in microglia and not infiltrated monocyte-derived macrophages.

23 l functions, but were less inflammatory than monocyte-derived macrophages.

24 eased phagocytosis of the AECs by autologous monocyte-derived macrophages.

25 rior Ab-dependent cellular phagocytosis with monocyte-derived macrophages.

26 promoters in immature dendritic cells and in monocyte-derived macrophages.

27 ace of naive monocytes, as well as in GM-CSF-monocyte-derived macrophages.

28 to study apoptotic cell uptake by autologous monocyte-derived macrophages.

29 flammatory cytokine profile similar to blood monocyte-derived macrophages.

30 to accumulation of extranuclear DNA in human monocyte-derived macrophages.

31 in both monocyte-derived dendritic cells and monocyte-derived macrophages.

32 stimulated against donor APCs in the form of monocyte-derived macrophages.

33 CR4 (CD11c/CD18) mediated infection of human monocyte-derived macrophages.

34 totic lymphocytes or neuronal cells by human monocyte-derived macrophages.

35 ritoneal mouse macrophages, as well as human monocyte-derived macrophages.

36 teady-state counterpart of these cells to be monocyte-derived macrophages.

37 iction factor targeting Env in primary human monocyte-derived macrophages.

38 lactate increases IL-10 production by human monocyte-derived macrophages.

39 ovide new insights into the heterogeneity of monocyte-derived macrophages.

40 e secretion in THP-1 cells and primary human monocyte-derived macrophages.

41 ately replaced by infiltrating monocytes and monocyte-derived macrophages.

42 om healthy nonsmokers and COPD donors and in monocyte-derived macrophages.

43 roduction of inflammatory cytokines in DADA2 monocyte-derived macrophages.

44 roinflammatory cytokines from mice and human monocyte-derived macrophages.

45 o regulated PKR phosphorylation during human monocyte-derived macrophage activation.

46 sident macrophage abundance, whereas CCR2(+) monocyte-derived macrophages adopted multiple cell fates

47 ed HIV-1 suppression in HIV-1-infected human monocyte-derived macrophages after crossing the BBB mode

48 Infiltrating monocyte-derived macrophages aggravate APAP hepatotoxici

49 In this article, we show the diversity of monocyte-derived macrophages along the course of experim

50 Additionally, monocyte-derived macrophages also interacted with HBsAg,

51 Human monocyte-derived macrophages and a mouse macrophage cell

52 hIL-10, and we identify CD14(+)monocytes and monocyte-derived macrophages and DCs as major sources of

53 In contrast, monocyte-derived macrophages and dendritic cells are per

54 Infection of X4 viruses in monocyte-derived macrophages and dendritic cells is enha

55 that different types of myeloid cells, i.e., monocyte-derived macrophages and dendritic cells, transf

56 nd interferon (IFN) gene expression in human monocyte-derived macrophages and dendritic cells.

57 sites of inflammation and differentiate into monocyte-derived macrophages and dendritic cells.

58 ite for enhancement of phagocytosis by human monocyte-derived macrophages and downregulation of IL-8

59 Studies using human monocyte-derived macrophages and endothelial cells furth

60 oregulatory cells, including IL-10-producing monocyte-derived macrophages and Foxp3(+) regulatory T c

61 sis factor and interleukin-6 are released by monocyte-derived macrophages and lymphocytes in the lung

62 ) and proinflammatory (GM-CSF-treated) human monocyte-derived macrophages and microglia using RNA seq

63 S accumulation of classically activated (M1) monocyte-derived macrophages and microglial expression o

64 e defective for replication in primary human monocyte-derived macrophages and murine J774 cells yet e

65 secretion, were conducted with primary human monocyte-derived macrophages and neutrophils and freshly

66 nd survives inside these cells as well as in monocyte-derived macrophages and neutrophils for at leas

67 In regenerative tissues, a central role of monocyte-derived macrophages and paracrine factors secre

68 investigated the apoptotic effect of Vpr on monocyte-derived macrophages and phorbol 12-myristate 13

69 e expression of SPIC transcription factor in monocyte-derived macrophages and promotes their differen

70 gate P2 receptor expression in primary human monocyte-derived macrophages and receptors that mediate

71 on CD11b, MerTK, and CD103 were reduced, and monocyte-derived macrophages and resident macrophages ex

