ライフサイエンスコーパス: motor function

1 ions) or shifting to another location (i.e., motor functions).

2 have sex-specific deficits in body mass and motor function.

3 nce of injury, without associated changes in motor function.

4 herapy has led to significant improvement in motor function.

5 ian deep cortical layers with involvement in motor function.

6 symptoms extends their lifespan and improves motor function.

7 ) injury on nerve regeneration and resulting motor function.

8 as language, primary sensory modalities, and motor function.

9 transition more easily to A-form would alter motor function.

10 ons that are associated with both limbic and motor function.

11 ed to reduce sedentary behaviour and enhance motor function.

12 organization of neural circuits controlling motor function.

13 oad impact of amblyopia on visual and visual-motor function.

14 did not support use of fluoxetine to improve motor function.

15 injury, which resulted in slowed recovery of motor function.

16 ay sitting, with very few options to improve motor function.

17 tal GABAergic microcircuits are critical for motor function.

18 weight and longer survival as well as better motor function.

19 fic location, which is associated with worse motor function.

20 in addition to its classic association with motor function.

21 mulation (DBS) within circuits that modulate motor function.

22 opment and degeneration in adulthood impairs motor function.

23 w interpretation of cerebellar structure and motor function.

24 ivation, reward-related learning, and normal motor function.

25 STN transmission and patterning and improved motor function.

26 lamic development, and thereby cognitive and motor function.

27 f cerebellar Purkinje cells and a decline in motor function.

28 This loss greatly affects full recovery of motor function.

29 ral ischemia, as well as muscle strength and motor function.

30 rking memory, learning, memory, fluency, and motor function.

31 deficits in APP/PS1, without altering gross motor function.

32 43, markedly increased survival and improved motor function.

33 on of Kv2.1 expression and an improvement in motor function.

34 ting cognitive development, respiration, and motor function.

35 rategies to achieve tunable and controllable motor function.

36 in brain 9cRA levels and greater recovery in motor function.

37 uces signatures of brain injury and impaired motor function.

38 g silencer (ISS) improves SMN expression and motor function.

39 nction, episodic memory, working memory, and motor function.

40 eration, alpha-syn aggregates and normalized motor function.

41 he need for intrinsic control over molecular motor function.

42 tor function and a ladder test to study fine motor function.

43 a range of conformational changes, blocking motor function.

44 branches and dendrites leading to decreased motor function.

45 ly-specific insertions in modulating kinesin motor function.

46 networks necessary for proper cognitive and motor function.

47 s, glial activation, and progressive loss of motor function.

48 ly, the sense of touch is a key component of motor function.

49 BI mice and led to transient improvements in motor function.

50 be resolved to restore complete respiratory motor function.

51 ostriatal circuit to mediate improvements in motor function.

52 which was in turn predictive of recovery of motor function.

53 le and male animals and did not change gross motor function.

54 ificantly improved recovery of affected limb motor function.

55 emisphere, is involved in maintaining normal motor function.

56 nce of Bmal1 may exacerbate circadian and PD motor function.

57 ning and establish subnuclear topography and motor function.

58 habilitation regimen for maximum recovery of motor function.

59 leotide nusinersen has been shown to improve motor function.

60 hip between dopamine and rhythmicity impacts motor function.

61 lved in the control of diverse motor and non-motor functions.

62 cognitive processes, reward mechanisms, and motor functions.

63 Cameroonian adult norms for assessing SIP or motor functions.

64 ce displayed only subtle impairment in their motor functions.

65 We assessed viral effects on SIP and motor functions.

66 ure that integrates cognitive, emotional and motor functions.

67 A) release in the striatum and thus impaired motor functions.

68 anent deficits of cognitive, sensory, and/or motor functions.

69 ed speed-of-information processing (SIP) and motor functions.

70 nt extension of life span and improvement of motor functions.

71 s that differ in vulnerability to damage and motor functions.

72 ic cells that perform sensory, relaying, and motor functions.

73 of age-related alterations in cognitive and motor functions.

74 logy of neurodegenerative diseases affecting motor functions.

75 hat male Synj1+/- mice display age-dependent motor function abnormalities as well as alpha-synuclein

76 re-emergence closely tracked the recovery of motor function across several movement qualities includi

77 ndent learning and memory deficits, restores motor function after brain trauma, and decreases brain l

78 ble to repair spinal cord tissue and restore motor function after complete spinal cord transection ow

79 Motor function after hemispheric lesions has been associ

80 nd trophic signaling can enhance recovery of motor function after injury in monkey primary motor cort

81 Effective therapies to support recovery of motor function after stroke are yet to be developed.

82 a brain-computer interface (BCI) to enhance motor function alone.

