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1 d invasive tumors with high disease-specific mortality.
2 tubation, ICU length of stay, and short-term mortality.
3 e common and contribute to organ failure and mortality.
4 er when pathogens acted as codrivers of tree mortality.
5 ED-LOS, HLOS, complications, and in-hospital mortality.
6 hat induced significant short- and long-term mortality.
7 ignificant variation was found for perinatal mortality.
8 cirrhosis with equivalence to LB in terms of mortality.
9 a lower risk of all-cause and cardiovascular mortality.
10 itation, plant compositional shifts and tree mortality.
11 ansmission lines, are substantial sources of mortality.
12 and the secondary outcome was cause-specific mortality.
13 Crs was independently associated with higher mortality.
14 tive defense strategy against viral-mediated mortality.
15 he PBS was not independently associated with mortality.
16 raft failure cause significant morbidity and mortality.
17 ns that most cost-effectively reduce PM(2.5) mortality.
18 k for a first hepatic decompensation and for mortality.
19 E hospitalizations, and 36% (IQR, 28-46) AGE mortality.
20 may be transient due to lagged increases in mortality.
21 y is associated with increased morbidity and mortality.
22 n expression, bacterial burden, and neonatal mortality.
23 cal care that directly affects morbidity and mortality.
24 ause a vast majority of cancer morbidity and mortality.
25 risk factor for cardiovascular morbidity and mortality.
26 extracutaneous manifestations, morbidity and mortality.
27 serious bleeding and increased morbidity and mortality.
28 OVID-19 to help predict disease severity and mortality.
29 is associated with substantial morbidity and mortality.
30 risk-adjusted predictors of post-transplant mortality.
31 , five for increased intervention-associated mortality.
32 a cytokine storm may increase morbidity and mortality.
33 is associated with substantial morbidity and mortality.
34 AVR, TAVR utilization rates, and in-hospital mortality.
35 ensity of stepping are associated with lower mortality.
36 ctors and phenotypes and between factors and mortality.
37 be vulnerable to COVID-related morbidity and mortality.
38 d to examine the association of ECLS use and mortality.
39 The primary endpoint was all-cause mortality.
40 were cardiovascular disease (CVD) and cancer mortality.
41 ibute substantially to overall morbidity and mortality.
42 personalized treatment regimens could reduce mortality.
43 diagnosis is an important cause of its high mortality.
44 th renal function decline and cardiovascular mortality.
45 management, respiratory failure, and patient mortality.
46 antioxidants was seen with CVD and all-cause mortality.
47 en the number of risk factors and subsequent mortality.
48 There was no toxic mortality.
49 rder to curb iatrogenic opioid morbidity and mortality.
50 e proportion of the population, causing high mortality.
51 cumulation of chronic diseases and all-cause mortality.
52 on associated with significant morbidity and mortality.
53 ted with increased vascular permeability and mortality.
54 erence between groups, including in-hospital mortality (1.7% for uncemented fixation vs 2.0% for ceme
55 for the background population; and all-cause mortality: 10.88% (95% CI: 10.23% to 11.55%) for sarcoid
59 ; 95% CI, -0.1 to 0.2; P = .58) or in 30-day mortality (34.7% vs 29.3%, respectively; hazard ratio, 1
61 le (ie, highest mortality) of fully adjusted mortality, 60% were located in 3 states: Oklahoma, Texas
62 rval: 0.68 to 0.88; p < 0.001) and all-cause-mortality (7.6% vs. 9.7%; adjusted hazard ratio: 0.61; 9
63 h COVID-19 who received ECMO, both estimated mortality 90 days after ECMO and mortality in those with
65 strated a significant reduction in all-cause mortality (ACM) risk with fluticasone furoate/umeclidini
66 omes did not show significant differences in mortality across teaching and nonteaching environments,
