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1 ted neurotropic coronavirus (rJ2.2 strain of mouse hepatitis virus).
2 ells were highly susceptible to a murine CoV-mouse hepatitis virus.
3 ing infection of p85beta-deficient mice with mouse hepatitis virus.
4 l infection by the neurotropic JHM strain of mouse hepatitis virus.
5 S infection by the neurotropic JHM strain of mouse hepatitis virus.
6 egion of the positive-stranded RNA genome of mouse hepatitis virus.
7 ternative receptor for the mouse coronavirus mouse hepatitis virus.
8 central nervous system by the JHM strain of mouse hepatitis virus.
9 bute to the persistence of the JHM strain of mouse hepatitis virus.
10 alphavirus chikungunya virus and coronavirus mouse hepatitis virus.
11 nucleocapsid protein of a model coronavirus, mouse hepatitis virus.
12 control acute infection with the cytopathic mouse hepatitis virus.
16 rmine the role of MDA5 during infection with mouse hepatitis virus, a murine coronavirus used to mode
17 ssibility was examined in mice infected with mouse hepatitis virus, a well-described model of virus-i
18 a mildly pathogenic neurotropic coronavirus (mouse hepatitis virus A59 strain [MHV-A59]) developed se
20 central nervous system (CNS) by neurotropic mouse hepatitis virus but do not suffice to achieve ster
21 I Ag presentation, H-2d-restricted HIV-1 and mouse hepatitis virus CTL epitopes were linked via vario
23 ysis of mice infected with the JHM strain of mouse hepatitis virus demonstrated that, in contrast to
26 d of the 3' untranslated region (UTR) of the mouse hepatitis virus genome contains two essential and
27 IP-10(-/-) mice infected with a neurotropic mouse hepatitis virus had an impaired ability to control
28 gnals of the SARS-associated coronavirus and mouse hepatitis virus have evolved to promote optimal fr
29 he pathogenesis of the neurotropic strain of mouse hepatitis virus in Fas-deficient mice suggested th
30 ein, encoded by ORF6, enhanced the growth of mouse hepatitis virus in tissue culture cells and in mic
31 rected against CCL5 to mice with established mouse hepatitis virus-induced demyelination and impaired
34 astrocytes during acute encephalomyelitis in mouse hepatitis virus-infected mice, and the majority of
37 infection with the neurotropic JHM strain of mouse hepatitis virus is inhibited in the absence of CD4
38 The MHV-JHM strain of the murine coronavirus mouse hepatitis virus is much more neurovirulent than th
40 For example, C57BL/6 (B6) mice infected with mouse hepatitis virus (JHM strain, JHMV) develop severe
41 ures containing CNS cells were infected with mouse hepatitis virus-JHM, which causes fatal encephalit
42 BL/6 mice with the neurotropic JHM strain of mouse hepatitis virus (JHMV) (a member of the Coronaviri
43 cephalomyelitis induced by the JHM strain of mouse hepatitis virus (JHMV) and sustained during viral
44 infected with the neurotropic JHM strain of mouse hepatitis virus (JHMV) clear infectious virus; nev
45 infected with the neurotropic JHM strain of mouse hepatitis virus (JHMV) develop acute and chronic d
46 ystem (CNS) by the neurotropic JHM strain of mouse hepatitis virus (JHMV) induces an acute encephalom
47 oculation with the neurotropic JHM strain of mouse hepatitis virus (JHMV) into the central nervous sy
48 al nervous system (CNS) by the JHM strain of mouse hepatitis virus (JHMV) is a rodent model of the hu
51 infection with the neurotropic JHM strain of mouse hepatitis virus (JHMV) resulted in an acute enceph
52 mouse CNS with the neurotropic JHM strain of mouse hepatitis virus (JHMV) results in an immune-mediat
60 here the outcome of viral encephalomyelitis [mouse hepatitis virus (MHV) A59, Theiler's encephalomyel
61 e that infection with the murine coronavirus mouse hepatitis virus (MHV) activated the NLRP3 inflamma
63 pathogens, including the murine coronavirus mouse hepatitis virus (MHV) and Haemophilus influenzae,
64 wo viruses within the genus Betacoronavirus, mouse hepatitis virus (MHV) and MERS-CoV, encode 2',5'-p
65 of the 5' untranslated regions (5'UTRs) from mouse hepatitis virus (MHV) and severe acute respiratory
66 evere acute respiratory syndrome (SARS)-CoV, mouse hepatitis virus (MHV) and the human CoV OC43 S2 su
70 e glycoprotein (S) of the murine coronavirus mouse hepatitis virus (MHV) binds to viral murine CEACAM
75 ve interfering (DI) RNA of the JHM strain of mouse hepatitis virus (MHV) consist of three discontinuo
76 2 (SL2) of the 5'-untranslated region of the mouse hepatitis virus (MHV) contains a highly conserved
78 positive-sense RNA genome of the coronavirus mouse hepatitis virus (MHV) contains sequences that are
79 spike glycoprotein from strain RSA59 (PP) of mouse hepatitis virus (MHV) contains two central, consec
