ライフサイエンスコーパス: monocytic cell

1 ntly suppressed exogenous viral infection in monocytic cells.

2 igration (85%), with similar results in U937 monocytic cells.

3 thelial cell lines (HeLa and C33A) and THP-1 monocytic cells.

4 that was reversed by GSK3beta suppression in monocytic cells.

5 ity as well as transendothelial migration of monocytic cells.

6 This was confirmed in primary human monocytic cells.

7 occurs indirectly by mediators released from monocytic cells.

8 selected mRNAs in interferon-gamma-activated monocytic cells.

9 an increased accumulation of S100A9-positive monocytic cells.

10 Ly6C(low) granulocytic and Ly6G(-)Ly6C(high) monocytic cells.

11 anscripts were identified in endothelial and monocytic cells.

12 ryo-1 up-regulates IFN-gammaR2 expression in monocytic cells.

13 ssion and regulation of IFN-gammaR chains in monocytic cells.

14 th IL-4 in enhanced CCL2 production in human monocytic cells.

15 expression in human PMA-differentiated THP-1 monocytic cells.

16 -mediated IL-1beta secretion requires ASC in monocytic cells.

17 ceptors (TLRs) ligands has been described in monocytic cells.

18 related cytokines from differentiated human monocytic cells.

19 lysaccharide-induced TNF-alpha production in monocytic cells.

20 transcription initiation between T cells and monocytic cells.

21 of innate immune responses in differentiated monocytic cells.

22 their effect on ALC/APAP-induced toxicity in monocytic cells.

23 nd APAP-induced toxicity in both hepatic and monocytic cells.

24 tective role for Panx1 in endothelial and/or monocytic cells.

25 yristate acetate (PMA) activated THP-1 human monocytic cells.

26 mpaired nuclear import of HIV-1 DNA in human monocytic cells.

27 atory host cells such as T cells and myeloid/monocytic cells.

28 ot contribute to IL-1beta release from human monocytic cells.

29 t to the secretome and the proteome of human monocytic cells.

30 ection with the activation status of porcine monocytic cells.

31 ge conversion from band-stage neutrophils to monocytic cells.

32 PRRSV is ideal for deciphering how monocytic cell activation statuses interact with antivir

33 at this contributes significantly to reduced monocytic cell adhesion to TNF-alpha-activated HAEC.

34 dothelial APP was also involved in mediating monocytic cell adhesion.

35 expression of adhesion molecules, leading to monocytic cell adhesion.

36 d that FKN was a major contributor to T- and monocytic-cell adhesion to HIMECs.

37 subsequent LPS challenge and is achieved by monocytic cells after prolonged exposure to LPS.

38 nd that HCV infects both CD14(+), CD16(+)(+) monocytic cells and CD14(+)(+), CD16(+)(+) monocytic cel

39 (AAT) decreases HIV replication in PBMCs and monocytic cells and decreases NF-kappaB activity, we pos

40 FXa) activity by IgG-containing ICs by THP-1 monocytic cells and human monocytes.

41 HoloBLG shuttled iron into monocytic cells and impaired their antigen presentation.

42 nd 15-prostaglandin dehydrogenase in primary monocytic cells and in a human Th2-related lung disease.

43 alis strain A7436 induces cell death in THP1 monocytic cells and in human primary peripheral blood ma

44 -1 transcription and replication in cultured monocytic cells and in primary monocyte-derived macropha

45 s (PRRSV), which directly infects subsets of monocytic cells and interferes with antiviral responses.

46 (Gal3), a pleiotropic lectin, is produced by monocytic cells and macrophages.

47 association with the cytoskeleton in myeloid/monocytic cells and modulates IL-1beta processing, NF-ka

48 re that oxLDL induced TF expression in human monocytic cells and monocytes.

49 R-297 and -299 are endogenously expressed in monocytic cells and negatively regulate VEGFA expression

50 vivo activated HIV-1 gene expression only in monocytic cells and not in CD4(+) T cells.

