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1 an factors (n = 348, approximately 1% of the mouse genome).
2 pa-Nppb, respectively, were deleted from the mouse genome.
3 the method to comprehensively map pAs in the mouse genome.
4 gth XP-MLV ERVs found in the sequenced C57BL mouse genome.
5 lite markers that are located throughout the mouse genome.
6 obacter sphaeroides and chromosome 16 of the mouse genome.
7 sposase to insert a large transgene into the mouse genome.
8 ll as introduction of polymorphisms into the mouse genome.
9 recombination, we totally deleted it in the mouse genome.
10 erminal repeats [sLTRs]) within the C57BL/6J mouse genome.
11 le-specific DNA methylation (ASM) map in the mouse genome.
12 and then mapping these sequences back to the mouse genome.
13 nd are normalized to the total PETs from the mouse genome.
14 iral superantigen (sag) genes encoded in the mouse genome.
15 ion of recombination initiation sites in the mouse genome.
16 he breaks are nonrandomly distributed in the mouse genome.
17 s, but a SHOX ortholog does not exist in the mouse genome.
18 ottest segment for DSB formation in the male mouse genome.
19 l CpG clusters that were integrated into the mouse genome.
20 g sites, are under negative selection in the mouse genome.
21 d in the rhesus monkey genome but not in the mouse genome.
22 ted and partially redundant MMP genes in the mouse genome.
23 r locus derived from ZIC target genes in the mouse genome.
24 map the orthologous promoter regions in the mouse genome.
25 functionally diverse set of genes across the mouse genome.
26 at the desired genomic locations within the mouse genome.
27 have applied it to the analysis of the whole mouse genome.
28 lotypes at a targeted location (Hprt) in the mouse genome.
29 ites are stronger for the human than for the mouse genome.
30 motif distinct from other CTCF sites in the mouse genome.
31 f heterozygous congenic strains spanning the mouse genome.
32 disease-causing I130T mutant allele into the mouse genome.
33 edite the functional characterization of the mouse genome.
34 d-like CVC (Prd-L:CVC) homeobox genes in the mouse genome.
35 stigating the recombination landscape of the mouse genome.
36 of DNA microarrays representing the complete mouse genome.
37 ith cis-regulatory motifs and modules in the mouse genome.
38 0 probes randomly distributed throughout the mouse genome.
39 we have inactivated this binding site in the mouse genome.
40 ing genomic region at insertion sites in the mouse genome.
41 ing evidence for periodicity in the human or mouse genome.
42 f antisense transcripts in the human and the mouse genome.
43 [CT(G/T)CCAGGACCT] occupied by ZBTB24 in the mouse genome.
44 fficiency for transgene integration into the mouse genome.
45 otropic murine leukemia virus content of the mouse genome.
46 ed deletions at 17 sites in four loci of the mouse genome.
47 e brought in cis-regulatory modules into the mouse genome.
48 ied, representing over 60% of the TFs in the mouse genome.
49 2 sgRNAs for human and 40,860 sgRNAs for the mouse genome.
50 acid sequence, using targeted editing of the mouse genome.
51 etic nephropathy since Nox5 is absent in the mouse genome.
52 p to 50% of CpG dinucleotides throughout the mouse genome.
53 e extent of molecular scars inflicted on the mouse genome.
54 lic nucleotide-gated channel 4) locus of the mouse genome.
55 t are located adjacent to one another in the mouse genome.
56 utionarily conserved TF binding sites on the mouse genome.
57 er unrepresented or totally absent, from the mouse genome.
58 e most recently active ERVs in the human and mouse genome.
59 of 41 imprinting gene clusters known in the mouse genome.
60 ssed Pol II-transcribed genes throughout the mouse genome.
61 and neurodegeneration when knocked into the mouse genome.
62 elerate tumorigenesis when engineered in the mouse genome.
63 iated from endogenous L1 elements across the mouse genome.
64 maintain silencing of a large portion of the mouse genome.
65 all the functional elements in the human and mouse genomes.
66 at are expressed across the entire human and mouse genomes.
