ライフサイエンスコーパス: msec

1 CV: -0.3% +/- 2.5), and per-segment (T2: 0.0 msec +/- 6.5; ECV: -0.4% +/- 3.5) analyses.

2 error of the mean]) than at 0 degrees (10.0 msec +/- 0.7, P < .001) or 90 degrees (9.9 msec +/- 0.4,

3 .001) and 40% +/- 10 lower in canines (23.0 msec +/- 4.0 vs 39.3 msec +/- 2.5, P < .001).

4 scle tissue: T1, 1417 msec +/- 106; T2, 31.0 msec +/- 2.4; T2*, 11.3 msec +/- 1.7; and ADC, 1.40 x 10

5 uscle tissue: T1, 1386 msec +/- 88; T2, 32.0 msec +/- 4.3; T2*, 10.8 msec +/- 0.8; ADC, 1.39 x 10(-3)

6 MOLLI had similar precision to ShMOLLI (4.0 msec vs 5.6 msec; P = .07) but higher precision than SAP

7 s (rest vs stress, 44.5 msec +/- 2.6 vs 49.0 msec +/- 5.5, respectively; P = .004; MBR, 1.1 msec +/-

8 f interest in an osteochondral lesion = 50.0 msec +/- 10.2) in comparison to adjacent intact cartilag

9 findings, mean T2 of both hemorrhagic (62.0 msec +/- 4.9) and nonhemorrhagic (71.7 msec +/- 7.3) inf

10 on with covariates of age (beta at 1.5 T = 0 msec per year, P = .70; beta at 3.0 T = 0 msec per year,

11 0 msec per year, P = .70; beta at 3.0 T = 0 msec per year, P = .83) or sex (beta at 1.5 T = -1 msec,

12 ectively, and T2-weighted VIP was equal to 0 msec and 303 msec (range, 39-1012 msec), respectively (P

13 with FA (b = -0.0004), T2(water) (b = -0.03 msec), and FF (b = -0.1%) at thigh level (P < .001).

14 3D UTE sequence at free breathing (TE, 0.03 msec).

15 ng single breath holds (echo time [TE], 0.05 msec) and with a self-navigated "Koosh ball" 3D UTE sequ

16 sotropic spatial resolution; echo time, 0.08 msec) were performed at 3 T.

17 2.9; ECV: -0.3% +/- 1.7), per-slice (T2: 0.1 msec +/- 4.6; ECV: -0.3% +/- 2.5), and per-segment (T2:

18 2 msec per heartbeat +/- 0.2 for T2 and -0.1 msec per heartbeat +/- 0.2 for T2*.

19 ec +/- 5.5, respectively; P = .004; MBR, 1.1 msec +/- 8.08).

20 rol subjects (1185.3 msec +/- 49.3 vs 1089.1 msec +/- 44.9, respectively; P < .001).

21 s of recipient mice (mean, 21.7 msec vs 27.1 msec, respectively; P = .0444).

22 ts revealed a mean standard deviation of 4.1 msec (0.412%).

23 lative to mean T2 of remote myocardium (52.1 msec +/- 4.8) by 18% +/- 9 and 38% +/- 13, respectively

24 er year, P = .83) or sex (beta at 1.5 T = -1 msec, P = .88; beta at 3.0 T = 6 msec, P = .42).

25 red by BF electrical stimulation within 5-10 msec.

26 Sokolow-Lyon voltage (beta = 15.1 microV/10 msec, P = .004), lower QRS Cornell voltage (beta = 9.2 m

27 er QRS Cornell voltage (beta = 9.2 microV/10 msec, P = .031), and shorter QRS duration (beta = 0.16 m

28 nd shorter QRS duration (beta = 0.16 msec/10 msec, P = .049).

29 ice were exposed to standard long pulses (10 msec, 0.5 Hz) containing 150 times more acoustic energy.

30 repetition time, 600-750 msec; echo time, 10 msec; in-plane resolution, 196 mm).

31 d in the bilateral hippocampus (1.68 MHz, 10-msec bursts, 1-Hz burst repetition frequency, 120-second

32 0 healthy control subjects to both long (100 msec) and short (50 msec) duration deviant sounds.