51 We treated human THP-1 monocytic cells and primary human blood monocytes with R

52 During differentiation of U937 monocytic cells and primary human CD14(+) monocytes, ST6

53 ty and proinflammatory cytokine secretion in monocytic cells and primary murine macrophages.

54 o HIV gp120, for the neurotoxic phenotype of monocytic cells and subsequent toxin-initiated neuronal

55 nity, because it directly infects subsets of monocytic cells and subverts overall immune responses.

56 jacked" leukocytes, previously identified as monocytic cells and T lymphocytes, transmit EHV1 to endo

57 Cooperative responses between monocytic cells and tissue cells are likely to be crucia

58 RANKL induced osteoclastogenesis in these monocytic cells, and curcumin inhibited both RANKL- and

59 af7, is expressed by resting primary myeloid/monocytic cells, and its expression in these cells is re

60 , survival and properties of agrin-deficient monocytic cells, and occur at stages later than stem cel

61 cell surface RON expression in HIV-infected monocytic cells, and Tat-mediated degradation of RON pro

62 owever, the presence and function of PKD1 in monocytic cells are currently unknown.

63 intact, demonstrating that G-CSFR signals in monocytic cells are sufficient to induce HSPC mobilizati

64 at short-term stimulation of human and mouse monocytic cells as well as mouse osteoblasts with P2RX7

65 cytokines, human TNFalpha, and IL-8 in THP-1 monocytic cells as well as mouse TNFalpha and MIP-2 in R

66 luble CD163 likely originates from activated monocytic cells, as serum from stroke patients stimulate

67 and microvesicles released by LPS-activated monocytic cells, as well as circulating microvesicles is

68 tion, variola was disseminated by means of a monocytic cell-associated viremia.

69 ills proliferating and nonproliferating U937 monocytic cells at a comparable specific activity, appro

70 Importantly, porcine monocytic cells at different activation states were susc

71 se pathways in uninfected and PRRSV-infected monocytic cells at different activation states.

72 Although it induces chemotaxis of monocytic cells at high concentrations, its physiologica

73 , we demonstrated that TM knockdown in human monocytic cells attenuated LPS-induced signaling pathway

74 describe a regulation of Dicer expression in monocytic cells, based on proteolysis.

75 d that miR-146a is critical for the in vitro monocytic cell-based endotoxin tolerance.

76 t miR-146a plays a crucial role for in vitro monocytic cell-based endotoxin-induced cross-tolerance.

77 from lipopolysaccharide (LPS)-treated THP-1 monocytic cells bind to and are internalized by human en

78 cells, CDK2 enhanced HIV-1 transcription in monocytic cells but inhibited it in T cells.

79 ) monocytic cells and CD14(+)(+), CD16(+)(+) monocytic cells but not CD14(+)(+), CD16- cells in indiv

80 ral proteins implicated in driving cancer in monocytic cells but only harbor limited activity in epit

81 ces the cytotoxicity of paclitaxel for human monocytic cells, but not for breast, lung, or pancreatic

82 ntially reactivated HIV-1 gene expression in monocytic cells, but not in CD4 T cells, in cART-treated

83 gnificantly increased HIV-1 transcription in monocytic cells, but not in HIV-1-infected CD4 T cells,

84 cell proliferation and ERK1/2 activation in monocytic cells, but the integrin binding-defective R74E

85 nhanced immune response that is dependent on monocytic cells, but this hyperimmune phenotype and its

86 we present evidence that upon stimulation of monocytic cells by M. tuberculosis a unique TNF-alpha en

87 type 1 (HIV-1) infection in noncycling human monocytic cells by reducing the intracellular deoxynucle

88 ssion with small interference RNA in myeloid/monocytic cells caused a dramatic increase in NFkappaB a