72 impaired the critical conditioning of these monocyte-derived macrophages and resulted in spontaneous

73 HBsAg-induced cytokine production by KCs and monocyte-derived macrophages and subsequent NK cell acti

74 lia is differentially regulated from that in monocyte-derived macrophages and the ramified microglia

75 a robust attenuation of liver recruitment of monocyte-derived macrophages and their repolarization to

76 te smoke on model THP-1 and peripheral blood monocyte derived macrophages, and discovered a marked in

77 ociated virulence was studied in guinea pig, monocyte-derived macrophage, and lysozyme resistance ass

78 ype 1 (HIV-1) infectivity in CD4(+) T cells, monocyte-derived macrophages, and dendritic cells.

79 is highly expressed in CD4(+) T lymphocytes, monocyte-derived macrophages, and dendritic cells.

80 owing three archetypes: activated monocytes, monocyte-derived macrophages, and heat shock-activated m

81 Steady-state cardiac macrophages, monocyte-derived macrophages, and locally sourced macrop

82 studies on human alveolar macrophages, human monocyte-derived macrophages, and murine bone marrow-der

83 umin in macrophage cell lines, primary human monocyte-derived macrophages, and murine bone marrow-der

84 Indeed, monocytes, monocyte-derived macrophages, and peripheral blood monon

85 nt cells (MGCs), a property not exhibited by monocyte-derived macrophages, and we detected MGCs of my

86 We show in this article that human monocyte-derived macrophages are able to engulf NETs in

87 Kupffer cells and monocyte-derived macrophages are critical for liver repa

88 Microglia and infiltrating monocyte-derived macrophages are now known to be functio

89 inflammation induced by obesity, bone marrow monocyte-derived macrophages are recruited to inflamed t

90 Monocyte-derived macrophages are the major innate immune

91 Monocyte-derived macrophages are the most abundant immun

92 ed the interactions of HCV EnvGPs with human monocyte-derived macrophages as antigen-presenting cells

93 otic GECs to THP-1 cells or peripheral blood monocyte-derived macrophages as assayed by confocal micr

94 We found that primary human monocyte-derived macrophages as well as THP-1 macrophage

95 abilization was detected in peripheral blood monocyte-derived macrophages at 2 to 24 h after infectio

96 ed inflammatory cytokine production by human monocyte-derived macrophages; autocrine IL-1 production

97 pectively, cannot replicate in primary human monocyte-derived macrophages because they trigger innate

98 anti-FcgammaR F(ab')2 fragments on uptake by monocyte-derived macrophages (both M1 and M2 macrophages

99 er in CNS-derived microglia than observed in monocyte-derived macrophages, both basally and under all

100 es readily completed its life cycle in human monocyte-derived macrophages but not in CD4(-) cells.

101 cium release (IC(50) 10 nM) induced in human monocyte-derived macrophages by 100 nM C3a, (b) inhibiti

102 IV-1 and other primate lentiviruses in human monocyte-derived macrophages by impairing reverse transc

103 Moreover, these donor monocyte-derived macrophages can themselves mediate cyto

104 ion is primarily based on the study of blood monocyte-derived macrophages, cells that have never been

105 T-A, and dexamethasone on Mtb containment in monocyte-derived macrophages co-incubated with purified

106 criptional and functional data revealed that monocyte-derived macrophages coordinate cardiac inflamma

107 tudies using primary cultures of bone marrow monocyte-derived macrophages, demonstrated that glatiram

108 monocyte plasticity and heterogeneity among monocyte-derived macrophages, describe possible mechanis

109 asing IL-10 secretion in donor-matched human monocyte-derived macrophages differentiated by GM-CSF or

110 Thus, monocyte-derived macrophages display distinct phenotypes

111 7A induced a unique transcriptome pattern in monocyte-derived macrophages distinct from known macroph

112 AdA exposure in human monocyte-derived macrophages enhanced efferocytosis of a

113 ith age and are progressively substituted by monocyte-derived macrophages, even in the absence of inf

114 extracellular trap (MET) formation by bovine monocyte-derived macrophages exposed to M. haemolytica o

115 wild-type (wt)HTT on the function of primary monocyte-derived macrophages from healthy, non-disease h

116 Although monocytes and monocyte-derived macrophages from humans and chimpanzees

117 ther supported by the skewed polarization of monocyte-derived macrophages from multicentric carpotars