67 KI-RRT patients had lower rates of long-term mortality (adjusted hazard ratio (HR), 0.473; 95% CI, 0.
68 ype interaction predicting time to all-cause mortality, adjusted for Meta-Analysis Global Group in Ch
69 nts with T2MI had higher long-term all-cause mortality after adjustment for age and sex, driven by ea
70 remains an important cause of morbidity and mortality after allogeneic hematopoietic cell transplant
71 aimed to report postoperative morbidity and mortality after esophagectomy and gastrectomy in the Net
72 a chemoprevention could reduce morbidity and mortality after hospital discharge in children younger t
73 %) were the most robust predictors of 1-year mortality after incident and repeated HFHs, respectively
74 lure (RVF) is a cause of major morbidity and mortality after left ventricular assist device (LVAD) im
76 ed hazard ratio (aHR) for MACCE or all-cause mortality (aHR, 1.71 [1.13-2.60]; P = 0.012) even after
78 no device, was associated with lower risk of mortality, all-cause hospital admission, and intubation,
80 n, and more intensive antifungals may reduce mortality among asymptomatic CrAg-positive patients iden
81 a worthwhile strategy for preventing CVD and mortality among older Mexican Americans with insufficien
82 conditions and all-cause and cause-specific mortality among survivors of early-adolescent and young
84 U survivor care process results in decreased mortality and a net annual cost savings to the insurer c
85 od concentrations are associated with higher mortality and a poorer physiological state, and (3) C15:
86 RBC transfusion correlates with increased mortality and acute kidney injury early after transcathe
90 was conducted to describe the trajectory of mortality and health-related quality of life morbidity f
91 class on presentation, and the end points of mortality and heart failure admissions in the CASTLE-AF
93 nakebite is a medical emergency causing high mortality and morbidity in rural tropical communities th
98 uptake, a primary determinant of prognosis, mortality and quality of life, is diminished in patients
99 en were limited to prevented cases of infant mortality and respiratory illnesses, with a monetized im
100 between handgun ownership and both all-cause mortality and suicide (by firearm and by other methods)
101 cluding a group closest to HFrEF with higher mortality, and a mostly female group with smaller hearts
103 e pregnancy outcomes, maternal TB, all-cause mortality, and liver injury during pregnancy through 12
104 on observed waitlist registrations, waitlist mortality, and living-donor and deceased-donor kidney tr
105 ate, intensive care unit mortality, hospital mortality, and physical function- and mental health-rela
106 ciation between BCG vaccination and COVID-19 mortality, and suggest that BCG could have a protective
108 -90 treatment targets on HIV-1 incidence and mortality, and to assess whether these interventions wil
109 link between regular yogurt consumption and mortality appears plausible, data are sparse and have yi
110 ment in a state with a high rate of dialysis mortality are at a higher risk for transplant failure co
112 ry disease was not associated with increased mortality, as has been suggested for peripheral arteries
113 the virus and the high rate of morbidity and mortality associated with COVID-19, developing effective
114 flated) estimate for the average increase in mortality associated with each hour until antibiotics, a
118 male sex was not an independent predictor of mortality at 10 years in patients with complex coronary
119 pothermia was associated with an increase in mortality at longest follow-up available (432/1,375 [31%
120 untries with large annual reductions in such mortality between 1990 and 2017 had achieved low levels
121 true magnitude of difference in HIV-related mortality between men and women receiving antiretroviral
122 nin-guided therapy trials have shown similar mortality, but the essential question is whether the sen
123 adiposity are independently associated with mortality, but there is no consensus on how best to asse
124 p<0.001) with a relative decrease in 30-day mortality by -1.6% (95% CI, -3.4 to -0.2, p = 0.039) and
125 (95% CI, -3.4 to -0.2, p = 0.039) and 90-day mortality by -2.3% (95% CI, -4.3 to -0.4, p = 0.018).
126 0, scaling up vaccination alone would reduce mortality by 61.7% (61.4-66.1), averting 4.8 million (4.
136 94 [95% CI, 0.73-1.21]; P = .61) and overall mortality (cumulative incidence at 1 year after operatio
140 000-2003 and 2012-2015, the adjusted overall mortality decreased by 24% (hazard ratio [HR] per year 0
142 Meta-regression of the effect of dose on mortality did not reveal an association, but reported do
143 For chronic limb-threatening ischemia, the mortality difference was not significant; paclitaxel (12
144 cally, tracheostomy complications, inpatient mortality, disposition of patients, and transmission of
145 rrence-free survival (RFS), disease-specific mortality (DSM), and time-to-recurrence, were reported.
