ライフサイエンスコーパス: mouse embryo

1 drogel, and lumen pressure in the developing mouse embryo.

2 sceral endoderm to the endoderm in the early mouse embryo.

3 s, especially in complex systems such as the mouse embryo.

4 d by two major routes, as illustrated by the mouse embryo.

5 it is the earliest expressed Hox gene in the mouse embryo.

6 enhancer activity and Shh expression in the mouse embryo.

7 human NC cells (hNCCs) into the gastrulating mouse embryo.

8 ak, mesoderm and anterior mesendoderm of the mouse embryo.

9 nding cells diminishes bone formation in the mouse embryo.

10 ta-gonad-mesonephros (AGM) of the developing mouse embryo.

11 ctories toward somite formation in the early mouse embryo.

12 nce, the plane of cell division in the early mouse embryo.

13 r visceral endoderm (AVE) cells in the early mouse embryo.

14 ssential to shape the mandibular arch of the mouse embryo.

15 ion of cell division in the pre-implantation mouse embryo.

16 struloids comparable with those found in the mouse embryo.

17 nitors by conditionally deleting Tfam in the mouse embryo.

18 pression during the onset of gastrulation in mouse embryos.

19 ically differentiate into the endocardium in mouse embryos.

20 e performed transcriptome profiling on whole mouse embryos.

21 racking individual cells in live analysis of mouse embryos.

22 early lineages in peri- and postimplantation mouse embryos.

23 s via injection into the cardiac crescent of mouse embryos.

24 ary to induce LHX3(+)/LHX4(+) RP identity in mouse embryos.

25 at this gene is essential for development of mouse embryos.

26 y establish hematopoietic fate in Gata2Venus mouse embryos.

27 and generation of the pro-amniotic cavity in mouse embryos.

28 (+) cells in a subdomain of the NMP niche in mouse embryos.

29 elevant oncogenes in utero into gastrulating mouse embryos.

30 ient incorporation of naive human cells into mouse embryos.

31 how germ cells differentiate into oocytes in mouse embryos.

32 ent was sufficient to extend trunk length in mouse embryos.

33 nd intact fluorescently-stained 12.5-day old mouse embryos.

34 nome activation timing between the human and mouse embryos.

35 es, and in the neural crest and limb buds of mouse embryos.

36 sues obtained from human fetuses with DS and mouse embryos.

37 ion has been studied extensively in frog and mouse embryos.

38 from both wild-type and Tdg-deficient E11.5 mouse embryos.

39 rior region of palatal shelves in Foxf2(-/-) mouse embryos.

40 to position the first inner cells of living mouse embryos.

41 t initiates TE segregation in human, cow and mouse embryos.

42 n guiding post-crossing axon trajectories in mouse embryos.

43 to the primitive endoderm in both human and mouse embryos.

44 cardiomyocytes (CMs) derived from mESCs and mouse embryos.

45 th analysis of Fat1 expression in developing mouse embryos.

46 ampal neurons from wild-type and Ctnnd2 null mouse embryos.

47 ical properties in cell lines and transgenic mouse embryos.

48 pes of RNA polymerase II (Pol II) binding in mouse embryos.

49 ism initiates a TE program in human, cow and mouse embryos.

50 targeting and tedious micromanipulations of mouse embryos.

51 hroid, and myeloid lineages in zebrafish and mouse embryos.

52 defect of the medial nasal process (Mnp) in mouse embryos.

53 s of neuronal and non-neuronal organs in SMA mouse embryos.

54 ly drove expression in the SAN of transgenic mouse embryos.

55 e findings were validated in cells and whole mouse embryos.

56 ociated cells from the diencephalon of E12.5 mouse embryos.

57 system can be used in medaka, killifish, and mouse embryos.

58 s and their descendants in post-gastrulation mouse embryos.

59 ls (EpiSCs) and compared them with E8.25 CLE mouse embryos.

60 retinal organoids and cell distributions in mouse embryos.

61 t expression in heart and skeletal muscle of mouse embryos.

62 t state linger within the inner cell mass of mouse embryos?

63 Additionally, by investigating mouse embryo 5fC profiles in a tissue-specific manner, w

64 vo labeling of single CN V axons in LgDel+/- mouse embryos, a genomically accurate 22q11.2DS model, a

65 art field (pSHF) of Tbx5 and Osr1 transgenic mouse embryos, a time-course gene expression change duri

66 ibody inhibited embryo implantation in vivo, mouse embryo adhesion and spreading in vitro, as well as

67 ess in three different types of fibroblasts (mouse embryo, adult cardiac, and tail tip).