118 PPBP expression by monocyte-derived macrophages from patients with ABPA or

119 with human macrophages (THP-1, U937, primary monocyte-derived macrophages from patients with inflamma

120 Monocyte-derived macrophages from subjects with the Fcga

121 e upon exposure to pathogenic stimuli, human monocyte-derived macrophages generated in the presence o

122 ne bone marrow-derived macrophages and human monocyte-derived macrophages generated mROS in response

123 imary human cancer cells, actively migrating monocyte-derived macrophages had greater redox ratios [N

124 Human monocyte-derived macrophages have been used to character

125 We show that monocyte-derived macrophages have elevated expression of

126 Although monocyte-derived macrophages have high dUTP levels, thes

127 observations, including those obtained from monocyte-derived macrophages, have argued that ribonucle

128 ow-derived and peritoneal macrophages, human monocyte-derived macrophages, HeLa cells, and mouse embr

129 to compare the secreted metabolomes of human monocyte-derived macrophages (hMDM) under normal conditi

130 eumophila to be defective in growth in human monocyte-derived macrophages (hMDMs) but not in Acantham

131 1q bound to immobilized C1q (imC1q) on human monocyte-derived macrophages (HMDMs) obtained from healt

132 regulating apoE secretion from primary human monocyte-derived macrophages (HMDMs) remain unclear.

133 em using primary human lymphocytes and human monocyte-derived macrophages (HMDMs) to characterize the

134 t upon attachment of L. pneumophila to human monocyte-derived macrophages (hMDMs), the host farnesyla

135 xpression of 5'-tRNA half molecules in human monocyte-derived macrophages (HMDMs).

136 pression of ABCA1 in comparison with intimal monocyte-derived macrophages, however, are unknown.

137 Monocyte-derived macrophages, however, encounter a gradu

138 ith their cardiac derivatives, monocytes and monocyte-derived macrophages in conventional cell cultur

139 also indicate that many cells identified as monocyte-derived macrophages in human atherosclerosis ar

140 nfiltration of monocytes and accumulation of monocyte-derived macrophages in inflammation.

141 ility complex class II on both monocytes and monocyte-derived macrophages in joints.

142 the individual role(s) of Kupffer cells and monocyte-derived macrophages in the induction of LPC pro

143 The demonstration of monocyte-derived macrophages in the steady state in huma

144 crophages in vivo and that it is produced by monocyte-derived macrophages in vitro, after Helicobacte

145 derived from human intestinal organoids with monocyte-derived macrophages, in a gut-on-a-chip platfor

146 n N-terminal kinase in Nod2-stimulated human monocyte-derived macrophages, in the absence of autocrin

147 pattern recognition receptors (PRR) in human monocyte-derived macrophages induces interleukin (IL)-1,

148 The temporal response of primary human monocyte-derived macrophages infected with highly pathog

149 Furthermore, IL-26, when added to human monocyte-derived macrophages infected with M. leprae, en

150 l replication or eliminate CD4(+) T cells or monocyte-derived macrophages infected with SIV variants

151 n cell death and apoptosis pathways in human monocyte-derived macrophages ingesting modified LDL; thi

152 moval of dead cells, prevented maturation of monocyte-derived macrophages into F4/80-expressing macro

153 lasmin in vivo also prevented trafficking of monocyte-derived macrophages into necrotic lesions after

154 Thus, they are markedly distinct from monocyte-derived macrophages invading after local distur

155 Proinflammatory activation of monocyte-derived macrophages is associated with a concom

156 e to IL-4 stimulation in tissue-resident and monocyte-derived macrophages is not understood.

157 production as treatment of proinflammatory, monocyte-derived macrophages, isolated from APAP-treated

158 marginal differences in LPS responses, human monocyte-derived macrophages killed Escherichia coli and

159 ng intracellular infection of peritoneal and monocyte-derived macrophages, known to secrete lysozyme,

160 In injured liver, infiltrating monocyte-derived macrophages largely augment the hepatic

161 Monocyte-derived macrophages, located in atherosclerotic

162 -HT(7) expression restricted to M-CSF-primed monocyte-derived macrophages (M-MO).