83 significant improvement in neurological and motor functions along with a decrease in infarct volume

84 piratory function, growth, and cognitive and motor function also improved, and asfotase alfa was gene

85 7, P <= 0.05) and negatively associated with motor function among HIV-negative women (b = -3.57, SE =

86 ed on an open-field grid test to study gross motor function and a ladder test to study fine motor fun

87 homozygous mutants in parallel with improved motor function and a reduction in neuroinflammation.

88 motor symptoms, selective deletion worsened motor function and accelerated the onset of paralysis.

89 5-HT2C receptors in the effects of SSRIs on motor function and affective behavior, highlighting the

90 onths with progression, correlations between motor function and biomarkers, and hazard ratios analyze

91 cular or respiratory function in addition to motor function and can be performed by trainees with som

92 altered basal ganglia activity disrupt both motor function and cognition.

93 Significant improvement of motor function and disability (as assessed by the Burke

94 circuit substrates that can be important for motor function and dysfunction.

95 ch was started at disease onset, ameliorated motor function and extended survival.

96 es showed significantly improved recovery of motor function and gait.

97 d SMN2 splicing in various tissues, restored motor function and improved the integrity of neuromuscul

98 ZPR1-dependent rescue improves growth and motor function and increases the lifespan of male and fe

99 tion and restored nerve conduction velocity, motor function and lifespan of the mice to wild-type lev

100 experienced a general post-flight malaise in motor function and motion perception, and a lack of cogn

101 Motor function and movement imitation are also altered i

102 t striatal pathway, which may be relevant in motor function and neurodegenerative diseases.

103 ntrol levels and significantly improves both motor function and neuronal viability.

104 ar junction, delayed disease onset, improved motor function and preserved motor neurons as well as ne

105 -specific caveolin-1 overexpression improves motor function and preserves memory in mice subjected to

106 ressive motor neuron disease causing loss of motor function and reduced life expectancy, for which li

107 led to exciting discoveries regarding dynein motor function and regulation.

108 n rural Bangladesh we examined cognitive and motor function and scholastic achievement in a cohort of

109 aling-mediated mechanism that contributes to motor function and social behavior.

110 However, delayed effect on motor function and some severely damaged capillaries wer

111 IBS pathophysiology since they regulate gut motor function and stool consistency, and targeted 5-HT4

112 sults indicate that changes in diaphragmatic motor function and strength among LOPD subjects could be

113 ) mouse model of ALS (G93A SOD1) would alter motor function and survival.

114 and present a detailed mechanistic model for motor function and switching of rotational direction.

115 n use a BCI to simultaneously reanimate both motor function and the sense of touch, leveraging residu

116 ion is required for regulation of cerebellar motor function and vocal communication, likely through d

117 oral domains including social communication, motor functioning and conditioned fear that are not expl

118 -generated PA in the regulation of kinesin-1 motor functions and breast cancer metastasis and suggest

119 -deficient (Nrros(-/-)) mice show defects in motor functions and die before 6 months of age.

120 e overall fundamental contribution of D2R in motor functions and explains some of the side effects el

121 llum regulates Purkinje cell development and motor functions and vocal communication, demonstrating e

122 progressively rob patients of their memory, motor function, and ability to perform daily tasks.

123 imaging findings, namely cognitive function, motor function, and brain volume (global and regional).

124 ional status, disability, ambulation status, motor function, and cardiac stress, with analysis by stu

125 ischemic limb perfusion, capillary density, motor function, and their amputation.

126 f the afferent mechanisms underlying healthy motor function, and their disruption in neurological con

127 e peripheral nervous system is essential for motor function, and uncontrolled SC proliferation occurs

128 within the thalamus, impaired cognitive and motor functions, and affected self-reports of mood/drug

129 of new synapses and the remapping of sensory-motor functions, and is associated with motor recovery.

130 rebral blood flow, improved neurological and motor functions, and reduced ischemic stroke infarct vol

131 a central role in the control of sensory and motor functions, and the disruption of its barriers can

132 eroon is associated with deficits in SIP and motor functions; ART and higher CD4 are associated with

133 is correlated with improved neurological and motor function, as well as with preservation of neuronal

134 ights into both filopodia formation and MYO6 motor function at endosomes and at the plasma membrane.