33 Ca(2+) is sufficient to trigger rapid (<100 msec) and synchronous fusion of the docked vesicles.

34 ncrease in females (1017 msec +/- 27 vs 1001 msec +/- 33, P = .066).

35 Myocardial T1 (978 msec +/- 23 vs 1006 msec +/- 29 vs 1044 msec +/- 14; P < .001) and T2 relaxa

36 Average native T1 was 1008 msec +/- 31, with a nonsignificant increase in females (

37 equal to 0 msec and 303 msec (range, 39-1012 msec), respectively (P < .001).

38 h a nonsignificant increase in females (1017 msec +/- 27 vs 1001 msec +/- 33, P = .066).

39 ardium across all examined subjects was 1031 msec +/- 33 (standard deviation).

40 (978 msec +/- 23 vs 1006 msec +/- 29 vs 1044 msec +/- 14; P < .001) and T2 relaxation times (56 msec

41 (mean, 1332 mec [95% CI: 1296, 1368] vs 1045 msec [95% CI: 1034, 1056]; P < .001) and T2 (72 msec [95

42 ed from 1286 msec +/- 99 at baseline to 1077 msec +/- 50 at 6 months (P < .0001), whereas T2 decrease

43 92 and 60 msec +/- 19; skeletal muscle, 1100 msec +/- 59 and 44 msec +/- 9; and fat, 253 msec +/- 42

44 for native T1 relaxation times (cutoff, 1140 msec) were equivalent compared with those of the establi

45 3 msec) at 1.5 T and 1159 msec (95% CI: 1143 msec, 1175 msec) at 3.0 T.

46 [CI]: 969 msec, 983 msec) at 1.5 T and 1159 msec (95% CI: 1143 msec, 1175 msec) at 3.0 T.

47 1.5 T and 1159 msec (95% CI: 1143 msec, 1175 msec) at 3.0 T.

48 erence was found in T1 relaxation time (1183 msec +/- 256; P = .37).

49 th SASHA (13 msec; P < .05) and SAPPHIRE (12 msec; P < .05).

50 msec +/- 77 and 47 msec +/- 10; spleen, 1232 msec +/- 92 and 60 msec +/- 19; skeletal muscle, 1100 ms

51 or fibroglandular tissues at 3.0 T were 1256 msec +/- 171 and 46 msec +/- 7, respectively.

52 of infarcted myocardium decreased from 1286 msec +/- 99 at baseline to 1077 msec +/- 50 at 6 months

53 sec) and MOLLI (44 msec) than with SASHA (13 msec; P < .05) and SAPPHIRE (12 msec; P < .05).

54 35% or less, a QRS duration of less than 130 msec, and echocardiographic evidence of left ventricular

55 failure and a QRS duration of less than 130 msec, CRT does not reduce the rate of death or hospitali

56 - 205 and 60 msec +/- 21; renal cortex, 1314 msec +/- 77 and 47 msec +/- 10; spleen, 1232 msec +/- 92

57 for mature skeletal muscle tissue: T1, 1386 msec +/- 88; T2, 32.0 msec +/- 4.3; T2*, 10.8 msec +/- 0

58 rence erector spinae muscle tissue: T1, 1417 msec +/- 106; T2, 31.0 msec +/- 2.4; T2*, 11.3 msec +/-

59 egory 3 lesions, T1 and ADC of cancers (1430 msec +/- 220 and [0.60 +/- 0.17] x 10(-3) mm(2)/sec, res

60 ADC in clinically significant cancers (1440 msec +/- 140 and [0.58 +/- 0.14] x 10(-3) mm(2)/sec, res

61 ely) were higher than those in cancers (1450 msec +/- 110, 36 msec +/- 11, and [0.57 +/- 0.13] x 10(-

62 ites were evident at 92 msec (C1) and at 146 msec (N1b).

63 entration-dependent with a QRS duration >150 msec independently highly predictive of mortality in chl

64 The mean arterial transit time was 1538 msec +/- 123 (standard deviation) in the pediatric cohor

65 se in clinically insignificant lesions (1580 msec +/- 120 and [0.75 +/- 0.17] x 10(-3) mm(2)/sec, res

66 .031), and shorter QRS duration (beta = 0.16 msec/10 msec, P = .049).