163 Infiltrating monocyte-derived macrophages (M-MPhi) influence stroke-i

164 Production of NO(-) by bovine monocyte-derived macrophage (MDM) and mouse peritoneal m

165 lease of infectious HIV-1 from primary human monocyte-derived macrophages (MDM) acutely infected with

166 Monocyte-derived macrophages (MDM) also expressed a mixe

167 f SPM-biosynthetic pathways in human M2-like monocyte-derived macrophages (MDM) and consequently for

168 o explain the presence of inflammatory CD14+ monocyte-derived macrophages (MDM) and immunoglobulin G+

169 Monocyte-derived macrophages (MDM) can polarize into dif

170 icantly up-regulated in HIV-1-infected human monocyte-derived macrophages (MDM) compared with bystand

171 ary cell-based coculture model, we show that monocyte-derived macrophages (MDM) efficiently transmit

172 erefore, ATM were isolated and compared with monocyte-derived macrophages (MDM) from the same obese p

173 onse to bacteria by alveolar macrophages and monocyte-derived macrophages (MDM) in COPD and healthy c

174 -meso-diaminopimelic acid (GM-triDAP), human monocyte-derived macrophages (MDM) produced an order of

175 ytes and that the bacteriostatic activity of monocyte-derived macrophages (MDM) requires NOX2 and gam

176 In vitro, HIV replication in monocyte-derived macrophages (MDM) selectively reduces H

177 lipins to analyze mediator profiles in human monocyte-derived macrophages (MDM) stimulated with HDM a

178 te in human macrophages, we infected primary monocyte-derived macrophages (MDM) that had been differe

179 duce early glycolytic reprogramming of human monocyte-derived macrophages (MDM), which is similar to

180 n a potent block to HIV-1 infection in human monocyte-derived macrophages (MDM).

181 role of PKCalpha in CRIg expression in human monocyte-derived macrophages (MDM).

182 drug particles readily accumulated in human monocyte-derived macrophages (MDM).

183 in experimentally infected human skin and in monocyte-derived macrophages (MDM).

184 cytic killing by primary human monocytes and monocyte-derived macrophages (MDM).

185 ng minimal effects on the viability of human monocyte derived macrophages (MDMs) and RBC integrity.

186 re analyzed in non-CF and F508del/F508del CF monocyte derived macrophages (MDMs) with and without cli

187 replication complexes in cell lines, primary monocyte-derived macrophages (MDMs) and CD4(+) T cells.

188 nerated primary myeloid cells, specifically, monocyte-derived macrophages (MDMs) and dendritic cells

189 aive and lipopolysaccharide (LPS) stimulated monocyte-derived macrophages (MDMs) and IPSDMs using RNA

190 NF-kappaB transcriptional activity in human monocyte-derived macrophages (MDMs) and the murine macro

191 Neutrophils and monocyte-derived macrophages (MDMs) are the main source

192 ts the first in vivo comparative analysis of monocyte-derived macrophages (MDMs) between rhesus macaq

193 es repressive effect on HIV in human primary monocyte-derived macrophages (MDMs) by promoting HIV sup

194 We report that a major subpopulation of monocyte-derived macrophages (MDMs) contains high levels

195 Here, we sought to determine whether monocyte-derived macrophages (MDMs) contribute to recove

196 d poorly to IFN-gamma, whereas monocytes and monocyte-derived macrophages (MDMs) did not.

197 (EBOV); knockdown of TIM-3 in differentiated monocyte-derived macrophages (MDMs) enhances HIV-1 produ

198 Monocyte-derived macrophages (MDMs) exhibit a high degre

199 ug levels reached 6 mug/10(6) cells in human monocyte-derived macrophages (MDMs) for nanoparticle tre

200 this article, we analyze CF and non-CF human monocyte-derived macrophages (MDMs) for ROS production,

201 ertook an in vitro comparative assessment of monocyte-derived macrophages (MDMs) from both nonhuman p

202 We found that human monocyte-derived macrophages (MDMs) from rs917997 AA ris

203 y over the past few years has involvement of monocyte-derived macrophages (MDMs) in CNS repair receiv

204 tor T cells to suppress viral replication in monocyte-derived macrophages (MDMs) in ESs and CPs.