135 stroke were based only on the initially lost motor function, at least for a specific fraction of pati

136 aggression was not a consequence of impaired motor function, because optogenetic stimulation did not

137 We assessed gross motor function before and after SDR and postoperative qu

138 on against the changes induced by ACR in the motor function, behavior, transcriptome and proteome.

139 tACS can modulate perception, cognition, and motor function but the underlying neural mechanism is po

140 BK/SK channel activator, partially improved motor function, but ataxia continued to progress.

141 Here we show in mice that SSRIs impair motor function by acting on 5-HT2C receptors in the subs

142 ons have demonstrated the ability to improve motor function by reengaging ipsilesional resources, whi

143 nges in corticospinal excitability (CSE) and motor function can be induced in a targeted and long-ter

144 in children with cerebral palsy and a Gross Motor Function Classification System (GMFCS) level of II

145 in terms of motor function, using the Gross Motor Function Classification System (GMFCS).

146 with severe dyskinetic cerebral palsy (Gross Motor Functioning Classification System level IV-V) aged

147 ommunication devices for people with limited motor function.Clinical Trial No: NCT00912041.

148 pressed in regions of the brain that control motor function, cognition, and motivation.

149 tment of symptomatic mice with ASO7 improved motor function compared to saline-treated mice.

150 by cerebrospinal fluid, yet R222 had normal motor function, could hear, see, smell, and respond to t

151 pha-actin switch coincided with the onset of motor function deficits and histopathological changes in

152 terest, motivation, emotional development or motor function depending on the animal model under inves

153 ce, relative to vehicle-treated animals, yet motor function did not improve in any of the treatment g

154 FTY720 significantly reduced the deficit of motor function, diminished the loss of tyrosine hydroxyl

155 rk is positioned at the nexus of sensory and motor functions, directing two tightly coupled processes

156 Explanations for the improved motor function during RBD episodes are evaluated in ligh

157 information provides insight into how these motors function during mitosis.

158 l relationship between pallidal activity and motor function/dysfunction.

159 for socioemotional behavior, cognitive, and motor function (e.g. amygdala, hippocampus, cerebellum).

160 nd thus likely linked to modulation of vocal motor function (e.g. KCNC1, GABRE), including a subset o

161 Human motor function emerges from the interaction between the

162 d risk of poor executive function and visual motor function, even if not detected clinically, and may

163 hortly after exposure (30 min), and impaired motor functions (falls: +83%; time top: -43%; time botto

164 re no differences in exploratory behavior or motor function, fasting lipid levels, or the inflammator

165 Gross motor function, feeding method, energy intake, and HPA l

166 Motor function findings in his right/left hand were: gri

167 tracts is associated with the improvement in motor function following gene therapy.

168 ys likely contribute to the recovery of hand motor function following spinal cord injury (SCI).

169 within the central nervous system to restore motor function following spinal cord injury, the role of

170 ergic agonist Quipazine, to enable hind limb motor functions following paralysis.

171 ) that is capable of rescuing wild-type (WT) motor function for cilium assembly and Hedgehog signalin

172 . crescentus, several CheY homologs regulate motor function for different aspects of the bacterial li

173 icates that subthalamic nucleus DBS improves motor function for up to 10 years, although the magnitud

174 33 um(2); p < 0.02); and greater recovery of motor function (grip strength: p < 0.001).

175 t-A (TMT-A), Color Trails-1 (CTT1)], and two motor function [Grooved Pegboard-dominant (GP-DH) and no

176 ease (PD) patients experience loss of normal motor function (hypokinesia), but can develop uncontroll

177 In addition to motor function impairment, HD mice also show impaired ci

178 amic radiation and callosal fibers involving motor function, improved after gene therapy.

179 showed better survival and neurological and motor function improvement than control.

180 teral parietal cortices also correlated with motor function improvement, consistent with the increase

181 he performance of a visuomotor grip task and motor function in a longitudinal (<5 days, 1 and 3 month

182 moter screen, prolongs survival and improves motor function in a mouse model of spinal muscular atrop

183 , with clinically meaningful improvements in motor function in a real-world cohort.

184 key factor in the detrimental outcome of the motor function in amyotrophic lateral sclerosis.

185 a Hfd, and synergistic impairment of sensory-motor function in APP/PS1 mice fed a Hfd.

186 re, FliL does not play a significant role in motor function in E. coli.

187 h clinical trials have shown improvements in motor function in infants and children treated with the

188 aim to restore lost function, most commonly motor function in paralyzed patients.

189 channels may improve ACh-DA reciprocity and motor function in Parkinson's disease (PD).

