戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 d why DENV reconfigures phospholipids in the mosquito vector.
2 ical role of the selective advantages in the mosquito vector.
3  formation and sporogonic development in the mosquito vector.
4  the microfilaria reservoir available to the mosquito vector.
5 n in humans and pathogen transmission to the mosquito vector.
6 cle of Plasmodium in the vertebrate host and mosquito vector.
7  the male gamete in the midgut of the female mosquito vector.
8 a persistent, nonpathogenic infection in the mosquito vector.
9  but essential for male gametogenesis in the mosquito vector.
10 , nor for gametogenesis and sporogony in the mosquito vector.
11 sion of parasites from the human host to the mosquito vector.
12 that are responsible for transmission to the mosquito vector.
13 ria parasite and occurs in the midgut of the mosquito vector.
14 cal interactions with the blood meal and the mosquito vector.
15 e population losses as it passes through its mosquito vector.
16 the difficulty of sustainable control of the mosquito vector.
17  parasites undergo a sporogonic cycle in the mosquito vector.
18 ametocytes, the only stages infective to the mosquito vector.
19 lead to infection even in the absence of the mosquito vector.
20 ncreasing due to geographic expansion of the mosquito vector.
21 y wide feeding host-range for the Haemagogus mosquito vector.
22  stages of parasite transmission through the mosquito vector.
23 hosts: a vertebrate and the female Anopheles mosquito vector.
24 e a platform to further study this important mosquito vector.
25 eld strain (FS) of Aedes aegypti, the dengue mosquito vector.
26 brate host and persistent replication in the mosquito vector.
27 icative sexual stage necessary to infect the mosquito vector.
28  completion of Plasmodium development in the mosquito vector.
29 tes and the ookinetes and sporozoites of the mosquito vector.
30 erythrocytes and hepatocytes, as well as the mosquito vector.
31 s a complex life cycle in a human host and a mosquito vector.
32 ission while minimizing fitness costs to its mosquito vector.
33 dition, was essential for development in the mosquito vector.
34 es, called gametocytes, are infective to the mosquito vector.
35 , oocyst and sporozoite formation inside the mosquito vector.
36 ular invasive stages that develop within the mosquito vector.
37 gional differences in the human host and the mosquito vector.
38 ol is reliant on insecticides to control the mosquito vector.
39 s with changing climatic suitability for the mosquito vector.
40 ed by insecticide resistance in some malaria mosquito vectors.
41 fitness, and likely would not be purified by mosquito vectors.
42 so block Plasmodium parasite transmission to mosquito vectors.
43 s essential for efficient virus infection of mosquito vectors.
44 s on the attractiveness of infected hosts to mosquito vectors.
45 must reduce malaria transmission through the mosquito vectors.
46 cytes of Anopheles gambiae and Aedes aegypti mosquito vectors.
47 smodium falciparum sexual stage parasites to mosquito vectors.
48 n feeding behavior and climate adaptation of mosquito vectors.
49 resistant strains might be more infective to mosquito vectors.
50 rotropic flavivirus transmitted to humans by mosquito vectors.
51 develop a persistent, life-long infection in mosquito vectors.
52 he coevolution between malaria parasites and mosquito vectors.
53 plification hosts for efficient infection of mosquito vectors.
54 remia that results in efficient infection of mosquito vectors.
55 fication of the Anopheles gambiae complex of mosquito vectors.
56 ndscape and abundance of medically important mosquito vectors.
57 bout how Plasmodium parasites adapt to their mosquito vectors.
58 ilability of high-quality genomes of several mosquito vectors.
59 tat conditions on the shape of the virome in mosquito vectors.
60  the way towards using CI to control malaria mosquito vectors.
61 lating chemosensory function and controlling mosquito vectors.
62 smissible stage than the average lifespan of mosquito vectors.
63  more rapidly than behavioural shifts within mosquito vectors.
64 y inadvertently promote the proliferation of mosquito vectors.
65 ce, and lack of transmissibility in the main mosquito vectors.
66 o block Plasmodium transmission to Anopheles mosquito vectors.
67 ed livestock products or multiple species of mosquito vectors.
68 n for up to 3 months without the presence of mosquito vectors.
69 ed vertically and sexually in addition to by mosquito vectors.
70 rtially restore pyrethroid susceptibility in mosquito vectors.
71 al network connecting flaviviruses and their mosquito vectors.
72 ion is the effective management of Anopheles mosquito vectors.
73 n monitoring and modeling arboviruses within mosquito vectors.
74 al prevalence of dengue virus (DENV) and its mosquito vectors.
75 tial to discover novel feeding deterrents of mosquito vectors.
