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1 ion may be an effective means of controlling motoneuronal activity in a behavior-dependent manner.
2                 We conclude that hypoglossal motoneuronal activity is more strongly influenced by che
3                                              Motoneuronal activity was recorded simultaneously from a
4 e central preparation; recruitment curves of motoneuronal activity were then generated.
5 that allows a temporally specific control of motoneuronal activity within a single phase of motor pro
6 ts of strong inspiratory bursts, which drive motoneuronal activity, and weaker burstlets, which we hy
7 muscle contractions are a graded function of motoneuronal activity, one consequence of the shortening
8 ators (CPGs) may be used to control rhythmic motoneuronal activity.
9 ating inspiratory drive to hypoglossal (XII) motoneuronal activity.
10 lospinal circuits, whereas corticospinal and motoneuronal adaptations are not dominant factors.
11           These results provide insight into motoneuronal adjustments during contraction tasks posing
12 e alterations in neuronal properties at both motoneuronal and pre-motoneuronal levels.
13  definitively that TASK channels account for motoneuronal, anesthetic-activated K(+) currents and to
14 he proximal femur and distal tibia, based on motoneuronal birth order.
15 ith labeled and unlabeled dendrites in these motoneuronal cell groups and contain large amounts of sp
16  NRA projects to a somewhat different set of motoneuronal cell groups compared with other species fit
17 nd contains premotor neurons that project to motoneuronal cell groups in the brainstem and spinal cor
18 oneurons involved in vocalization, i.e., the motoneuronal cell groups innervating soft palate, pharyn
19                                  To identify motoneuronal cell groups receiving input from the NRA, t
20 behavior by means of projections to distinct motoneuronal cell groups.
21 ce in the iliopsoas, axial, and pelvic floor motoneuronal cell groups.
22 hat under certain circumstances even cortico-motoneuronal cells, which make monosynaptic connections
23                           Motor cortical and motoneuronal changes were similar during electrically ev
24 uscles of rhesus monkeys to identify cortico-motoneuronal (CM) cells in the primary motor cortex (M1)
25            This enabled us to define cortico-motoneuronal (CM) cells that make monosynaptic connectio
26 rged together with the appearance of cortico-motoneuronal (CM) connections during the evolution of th
27 al system and, in particular, direct cortico-motoneuronal (CM) connections to hand muscles.
28          The significance of direct, cortico-motoneuronal (CM) connections, which were discovered a l
29 and bilateral projections to the respiratory motoneuronal columns.
30 axon initial segment (AIS), but not on other motoneuronal compartments, inhibited the action potentia
31 we show using electrophysiology that cortico-motoneuronal connections from fast conducting CST fibers
32 how that all parts of M1 and 3a have cortico-motoneuronal connections over more slowly conducting CST
33 w M1") and area 3a make monosynaptic cortico-motoneuronal connections with limb motoneurons.
34 nd pharmacological understanding of specific motoneuronal contributions to eye movements might help i
35  a controlling signal of pre- and post-alpha motoneuronal control of the soleus H-reflex.
36                                          The motoneuronal control provides comprehensive and separate
37 rolled by a dedicated experimentally derived motoneuronal control.
38 time course were investigated during ongoing motoneuronal degeneration.
39 ith retrogradely labeled genioglossus muscle motoneuronal dendrites and perikarya in the hypoglossal
40 cal properties, and consequently altered the motoneuronal dendritic processing of synaptic inputs.
41 ic connections between the interneuronal and motoneuronal elements that generate the two behaviors.
42 vel of the pTRG diminished the contralateral motoneuronal EPSPs as well as a local injection of 6-cya
43 ting a role of phosphorylation in modulating motoneuronal excitability affecting behaviorally relevan
44          These effects of halothane decrease motoneuronal excitability and may contribute to the immo
45 ic lateral sclerosis (ALS), abnormalities in motoneuronal excitability are seen in early pathogenesis
46 , based on differences in motor cortical and motoneuronal excitability between exercise modalities (e
47              Electrically evoked KE impaired motoneuronal excitability but not motor cortical excitab
48                   Neurotransmitters increase motoneuronal excitability by inhibiting TWIK-related aci
49 erents do not contribute to the cortical and motoneuronal excitability changes during this exercise m
50 Since protein kinase-dependent modulation of motoneuronal excitability contributes to adaptive change
51 not exclusively, account for the decrease in motoneuronal excitability during fatiguing KE.
52 V leg muscle afferents on motor cortical and motoneuronal excitability during fatiguing knee-extensor
53 inducing long-term increases in genioglossal motoneuronal excitability to AMPA-mediated drive may hel
54 latile anaesthetics have opposing effects on motoneuronal excitability which appear to reflect contra
55  motor cortical excitability but compromises motoneuronal excitability, with the combined effect of a
56 y KE enhanced motor cortical but compromised motoneuronal excitability, with the net effect of an ove
57 e--activated 5-HT1A receptors that decreased motoneuronal excitability.
58 ced membrane hyperpolarization and increased motoneuronal excitability.
59 examines the role of polyamine modulation of motoneuronal excitation in situ, with an emphasis on pos
60 otoneurons, thereby diminishing serotonergic motoneuronal excitation.
61 pains reduced this proteolysis, restored the motoneuronal expression of Nav and KCC2, normalized I(Na
62  relevant role of the MLF pathway in driving motoneuronal firing and evidenced compensatory mechanism
63 unced temporal precision in population-level motoneuronal firing depends on gap junction-mediated, gl
64 th motoneurons and were active just prior to motoneuronal firing in each segment.