80 ve (ts) mutant helper virus, two coronavirus mouse hepatitis virus (MHV) defective interfering (DI) R
83 tep toward understanding the function of the mouse hepatitis virus (MHV) E protein, we carried out cl
84 on with the recombinant JHM (RJHM) strain of mouse hepatitis virus (MHV) elicits a weak CD8(+) T-cell
85 e replicase gene (gene 1) of the coronavirus mouse hepatitis virus (MHV) encodes two co-amino-termina
89 Using the recombinant murine coronavirus mouse hepatitis virus (MHV) expressing the T cell-chemoa
92 ithin the 3'-terminal 166 nucleotides of the mouse hepatitis virus (MHV) genome and assessed their fu
95 ream of the leader at the 5' terminus of the mouse hepatitis virus (MHV) genomic RNA, contains a sequ
97 Previous work with the prototype coronavirus mouse hepatitis virus (MHV) has shown that a major deter
100 o the nonessential gene 4 of the coronavirus mouse hepatitis virus (MHV) in order to test the applica
101 constructed mutants of the model coronavirus mouse hepatitis virus (MHV) in which all or part of the
102 , we constructed a mutant of the coronavirus mouse hepatitis virus (MHV) in which the ectodomain of t
103 of 17Cl-1 cells with the murine coronavirus mouse hepatitis virus (MHV) induced caspase-dependent ap
112 RNA synthesis by the prototype coronavirus mouse hepatitis virus (MHV) is carried out by a replicas
113 processing of the replicase gene product of mouse hepatitis virus (MHV) is essential for viral repli
117 rotective roles of specific ISGs against the mouse hepatitis virus (MHV) members of the coronaviruses
119 ave demonstrated that the murine coronavirus mouse hepatitis virus (MHV) nonstructural protein 2 (ns2
120 uclear magnetic resonance (NMR) structure of mouse hepatitis virus (MHV) nsp3a and show, using isothe
128 on of cultured cells with murine coronavirus mouse hepatitis virus (MHV) resulted in activation of th
129 ed that infection of rat oligodendrocytes by mouse hepatitis virus (MHV) resulted in apoptosis, which
130 Intracranial infection of C57BL/6 mice with mouse hepatitis virus (MHV) results in an acute encephal
132 (hnRNP) A1 has previously been shown to bind mouse hepatitis virus (MHV) RNA at the 3' end of both pl
134 shown to function as regulatory factors for mouse hepatitis virus (MHV) RNA synthesis as a result of
139 accumulation kinetics of murine coronavirus mouse hepatitis virus (MHV) RNAs early in infection by u
142 s are important for clearance of neurotropic mouse hepatitis virus (MHV) strain A59, although their p
149 erfering (DI) RNAs of the murine coronavirus mouse hepatitis virus (MHV) suggest that a 69-nucleotide
150 have previously generated E gene mutants of mouse hepatitis virus (MHV) that had marked defects in v
151 the current studies, neurotropic strains of mouse hepatitis virus (MHV) that induce meningitis, ence
152 previously generated E gene point mutants of mouse hepatitis virus (MHV) that were defective in growt
153 s, we previously generated E gene mutants of mouse hepatitis virus (MHV) that were defective in viral
155 ort that infection by the murine coronavirus mouse hepatitis virus (MHV) triggers the proximal UPR tr
160 ic packaging signal (PS) for the coronavirus mouse hepatitis virus (MHV) was originally identified as
162 that AhR is activated in cells infected with mouse hepatitis virus (MHV), a coronavirus (CoV), and co
168 is question, we used the rJHM strain (rJ) of mouse hepatitis virus (MHV), a neurotropic coronavirus t
169 he N-terminal domain (NTD) of N protein from mouse hepatitis virus (MHV), a virus most closely relate
170 a Sarbecovirus, and the packaging signal of mouse hepatitis virus (MHV), an Embecovirus, which is a
171 encoded by the prototypical Betacoronavirus, mouse hepatitis virus (MHV), and by Middle East respirat
173 spectively, including the murine coronavirus mouse hepatitis virus (MHV), Haemophilus influenzae, Nei
177 epitope of a highly neurovirulent strain of mouse hepatitis virus (MHV), JHM, is thought to be essen
179 ells infected with a prototypic coronavirus, mouse hepatitis virus (MHV), resulting in the expression
182 nhibited virus entry and cell-cell fusion of mouse hepatitis virus (MHV), suggesting the importance o
185 s study, we exploited the model coronavirus, mouse hepatitis virus (MHV), to investigate the genotype
186 we report genetic analysis of a ts strain of mouse hepatitis virus (MHV), tsNC11, focusing on the rol
187 ns in the N protein of the model coronavirus mouse hepatitis virus (MHV), we constructed mutants in w
188 ions of N protein domains in the coronavirus mouse hepatitis virus (MHV), we replaced the MHV N gene