67 genome-wide screening for both the human and mouse genomes.
68 e organization and function of the human and mouse genomes.
69 -responsive elements (EREs) in the human and mouse genomes.
70 f hybrid cells with respect to the human and mouse genomes.
71 annotation of bimodal genes in the human and mouse genomes.
72 ranscripts have been identified in human and mouse genomes.
73 annel subunits encoded by the human, rat and mouse genomes.
74 comparing the 129X1/SvJ and C57BL/6J inbred mouse genomes.
75 s comprise a large fraction of the human and mouse genomes.
76 d its targets-are prevalent in the human and mouse genomes.
77 l known and predicted genes in the human and mouse genomes.
78 the RE1 and used it to search the human and mouse genomes.
79 istribution of retroposed genes in human and mouse genomes.
80 Ri and CRISPRa libraries targeting human and mouse genomes.
81 des with most core origins in both human and mouse genomes.
82 port a novel DNA sequence motif in human and mouse genomes.
83 equence similarities to that region in other mouse genomes.
84 n evolution of GGI networks in the human and mouse genomes.
85 rtebrates, with an emphasis on the human and mouse genomes.
86 caspase that is found in the human, and not mouse, genome.
87 f their target genes in mammalian (human and mouse) genomes.
92 of this Mef2c neural crest enhancer from the mouse genome abolishes Endothelin induction of Mef2c exp
93 erformed genomic DNase I footprinting of the mouse genome across 25 cell and tissue types, collective
95 ely 1,200 odorant receptor (OR) genes in the mouse genome, an olfactory sensory neuron is thought to
96 study, we collected 73643859 SNPs across the mouse genome, analyzed 1187 pre-miRNAs and 2027 mature m
98 th K-ras and p53 missense mutations into the mouse genome and established a more faithful genetic mod
99 human mutant huntingtin allele in the native mouse genome and gradually manifest symptoms late in lif
100 lion copies, B2 accounts for ~350,000 in the mouse genome and has garnered special interest because o
101 or site within intron 1, is conserved in the mouse genome and is widely expressed in mouse tissues.
102 ional Nna1-like genes were identified in the mouse genome and named cytosolic carboxypeptidase (CCP)
103 that the ERbeta gene was eliminated from the mouse genome and that no ERbeta mRNA or protein was dete
104 alysis to identify similar structures in the mouse genome and validated that these host structures al
105 inding sites for RBP motifs across human and mouse genomes and allows large scale querying of predict
106 enes have alternative promoters in human and mouse genomes and approximately 40% of promoters are tis
107 earch in the intergenic regions of human and mouse genomes and between the Burkholderia cenocepacia J
108 We tested GRAPeFoot using the human and mouse genomes and compared its performance to a set of p
110 -length DNA sequence motifs in the human and mouse genomes and sub-selected those with multiple recur
111 s approaching that of the finished human and mouse genomes and suggests an affordable roadmap to high
112 The annotated sequences add up to 11% of the mouse genome, and include more than 70% of conserved non
113 ci of both proviruses were determined in the mouse genome, and integration site information was used
115 cisely mapped, and their distribution in the mouse genome appears random relative to transcription un
116 Most transposable elements (TEs) in the mouse genome are heavily modified by DNA methylation and
119 2 (major capsid protein) EVE sequence from a mouse genome assembled into capsids that had a similar s
121 50 kb and map to over 95.6% of the reference mouse genome assembly (NCBIm37), covering 98.8% of Ensem
122 of 205 kb and map to 93.9% of the reference mouse genome assembly, covering 95.7% of Ensembl genes.
123 new interfaces for exploring regions of the mouse genome associated with cancer phenotypes and incor
126 provements include integration of the latest mouse genome build (GRCm38), improved access to comparat
128 ir genetic blueprint is still present in the mouse genome but is normally modified to the needs of th
131 extend our initial observation to the entire mouse genome by enriching for methylated DNA with the Me
132 e report a cloning-free method to target the mouse genome by pronuclear injection of a commercial Cas
133 sults suggest that SB Tn insertions into the mouse genome can be discriminated by DNA methylation mac
135 oadmap of hepatic PXR bindings in the entire mouse genome [chromatin immunoprecipitation (ChIP)-Seq].