205 rts demonstrating the presence of CD14+CD16+ monocyte-derived macrophages (MDMs) in the gingival tiss

206 ripheral blood mononuclear cells (PBMCs) and monocyte-derived macrophages (MDMs) in vitro and induces

207 vary continuously in their ability to enter monocyte-derived macrophages (MDMs) in vitro, and MDMs v

208 miRNA expression profiles were determined in monocyte-derived macrophages (MDMs) incubated in conditi

209 to T-tropic viruses, M-tropic viruses infect monocyte-derived macrophages (MDMs) on average 28-fold m

210 in low-CD4-expressing Jurkat cells and human monocyte-derived macrophages (MDMs) through downregulati

211 igen presentation in MERS-CoV-infected human monocyte-derived macrophages (MDMs) versus SARS-CoV-infe

212 ripheral blood mononuclear cells (PBMCs) and monocyte-derived macrophages (MDMs) were isolated from p

213 Human monocyte-derived macrophages (MDMs) were studied in nonc

214 Monocyte-derived macrophages (MDMs) were treated with M-

215 We hypothesize that infection of human monocyte-derived macrophages (MDMs) with Salmonella ente

216 epithelial cell monolayer, (ii) attached to monocyte-derived macrophages (MDMs), (iii) bound monocyt

217 31461630

218 Using lung epithelial cells, primary human monocyte-derived macrophages (MDMs), and neutrophils fro

219 ognition receptor (PRR) stimulation of human monocyte-derived macrophages (MDMs), decreasing STAT3, S

220 We found that monocyte-derived macrophages (MDMs), including granulocy

221 d a subset of interferon-stimulated genes in monocyte-derived macrophages (MDMs), which differed from

222 IV-1 infections of IFN-alpha-treated primary monocyte-derived macrophages (MDMs).

223 to EBOV and RESTV infection in primary human monocyte-derived macrophages (MDMs).

224 ime course of IAV infection in primary human monocyte-derived macrophages (MDMs).

225 5-tropic) HIV-1 to induce BAFF production by monocyte-derived macrophages (MDMs).

226 everal microRNAs (miRNAs) in polarized human monocyte-derived macrophages (MDMs).

227 rowth of Mycobacterium tuberculosis in human monocyte-derived macrophages (MDMs).

228 lood mononuclear cells and differentiated to monocyte-derived macrophages (MDMs).

229 is in Vpx+ virus-like particle (VLP)-treated monocyte-derived macrophages (MDMs).

230 rphine may modulate HIV-1 infection of human monocyte-derived macrophages (MDMs).

231 sed both STAT1 and STAT4 expression in human monocyte-derived macrophages (MDMs).

232 nd trafficking to lysosomes in primary human monocyte-derived macrophages (MDMs).

233 Unlike traditional monocyte-derived macrophages, microglia originate from p

234 How monocyte fate is directed toward monocyte-derived macrophages (mo-Macs) or monocyte-deriv

235 cluding activated microglia and infiltrating monocyte-derived macrophages (mo-MPhi).

236 Monocyte-derived macrophages (mo-MPhis) and T cells have

237 ssion in human alveolar macrophages (AM) and monocyte-derived macrophages (MoDM).

238 Upon experimental APAP overdose in mice, monocyte-derived macrophages (MoMFs) massively accumulat

239 To generate monocyte-derived macrophages, monocytes were cultured fo

240 n (myeloid-derived suppressor cells [MDSCs], monocyte-derived macrophages [Mphis], and dendritic cell

241 e of tissue-resident microglia, infiltrating monocyte-derived macrophages, neutrophils, and T cells.

242 phages, Kupffer cells (KCs), but not hepatic monocyte-derived macrophages or dendritic cells (DCs), a

243 primary human neutrophils with primary human monocyte-derived macrophages or dendritic cells (DCs).

244 lls (PBMC) and a CD4 T-cell line compared to monocyte-derived macrophages, or dendritic cells (DC).

245 e role for neutrophils during a UTI, whereas monocyte-derived macrophages orchestrate a strong, but i

246 In conclusion, HCVc inhibits monocyte-derived macrophage polarization via TLR2 signal

247 tion, local arterial inflammation, driven by monocyte-derived macrophages, predicts future cardiovasc

248 virus maintained the ability to replicate in monocyte-derived macrophages, primary CD4(+) T cells, an

249 Monocyte-derived macrophages prime acute cellular reject

250 Monocytes and monocyte-derived macrophages promote atherosclerosis thr

251 Specifically, GH treatment of GM-CSF-primed monocyte-derived macrophages promotes a significant enri

252 erstood about the diversity of monocytes and monocyte-derived macrophages recruited to the heart afte

253 to determine the effect of CD40L absence on monocyte-derived macrophage responses.