89 We observed that monocytic cells cocultured (but not in contact) with ECs

90 els of CYP2E1 caused significant toxicity in monocytic cells compared to exosomes derived from contro

91 d TLR4 surface expression in HIV-infected U1 monocytic cells compared to the uninfected parental U937

92 A forms and induced less TNF-alpha in THP-1 monocytic cells compared with LOS from group 1.

93 se of the autofluorescent signal recorded in monocytic cells could be correlated with the IR detectio

94 ytic cells with nonrespiring mitochondria or monocytic cells cultured in the presence of pyruvate or

95 norrhoeae infection promotes NLRP3-dependent monocytic cell death via pyronecrosis, a recently descri

96 sisting after the infection resolves, and is monocytic cell dependent.

97 solution of inflammation is characterized by monocytic cell-dependent production of proangiogenic fac

98 feasibility and efficacy of IU injections of monocytic cells (derived from normal marrow) in feline a

99 s relayed by the MKK6-p38MAPK cascade induce monocytic cell differentiation from band-stage neutrophi

100 Monocytic cell differentiation potential was retained in

101 C/EBPalpha, which enabled the induction of a monocytic cell differentiation program.

102 d the effects of ex vivo-derived exosomes on monocytic cell differentiation/activation using THP-1 ce

103 fect of Panx1 deletion in endothelial and/or monocytic cells during atherogenesis is counterbalanced

104 respiratory epithelial cells and circulating monocytic cells efficiently transferred virions to T lym

105 We show that the donor monocytic cells engraft and persist (for up to 125 days)

106 elate with inefficient HIV-1 transmission by monocytic cells expressing DC-SIGN.

107 pon primary but not secondary stimulation of monocytic cells (FimA tolerance).

108 X-2 expression and PGE(2) production in THP1 monocytic cells following infection with Porphyromonas g

109 We found that migration of monocytic cells, formation of L. monocytogenes-containin

110 In particular, miR-10a was transferred to monocytic cells from EC-EVs and could repress inflammato

111 re normal in KLF4(-/-) chimeras, bone marrow monocytic cells from KLF4(-/-) chimeras expressed lower

112 In monocytic cells, geldanamycin (GA), an Hsp90 inhibitor,

113 present study was to examine changes in the monocytic cell gene-expression profile in response to C.

114 A new population of monocytic cells has been found to express LYVE-1 in norm

115 Monocytic cells have been found to be an important part

116 us cellular ecosystems, where non-neoplastic monocytic cells have emerged as key regulators of tumor

117 n gingival fibroblasts (HGFs) and U937 human monocytic cells; however, the responsible mechanisms hav

118 been implicated in neurotoxin production by monocytic cells (i.e., macrophages and microglia), as we

119 The reduction of monocytic cells in Ccr2(-/-) mice or after Gr-1 antibody

120 nsfusion of haematopoietic-stem-cell-derived monocytic cells in newborn mice is sufficient to rescue

121 r, our study reveals a dynamic modulation of monocytic cells in response to varying dosages of endoto

122 7 increases the number of mature myeloid and monocytic cells in spleen and peripheral blood.

123 pports a role for IL-27 in the activation of monocytic cells in terms of inflammatory responses.

124 ompanied by increased numbers of myeloid and monocytic cells in the bone marrow resembling the myelop

125 -type mice is associated with marked loss of monocytic cells in the bone marrow.

126 ecreted by primary human monocytes and THP-1 monocytic cells in the presence of alcohol in a concentr

127 aneously measure the mechanical stiffness of monocytic cells in three rotational and two translationa

128 in the gastrointestinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaini

129 in the gastrointestinal tract) and lymphoid/monocytic cells in tonsils and Peyer's patches (explaini

130 ctures in the ECM, providing a substrate for monocytic cells in vitro, whereas lung fibroblasts from

131 ts of DNA and DNA-coated beads by U937 human monocytic cells, in vitro.