68 different chromosomes, in cultured cells and mouse embryos alike.

69 struloids, together with the fate map of the mouse embryo, allows the organization of these subpopula

70 essential for any biologist working with the mouse embryo, although the last revision dates back to 1

71 stem cells (TSCs) are derived from the early mouse embryo and can substantially contribute to placent

72 is challenging due to the small size of the mouse embryo and rapid heart rate.

73 tion and morphogenesis: the pre-implantation mouse embryo and the developing mouse olfactory epitheli

74 The specimens are an early mouse embryo and two different cellular spheroids.

75 for anterior-posterior axis formation of the mouse embryo and was shown to promote posterior neuroect

76 endocrine progenitors from different staged mouse embryos and analyzed H3K27me3 genome-wide.

77 ogen signaling and target gene expression in mouse embryos and chick ex vivo assays.

78 nal and naive-like types) were injected into mouse embryos and cultured, some human cells survived bu

79 to the onset of the developmental program in mouse embryos and demonstrate a role for broad H3K4me3 d

80 Here, we show in mouse embryos and differentiating embryonic stem cells t

81 multiple imprinting control regions in early mouse embryos and embryonic stem (ES) cells.

82 and time and has been studied previously in mouse embryos and embryonic stem cell models.

83 d isolated populations of Cdh5(+) cells from mouse embryos and embryonic stem cells can be differenti

84 al and mesodermal cell fate determination in mouse embryos and ESCs.

85 ae, whole-mount chicken embryos, whole-mount mouse embryos and formalin-fixed paraffin-embedded human

86 ncing on the X chromosome in preimplantation mouse embryos and in embryonic stem cells.

87 ndependent off-target C*G-to-T*A mutation in mouse embryos and in rice.

88 the tooth bud mesenchyme in Osr2(-/-) mutant mouse embryos and is partially restored in the tooth mes

89 ositioning during EMT in vivo, in developing mouse embryos and mammary gland, and in vitro, in cultur

90 o obtain high-speed image data from in utero mouse embryos and multi-angle, vector-flow algorithms we

91 and E13.5 Osr2(RFP/+) and Osr2(RFP/-) mutant mouse embryos and performed whole transcriptome RNA sequ

92 s (rsPSCs), can be efficiently obtained from mouse embryos and primate pluripotent stem cells, includ

93 when epigenetic reprogramming occurs: early mouse embryos and primordial germ cell development.

94 using single cells from Etv2-EYFP transgenic mouse embryos and reveal specific molecular pathways tha

95 the craniofacial mesenchyme of mid-gestation mouse embryos and that ablation of Pdgfrb in the neural

96 cause hearing loss, we evaluated Esrp1(-/-) mouse embryos and uncovered alterations in cochlear morp

97 e region of active spinal neurulation in the mouse embryo as a prerequisite for successful NT closure

98 ut 9400 individual cerebellar cells from the mouse embryo at embryonic day 13.5.

99 In the mouse embryo at embryonic day 6.5, cells located at the

100 uthors show that de novo polarisation of the mouse embryo at the 8-cell stage is directed by Phosphol

101 n accessibility in 19,453 single nuclei from mouse embryos at 8.25 days post-fertilization.

102 sl1-expressing cells in the urinary tract of mouse embryos at E10.5 and distributed in the bladder at

103 halon was significantly decreased in Cln5-/- mouse embryos at embryonic day 14.5 (E14.5), and express

104 ally induces right ventricular dilatation in mouse embryos at embryonic day 16.5.

105 type (WT) and Tgf-beta3 -/- homozygous (HM) mouse embryos at the crucial palatogenesis stages of E14

106 cted palatal epithelial cells from embryonic mouse embryos at various palate development stages.

107 epiblast and extraembryonic ectoderm of the mouse embryo become enveloped by a basement membrane.

108 In wild-type mouse embryos, beta-actin expression was prominent in th

109 emonstrate a gene-environment interaction in mouse embryos between Notch1 heterozygosity and low oxyg

110 llular heterogeneities detected in four-cell mouse embryos bias the process of cell fate acquisition

111 rces axial bias in skin spreading around the mouse embryo body.

