戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 nicity of ectromelia, the causative agent of mousepox.
2  infection with ECTV, the causative agent of mousepox.
3  capable of protecting susceptible mice from mousepox.
4 nant type I IFN bp protects mice from lethal mousepox.
5 ovided significant protection against lethal mousepox.
6  the susceptibility of CL13-infected mice to mousepox.
7  by long-lived memory CD8(+) T cells prevent mousepox, a disease caused by ectromelia virus, a close
8 ential for the resistance of C57BL/6 mice to mousepox, a disease caused by the Orthopoxvirus ectromel
9 V) clone 13 (CL13) are highly susceptible to mousepox, a disease that is caused by ectromelia virus a
10 nown that C57BL/6 (B6) mice are resistant to mousepox, a lethal disease caused by the orthopoxvirus e
11 lls protect susceptible strains of mice from mousepox, a lethal viral disease caused by ectromelia vi
12               Ectromelia virus (ECTV) causes mousepox, a surrogate mouse model for smallpox caused by
13 +) T cells can protect susceptible mice from mousepox, an acute lethal viral disease.
14 t NK cells efficiently protected from lethal mousepox and controlled MCMV titers in the spleen.
15 ge with ectromelia virus (ECTV; the agent of mousepox) and that natural killer (NK) cells in CL13-inf
16 ia, monkeypox, camelpox, cowpox, ectromelia (mousepox), and variola viruses.
17 RNA-LNP protected most susceptible mice from mousepox, and booster vaccination increased the memory C
18 ce chronically infected with CL13 succumb to mousepox but that most of those that recovered from acut
19 oxvirus species variola, vaccinia, camelpox, mousepox, cowpox, and monkeypox virus.
20 rus infection of humans (smallpox) and mice (mousepox [ectromelia virus {ECTV}]) despite the lack of
21 esented by HLA-B*07:02 (B7.2) molecules in a mousepox/ectromelia virus (ECTV)-infection, B7.2 transge
22 ss II-restricted TCD4+ repertoire induced by mousepox (ECTV) infection and the functional profile of
23 uivalent ectromelia virus (ECTV; an agent of mousepox), encode immune response modifiers (IRMs) that
24 virus, which causes a smallpox-like disease (mousepox) in mice, was determined and allows for classif
25    EVM1 is abundantly expressed early during mousepox infection and is able to selectively bind CC ch
26 te resistance of C57BL/6 (B6) mice to lethal mousepox is controlled by multiple genes.
27 log in ectromelia virus (ECTV), the agent of mousepox, is EVM036.
28 d NKT cells are not important in the C57BL/6 mousepox model and CD4+ and CD8+ T cells do not exhibit
29 required for CD4-CTL differentiation and for mousepox resistance.
30   We show that mice genetically resistant to mousepox (the mouse parallel of human smallpox) lose res
31 rotypic immunity to smallpox, monkeypox, and mousepox, the mechanistic basis for which is poorly unde
32 t at protecting susceptible mice from lethal mousepox, the mouse equivalent of human smallpox.
33 shed importance of NK cells in resistance to mousepox, these data suggest that the NK cell dysfunctio
34 T(m)s, 61.3 to 63.7 degrees C), 8 strains of mousepox virus (T(m), 61.9 degrees C), and 8 strains of
35 of viral virulence for the ectromelia virus (mousepox virus) in a murine infection model.
36 ts are immunogenic in cowpox and ectromelia (mousepox) virus infections, and immunization with the im
37 ctive variant peptide encoded by ectromelia (mousepox) virus.
38 ural resistance of nonimmune C57BL/6 (B6) to mousepox, we show that memory CD8(+) T cells of single s