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1 mental MI can accelerate atherosclerosis via monocytosis.
2 00a8, or its cognate receptor Rage prevented monocytosis.
3 , with prefibrotic myelofibrosis or reactive monocytosis.
4 levels and subsequent hyperlipidemia-induced monocytosis.
5 am mice, neither diabetes nor MI resulted in monocytosis.
6  morphological features of myelodysplasia or monocytosis.
7 pment of severe anemia with macrocytosis and monocytosis.
8 ype mice with artificially induced Ly-6C(hi) monocytosis.
9 ral infections like Epstein-Barr virus cause monocytosis.
10 and in cDC1s, a mild neutrophilia and a mild monocytosis.
11  characterized by sustained peripheral blood monocytosis.
12 oietic hypocretin-receptor-null mice develop monocytosis and accelerated atherosclerosis, sleep-fragm
13 l monocytosis include clonal cytopenias with monocytosis and acute and chronic myeloid neoplasms.
14 nal whole blood leukocyte analysis indicated monocytosis and classical M1 phenotypic shifts.
15 cing panels that effectively identify clonal monocytosis and complement clinical prognostic scoring s
16 terleukin (IL)-2 production were followed by monocytosis and eosinophilia.
17              Burn sepsis induced circulating monocytosis and granulocytopenia.
18 nant condition that presents with persistent monocytosis and is often associated with leukocytosis, l
19                                Understanding monocytosis and its consequences in disease will reveal
20 oth CCL2 and CCL7 are required for efficient monocytosis and monocyte accumulation in the CNS, only C
21 rom two unrelated kindreds with intermittent monocytosis and mycobacterial disease, including bacillu
22 )/(-) mice fed a chow or Western- type diet, monocytosis and neutrophilia developed in association wi
23         We found that chronic stress induced monocytosis and neutrophilia in humans.
24 KO) BM-transplanted Ldlr(-/-) mice displayed monocytosis and neutrophilia in the absence of hematopoi
25 ases in IL-23 and IL-17A and proinflammatory monocytosis and neutrophilia that precedes development o
26        Stx type 2-triggered neutrophilia and monocytosis and polymorphonuclear neutrophil and monocyt
27 ted with a blunting of hyperglycemia-induced monocytosis and reduced monocyte recruitment to the plaq
28  a mouse model of human JMML equally reduces monocytosis and splenomegaly; however, the combined trea
29 phage proinflammatory responses, Ly-6C(high) monocytosis, and atherosclerosis in the settings of hype
30 ABCA1/ABCG1 and decrease cell proliferation, monocytosis, and atherosclerosis.
31 y via amelioration of hyperlipidemia-induced monocytosis, and can be augmented with a synthetic CXCR7
32 uppressed hyperglycemia-driven myelopoiesis, monocytosis, and neutrophilia.
33 (+/-) BM cells, led to increased WBC counts, monocytosis, and splenomegaly in WT recipient mice.
34 ow myeloid progenitor cell proliferation and monocytosis, as well as reduced atherosclerosis and a st
35                                          The monocytosis characteristic of older BXSB mice was also r
36     In contrast, Apoa1(-)/(-) mice showed no monocytosis compared with controls.
37      H. polygyrus infection induces systemic monocytosis contributing to elevated mononuclear phagocy
38                                    Ly-6C(hi) monocytosis disturbs resolution of inflammation in murin
39 2 mediates highly selective peripheral blood monocytosis during WNV infection of mice and that this i
40  the development of a macrocytic anemia with monocytosis, early features of t-MN.
41  cells revealed an "inflammatory" peripheral monocytosis enriched for the expression of S100A family
42 atory markers were largely nonspecific, with monocytosis frequently observed (n = 36/75; 48%).
43     Defined by a persistent peripheral blood monocytosis >/=1 x 10(9)/L and monocytes accounting for
44              Hypercholesterolemia-associated monocytosis (HAM) developed from increased survival, con
45 e/monocyte inflammatory activation patterns (monocytosis, high-inflammatory gene expression, raised g
46 reatment-induced resolution of lymphopaenia, monocytosis, hypercytokinaemia, and hyperchemokinaemia.
47 reatment each reduced HSPC proliferation and monocytosis in Apoe(-)/(-) mice.
48  not Ccr6(-/-) mice; moreover, Ccl20 induced monocytosis in ApoE(-/-) mice in vivo.
49 which prompts the question as to whether the monocytosis in childhood obesity contributes to atheroge
50  aim of this study was to test whether blood monocytosis in mice with atherosclerosis affects infarct
51 ) mice induced a strong and highly selective monocytosis in peripheral blood that was absent in Ccr2(
52 nk4b(fl/fl)-LysMcre mice develop nonreactive monocytosis in the peripheral blood accompanied by incre
53                             Causes of clonal monocytosis include clonal cytopenias with monocytosis a
54 gies of kidney injury associated with clonal monocytosis include direct renal infiltration by monocyt
55 neoplasm characterized by myelodysplasia and monocytosis, including but not limited to, chronic myelo
56 t high frequency in patients referred with a monocytosis, irrespective of diagnosis.
57 meaning that the distinction from a reactive monocytosis is challenging.
58           Pathomorphologic features included monocytosis, monocytoid blasts, aberrant expression of p
59 asm (MPN) overlap disorders characterized by monocytosis, myelodysplasia, and a characteristic hypers
60  autonomously to control HSPC proliferation, monocytosis, neutrophilia, and monocyte accumulation in
61 oduction, glomerulonephritis, and the unique monocytosis of BXSB males, were severely reduced or abse
62 feeding prompted mobilization culminating in monocytosis of chronologically older and transcriptional
63 nistic etiologies of kidney injury as clonal monocytosis of renal significance and provide guidance o
64 th healthy donors and patients with reactive monocytosis or another hematologic malignancy, CMML pati
65 cutive samples referred for investigation of monocytosis over a 2-year period (N = 283).
66 verse prognostic impact, whereas response of monocytosis proved to be a positive on-treatment paramet
67 ctional HDL levels in diabetic mice prevents monocytosis, reduces the quantity and inflammation of pl
68                                       Clonal monocytosis reflects a preneoplastic or neoplastic susta
69          In this review, we highlight clonal monocytosis-related etiologies that give rise to acute k
70 ced frequencies of marginal zone B cells and monocytosis, renal disease, and premature morbidity.
71  atherosclerotic mice with chronic Ly-6C(hi) monocytosis results in impaired healing, underscoring th
72  atypical MPN characterized by leukocytosis, monocytosis, splenomegaly, and progressive anemia.
73  that WNV infection induces a CCR2-dependent monocytosis that precedes monocyte migration into the CN
74                                Unexpectedly, monocytosis was more frequent in GATA2-mutated patients.
75                                  Significant monocytosis was observed at 24 hours (729 cells/muL [IQR
76                                   Lm-induced monocytosis was similar for wild-type and MSKO mice; how
77  attributed in part to hyperglycemia-induced monocytosis, which increases monocyte entry into plaques