142 mutation into the homologous position in the mouse genome, creating mice that were heterozygous and h
150 currence, we surveyed the Jackson Laboratory Mouse Genome Database for knockout mouse strains and the
159 P-MLV ERVs map to the 95% of the laboratory mouse genome derived from P-MLV-infected M. m. domesticu
160 and are embedded in the 5% of the laboratory mouse genome derived from the Asian Mus musculus musculu
161 Individual deletion of these sites from the mouse genome did not alter expression of any of the gene
162 se divergent intervals span 19% of the house mouse genome, disproportionately aggregating on the X ch
165 d probes, optical microscopy techniques, and mouse genome editing hold great potential over the next
167 sceptible to elimination, as occurred in the mouse genome, either by chance or selection on one of th
168 r in mediating this phenotype.IMPORTANCE The mouse genome encodes a family of Oas proteins that synth
169 nd molecular functions in both the human and mouse genomes even though the underlying sequences are n
170 ed survey of asynchronous replication in the mouse genome, excluding known asynchronously replicated
171 rehensive, unified, non-redundant catalog of mouse genome features generated by distilling gene predi
172 high-density BAC arrays covering >95% of the mouse genome for analysis of genomic patterns of aberrat
173 The binding motif was used to search the mouse genome for candidate 2.102 reactive allopeptides t
174 ces and/or the vast noncoding regions of the mouse genome for large-scale functional genomic analysis
179 of variation found in the inbred laboratory mouse genome has increased to 71 M SNPs and 12 M indels.
180 e immense challenge of annotating the entire mouse genome has stimulated the development of cutting-e
182 far, most of the functional sequences in the mouse genome have yet to be found, and the cis-regulator
183 the transcriptional output of the human and mouse genomes have revealed that there are many more tra
186 ee-dimensional organization of the human and mouse genomes in embryonic stem cells and terminally dif
187 the structure and dynamics of the human and mouse genomes in space and time with the goal of gaining
188 of parameters, our designs cover 78% of the mouse genome including many regions previously considere
189 tially in putative regulatory regions of the mouse genome, including CpG islands and non-exonic ultra
190 ave constructed a revised genetic map of the mouse genome, incorporating 10,195 single nucleotide pol
192 combined with genetic and phenotypic data in Mouse Genome Informatics (MGI) and made readily accessib
193 e Database (MGD) is a major component of the Mouse Genome Informatics (MGI) database resource and ser
197 gical context through integration with other Mouse Genome Informatics (MGI) resources and interconnec
198 rmation is integrated with the other data in Mouse Genome Informatics and interconnected with other d
199 type Ontology, EBI's Ontology Working Group, Mouse Genome Informatics and the Monarch Initiative amon
200 tools that included GeneWeaver, String, and Mouse Genome Informatics identified a total of nine addi
203 to generate mutations for every gene in the mouse genome is a first and essential step in this endea
205 implies that a near-complete coverage of the mouse genome is obtainable with this approach using 20 g
208 al collaborative efforts, a large portion of mouse genome is still poorly characterized for cellular
210 nd description of the genes in the human and mouse genomes is a fundamental requirement for high qual
211 n variable-gene repertoire (2.7 Mb) into the mouse genome, leaving the mouse constant regions intact.