112 rimary cortical neurons, derived from E16-18 mouse embryos (both sexes), with mito-GFP allowed monito

113 aging and electrophysiological recordings in mouse embryo brainstem slices together with computationa

114 (ICM) and epiblast of the peri-implantation mouse embryo, but its function has not been investigated

115 ired for the development of pre-implantation mouse embryos, but the mechanism accounting for such a r

116 enotypes and hepatitis in late organogenesis mouse embryos, but the molecular and cellular mechanisms

117 l forebrain angiogenesis and BBB function in mouse embryos, but the role of this receptor in adult an

118 Global mapping of STRAP-RNA binding in mouse embryos by enhanced-CLIP sequencing (eCLIP-seq) re

119 e) of 9.5 days post coitus (dpc) to 11.5 dpc mouse embryos by single-cell RNA sequencing and single-c

120 cribes how to observe gastrulation in living mouse embryos by using light-sheet microscopy and comput

121 RNA-sequencing platform in which many mutant mouse embryos can be assayed simultaneously, recovering

122 ectoderm cells of pre-implantation human and mouse embryos can be infected with ZIKV, and propagate v

123 The development of mouse embryos can be partially recapitulated by combinin

124 Mouse embryos carrying Reck(P256A,W261A) have severe def

125 Ablation of Dnmt1 in mouse embryos causes death at the post-gastrulation stag

126 We show that mutation of Fat1 in mouse embryos causes defects in cranial neural tube clos

127 In the CNS of embryonic day 10.5 mouse embryos, CD31(+)F4/80(+) hematopoietic lineage cel

128 tional profiles of 116,312 single cells from mouse embryos collected at nine sequential time points r

129 Mouse embryos conditionally lacking Tgif1 and Tgif2 have

130 nail1/E-cadherin axis described in the early mouse embryo corresponds to Snail2/P-cadherin in the chi

131 lin-like growth factor 1 receptor (Igf1r) in mouse embryos creates a permissive "cell-competitive nic

132 and fates of basal progenitors, we find that mouse embryos defective for the planar cell polarity (PC

133 Here, we examined mouse embryos deficient for the chromatin remodeling pro

134 Accordingly, mouse embryos deficient in Orai1, Klf2, or Klf4 showed a

135 Analysis of miR-34/449 knockout (KO) mouse embryos demonstrates significant spindle misorient

136 artially rescued the phenotypes in Chd7-null mouse embryos, demonstrating that p53 contributes to the

137 a robust BMP signaling gradient in the early mouse embryo depends on the restricted, basolateral loca

138 Here we show that mesoderm organization in mouse embryos depends on beta-Pix (Arhgef7), a guanine n

139 During post-implantation development of the mouse embryo, descendants of the inner cell mass in the

140 Wt1-null mouse embryos develop CDH but the mechanisms regulated b

141 eover, we report that LEC-specific Reln-null mouse embryos develop smaller hearts, that RELN is requi

142 In the absence of liver sinusoidal GATA4, mouse embryos developed hepatic capillaries with upregul

143 cover a crucial role of Brpf1 in controlling mouse embryo development and regulating cellular and gen

144 nutrient concentrations in culture medium on mouse embryo development, metabolism, and quality as a p

145 we show that induced disruption of Rasa1 in mouse embryos did not affect initial specification of LV

146 Yap and Taz in the lymphatic vasculature of mouse embryos did not affect the formation of LVs or LVV

147 iCPCs injected into the cardiac crescent of mouse embryos differentiated into cardiomyocytes.

148 Snx3(-/-) mutant mouse embryos display a fully-penetrant cranial NTD.

149 iac differentiation, whereas Hoxb1-deficient mouse embryos display premature cardiac differentiation.

150 Nkx2-5(-/-) null mouse embryos display severe OFT and RV hypoplasia and a

151 Placentas from Grhl2-deficient mouse embryos displayed defects in BCT cell polarity and

152 Depletion of Unkempt in mouse embryos disrupts the shape of migrating neurons, w

153 rnal provision of embryonic vitamin E to the mouse embryo during neural tube closure.

154 cord tissues after intraspinal injection of mouse embryos (E16).

155 Here we show that CLIC4-null mouse embryos exhibit impaired renal tubulogenesis.

156 an essential gene, as homozygous null mutant mouse embryos exhibit multiple developmental abnormaliti

157 80% of Pax9(del/del);Wise(-/-) double-mutant mouse embryos exhibit rescued palatal shelf elevation/re

158 Setd5-deficient mouse embryos exhibit severe defects in neural tube form

159 nistic changes in early brain development in mouse embryos exposed to this maternal gene-environment

160 ne embryonic stem cell (ESC) line that, like mouse embryos, expresses functional GLUT2 transporters.

