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1 eiotic arrest genes, always early and cookie monster.
2 atural families, and are not involved in the monster.
3 nd map out some of the territories where lie monsters.
4 trum gregarium, popularly known as the Tully monster, a large soft-bodied organism from the late Carb
5 enhance resistance to the venom of the Gila monster, a toxic component of that venom (helodermin), a
7 ne the phylogenetic affinities of the 'Tully monster', and although it has been allied to such dispar
8 the discovery of new examples, including the monster, and six pariah groups which do not belong to an
9 with this idea, both always early and cookie monster are still expressed in flies deficient in vismay
10 ations support Alberch's ill-named "logic of monsters" - as a byproduct of strong developmental/topol
11 similar phenotypes, always early and cookie monster, as components of the machinery required for the
12 iological advantage of exendin-4 to the Gila monster, but for humankind GLP-1RAs are peptides with si
15 e deregulation leads to enlarged, misshapen "monster" cells with increased DNA content and apparent d
17 novel Drosophila meiotic arrest gene, cookie monster (comr), that has a mutant phenotype indistinguis
19 on/GLP-1 cDNAs and Southern blotting of Gila monster DNA demonstrate the coexistence of separate gene
22 isolated from the salivary gland of the Gila monster, Heloderma suspectum, are approximately 50% homo
24 ese specimens strongly support the "logic of monsters" hypothesis, in the sense that there is an 'ord
31 nstrous moonshine, which is an appearance of monster symmetry in number theory that catalysed develop
33 ogically unlikely, alternative is a "hopeful monster" that transforms in a single step from the ances
34 n-polymerase chain reaction of multiple Gila monster tissues identified approximately 500-base pair t
35 k on the vertex operator construction of the Monster to give a geometric definition of vertex operato
36 iting or stabilizing Always early and Cookie monster to specific target genes that need to be transcr
37 peptide-1 receptor agonist, GLP-1RA) in Gila monster venom may be regarded as one of the most serendi
38 re consistent with the observation that Gila monster venom PLA2 (Pa2), which is homologous to bvPLA2,