254 g HDAC1 and HDAC3 in Twist1/Twist2-deficient monocyte-derived macrophages restored the reduced histon

255 stimulation of the PRR NOD2 in primary human monocyte-derived macrophages resulted in increased H3 an

256 ccessful efferocytosis of apoptotic cells by monocyte-derived macrophages resulted in the suppression

257 ciple study was conducted with primary human monocyte-derived macrophages, seeded in decreasing numbe

258 Also, monocyte-derived macrophages showed intact phagocytosis,

259 In monocyte-derived macrophages, SSRI treatment decreased e

260 g the roles of tissue-resident and recruited monocyte-derived macrophage subsets.

261 eover, these results were confirmed in human monocyte-derived macrophages, suggesting that global imm

262 , after injury, these cells were replaced by monocyte-derived macrophages that are proinflammatory an

263 activated T cells lead to the recruitment of monocyte-derived macrophages that become highly activate

264 Moreover, and unlike in the CNS, monocyte-derived macrophages that develop during injury

265 ell loss was compensated by gain of adjacent monocyte-derived macrophages that exhibited convergent e

266 on in the retina, and in the infiltration of monocyte-derived macrophages that were absent from contr

267 In peripheral monocyte-derived macrophages, the expression of the M1,

268 ) modulates innate immunity in human primary monocyte-derived macrophages through toll-like receptor

269 se cells were preferentially phagocytosed by monocyte-derived macrophages, thus linking proteasome ac

270 clear cells, purified monocytes, T cells and monocyte-derived macrophages to examine functional immun

271 on and subsequent territorial restriction of monocyte-derived macrophages to infarct tissue.

272 T-Regs decrease the ability of alveolar and monocyte-derived macrophages to restrict the growth of M

273 We used the human monocyte-derived macrophages to study the role of M. tub

274 nhibit astroglial CCL2-driven trafficking of monocyte-derived macrophages to the CNS during acute MS

275 ) on the pro-inflammatory responses of human monocyte-derived macrophages to the saturated fatty acid

276 In monocyte-derived macrophages, URMC-099 induction of auto

277 the signaling and function of primary human monocyte-derived macrophages, using a C5aR2 agonist (Ac-

278 Expression of DHRS9 within a panel of monocyte-derived macrophages was investigated by quantit

279 ycobacterial growth in infected alveolar and monocyte-derived macrophages was significantly diminishe

280 gulfment of EMPs or cMPs by peripheral blood monocytes-derived macrophages was associated with signif

281 and transient knockdown of TMEM203 in human monocyte-derived macrophages, we show that TMEM203 prote

282 rophage-mediated cancer cell activity, human monocyte derived-macrophages were subjected, for a week,

283 Monocyte-derived macrophages were also infected with NTH

284 Monocyte-derived macrophages were chosen as natural targ

285 H37Rv-infected alveolar and monocyte-derived macrophages were cocultured with autolo

286 Monocyte-derived macrophages were collected from healthy

287 Pulmonary and monocyte-derived macrophages were cultured in vitro with

288 es isolated from peripheral blood, and human monocyte-derived macrophages were either coincubated wit

289 eboid migration mode of HIV-1-infected human monocyte-derived macrophages were inhibited, whereas the

290 perfusate-derived KCs and in vitro-generated monocyte-derived macrophages were investigated for funct

291 d enteroid monolayers co-cultured with human monocyte-derived macrophages were used to evaluate barri

292 Unlike recruited monocyte-derived macrophages, which actively contribute

293 icroglia, an endogenous cell population, and monocyte-derived macrophages, which infiltrate from the

294 the peripheral nervous system, infiltrating monocyte-derived macrophages, which use the chemokine re

295 M-CSF drives the generation of human monocyte-derived macrophages with a potent anti-inflamma

296 +) cells are a tissue-resident population of monocyte-derived macrophages with a short half-life of <

297 onstrated an association of CNS-infiltrating monocyte-derived macrophages with disease severity.

298 We sought to compare pair-matched monocyte-derived macrophages with m isolated from chroni

299 Incubation of human monocyte-derived macrophages with the isolated extracell

300 imics induced a proinflammatory phenotype in monocyte-derived macrophages, with enhanced expression a