161 bl/6NJ genetic background, homozygous Dnaaf2 mouse embryos fail to progress beyond organogenesis stag

162 ontrol fetal human biopsies or in developing mouse embryos, FAT1 is expressed at lower levels in musc

163 by mild chromosome instability in genomes of mouse embryo fibroblast cells from Wwox-knockout mice.

164 ular structures of lamin A, C, B1, and B2 in mouse embryo fibroblast nuclei.

165 C12 activated apoptosis in mouse embryo fibroblasts (MEF) from both wild type (WT)

166 Accordingly, p19(Arf)-deficient mouse embryo fibroblasts (MEFs) arrest in response to ac

167 show that disruption of Cul9-p53 binding in mouse embryo fibroblasts (MEFs) by a knock-in mutation i

168 a, and kidney tumor and that PCBP4-deficient mouse embryo fibroblasts (MEFs) exhibit enhanced cell pr

169 In this report, we demonstrate that mouse embryo fibroblasts (MEFs) lacking all three isofor

170 In mouse embryo fibroblasts (MEFs) lacking CPEB, many mRNAs

171 Mouse embryo fibroblasts (MEFs) lacking Tm5NM1, which ha

172 Furthermore, R137Q mouse embryo fibroblasts (MEFs) were more sensitive to D

173 nd integrin alpha5 proteins in K41 wild-type mouse embryo fibroblasts (MEFs), CRT null MEFs were unre

174 Tgif1; Tgif2-null embryos and in double-null mouse embryo fibroblasts (MEFs).

175 was notably higher than in similarly treated mouse embryo fibroblasts (MEFs).

176 Using genetic deletion in mouse embryo fibroblasts and a combination of CRISPR-med

177 Approximately 50% of reprogrammed mouse embryo fibroblasts displayed spontaneous beating a

178 Genetic inactivation in mouse embryo fibroblasts or CUX2 knockdown in HCC38 cell

179 Here, we show that Rev1-deficiency in mouse embryo fibroblasts or mouse liver tissue is associ

180 The loss of PRMT5 activity in mouse embryo fibroblasts results in almost complete loss

181 Although cultures of wild-type mouse embryo fibroblasts set Myc levels precisely, Myc l

182 ermore, cell-cycle progression of Rev3L(-/-) mouse embryo fibroblasts was arrested in late S/G2 follo

183 tes revealed that the majority of editing in mouse embryo fibroblasts was carried out by ADAR1.

184 ate ADAR1 p150 as the major A-to-I editor in mouse embryo fibroblasts, acting as a feedback suppresso

185 was insufficient to impair 60S biogenesis in mouse embryo fibroblasts, but a profound reduction of BC

186 bryonic stem cells, epiblast stem cells, and mouse embryo fibroblasts, derived from mice of the same

187 ssociates with mitochondria in MAVS knockout mouse embryo fibroblasts.

188 on of IFN-induced, RNA-mediated responses in mouse embryo fibroblasts.

189 ut not in undifferentiated neuronal cells or mouse embryo fibroblasts.

190 ive beta-catenin levels in HEK293T cells and mouse embryo fibroblasts.

191 discuss chromatin reprogramming in the early mouse embryo, focusing on DNA methylation, chromatin acc

192 in is involved in chromatin opening in early mouse embryos for normal development.

193 In mouse embryo gastrulation, epiblast cells delaminate at

194 ich further reveals differences in human and mouse embryo gene expression.

195 The first lineage segregation in the mouse embryo generates the inner cell mass (ICM), which

196 effects of short-term gestational hypoxia on mouse embryos genetically predisposed to heart defects.

197 In the mouse embryo, global epigenetic changes occur during zyg

198 Decades of research in the mouse embryo have revealed that a signaling cascade invo

199 nic and extraembryonic lineages of the early mouse embryo have seemingly co-opted TEs as enhancers, b

200 Moreover, we found that Fkbp8 knockout mouse embryos have abnormal expression of Wnt3a and Nkx2

201 MEFs derived from LCMT-1 knock-out mouse embryos have reduced levels of PP2A B regulatory s

202 Exposure of mouse embryos heterozygous for Tbx1 or Fgfr1/Fgfr2 to hy

203 Here we identify the mouse embryo hindbrain as a powerful model to study embr

204 depletion of integrin-linked kinase (ILK) in mouse embryos hyper-activates VEGFR3 signalling and lead

205 ation from entire Drosophila, zebrafish, and mouse embryos imaged with confocal and light-sheet micro

206 In the mouse embryo in vivo, Ybx1 is required for forebrain spe

207 e expression in both the wrist and digits of mouse embryos in patterns that are nearly indistinguisha

208 ere challenge this view and demonstrate that mouse embryos in the mitotic cell cycle can also directl

209 ng, human embryonic stem cells in vitro, and mouse embryos in vivo, we find that the geometric compar

210 rogeneity was also evident in human ESCs and mouse embryos in vivo.