76 , their management relies on controlling the mosquito vectors.
77 iology, evolution, and dispersal of the main mosquito vectors.
78 ad by the Aedes aegypti and Aedes albopictus mosquito vectors.
79 and use them to infect native and non-native mosquito vectors.
80 e first in the United States to characterize mosquito vector abundance and diversity on commercial sw
81                            In females of the mosquito vector Aedes aegypti (L.), aedeskinins are know
82 incompatible insect technique (IIT) with the mosquito vector Aedes aegypti in northern Australia in a
83 igenaemia determines ZIKV infectivity in its mosquito vector Aedes aegypti, which acquires ZIKV via a
84  viruses (VEEV) readily infect the epizootic mosquito vector Aedes taeniorhynchus.
85 EN) that are transmitted among humans by the mosquito vectors Aedes aegypti and Aedes albopictus are
86          Dengue virus (DENV) and its primary mosquito vectors Aedes spp. have spread to every contine
87  restricted range of movement of the primary mosquito vector, Aedes aegypti, local human movements ma
88  for its efficient maintenance in the dengue mosquito vector, Aedes aegypti.
89 otypically structured model for the invasive mosquito vector, Aedes albopictus, and dengue, the secon
90 approach to control DENV transmission by the mosquito vectors, Aedes aegypti and Aedes albopictus, us
91 ns worldwide, and the expanding range of the mosquito vector, affected in part by climate change, inc
92 n and genotype-phenotype associations in the mosquito vector, along with sexual reproduction, have hi
93  primarily transmitted by Aedes aegypti, the mosquito vector also important in transmission of the fl
94   Here, we use a single pairing of the Asian mosquito vector, An. stephensi and the human malaria par
95 ct of a complex developmental process in the mosquito vector and are destined to infect the mammalian
96 nthase completely blocks transmission to the mosquito vector and could potentially be targeted for di
97 equisite for transmission from humans to the mosquito vector and has emerged as a target for interven
98 at body of knowledge about the parasite, its mosquito vector and human host.
99 e key ecological links between the Aedes sp. mosquito vector and humans.
100 opmental stages of the parasite, both in the mosquito vector and in the human host.
101 ell invasion and replication within both the mosquito vector and mammalian host.
102  its effects on the population growth of the mosquito vector and on pathogen development within the v
103 e host, through the different tissues of its mosquito vector and onwards to infect a new vertebrate h
104           Interactions between the Anopheles mosquito vector and Plasmodium parasites shape how malar
105 for malaria control in the mammalian and the mosquito vector and provide a molecular basis for the st
106  to reduce malaria parasite infection in the mosquito vector and provide new insight into the mechani
107 l factors, including increased spread of the mosquito vector and rapid urbanization.
108 , successively invade salivary glands in the mosquito vector and the liver in the mammalian host.
109 oites must infect the salivary glands of the mosquito vector and the liver of the mammalian host; bot
110 site that invades the salivary glands of the mosquito vector and the liver of the vertebrate host, ex
111              During their passage within the mosquito vector and the vertebrate host, sporozoites dis
112 amics in that it is concurrently spread by a mosquito vector and through sexual contact.
113 geneity, attributable to the dynamics of the mosquito vector and to the characteristics and mobility
114 ity for surveillance of WN virus activity in mosquito vectors and avian hosts, and, in addition, it i
115  malaria transmitted by pyrethroid-resistant mosquito vectors and could thus be a crucial addition to
116 d that WNV is well adapted to the ecology of mosquito vectors and diverse avian hosts in the United S
117 al heterogeneity on the contact rate between mosquito vectors and hosts.
118 ria transmission by limiting contact between mosquito vectors and human hosts when mosquitoes feed du
119                               The ecology of mosquito vectors and malaria parasites affect the incide
120 e result of parasite adaptation to sympatric mosquito vectors and may be an important factor driving
121  our understanding of viral diversity within mosquito vectors and provides information that opens the
122 cific region where the presence of competent mosquito vectors and suitable climatic conditions could
123 insecticide resistance that has developed in mosquito vectors and the drug resistance of pathogens.
124 ave facilitated the spread of both efficient mosquito vectors and the four dengue virus serotypes bet
125 e these viruses incapable of transmission by mosquito vectors and to differentially regulate expressi
126                           The spread of both mosquito vectors and viruses has led to the resurgence o
127  cell invasion and block transmission to the mosquito vector, and to vaccines to other extracellular
128 piled infection data from sentinel chickens, mosquito vectors, and human cases in Iowa over a 15 year
129 ria and the greatest diversity of parasites, mosquito vectors, and human victims.