132 g studies revealed SAMHD1 tetramers in human monocytic cells, in which it strongly restricts HIV-1 in

133 Monocytic cells, including macrophages and dendritic cel

134 Activation statuses of monocytic cells, including monocytes, macrophages (Mvarp

135 We found that cross-linking of CD13 on U937 monocytic cells induced phosphorylation of a number of p

136 hat M. tuberculosis infection of human THP-1 monocytic cells induced the down-regulation of CatG mRNA

137 n cellular iron release was examined in U937 monocytic cells induced to differentiate to the macropha

138 We observed spontaneous monocytic-cell infiltration in the lungs of Serpine2-def

139 model in which G-CSFR signals in bone marrow monocytic cells inhibit the production of trophic factor

140 n a mixed culture containing fibroblasts and monocytic cells, interferon-gamma stimulated Trx release

141 We showed that in monocytic cells KLF4 has to be repressed to allow their

142 g various phases of HIV-1 infection using U1 monocytic cells (latently infected U937 cells with HIV-1

143 stimulation of TLR2 in human epithelial and monocytic cells leads to rapid and transient activation

144 High glucose treatment of THP-1 monocytic cells led to a significant three- to fivefold

145 tional changes was carried out using a human monocytic cell line (THP-1 cells) in response to differe

146 to induce inflammatory responses in a human monocytic cell line (THP-1 cells).

147 ed with TLR2/4-mediated responses in a human monocytic cell line (THP-1) and in human primary monocyt

148 a fully endotoxin tolerant state in a human monocytic cell line (THP-1) and mouse bone marrow-derive

149 ecretion were conducted with the human THP-1 monocytic cell line and corroborated in primary human pe

150 r of cytokine signaling 1 (SOCS1) in a human monocytic cell line and in HEK293-TLR4/MD2 cells stably

151 Also, overexpression of miR-155 in the THP1 monocytic cell line decreases PU.1 protein levels and DC

152 d IL-12 production in a NEMO knockdown human monocytic cell line following LPS treatment.

153 T. gondii, we genetically engineered a human monocytic cell line for NALP1 gene knockdown by RNA inte

154 We report the establishment of a monocytic cell line latently infected with KSHV (KSHV-TH

155 we knocked down these proteins in the human monocytic cell line Mono Mac 6 (MM6).

156 of the pro-inflammatory cytokine IL-6 in the monocytic cell line Mono Mac 6, induction of the Toll-in

157 Similar regulation was observed in the monocytic cell line RAW264.7.

158 e resulting products against the THP-1 human monocytic cell line revealed hitherto unrecognized trend

159 y, we observe that depletion of BICD2 in the monocytic cell line THP-1 results in an induction of IFN

160 CD1d ligation on the human monocytic cell line THP-1 similarly specifically stimula

161 Here, we investigated the adhesion of the monocytic cell line THP-1 to a surface coated with inter

162 The human monocytic cell line THP-1 treated with lipopolysaccharid

163 -triggered antimicrobial activity, the human monocytic cell line THP-1 was infected with M. tuberculo

164 nalysis revealed that treatment of the human monocytic cell line THP-1 with LPS induced a rapid and t

165 IL-18BPa secretion in cultures of the human monocytic cell line THP-1, as measured by specific ELISA

166 nhibited CCL2-driven chemotaxis of the human monocytic cell line THP-1, CCL3-driven chemotaxis of per

167 Using the monocytic cell line THP-1, we show that angiocidin induc

168 phabeta signaling in human APC and the human monocytic cell line THP-1, which provides a model for th

169 A3A in CD14(+)-enriched primary cells or the monocytic cell line THP-1.

170 ing on E. chaffeensis infection of the human monocytic cell line THP-1.

171 osa-phagocyte interaction by using the human monocytic cell line THP-1.

172 to lipid rafts in blood monocytes and in the monocytic cell line THP-1.

173 q and bacA mutants was followed in the human monocytic cell line THP-1.