190 structure to measures of proximal and distal motor function in patients after hemispherotomy.

191 alyses suggest that olesoxime might maintain motor function in patients with type 2 or type 3 SMA ove

192 ding frame programme significantly increased motor function in people with severe progressive multipl

193 ed significant improvements in life span and motor function in Ppt1(-/-) mice.

194 a method that can be used to assess cortical motor function in RTT patients.

195 Motor function in SMA patients may be additionally impro

196 ensatory mechanism associated with preserved motor function in the dopamine depleted OFF state.

197 des Class III evidence to support benefit of motor function in young boys with DMD treated with vamor

198 measures of cognition, perception, gaze and motor functioning in a large general population (n = 92;

199 tion of secondary (M2) motor cortex improves motor functions in dopamine-depleted male mice.

200 atal axis and neurotransmitters and improved motor functions in hemiparkinsonian monkeys.

201 rotransmitters, and continuous impairment of motor functions in MPTP-intoxicated mice.

202 cance of lobules implicated in cognitive and motor functions in normal subjects.

203 ase leaded to a near complete restoration of motor functions in Parkin Q311X(A) mice and improved bra

204 quality and child behavioral, language, and motor functions in the Norwegian Mother and Child Cohort

205 s hindered an understanding of how kinesin-2 motors function in cilium assembly and IFT.

206 ersive learning as well as markedly affected motor function including disordered coordination.

207 tropy of the PT explained (p = 0.050) distal motor function including finger tapping rate (p = 0.027)

208 the cerebellum is involved in motor and non-motor function, including cognition and emotion.

209 al states may be able to rescue the impaired motor function induced by disease mutations.

210 he degeneration of motor neurons and loss of motor function is a fundamental question to comprehend a

211 terestingly, earlier in dSod1 (G85R) larvae, motor function is also compromised, but their motor neur

212 Previous reports raise the question if motor function is modulated by substrate DNA sequence: (

213 ion, a R502P amino acid substitution, on the motor function is unclear.

214 ance, yet its impact on human cardiac myosin motor function is unclear.

215 can be achieved if the user, in addition to motor functions, is provided with the sensations that ar

216 is traditionally associated with balance and motor function, it also plays wider roles in affective a

217 was used to identify lobules associated with motor function, language, executive function, memory, ve

218 associated with a loss of somatosensory and motor function, leading to impairments in gait, balance,

219 ion of microtubule (MT) stability and dynein motor function/localization that alters mitotic spindle

220 tcome was score changes in the 66-item Gross Motor Function Measure (GMFM-66) and seven domains of th

221 The primary clinical outcome was motor function measured by the Amended Motor Club Assess

222 baseline (pre-CMR), participants improved on motor function (MESUPES arm p = 0.02; MESUPES hand p = 0

223 ed important hallmarks of disease, including motor function, NMJ pathology and motor neuron cell pres

224 sive manganese accumulation was the impaired motor function observed in the Zip14 KO mice.

225 lopodial tips was hampered by the diminished motor function of a dimeric construct of the shaker-1 mu

226 The motor function of rats was assessed by the rotarod test.

227 e rich insights into the emotional state and motor function of the test animal.

228 exocytosis in MB neurons and alters specific motor functions of 1-year-old male mice.

229 oral delivery of gold nanocrystals improved motor functions of cuprizone-treated mice in both open f

230 tention control are also seen on lower level motor functions of dexterity and strength-by examining r

231 tance of ERK/MAPK signaling in governing the motor functions of the striatal direct and indirect path

232 in SOD1(G93A) mice, we found no benefits on motor function or disease pathology, including muscle de

233 d no reproducible abnormalities in survival, motor function, or neurodegeneration.

234 agnosed with additional conditions affecting motor function other than cerebral palsy (eg, spina bifi

235 or PITX3-eGFP(-) cell grafts showed improved motor function over 6 months.

236 to be the only clinical variable to explain motor function (p < 0.001).

237 faster and more complete recovery of forepaw motor function (P < 0.05).

238 al hemisphere in the pons explained proximal motor function (p = 0.001).

239 en food insecurity and learning, memory, and motor function (P values <0.05).

240 ed (p=0.002), executive function (p<0.0001), motor function (p<0.0001), and working memory (p=0.001).

241 the domains of episodic memory (p=0.03) and motor function (p=0.02).

242 blood draw, clinical assessment of strength, motor function, quality of life, and adverse effect asse

243 d RR, 2.32; 95% CI, 1.17 to 4.59) and visual motor function (RD, 0.03; 95% CI, 0.01 to 0.06 and RR, 3

244 In both studies, TBI + Sp mice had poorer motor function recovery compared with TBI + PBS mice.

245 lay an important role in symptom control and motor function recovery during deep brain stimulation.