211 ny features of the pre- and postimplantation mouse embryo, including gastrulation.

212 embryos have several features in common with mouse embryos, including a stage-related initiation of l

213 we generated a variety of editing schemes in mouse embryos, including indel (insertion/deletion) muta

214 ay during preimplantation development of the mouse embryo is known to be essential for differentiatio

215 Here, we show that the first M-phase in the mouse embryo is significantly extended due to a delay in

216 e cells along the anterior-posterior axis of mouse embryos is responsible for left-right symmetry bre

217 orter, GLUT2 (SLC2A2), which is expressed by mouse embryos, is important for survival before embryoni

218 Here, we discovered that female mouse embryos lacking Coup-tfII (chicken ovalbumin upstr

219 Here, we examine the NMJs in mutant mouse embryos lacking either synaptobrevin 1 (Syb1(lew/l

220 Mouse embryos lacking intact maternal/zygotic CTNNB1 fro

221 Heterozygous mouse embryos lacking maternal Plag1 showed disrupted re

222 Here we report that mouse embryos lacking OLA1 have stunted growth, delayed

223 Mouse embryos lacking Shh or its essential signal transd

224 ablation of the entire Id (Id1-4) family in mouse embryos leads to failure of anterior cardiac proge

225 disruption of Wnt/beta-catenin signalling in mouse embryos led to conversion of fundic to antral epit

226 in lymphatic endothelial cells of developing mouse embryos led to defective lymphovenous valve format

227 rized cilia positioning that is essential to mouse embryo left-right asymmetry establishment.

228 Treatment of mouse embryo limb mesenchymal micromass cultures with ex

229 By contrast, Pou5f1-null mouse embryos maintained the expression of orthologous g

230 that cause genomic instability render female mouse embryos markedly more susceptible than males to em

231 al proteins and in-depth characterization of mouse embryos mutant for the Mrp genes Mrpl3, Mrpl22, Mr

232 of both mixed-ratio (1:1 and 10:1) yeast and mouse embryo myogenesis proteomes.

233 cal analysis of cardiac tissues from C57BL/6 mouse embryos, neonatal mice, and adult mice was perform

234 placental defects in the development of CdLS mouse embryos (Nipbl and Hdac8).

235 re, we show that de novo polarisation of the mouse embryo occurs in two distinct phases at the 8-cell

236 Transgenic and mutant studies performed in mouse embryos of either sex and adult males showed that

237 of mTOR complex 1 (mTORC1), was expressed in mouse embryos of either sex via in utero electroporation

238 and GTP levels compared with those in whole mouse embryos or murine cell cultures.

239 tomeres or whole eight-cell pre-implantation mouse embryos, or by conversion of mouse ES cells or ind

240 We showed that in chicken and mouse embryos, PAPC expression is tightly regulated by t

241 nd resolution of tetrads and rosettes in the mouse embryo, possibly in part by spatially restricting

242 oss of potency that takes place in the early mouse embryo prior to the first lineage decisions.

243 nct endodermal cell populations in the early mouse embryo remain ill defined.

244 on precursors in Cdhr23(-/-) and Cdhr15(-/-) mouse embryos, respectively, failed to enter the embryon

245 Hepatocyte-specific deletion of PR-SET7 in mouse embryos resulted in G2 phase arrest followed by ma

246 Vegfc gene deletion in mouse embryos results in failure of lymphangiogenesis, f

247 Analysis of mouse embryos revealed a localized domain of Zic1 expres

248 ad/mesonephros (AGM) regions of midgestation mouse embryos revealed a robust innate immune/inflammato

249 Application to mouse embryos revealed major tissue types in early organ

250 Determination of dNTP pools in mouse embryos revealed that inactivation of one SAMHD1 a

251 Their work gives insight into how the mouse embryo selects cells with the highest future poten

252 In situ hybridization analyses of mouse embryos show that Cnot1 is expressed in the prosen

253 Fkbp8-/- mouse embryos showed posterior NTDs consistent with a di

254 e applied IGS to human fibroblasts and early mouse embryos, spatially localizing thousands of genomic

