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1 a conserved organization of human and mouse mononuclear phagocyte system.
2 to unravel the functional complexity of the mononuclear phagocyte system.
3 of immune complexes (ICs) bearing C3 by the mononuclear phagocyte system.
4 ndency to be removed from circulation by the mononuclear phagocyte system.
5 its role to traffic immune complexes to the mononuclear phagocyte system.
6 inite period in tissues rich in cells of the mononuclear phagocyte system.
7 ted tissue, most often localized in cells of mononuclear phagocyte system.
8 ry plasticity in differentiated cells of the mononuclear phagocyte system.
9 en due to resident macrophages that form the mononuclear phagocyte system.
10 dence that dendritic cells are a part of the mononuclear phagocyte system and are derived from a comm
11 show minimal uptake of pegylated NCPs by the mononuclear phagocyte system and excellent blood circula
12 an ontogenically distinct population in the mononuclear phagocyte system and have implications for t
13 ays in which nanoparticles interact with the mononuclear phagocyte system and impact its function dur
14 on of liposomes and silicon particles in the mononuclear phagocyte system and improved tumoritropic a
15 in in circulation, minimize clearance by the mononuclear phagocyte system, and limit uptake in health
16 process in immunobiology, with cells of the mononuclear phagocyte system being critical in mediating
17 the complexity of a central component of the mononuclear phagocyte system by using a new in vivo appr
21 Brucella melitensis chronically infects the mononuclear phagocyte system in BALB/c mice, but it caus
22 sis, thus compromising the efficiency of the mononuclear phagocyte system in dealing with infection.
25 endritic cells (DCs) from other cells of the mononuclear phagocyte system is complicated by the share
27 nduced depression in IL-12 production by the mononuclear phagocyte system may promote T-cell commitme
28 ffect of polymer structure in modulating the mononuclear phagocyte system (MPS) accumulation of polyi
31 brane impermeability, enzymatic degradation, mononuclear phagocyte system (MPS) entrapment, fast rena
32 -1 particles effectively avoid uptake by the mononuclear phagocyte system (MPS) in vivo with a long b
36 ls and a high proportion of clearance by the mononuclear phagocyte system (MPS) of the liver and sple
38 Ps and AuNPs were primarily deposited in the mononuclear phagocyte system (MPS) such as the liver and
39 he innate immune system, particularly of the mononuclear phagocyte system (MPS), its ontogeny and pot
45 ifferentiation, and survival of cells of the mononuclear phagocyte system (MPS; progenitors, monocyte
46 vivo and their rapid uptake by cells of the mononuclear phagocyte system on intravenous administrati
50 factor receptor GFP transgene throughout the mononuclear phagocyte system), quantitative analysis of
51 ted hemoglobin modulates the response of the mononuclear phagocyte system to endotoxin, possibly thro
52 due to enhanced off-target clearance by the mononuclear phagocyte system, with elevated accumulation