246 dependent fear learning and memory, improved motor function recovery, and decreased brain lesion volu

247 d that, once implanted in SCI rats, improves motor function recovery.

248 ing leads to pathway-specific alterations in motor function, reduced neuronal excitability, and the i

249 and attenuation of Mn-induced impairment of motor functions, reduction of TH-expressing cells in SN,

250 bserved during RBD episodes exhibit improved motor function, relative to baseline states during wake

251 uch trauma on objective metrics of cognitive-motor function remain poorly understood.

252 tes from the OEM (fulfilling conductance and motor functions, respectively), enabling degradation of

253 cts of liraglutide versus placebo on gastric motor functions, satiation, satiety, and weight in obese

254 Infant motor function scales (Test of Infant Motor Performance

255 pyramidal motor side effects; mean scores on motor function scales indicated no motor disturbances wi

256 Motor function scores (MFS) and compound muscle action p

257 ility of the graft composition, and enhanced motor function.SIGNIFICANCE STATEMENT Clinical trials ha

258 lizing activity within cortical networks for motor function.SIGNIFICANCE STATEMENT Neuronal plasticit

259 deficits in mitochondrial bioenergetics and motor function.SIGNIFICANCE STATEMENT The present work p

260 h, and improved recovery of some sensory and motor function.SIGNIFICANCE STATEMENT The studies shed l

261 Findings suggest that measures of motor function speed and accuracy within a more practica

262 Motor function speed and variability were recorded and c

263 postnatal week followed by a degeneration of motor function starting at periadolescence in the settin

264 d disability scale evaluating fine and gross motor function, strength, sensation, and balance.

265 Despite its importance for motor function, structural information about the interac

266 Furthermore, disorders of colonic motor function, such as irritable bowel syndrome, are mu

267 Progress was assessed by monthly sensory and motor function tests during routine clinic visits and wi

268 achievement of motor milestones, and better motor function than in historical cohorts.

269 likely to be alive and have improvements in motor function than those in the control group.

270 with larger decreases seen for language and motor functions than for behavioral functions.

271 mulation of D1 receptors is known to enhance motor function, the global effect of D2 receptor (D2R) s

272 r switching is involved, with improvement of motor function through physical activity and cure of sea

273 otor defects and partially rescue late-stage motor function to extend lifespan.

274 y, including the localisation of sensory and motor functions to the brain.

275 working memory, learning ability, and visual-motor function trajectories were not statistically diffe

276 sensitivity and effectiveness), and colonic motor function (transit time) examinations.

277 usion and load-detachment kinetics on single-motor functions under physiologically relevant condition

278 t Diffusion Tensor Imaging and evaluation of motor function using the Fugl-Meyer Assessment and the i

279 ve functioning, learning ability, and visual-motor function, using linear mixed models.

280 f cerebral palsy was represented in terms of motor function, using the Gross Motor Function Classific

281 l known to play a key role in the control of motor function via balanced output from the indirect (iS

282 Motor function was also impaired in rats with HE (P < .0

283 Motor function was altered, and specific effects were fo

284 Motor function was evaluated by a licensed occupational

285 se of SSRIs to prevent depression or improve motor function was not supported.

286 Importantly, this improvement in motor function was obtained without any adverse dyskinet

287 7-day continuous recording of motor function was used to determine the Parkinson's dis

288 on, executive function, visual function, and motor function were assessed at 4.5 years.

289 Disruptions in motor function were associated with increases in cerebel

290 Four assessments of gross motor function were carried out for each participant ove

291 spiratory support, growth, and cognitive and motor functions were also evaluated.

292 early onset and severe disease, survival and motor functions were better than historically untreated

293 After Mn exposure, motor functions were determined in open-field and rotaro

294 pectively.General intelligence or memory and motor functions were not affected by antenatal or newbor

295 ical structures (associative, sensorial, and motor functions), which was not observed for marmosets (

296 sed on a well-supported theory of cerebellar motor function, which ascribes to the cerebellum a role

297 cted effect of ageing on episodic memory and motor function with advanced stages of HIV infection sug

298 ced dramatic and synergistic improvements in motor function with an unprecedented increase in life sp

299 y for spinal cord injury (SCI) is to restore motor function without exacerbating pain.

300 ed hiNPC demonstrated significantly improved motor function, without exacerbating pain.