255 structure of IHC synapses from late prenatal mouse embryo stages (embryonic days 14-18) into adulthoo

256 In an early postimplantation mouse embryo, TEAD4 is selectively expressed in trophobl

257 Mouse embryos that lack all p120-catenin, or that lack p

258 Here we show that mouse embryos that lack the small guanosine triphosphata

259 into regulation of gene expression in 1-cell mouse embryos that may confer a protective mechanism aga

260 w that, contrary to the current view, in the mouse embryo the patella initially develops as a bony pr

261 In the developing mouse embryo, the first hematopoietic stem cells (HSCs)

262 In R137Q knock-in mouse embryos, the BER efficiency was severely impaired,

263 rk that triggers de novo polarisation of the mouse embryo.The molecular trigger that establishes cell

264 large cleared samples ranging from perinatal mouse embryos to adult organs, such as brains or kidneys

265 workflow with laser ablation of live-imaged mouse embryos to investigate the biomechanics of mammali

266 We show that exposure of mouse embryos to short-term gestational hypoxia induces

267 ional analysis focuses on the 32- to 64-cell mouse embryo transition, Embryonic day (E3.25), whose st

268 formation in genetically defective recipient mouse embryos unable to specify (due to Ctnnb1(cnull) mu

269 is sufficient to accommodate an organ-stage mouse embryo under live conditions.

270 The mouse embryo undergoes compaction at the 8-cell stage, a

271 MO-mediated gene knockdown frog and knockout mouse embryos unearthed PCP/CE-related phenotypes as wel

272 epresent all endoderm populations within the mouse embryo until midgestation.

273 ally targeting ZFP57 to reprogrammed loci in mouse embryos using a dCas9 approach, we confirm that ZF

274 ations by editing one blastomere of two-cell mouse embryos using either CRISPR-Cas9 or base editors.

275 ations by editing one blastomere of two-cell mouse embryos using either CRISPR-Cas9 or base editors.

276 pituitary, pancreas, lungs and oesophagus of mouse embryos using in situ hybridization.

277 fluorescent protein tags in human cells and mouse embryos using PCR fragments.

278 Here we compare mouse gastruloids with mouse embryos using single-cell RNA sequencing and spati

279 and nascent Flk1(+) mesoderm of gastrulating mouse embryos using single-cell RNA sequencing, represen

280 ted and free hsa-miR-30d was internalized by mouse embryos via the trophectoderm, resulting in an ind

281 y cilia on neuroepithelial cells in Pam(-/-) mouse embryos was also aberrant.

282 ough experiments conducted in both chick and mouse embryos we have developed a model explaining simul

283 approach to hematopoietic development in the mouse embryo, we map the progression of mesoderm toward

284 seq) of skin in wild-type and IRF6-deficient mouse embryos, we define the transcriptional programs th

285 Nkx2-1 mutants and NKX2-1 ChIP-sequencing in mouse embryos, we delineate the NKX2-1 transcriptional p

286 Using cell lines from Hat1+/+ and Hat1-/- mouse embryos, we demonstrate that Hat1 is not required

287 Inspecting ICM organoids and mouse embryos, we describe a so far unknown local cluste

288 tem cells and Pax3-null embryonic day (E)9.5 mouse embryos, we identified conserved Pax3 functions in

289 Using high-resolution live imaging of mouse embryos, we observed randomly distributed spontane

290 In live mouse embryos, we observed transient neurotransmission a

291 Using mouse embryos, we show that cell protrusions emanating f

292 -in mice, and found that homozygous knock-in mouse embryos were typically small in size and had a hig

293 T1 at an early postimplantation stage of the mouse embryo, when its paralogs Tet2 and Tet3 are not de

294 Usp36 depletion is lethal in preimplantation mouse embryos, where it blocks the transition from morul

295 d the same phenomena in Huntington's disease mouse embryos, where we linked these abnormalities to de

296 ction of RPGRIP1L, we analyzed Rpgrip1l(-/-) mouse embryos, which display a ciliopathy phenotype and

297 chromatin remodeling ATPase LSH (HELLS)-null mouse embryos, which lack DNA methylation from centromer

298 ith special focus on palate formation, using mouse embryos with a complete knockout of Tmem107.

299 malformations observed in human patients and mouse embryos with mutations in Foxc1.

300 siological conditions, including how to hold mouse embryos without agarose embedding, how to transfer