130  glaciation, increased anthropophilia of the mosquito vectors, and the spread of agriculture.
131 the same natural macaque hosts and Anopheles mosquito vectors, and therefore have a similar epidemiol
132 smodium parasite transmission by the African mosquito vector Anopheles gambiae depends on finely tune
133 asite-resistance island (PRI) of the African mosquito vector, Anopheles gambiae, was mapped to five g
134  implications of a range expansion of dengue mosquito vectors are severe.
135 an/reptilian Plasmodium parasites, spread by mosquito vectors, are ancestral sister taxa, from which
136 rong surveillance system for malaria and its mosquito vector as an essential pillar of the malaria el
137 species, and could be transferred into other mosquito vectors as part of control programs.
138 sistance to pathogens, or even eliminate the mosquito vectors, as a means of control.
139 rrent malaria-control methods, several novel mosquito vector-based control strategies have been propo
140 he parasite stages transmitted to and by the mosquito vector because they may represent more vulnerab
141 acilitated virus acquisition by their native mosquito vectors because the protein enabled the virus t
142              Blocking these pathogens in the mosquito vector before they are transmitted to humans is
143                           Given that malaria mosquito vectors belong to a species complex comprising
144 d animal pathogens, which are transmitted by mosquito vectors between vertebrate hosts.
145  on the regulatory elements is available for mosquito vectors but is scant for other vectors.
146 ansion is attributed to the success of Aedes mosquito vectors, but local epidemiological drivers are
147 laria is transmitted from vertebrate host to mosquito vector by mature sexual blood-living stages cal
148  Dengue Virus (DENV), a flavivirus spread by mosquito vectors, can cause vascular leakage and hemorrh
149 ite's interplay with both the human host and mosquito vector cannot be underestimated.
150 nding of the roles of mosquito phagocytes in mosquito vector competence and demonstrates the utility
151 ssible biological significance and effect on mosquito vector competence for arboviruses.
152 les of apoptosis and caspases in determining mosquito vector competence for arboviruses.
153 veral insect pest species, but its effect on mosquito vector control has been limited.
154 rea-wide application of combined IIT-SIT for mosquito vector control.
155  the orthologues of mosGCTL in another major mosquito vector (Culex pipiens pallens) also impairs the
156 ture and moisture are favorable for the sole mosquito vector, Culex quinquefasciatus, and extrinsic s
157  that the viruses initially delivered by the mosquito vector differ from those generated in subsequen
158 the successful reduction of infection in the mosquito vector, direct evidence that there is an onward
159  novel approaches to combating the spread of mosquito-vectored diseases.
160 in parts of Africa where climate drives both mosquito vector dynamics and parasite development rates.
161 ite drug resistance and host susceptibility, mosquito vector ecology and transmission seasonality.
162 ansition through multiple mammalian host and mosquito vector environments.
163 ent and more recent human interactions shape mosquito vector evolution.
164 risk of dengue transmission should competent mosquito vectors expand their range.
165 ia parasites occurs when nocturnal Anopheles mosquito vectors feed on human blood.
166                    The wide endemic range of mosquito-vectored flaviviruses-such as Zika virus and de
167 l and apply it to Aedes aegypti, an invasive mosquito vector for arboviruses (e.g. dengue, zika and y
168  Aedes aegypti (Linnaeus, 1762), the primary mosquito vector for dengue and other arboviral pathogens
169                 Here, we show that the major mosquito vector for dengue virus uses the JAK-STAT pathw
170 site Brugia malayi that requires a competent mosquito vector for its development and transmission.
171               Aedes aegypti is the principal mosquito vector for many arboviruses that increasingly i
172 m parasites must complete development in the mosquito vector for transmission to occur.
173                                          The mosquito vectors for this virus are globally distributed
174                                              Mosquitoes vector harmful pathogens that infect millions
175 ia ookinetes to cross the midgut wall of the mosquito vector has been (and continues to be) controver
176              Surveillance of avian hosts and mosquito vectors has been insufficient to elucidate the
177 tomics and the complete genome sequencing of mosquito vectors have increased our knowledge of the SG
178 erefore, we hypothesized that changes in the mosquito vector host range might have contributed to the
179 e part to its replication in and spread by a mosquito vector host.
180  the infective stage injected by bite of the mosquito vector, however, whole parasite vaccines presen
181 inea, where the level of transmission by the mosquito vector, human infection rates and clinical morb
182                 In addition to spreading via mosquito vectors, human WEEV infections can potentially
183 evented parasites from developing within the mosquito vector in standard membrane feeding assays.
184 e and therefore drive the persistence of the mosquito vector in the context of ZIKV infection.