174 To model systemic effects, we used the human monocytic cell line THP-1; to model effects in the centr

175 vity in a time-dependent manner in the human monocytic cell line THP1.

176 broma cells stimulated chemotaxis of a human monocytic cell line to a greater extent than conditioned

177 uses a minimal defect in growth in the human monocytic cell line U937 and the environmental host Acan

178 trophils and diminished calcium signaling in monocytic cell line U937 transfected with the C5a recept

179 human cells, we stably transfected the human monocytic cell line U937 with murine IL-4R(alpha) cDNA b

180 creases TET2 protein expression in the human monocytic cell line U937.

181 A human monocytic cell line was genetically engineered using len

182 Knocking down DJ-1 (siRNA) in THP-1 (human monocytic cell line) and polymorphonuclear cells from pa

183 mooth muscle cells and THP-1 (human leukemia monocytic cell line) macrophages stimulated with lipopol

184 cytokine induced IL-6 expression in a human monocytic cell line, and (3) AhR-deficient mice are resi

185 In the THP-1 human monocytic cell line, reducing AIRE expression resulted i

186 In peripheral blood monocytes and the THP-1 monocytic cell line, SAA induces the expression of IL-12

187 We used a human monocytic cell line, THP-1, and dendritic cells to study

188 ome, but not the plasma membrane, in a human monocytic cell line, THP-1, and primary human monocyte-d

189 Using primary mouse microglia and the human monocytic cell line, THP-1, we have begun investigating

190 man airway smooth muscle cells and the human monocytic cell line, THP-1.

191 r in stably transfected CHO cells and in the monocytic cell line, THP-1.

192 Using a monocytic cell line, THP1, we show that LPS activates en

193 itself, on adhesion of THP-1 cells, a human monocytic cell line, to vascular endothelial cells (ECs)

194 At the molecular level, in a human monocytic cell line, U937 IL-10 suppressed LPS-induced m

195 Using a human monocytic cell line, we demonstrate that Porphyromonas g

196 an undifferentiated, normally nonpermissive monocytic cell line.

197 effects on migration of THP-1 cells, a human monocytic cell line.

198 MCs from patients with APECED syndrome and a monocytic cell line.

199 stimulated potent calcium flux in the human monocytic cell line.

200 tic differentiation using RAW264.7, a murine monocytic cell line.

201 eoclastogenesis in RAW 264.7 cells, a murine monocytic cell line.

202 ester accumulation in foam cells of the THP1 monocytic cell line.

203 ased consequent late cell death of the human monocytic cell line.

204 own in primary human microglia and the human monocytic cells line THP-1 cells, using diverse cell sti

205 transcription in microglial cells (HC69) and monocytic cell lines (U1 and OM10.1).

206 nse to various inflammatory stimuli in human monocytic cell lines and murine bone marrow-derived macr

207 induces less inflammatory signaling in human monocytic cell lines and murine macrophages than the par

208 lial cells and measured the binding of human monocytic cell lines and peripheral blood monocytes.

209 s of vitamin A, repress HIV-1 replication in monocytic cell lines and primary macrophages by blocking

210 vatives of the A549 lung carcinoma and THP-1 monocytic cell lines and used these to study T cell resp

211 neal exudate cells, and adoptive transfer of monocytic cell lines engineered to express the mutant CD

212 revealed that Bryo-1 treatment of the human monocytic cell lines MonoMac6 and THP-1 and human monocy

213 observed similar recombination rates in two monocytic cell lines regardless of the differentiation s

214 een A1PI expression in primary monocytes and monocytic cell lines, and inflammatory cytokine expressi

215 e suppression of HIV in human microglial and monocytic cell lines.

216 nduced by P. gingivalis LPS in a human THP.1 monocytic cell lines.

217 cycle in primary human T lymphocytes and in monocytic cell lines.

218 uence of HIV on global lncRNAs expression in monocytic cells lines.

