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1 n PLRP2 hydrolyzed long-chain tri-, di-, and monoglycerides.
2 to a broader knowledge of immune modulating monoglycerides.
3 hibited greater hardness (5.9 N) than either monoglyceride (1.7 N), stearic acid (0.7 N) or carnauba
4 ides, and other minor ones, such as 1- and 2-monoglycerides, 1,2-diglycerides, vegetable stanols and
8 ysis reactions that lead to a reduction in 1-monoglycerides and an increase in 1,2-diglycerides, espe
10 ng the distribution of sterols, fatty acids, monoglycerides, and diglycerides that are not detected i
11 t was noticed, however, that in the thickest monoglyceride bilayer used in this study, both the SS- a
14 The differential effects of relatively thick monoglyceride bilayers on proton transfer in both dioxol
18 ith specific emulsifiers (citric acid esters monoglycerides (CITREM), whey protein isolate (WPI), and
21 presence of diacetyl tartaric acid ester of monoglycerides (DATEM) and glycerol monostearate (GM).
22 , ionization of ammonium adducts of diacetyl monoglyceride derivatives in positive-ion mode markedly
25 SFA intake on the fatty acid (FA) profile of monoglycerides, diglycerides and cholesteryl esters from
27 s of different natures, fatty acids, esters, monoglycerides, fatty amides, aldehydes, ketones, alcoho
28 hydrolysis products were 1,2-diglycerides, 2-monoglycerides, glycerol and fatty acids, although small
30 lication of this approach in the analysis of monoglycerides in multiple biologic tissues demonstrated
31 tep in triglyceride degradation, hydrolyzing monoglycerides into glycerol and fatty acids (FAs) and c
32 rol monolaurate (GML), a naturally occurring monoglyceride, is widely used commercially for its antim
38 cid amide hydrolase paralogs (FAAH1, FAAH2), monoglyceride lipase, N-acylethanolamine acid amidase, N
39 eroxisomal genes, such as CD36, Ly-6D, Rbp7, monoglyceride lipase, pyruvate dehydrogenase kinase-4, a
40 , which inhibits the 2-AG degradative enzyme monoglyceride lipase, restored both LPP-LTP and episodic
41 nterneurons; and 4) is metabolized by either monoglyceride lipase, which is located in the inhibitory
42 hrough measures of diacylglycerol lipase and monoglyceride lipase, which synthesize and degrade 2-ara
48 CUs) was gelled by adding 5% (w/w) saturated monoglycerides (MG), rice bran waxes (RW) or a mixture o
49 nol levels were gelled by using 10% (w/w) of monoglycerides (MG), rice wax (RW), sunflower wax (SW),
50 virgin olive oil (EVOO) was gelled with 10% monoglycerides, (MG), rice wax (RW), gamma-oryzanol, and
51 virgin olive oil (EVOO) was gelled with 10% monoglycerides, (MG), rice wax (RW), y-oryzanol, and B-s
55 markably, the regiospecificity of individual monoglyceride molecular species is also diverse from tis
57 toxicity of microemulsions composed of a 30% monoglyceride oil, 20% Tween 80 and 50% aqueous buffer w
58 The influence of palm oil replacement with a monoglyceride-palm oil-water gel (hydrogel) on physical
62 rofiling, identification and quantitation of monoglyceride regioisomers directly from tissue extracts
63 tion enables the differentiation of discrete monoglyceride regioisomers without chromatography throug
65 pase (MGL), a serine hydrolase that converts monoglycerides to fatty acid and glycerol, participates
66 ex endothelial cell glycerolipids, including monoglycerides, triglycerides, phosphatidylcholine, and
68 ternary phase diagram containing short chain monoglycerides was larger than for di- and triglycerides
70 tion can be absorbed intact, i.e. as the sn2 monoglyceride, whereas the sn1,3 fatty acids are absorbe
71 re formed from an appropriate combination of monoglyceride with various fatty acid lengths and solven