189 terfering (DI) RNA of the murine coronavirus mouse hepatitis virus (MHV), when introduced into MHV-in
190 tory syndrome coronavirus 2 (SARS-CoV-2) and mouse hepatitis virus (MHV)-A59 and identified conserved
191 ocalization and expression is altered due to mouse hepatitis virus (MHV)-A59 infection both in vivo a
192 a group of mutants of the murine coronavirus mouse hepatitis virus (MHV)-A59, isolated from persisten
195 togen-activated protein kinases (MAPKs) in a mouse hepatitis virus (MHV)-infected macrophage-derived
209 ralized the infectivity of the A59 strain of mouse hepatitis virus (MHV-A59) in a concentration-depen
211 N did not serve as a functional receptor for mouse hepatitis virus (MHV-A59), which is in serogroup I
215 n of C57BL/6 mice with the V5A13.1 strain of mouse hepatitis virus (MHV-V5A13.1) results in an acute
218 uced into a heterologous murine coronavirus (mouse hepatitis virus [MHV]) but is not essential for op
219 mice with a recombinant murine coronavirus (mouse hepatitis virus [MHV]) expressing the T-cell chemo
220 vation of RNase L during murine coronavirus (mouse hepatitis virus [MHV]) infection of myeloid cells
222 infected with the neurotropic JHMV strain of mouse hepatitis virus mount potent regional CTL response
223 udied the interaction between coronaviruses (mouse hepatitis virus) of different neurovirulences with
224 Early during clearance of the JHM strain of mouse hepatitis virus, only few virus-specific Ab-secret
225 ce show reduced inflammasome activation upon mouse hepatitis virus or rotavirus infection, and in ste
226 utralization is critical for maintaining JHM mouse hepatitis virus persistence within the central ner
227 cellular immunity in controlling neurotropic mouse hepatitis virus persistence within the CNS were de
228 The neurotropic coronavirus JHM strain of mouse hepatitis virus persists in oligodendroglia despit
229 tem (CNS) with the neurotropic JHM strain of mouse hepatitis virus produces acute and chronic demyeli
230 Infection by the neurotropic JHM strain of mouse hepatitis virus produces an acute demyelinating en
233 we found that proteasome inhibitors blocked mouse hepatitis virus replication at an early step in th
234 binant CXCL10-expressing murine coronavirus (mouse hepatitis virus) resulted in protection from disea
235 most neurotropic strains of the coronavirus mouse hepatitis virus results in an immune response-medi
236 tion of the neuroattenuated OBLV60 strain of mouse hepatitis virus results in infection of mitral neu
237 vel 2 conditions, we constructed recombinant mouse hepatitis viruses (rMHVs) (rMHV strain A59) that e
238 on activation gene 2(-/-) mice with only non-mouse hepatitis virus-specific T cells, we show that CD8
239 al (i.n.) infection of A/J mice with the CoV mouse hepatitis virus strain 1 (MHV-1) induces an acute
240 here investigation into the genetic basis of mouse hepatitis virus strain 1 (MHV-1) pneumovirulence.
241 ially induced in primary mouse astrocytes by mouse hepatitis virus strain A59 (MHV-A59) and MHV-2.
245 ersistently infected with murine coronavirus mouse hepatitis virus strain A59 (MHV-A59), expression o
246 was recently established for the coronavirus mouse hepatitis virus strain A59 (MHV-A59), in which cDN
248 se data suggest that clearance of infectious mouse hepatitis virus strain A59 from the CNS requires A
250 hether B cells are important for controlling mouse hepatitis virus strain A59 infection, we infected
255 ce infected with the neurotropic coronavirus mouse hepatitis virus strain JHM (MHV-JHM) develop a chr
258 ons, C57BL/6 mice persistently infected with mouse hepatitis virus strain JHM (MHV-JHM) develop clini
266 Mice infected with the murine coronavirus, mouse hepatitis virus, strain JHM (MHV) develop an immun
268 Mice infected with attenuated strains of mouse hepatitis virus, strain JHM, develop a chronic inf
269 pes recognized in C57BL/6 mice infected with mouse hepatitis virus, strain JHM, or lymphocytic chorio
273 t has recently been shown that cell entry of mouse hepatitis virus type 2 (MHV-2) is mediated through
274 ctivity of PLPs from SARS-CoV, MERS-CoV, and mouse hepatitis virus was evaluated against seven ISG15s
275 The importance of the CD (SWWSFNPETNNL) in mouse hepatitis virus was investigated with a panel of m
277 nonpathogenic (V-2) variant of a neurotropic mouse hepatitis virus, which differ in the ability to pe
278 well as mice infected with the JHM strain of mouse hepatitis virus, which exhibit CTL escape variants
279 embrane (M) protein of the model coronavirus mouse hepatitis virus with its counterpart from a hetero
280 Replication of the neurotropic JHM strain of mouse hepatitis virus within the central nervous system