ライフサイエンスコーパス: motion sickness

1 aines) share underlying genetic factors with motion sickness.

2 entering and leaving curves had induced the motion sickness.

3 and how to maintain speed while eliminating motion sickness.

4 es can go faster, but passengers complain of motion sickness.

5 ssenger yaw and roll, and a survey evaluated motion sickness.

6 significantly higher ratings of vection and motion sickness.

7 ndividuals experiencing increasing levels of motion sickness.

8 The present study tested the hypothesis that motion sickness affects thermoregulatory responses to co

9 A study of motion sickness and allodynia in migraine patients suppo

10 rovided ratings of their perceived levels of motion sickness and dizziness.

11 Visual-vestibular conflicts can induce motion sickness and further postural instability.

12 itions provocative of and protective against motion sickness and how vestibular disease may sensitize

13 tilt with yaw velocity on curves will reduce motion sickness and improve passenger comfort on tilting

14 ion is recommended to reduce the symptoms of motion sickness and improve postural stability with an a

15 ng brain mechanisms and loci associated with motion sickness and nausea per se.

16 t to the importance of the nervous system in motion sickness and suggest a role for glucose levels in

17 n three individuals is highly susceptible to motion sickness and yet the underlying causes of this co

18 bo intervention improved nausea, symptoms of motion sickness, and gastric myoelectrical activity (nor

19 he absence of real motion), visually-induced motion sickness, and one's sense of presence in a passiv

20 his study compares vection, visually induced motion sickness, and presence among participants experie

21 s associated with higher ratings of vection, motion sickness, and presence at slow speeds and with ve

22 tion) resulted in higher ratings of vection, motion sickness, and presence compared to contracting cu

23 at placebo effects on symptoms of nausea and motion sickness are resistant to experimentally-induced

24 manifestations of vestibular dysfunction and motion sickness are well established in the clinical lit

25 thermore, behavioral data recorded using the motion sickness assessment questionnaire (MSAQ) showed s

26 de polymorphisms (SNPs) were associated with motion sickness at a genome-wide-significant level (P <

27 pharmacological neuromodulation tool to keep motion sickness at bay.

28 Motion sickness attenuates the vasoconstrictor response

29 We searched for comorbid phenotypes with motion sickness, confirming associations with known como

30 ure perception, thermal comfort and level of motion sickness discomfort at regular intervals.

31 y and feasibility, with minimal incidence of motion sickness during the VR headset use.

32 g. being a poor sleeper) that correlate with motion sickness, findings that could help identify risk

33 s a potential candidate marker of successful motion sickness habituation.

34 and how vestibular disease may sensitize to motion sickness has increased.

35 Secondary outcomes included differences in motion sickness, headache burden, and migraine disabilit

36 However, the polysymptomatic nature of motion sickness, high interindividual variability, and t

37 d the first genome-wide association study on motion sickness in 80 494 individuals from the 23andMe d

38 ure immersion was preceded by provocation of motion sickness in a human centrifuge.

39 st that has traditionally been used to treat motion sickness in humans.

40 Motion sickness is an emerging hazard in information tec

41 ncreased, and autonomic imbalance induced by motion sickness is restored.

42 Motion sickness may predispose individuals to hypothermi

43 physiology and brain regions associated with motion sickness may provide for more effective medicatio

44 Travel can induce motion sickness (MS) in susceptible individuals.

45 No differences in visually induced motion sickness or presence were found between condition

46 discomfort (P = .03), fatigue (P = .03), and motion sickness (P = .01).

47 In the motion sickness procedure, the DeltaT(forearm-fingertip)

48 In the 'motion sickness' procedure immersion was preceded by pro

49 exposed to similar G-stress, but without the motion sickness provocation.

50 ts of the placebo intervention on nausea and motion sickness remained unchanged, whereas no improveme

51 Motion sickness remains bothersome in conventional trans

52 iveness of some anti-mAChR drugs in treating motion sickness suggest that we may, in fact, already be

53 We found that tES reduced motion sickness symptoms by significantly increasing nor

54 In the case of vestibular dysfunction or motion sickness, the unpleasant visceral manifestations

55 rls reported discomfort consistent with mild motion sickness; the boy said he was bored and the heads

56 public's longstanding resigned tolerance to motion sickness threatens to change, due to the widespre

57 Other SNPs may affect motion sickness through nearby genes with roles in the n

58 n autonomic function during visually induced motion sickness, through the analysis of spectral and ti

59 ogical changes accompanying visually induced motion sickness, using a motion video, hypothesizing tha

60 All were exposed to a 10-min motion-sickness video during fMRI.

61 Concurrently, motion sickness was greater (P<0.001) in the reactive mo

62 No motion sickness was induced in the untilted mode, but th

63 Visually induced motion sickness was probed using the Simulator Sickness

64 80 healthy female volunteers susceptible to motion sickness were randomly assigned to either the Maa

65 tory of postoperative nausea and vomiting or motion sickness, young age, volatile anesthetic agents,