185 e a measurable effect on transmission by the mosquito vector in vivo.
186  data relating to monkey hosts and Anopheles mosquito vectors in Indonesian Borneo.
187 diagnosis but also virologic surveillance of mosquito vectors in the field.
188 h it is transmitted, and the role of various mosquito vectors in the recent outbreaks.
189 sage has resulted in increased resistance in mosquito vectors, including C. quinquefasciatus.
190 ributions, and are strongly predicted by the mosquito vector index.
191 ria, undergo crucial developments within the mosquito vector, initiated by the formation of male and
192 e Plasmodium falciparum within a susceptible mosquito vector is a prerequisite for the transmission o
193 he life cycle of the malaria parasite in its mosquito vector is essential for developing new strategi
194                                         This mosquito vector is highly domesticated, living in close
195  we consider important because combating the mosquito vector is the only way to contain dengue transm
196 virus infection in southeast Asia, where the mosquito vector is widespread and other arboviruses circ
197 comprehensive understanding of the virome in mosquito vectors is crucial for assessing the potential
198 , close surveillance in animals, humans, and mosquito vectors is necessary to detect the presence of
199          As pyrethroid resistance evolves in mosquito vectors, it will be useful to understand how th
200                        It circulates through mosquito vectors mainly of the Aedes family and a mammal
201 lection for efficient infection of epizootic mosquito vectors may mediate VEE emergence.
202 n, the virus quickly adapted to infect local mosquito vectors more efficiently than the originally in
203 ons do not naturally occur in some important mosquito vectors, notably Aedes aegypti.
204                              Saliva from the mosquito vector of flaviviruses is capable of changing t
205 s of Anopheles stephensi, a major anopheline mosquito vector of human malaria in Asia.
206 d humidity adaptation of A. gambiae, a major mosquito vector of human malaria in sub-Saharan Africa.
207 e, and Anopheles gambiae, the most important mosquito vector of malaria in sub-Saharan Africa.
208  Anopheles gambiae, which has been the major mosquito vector of the malaria parasite Plasmodium falci
209                              We find a major mosquito vector of these viruses-Aedes aegypti-can carry
210 the southern house mosquito) is an important mosquito vector of viruses such as West Nile virus and S
211 cits a response from many insects, including mosquito vectors of diseases such as malaria and yellow
212 ic modification of the vectorial capacity of mosquito vectors of human disease requires promoters cap
213 standing regulation of gene transcription in mosquito vectors of human diseases.
214 s gambiae species complex, the major African mosquito vectors of human malaria.
215 ration of synthetic repellents against major mosquito vectors of infectious diseases.
216         During the long Sahelian dry season, mosquito vectors of malaria are expected to perish when
217                                              Mosquito vectors of malaria in Southeast Asia readily fe
218 e plantations provide ideal habitats for the mosquito vectors of malaria, dengue, and chikungunya.
219 , are capable of generating large numbers of mosquito vectors of malaria, which causes more than 400,
220 gies of major agricultural pests but also on mosquito vectors of serious human diseases such as Dengu
221 ntribute to our understanding of ISVs within mosquito vectors of the Culicidae family in the Eastern
222    Besides its strong insecticidal effect on mosquito vectors of the disease, ivermectin inhibits Pla
223 aised concerns that climate change may cause mosquito vectors of these diseases to expand into more t
224 hird, highly focused feeding patterns of the mosquito vectors of WNV result in unexpected host specie
225 est connectivity, factors that may favor the mosquito vectors of yellow fever.
226  against Zika virus and to identify possible mosquito vectors of Zika virus.
227  architecture of osmoregulation in Anopheles mosquitoes, vectors of human malaria.
228 hinery in cells of both mammalian (host) and mosquito (vector) origin.
229 possible for marine mammals to be exposed to mosquito-vectored pathogens through direct interactions
230 ns unknown how wild cetaceans are exposed to mosquito-vectored pathogens.
231       This model interconnects a homogeneous mosquito vector population with a heterogeneous human ho
232 deforestation and agriculture, can influence mosquito vector populations and malaria transmission.
233              The use of pesticides to reduce mosquito vector populations is a cornerstone of global m
234 rile insect technique does not only suppress mosquito vector populations through the induction of ste
235 ggests adaptation to different human host or mosquito vector populations.
236 luding increased air travel and uncontrolled mosquito vector populations.
237 tion of whether malaria parasites kill their mosquito vectors remains open.