146 n their vulnerability to different causes of mortality due to traits, life history stages, or locatio
147 nic inflammation and increased morbidity and mortality during antiretroviral-treated HIV disease.
150 tients with septic shock), blending together mortality estimates from patients with very long interva
154 cess in composite 30-day HF readmissions and mortality for Black patients ranged from 3.9% (95% CI, 1
155 relationship between time-to-antibiotics and mortality for patients with possible sepsis is therefore
160 h presumptive COVID-19 and low likelihood of mortality from STEMI and use of preventive strategies su
161 r profile, the absolute age-standardized CVD mortality gap would decline by 33.3% (95% CI 25.1-40.1)
165 agrelor significantly reduced cardiovascular mortality (hazard ratio [HR], 0.82 [95% CI, 0.72-0.92])
166 was significantly associated with all-cause mortality (hazard ratio [HR], 1.49 [CI, 1.15 to 1.94]) a
167 te of myocardial infarction, stroke, and CVD mortality; hazard ratio [HR], 0.92 [95% CI, 0.80-1.06])
168 strength, delirium rate, intensive care unit mortality, hospital mortality, and physical function- an
169 HR], 0.82 [95% CI, 0.72-0.92]) and all-cause mortality (HR, 0.83 [95% CI, 0.75-0.92]), whereas there
170 1.49 [CI, 1.15 to 1.94]) and cardiovascular mortality (HR, 1.66 [CI, 1.07 to 2.57]) in participants
171 nts' MELD) was associated with postoperative mortality (HR: 8.027; 95% CI: 2.387-18.223; P = 0.026) a
172 aminase (ALT) and clinical liver disease and mortality in 111,612 individuals from the Danish general
177 The FLIS was an independent risk factor for mortality in both patients with CACLD (adjusted hazard r
183 RS were moderately associated with all-cause mortality in independent data within the UK Biobank: the
190 emia ratio was significantly associated with mortality in patients with glycosylated hemoglobin less
193 Results were also null for cardiovascular mortality in the 2 external cohorts (eg, HRs of IDH by t
194 risk factors predisposing to severe disease/mortality in the general population also seemed to affec
197 -19) pandemic has led to a large increase in mortality in the United States and around the world, lea
198 ve alcohol use is the third leading cause of mortality in the United States, where alcohol use consis
200 on tension was independently associated with mortality in this cohort of venoarterial extracorporeal
201 h estimated mortality 90 days after ECMO and mortality in those with a final disposition of death or
202 20 combinations most highly associated with mortality in UK Biobank and 12 of the top combinations i
203 est the implications of parasites for annual mortality in wild bird populations using phylogenetic co
204 tree was constructed with associated 2-year mortality incorporating an LB or NIT strategy to diagnos
205 periencing a complication, as well as 30-day mortality, independent of other hospital level character
206 nt and is an independent predictor of 30-day mortality, irrespective of periprocedural major bleeding
207 th acute kidney injury (AKI) and the risk of mortality is high, especially if renal replacement thera
209 d sex and the composite outcome of all-cause mortality, LV assist device implantation, or heart trans
210 mplications in determining urgency of LT and mortality models in cirrhosis and LT waitlisting, especi
211 pose that to understand causes of sex-biased mortalities, more complex analyses are needed that incor
215 mitted stroke patients was high with 30 days mortality of 31% in ischemic stroke and 42% in intracere
216 -based estimate of on-board and post-release mortality of bycaught marine turtles that has until now
217 timated harvest and background (other cause) mortality of landlocked migratory salmonids over half a
222 COVID-19 pandemic (compared with background mortality) of 1.5, 2.0, and 3.0 at differing infection r
223 ies in the top fifth percentile (ie, highest mortality) of fully adjusted mortality, 60% were located
224 ionally, we analyzed the effects of drought- mortality on second-entry burn emissions and compared em
226 sis among patients with hypertension or with mortality or severe disease among patients diagnosed as
227 67 was also associated with increased 30-day mortality (OR, 8.4 [95% confidence interval, 2.23-31.7])
228 spital mortality (29.75% vs 21.1%), combined mortality, or discharge to hospice (37.2% vs 25.3%), ext
229 sociated with significant decrease in day 30 mortality over time (odds ratio, 0.96; 95% CI, 0.93-0.98
230 tion)<0.0001; 1.9% versus 0.6% for all-cause mortality, P(interaction)=0.02; 2.7% versus 1.7% for maj
231 he magnitude of sex differences in mammalian mortality patterns is likely shaped by local environment
233 overall survival, with a 2-fold decrease in mortality rate (HR, 0.50; 95% CI, 0.27 to 0.90), were ob
236 Consistent with late presentations, the mortality rate was high, whereas frequencies of LVRR and
238 quiring two treatments (ii) complication and mortality rates (iii) local and distant recurrence (LR),
239 64 days), child (age 1-4 years), and under-5 mortality rates (U5MRs) for each ethnic group within eac
250 ereas there was no statistically significant mortality reduction with prasugrel (HR, 0.90 [95% CI, 0.