65                  (2) The kt value (change in motoneuronal firing necessary to increase motor unit for
66  permanent, 2 months after ATD lesioning all motoneuronal firing parameters were similar to the contr
67  laminae V-VIII, as well as the laterodorsal motoneuronal group of lamina IX (which innervates distal
68  to Ca excitotoxicity theory, predictions of motoneuronal hyper-excitability, normal and hypo-excitab
69 nce of phosphorylated eIF-2a should minimize motoneuronal injury in obstructive sleep apnea.
70 entation of eIF-2a phosphorylation minimizes motoneuronal injury in this model.
71  muscle model is generally used to transform motoneuronal input into muscle force.
72  in motor unit discharge rate and an altered motoneuronal input-output gain.
73 ckroach Blaberus discoidalis; in particular, motoneuronal inputs and muscle force levels are chosen t
74 s activating protein kinase G (PKG) modulate motoneuronal inspiratory drive currents and long-term pl
75 espiratory-related rhythm, hypoglossal (XII) motoneuronal inspiratory drive currents and respiratory-
76 ruthenium red, we found a major component of motoneuronal isoflurane-sensitive TASK-like current that
77  these transmitters might be detected at the motoneuronal level during muscle tone suppression elicit
78 rd by acting at both the premotoneuronal and motoneuronal levels.
79 onal properties at both motoneuronal and pre-motoneuronal levels.
80 s maintaining muscle strength in the face of motoneuronal loss caused by injury or disease.
81 sis, a disorder characterized by progressive motoneuronal loss; however, whether these changes relate
82 e association of SOD1-G85R or SOD1-G93A with motoneuronal mitochondria is reduced capacity of the ETC
83 iated by inhibition of rhythmogenic and (pre)motoneuronal networks; and (iii) pre-inspiratory (Pre-I)
84 cytoplasmic ribonucleoprotein complexes from motoneuronal NSC-34 cells.
85 lar circuitry makes it likely that a similar motoneuronal organization is also implemented in other v
86 d show that Zfh1 cell autonomously regulates motoneuronal outgrowth and larval growth of neuromuscula
87  released from neurones, can directly affect motoneuronal output and central fatigue.
88 tion to enhance motor cortical excitability, motoneuronal output and, ultimately, exercise performanc
89                Synchrony strength in cortico-motoneuronal output neurons in primary motor cortex depe
90 edback to the CNS which, in turn, constrains motoneuronal output to the active skeletal muscle.
91                                     Although motoneuronal output was 21 +/- 12% higher during FENT co
92                                              Motoneuronal output was estimated through vastus lateral
93 rones and plays an important role in shaping motoneuronal output.
94 ization by activating the prefrontal-PAG-NRA-motoneuronal pathway, and, at the same time, they modula
95 ising limb muscles through the corticospinal-motoneuronal pathway, which links the motor cortex and s
96  receptors were localized at the membrane of motoneuronal perikarya and dendrites.
97 o enriched at glutamatergic synapses in both motoneuronal perikarya and dendrites.
98                                              Motoneuronal persistent inward currents (PICs) are facil
99         pTRG neurons projecting to the vagal motoneuronal pool were identified in a restricted area o
100 n part, through activation of nonhypoglossal motoneuronal pools innervating the UA muscles.
101 A(A) gamma2 levels towards control values in motoneuronal pools of both muscles.
102  neurons terminate preferentially within the motoneuronal pools of the lumbosacral spinal cord that i
103  study demonstrates the presence of specific motoneuronal populations with pharmacological profiles t
104                                              Motoneuronal potassium currents were unaffected.
105 r formation prolonged the inhibitory phrenic motoneuronal response to superior laryngeal nerve stimul
106 imulation and abolished or reduced abdominal motoneuronal responses during respiration, vomiting, and
107                                   Defects in motoneuronal retrograde axonal transport may critically
108      Because both the reticulospinal and the motoneuronal segmental patterns persist in the hindbrain
109 he inability to meet the surge in demand for motoneuronal SMN that was seen in controls.
110 king a functional Scn8a sodium channel gene, motoneuronal sodium current density was comparable at P0
111                                              Motoneuronal soma size in Nissl stain revealed the same
112 hin Rexed's lamina IX, in close proximity to motoneuronal somata.
113                        Because of the spinal motoneuronal somatotopic organization, motor coordinatio
114                         In contrast, primary motoneuronal specification depends on the presence of so
115  In addition, a positive correlation between motoneuronal survival and voiding efficiency was observe
116 ypothesized that strengthening corticospinal-motoneuronal synapses at multiple spinal cord levels thr
117         Modulation of residual corticospinal-motoneuronal synapses may present a novel therapeutic ta
118 ding and peripheral volleys at corticospinal-motoneuronal synapses of an intrinsic finger muscle in h
119  Hebbian stimulation targeting corticospinal-motoneuronal synapses of multiple arm and leg muscles fo
120  or sham stimulation targeting corticospinal-motoneuronal synapses of multiple leg muscles followed b
121 pendent plasticity of residual corticospinal-motoneuronal synapses provides a mechanism to improve mo
122 ntly reversed by strengthening corticospinal-motoneuronal synaptic connectivity via targeted, tempora
123 t VEGF is an essential retrograde factor for motoneuronal synaptic drive and discharge activity.
124 ion to the nRO spinal autonomic and pudendal motoneuronal targets, projections were observed to regio
125                             Some cholinergic motoneuronal terminals on both Renshaw cells and motoneu
126 , and Shh to NSFCs induced the expression of motoneuronal transcription factors, tyrosine hydroxylase

 
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