219 ntiviral infection with activation status of monocytic cells may provide a means of potentiating anti

220 sue factor procoagulant activity (TF PCA) on monocytic cells more potently than other stimuli that de

221 port that STAT3 is activated in infiltrating monocytic cells near active MS lesions and that activati

222 ured by flow cytometry using uptake by THP-1 monocytic cells of fluorescent beads coated with gp120,

223 ortunately, the activation status of porcine monocytic cells or how cell activation status functional

224 tory cytokine release was evaluated in THP-1 monocytic cells or THP-1 cells lacking the inflammasome

225 ganism has been detected in peripheral blood monocytic cells (PBMCs).

226 Monocytic cells perform crucial homeostatic and defensiv

227 The discovery of this novel adipose tissue monocytic cell population provides advances toward under

228 Selective depletion of this monocytic cell population, using either clodronate-lipos

229 se receptors in establishing tissue-resident monocytic cell populations.

230 abetes resulted in increased accumulation of monocytic cells positive for S100A9, a proinflammatory b

231 erentially enhanced HIV-1 gene expression in monocytic cells rather than in T cells and that whereas

232 LPS (lipopolysaccharide)-stimulated monocytic cells release free mitochondria and microvesic

233 These monocytic cells represent one of the best-defined human

234 cation by phorbol ester in latently infected monocytic cells requires the posttranscriptional inducti

235 ntal antagonism of miR-452 in differentiated monocytic cells resulted in increased expression of MMP1

236 gest that M. tuberculosis infection of human monocytic cells results in a "cathepsin switch" with dow

237 a consequence, parabiosis or transfusion of monocytic cells results in long-term gene transfer in os

238 f infected human, primary, neuronal stem and monocytic cells revealed effects on neurodevelopment and

239 peripheral-blood mononuclear cells and THP-1 monocytic cells secreted TNF- alpha in response to cervi

240 Thus, incubation of U937 monocytic cells stably expressing L-selectin (U937LAM) w

241 ll-like receptor (TLR) 2 in human and murine monocytic cells stimulated with Borrelia burgdorferi.

242 s expression of APOL1 in differentiated U937 monocytic cells stimulated with IFN-gamma resulted in a

243 otein (AP)-1 was reduced in TLR2-neutralized monocytic cells, suggesting that AP-1 plays a role in th

244 plasminogen activator inhibitor-1 (PAI-1) by monocytic cells, suggesting that TSP1-induced macrophage

245 Enforced expression of human ABIN-3 in human monocytic cells suppressed the cytoplasmic degradation o

246 ) greater opsonization of these IEs by human monocytic cells than IgGs from malaria-exposed Malian me

247 linically characterized by overproduction of monocytic cells that can infiltrate organs, including th

248 ldren characterized by the overproduction of monocytic cells that infiltrate the spleen, lung, and li

249 olar and interstitial macrophages as well as monocytic cells that survey lung tissues.

250 ation was seen especially in lymphocytes and monocytic cells, the natural hosts for HIV-1 infection.

251 thia, stimulate cytokine production in human monocytic cells (THP-1) through Toll-like receptor 9 (TL

252 Monocytic cells (THP-1) were cultured in the presence of

253 ocaine treatment increased transmigration of monocytic cells through the HBMEC barrier.

254 aneous psoriatic lesions, and BMP7 instructs monocytic cells to acquire characteristics of psoriasis-

255 , we showed that Slit2 inhibited adhesion of monocytic cells to activated human endothelial cells, as

256 cs approach to assess the responses of human monocytic cells to Au-NPs of two different sizes with th

257 investigate the activation status of porcine monocytic cells to determine the intricate interaction o

258 nfluence the antigen presenting phenotype of monocytic cells to determine the nature of T cell respon

259 nt lipid mediators that activate THP-1 human monocytic cells to generate tumor necrosis factor alpha

260 aB epigenetically reprograms the response of monocytic cells to IFNgamma toward an immunosuppressive

261 th ALF significantly reduced the adhesion of monocytic cells to immobilized P-selectin.Fc.