212 t1 transcripts or deletion of Mist1 from the mouse genome led to increased cell proliferation and a c
213 (Hsa21) are syntenic to three regions in the mouse genome, located on mouse chromosome 10 (Mmu10), Mm
216 sequences, we steadily characterized a whole mouse genome miRNA related SNPs, analyzed their effects
218 ntly contains binding sites across the whole-mouse genome of 8 NRs identified in 40 chromatin immunop
221 principal technology for manipulation of the mouse genome, offering unrivalled accuracy in allele des
222 s concluded that paralogous genes within the mouse genome or the human genome are more functionally s
223 vidence of PXR DNA-binding signatures in the mouse genome, paving the path for predicting and further
226 xed models using worked examples from Sanger Mouse Genome Project focusing on Dual-Energy X-Ray Absor
227 etions (indels) present in SPRET/Ei from the Mouse Genomes Project (Wellcome Trust Sanger Institute)
230 g human FcgammaRIIA gene, not present in the mouse genome, recapitulated the human situation and resc
234 m line, occupy ~8% and ~10% of the human and mouse genomes, respectively, and affect their structure,
235 We identified 1301 and 997 RE1s inhuman and mouse genomes, respectively, of which >40% are novel.
237 d as transgenes, have been inserted into the mouse genome resulting in recapitulation of the unique p
244 pping the BAC end-sequences to the reference mouse genome sequence, was 7.1-fold across the autosomes
245 an histone acetylation island sequences with mouse genome sequences, we find that despite the conserv
246 ject was launched a decade ago to complement mouse genome sequencing efforts by promoting new phenoty
250 leases of the human, chimp, dog, macaque and mouse genomes, so that the results can be compared direc
251 than 50 class I MHC (MHCI) molecules in the mouse genome, some of which are now known to be expresse
252 2 PDZ domains and 93 peptides encoded in the mouse genome, successfully predicts interactions involvi
253 ne targeting allows precise tailoring of the mouse genome such that desired modifications can be intr
254 efficiency between two L1 variants from the mouse genome, T(FC) and T(Fspa), to a single amino acid
256 s and fewer losses of duplicate genes in the mouse genome than in the human genome, although the popu
257 troducing an X chromosome duplication in the mouse genome that contains Plp1 and five neighboring gen
258 million primer pairs targeting the human and mouse genomes that is readily usable for rapid and flexi
263 combining chromatin immunoprecipitation with mouse genome tiling array profiling, we identify Foxp3 b
264 used CRISPR-Cas9 gene editing to modify the mouse genome to encode two amino acids (positions 288 an
267 length Emu region (core plus MARsEmu) in the mouse genome to study VH gene repertoire and IgH express
269 annotated conserved islands in the human and mouse genomes to transcribed regions and to RFBSs, relyi
270 y (which allows for rapid engineering of the mouse genome) to quickly develop and evaluate antibodies
271 nnotated bimodal or switch-like genes in the mouse genome using a large collection of microarray data
272 (TDMRs) were identified and localized in the mouse genome using second generation virtual RLGS (vRLGS
276 ion of cytosine methylation in 6.2 Mb of the mouse genome was tested using cohybridization of genomic
279 fying thousands of TERRA target sites in the mouse genome, we demonstrate that TERRA can bind both in
280 roughput sequencing of a 2.5Mb region of the mouse genome, we discovered a mutation in the grainyhead
281 robable transcription factors encoded in the mouse genome, we identify 12 novel transcription factors
282 ity candidate genes in a small region of the mouse genome, we noticed that many genes when knocked ou
283 istribute loxP sites randomly throughout the mouse genome, we present a simple but comprehensive meth
284 ated CpG islands and promoter regions in the mouse genome, we report the genome-wide DNA methylation
285 culin tension sensor that we knock-in to the mouse genome, we show that cortical force oscillations p
287 Using markers distributed throughout the mouse genome, we use a hybrid zone between two recently
289 affinities for core histone proteins in the mouse genome were not altered by removal of core histone
292 used a glioma model to identify loci in the mouse genome, which were repeatedly targeted by platelet
293 allelic variants of one or more genes in the mouse genome whose human orthologues should be important
294 vised sgRNA design rules to create human and mouse genome-wide libraries, perform positive and negati
295 In contrast to human population studies, our mouse genome-wide searches find loci that individually e
297 uman genome, so a thorough annotation of the mouse genome will be of significant value to understandi
299 e map of genetic variation in the laboratory mouse genome will provide insights into the evolutionary
300 positions of practically all hotspots in the mouse genome, with the exception of the pseudo-autosomal