252 D-19 pandemic and to closely examine the Crs-mortality relationship.Methods: We undertook a secondary
253 ore per nurse ratio on day 1 and in-hospital mortality remained significant (odds ratios, 1.29 and 1.
256 e mortality decreased 16.4% across all ages, mortality resulting from CHD declined 39.4% overall.
257 ]: 2.493.494.89, P < 0.001), but a 62% lower mortality risk (aHR: 0.310.380.46, P < 0.001) beyond thi
259 Higher nut intake was associated with lower mortality risk from both cardiovascular and noncardiovas
260 stic value, although its capacity to predict mortality risk in HIV-HCV-coinfected patients has never
263 ed with expected population survival, excess mortality risks of pure AR, AR + OMR, and AR + FMR were
266 days after symptom onset led to lower 90-day mortality than use of placebo (relative risk, 0.19; 95%
267 jury remains a major driver of morbidity and mortality, the ability to accurately identify patients a
268 an established risk factor for morbidity and mortality, the minimum amount of weight loss to have a m
271 hese parasites can cause major pathology and mortality to livestock leading to significant welfare an
272 llapsibility were independent predictors for mortality, together with base excess and Glasgow Coma Sc
273 fered among family lineages, and the highest mortality under OA occurred in the fastest growing cross
274 nal observational study of COVID-19 hospital mortality using data from the SIVEP-Gripe (Sistema de In
275 short-term (mean 5 y, range 1 to 23 y after mortality) vegetation-type conversion in multiple biomes
276 timately, C starvation may lead to increased mortality vulnerability, but hydraulic failure or biotic
278 obactam for both endpoints: day 28 all-cause mortality was 15.9% with imipenem/cilastatin/relebactam
282 t quintiles were compared; however, neonatal mortality was significantly associated with SO2 (1.207 [
283 of follow-up (n = 1,775, 9 RCTs), all-cause mortality was significantly lower in the DCB group when
286 mission sector contribution to PM2.5-related mortality, we found that reductions in sulfur-dioxide em
289 stay (HLOS); complications; and in-hospital mortality were compared before (PRE) and after (POST)imp
290 tive incidence of oncologic and nononcologic mortality were compared using competing risks approaches
291 I 1 year and beyond, the effects on MACE and mortality were consistent irrespective of time since las
293 Factors significantly associated with 90-day mortality were: P: age, gender and ACLF type; I: drug, i
294 1) were independently associated with day 30 mortality, whereas underlying malignancy, allogeneic hem
296 0.93]) had the best predictive value for ICU mortality with cutoff values less than or equal to 1.25
298 , 0.96-0.97) lower adjusted critical illness mortality within a non-minority-serving hospital, but no
299 ic ethanol consumption is a leading cause of mortality worldwide, with higher risks to develop pulmon
301 le (i.e. high bacillary load) had 57% 2-week mortality; worse than the intermediate (17%) and high (2