262 rom erythroid cells, activated MNC and THP-1 monocytic cells to induce LT production.

263 ower concentrations of human GzmA stimulated monocytic cells to secrete proinflammatory cytokines (in

264 tease thrombin in regulating the adhesion of monocytic cells to smooth muscle cells producing an infl

265 n and the recruitment of bone marrow-derived monocytic cells to the hypothalamus; in addition, this i

266 rom CD11c(dim)CD11b(int)Gr1(-) lung-resident monocytic cells transformed by KRAS(G12D) In contrast, B

267 human epithelial pulmonary cells (A549) and monocytic cells (U937) upon IAV infection using synchrot

268 Cultured human monocytic cells, U937, were differentiated into macropha

269 pression and mechanism of their induction in monocytic cells under high-glucose conditions.

270 A and IL-15Ralpha appeared on the surface of monocytic cells upon activation.

271 These data combine to suggest that monocytic cells use a cytoplasmic retention mechanism to

272 apable of extensive replication within human monocytic cell vacuoles and induces apoptotic death via

273 ion-channel function of ATP receptor P2X7 in monocytic cells via nAChR containing alpha9 and alpha10

274 Inflammatory signaling in human THP-1 monocytic cells was much greater with pathogenic than wi

275 Furthermore, KYNA-triggered adhesion of monocytic cells was reduced by short hairpin RNA-mediate

276 study antiviral responses in PRRSV-infected monocytic cells, we characterized inflammatory cytokine

277 ions of pyrin and caspase-1 from THP-1 human monocytic cells were consistent with the interaction of

278 At 48 hpf, monocytic cells were evident in both the ICM and circula

279 CD19+ B cells and CD11c+ monocytic cells were found in the spleen, lung, liver, a

280 Inflammatory monocytic cells were not observed infiltrating the skin

281 the two genomovars was preserved when human monocytic cells were stimulated with purified LPS.

282 ll types, primary human CD4 T-cells and U937 monocytic cells, were used to compare differences in cyt

283 t in apoptosis in many cell types, including monocytic cells, whereas transient activation of P2RX7 i

284 te receptor ET-BR in Kupffer cells and THP-1 monocytic cells, which also involved HIF-1alpha activati

285 and other galactose-related genes in myeloid-monocytic cells, which could affect energy utilization a

286 rst genome-wide analysis of TNF tolerance in monocytic cells, which differentially inhibits NF-kappaB

287 Using RAW 264.7 mouse monocytic cells, which express endogenous MCH receptor,

288 n, it is dispensable in B, T, dendritic, and monocytic cells, which is in contrast with an essential

289 receptor, purinergic receptor X7 (P2RX7), on monocytic cells, which promotes the processing/release o

290 Infection of human THP-1 monocytic cells with a MYXV construct deleted for the M0

291 Treatment of monocytic cells with ALF for 2 h significantly reduced t

292 se to CCL5 demonstrated that pretreatment of monocytic cells with ALF reduced migration (p = 0.0004)

293 paB proteins before and after stimulation of monocytic cells with bacterial lipopolysaccharide (LPS).

294 nvolve iterative fusion of circulating blood monocytic cells with long-lived osteoclast syncytia.

295 upon T. gondii infection was not observed in monocytic cells with NALP1 knockdown.

296 stically reduced when they were derived from monocytic cells with nonrespiring mitochondria or monocy

297 vels are stable upon infection of human THP1 monocytic cells with Pg but decrease after cytokine indu

298 sialylation in macrophages, we treated U937 monocytic cells with PMA, which stimulates both macropha

299 We treated hepatic and extra-hepatic (monocytic) cells with exosomes +/- ALC/APAP.

300 pe 1 (HIV-1) coreceptors (CCR5 and CXCR4) by monocytic cells within human genital ulcers.