238 Transmission from the vertebrate host to the mosquito vector represents a major population bottleneck
239 red blood cells, while transmission to their mosquito vector requires asexual blood-stage parasites t
240 rasites (Plasmodium spp.) from humans to the mosquito vector requires differentiation from asexual st
241 aria parasite from the mammalian host to the mosquito vector requires the formation of adequately ada
242                 Anopheles gambiae is a major mosquito vector responsible for malaria transmission, wh
243 ed cells of Anopheles gambiae, the principle mosquito vector responsible for the transmission of over
244                     Anopheles gambiae is the mosquito vector responsible for transmitting Plasmodium
245 e emergence of insecticide resistance within mosquito vectors risks jeopardizing the future efficacy
246 ssee, with special emphasis on the potential mosquito vector(s).
247 ific interactions of the sporozoite with the mosquito vector salivary glands or the mammalian host he
248 ites' transmission stage that resides in the mosquito vector salivary glands, can transform into earl
249                                Aedes aegypti mosquitoes vector several arboviruses of global health s
250 due to the co-prevalence of the transmitting mosquito vector species Aedes and Culex.
251 nization often result in increased levels of mosquito vector species and vector-borne pathogen transm
252                              Determining how mosquito vector species composition and abundance depend
253              However, longitudinal shifts in mosquito vector species composition were evident within
254                          We hypothesize that mosquito vector species, especially Aedes aegypti, are l
255  and stable transformation of this important mosquito vector species.
256 irus (ZIKV) that explicitly includes two key mosquito vector species: Aedes aegypti and Aedes albopic
257 iency of malaria parasite development within mosquito vectors (sporogony) is a critical determinant o
258 gly significant public health problem as its mosquito vectors spread over greater areas; no vaccines
259         Given the wide distribution of CHIKV mosquito vectors, studies to determine the geographic ex
260 aging tool, is promising in transforming the mosquito vector surveillance programs by reducing the bu
261 ckage in terms of the fitness imposed to the mosquito vector that expresses either molecule.
262                                          The mosquito vector that transmits this virus is widespread
263 hat is spread by travelers and adapts to new mosquito vectors that live in temperate climates.
264 ly capture the biology and behaviour of many mosquito vectors that refeed frequently (every 2-3 d)(4)
265 ients for use in novel biopesticides against mosquito vectors that transmit malaria.
266 trol malaria is by controlling the Anopheles mosquito vectors that transmit the parasites.
267                               Along with its mosquito vector, the Alphavirus chikungunya virus (CHIKV
268 EV strains are opportunistic in their use of mosquito vectors, the most widespread outbreaks appear t
269 velopment of malarial parasites within their mosquito vector, thereby abrogating the cascade of secon
270 n an effort to limit ZIKV replication in the mosquito vector, thereby interrupting the transmission a
271  rapidly lose the capacity to develop in the mosquito vector through a defect in exsheathment and ina
272 ch causes febrile illness, is transmitted by mosquito vectors throughout tropical and subtropical reg
273  Plasmodium, must survive and develop in the mosquito vector to be successfully transmitted to a new
274 uality that the parasite must sustain in the mosquito vector to complete its life cycle.
275 e pathogens successfully transition from the mosquito vector to the vertebrate host is an important a
276 volving human amplification and peridomestic mosquito vectors to cause major epidemics.
277 ncidence because of the sensitivity of their mosquito vectors to climate.
278 timately require the genetic manipulation of mosquito vectors to disrupt virus transmission to human
279 ing the feasibility of genetically modifying mosquito vectors to impair their ability to transmit the
280 have been suggested to alter the behavior of mosquito vectors to increase the likelihood of transmiss
281 mic regions, where water availability limits mosquito vectors to only part of the year.
282 is the first to consider the spread of Aedes mosquito vectors to project dengue suitability.
283 ing the temperature from that present in its mosquito vectors to that of its human host.
284 outbreaks have been contained by controlling mosquito vectors using insecticide-based approaches.
285 rus genus of the family Togaviridae contains mosquito-vectored viruses that primarily cause either ar
286 on due to its profound effect on ectothermic mosquito vectors, viruses, and their interaction.
287 d in widespread development of resistance in mosquito vectors, warranting continuous monitoring and i
288 gunya virus (CHIKV) to study adaptation to a mosquito vector, we evaluated mutations associated with
289                                      Malaria mosquito vectors were captured 3 months before, during,
290 ation between Plasmodium parasites and their mosquito vectors, which shapes parasite virulence both i
291 clear picture into the role of small RNAs in mosquito vectors will pave the way to the utilization of
292 cal model for the population dynamics of the mosquito vector with the temperature time series and the
293 omplex interplay between bird reservoirs and mosquito vectors, with human cases the result of epizoot
294 P. falciparum to a new human host requires a mosquito vector within which sexual replication occurs.

 
Page Top