67 rs were lower than those in noncancers (1620 msec +/- 120, 47 msec +/- 16, and [0.82 +/- 0.13] x 10(-

68 T1, T2, and ADC from cancer (mean, 1628 msec +/- 344, 73 msec +/- 27, and 0.773 x 10(-3) mm(2)/s

69 y) were lower than those of noncancers (1630 msec +/- 120 and [0.81 +/- 0.13] x 10(-3) mm(2)/sec, res

70 nd T2 in metastatic adenocarcinoma were 1673 msec +/- 331 and 43 msec +/- 13, respectively, significa

71 those from NPZ (mean, 2247 msec +/- 450, 169 msec +/- 61, and 1.711 x 10(-3) mm(2)/sec +/- 0.269) (P

72 P < .001) and fetal brain (3.7 msec vs 7.17 msec; P = .02), whereas there was no significant differe

73 tion) and 31 msec +/- 6; renal medulla, 1702 msec +/- 205 and 60 msec +/- 21; renal cortex, 1314 msec

74 erence in the fetal liver (2.72 msec vs 3.18 msec; P = .47).

75 The T1, T2, and ADC of NTZ (1800 msec +/- 150, 65 msec +/- 22, and [1.13 +/- 0.19] x 10(-

76 reath hold with a mean positive slope of 0.2 msec per heartbeat +/- 0.1 for T2 and 0.2 msec per heart

77 .2 msec per heartbeat +/- 0.1 for T2 and 0.2 msec per heartbeat +/- 0.1 for T2*, whereas for particip

78 T2* values had a mean negative slope of -0.2 msec per heartbeat +/- 0.2 for T2 and -0.1 msec per hear

79 the same area before the transfer (T1, 137.2 msec +/- 39.3 and 239.5 msec +/- 17.6, respectively; P <

80 MASIs showed significant T2 shortening (22.2 msec +/- 3.2 vs 27.9 msec +/- 1.8; P < .001) and no diff

81 1 compared with group 3 (difference of -26.2 msec; P = .002), which correlated with the number of pre

82 lower in patients (15.9 msec +/- 4.5 vs 35.2 msec +/- 2.1, P < .001) and 40% +/- 10 lower in canines

83 psychomotor speed (change in raw score, 5.2 msec and 0.9 msec, respectively).

84 ely; P = .006) and increased T2 values (64.2 msec +/- 10.9 vs 76.2 msec +/- 13.7, respectively; P = .

85 reased T2 values (64.2 msec +/- 10.9 vs 76.2 msec +/- 13.7, respectively; P = .01) in the proximal ul

86 -1.30) for malignant lesions, and 1.13 um(2)/msec (range, 0.35-2.63 um(2)/msec), 1.45 um(2)/msec (ran

87 ec (range, 0.35-2.63 um(2)/msec), 1.45 um(2)/msec (range, 0.44-3.34 um(2)/msec), and 0.65 (range, 0.4

88 dian ADC, D(app), and K(app) were 0.74 um(2)/msec (range, 0.52-1.44 um(2)/msec), 0.98 um(2)/msec (ran

89 ec (range, 0.52-1.44 um(2)/msec), 0.98 um(2)/msec (range, 0.63-2.12 um(2)/msec), and 1.01 (range, 0.6

90 were 0.74 um(2)/msec (range, 0.52-1.44 um(2)/msec), 0.98 um(2)/msec (range, 0.63-2.12 um(2)/msec), an

91 and 1.13 um(2)/msec (range, 0.35-2.63 um(2)/msec), 1.45 um(2)/msec (range, 0.44-3.34 um(2)/msec), an

92 ec), 1.45 um(2)/msec (range, 0.44-3.34 um(2)/msec), and 0.65 (range, 0.44-1.43) for benign lesions (P

93 ec), 0.98 um(2)/msec (range, 0.63-2.12 um(2)/msec), and 1.01 (range, 0.69-1.30) for malignant lesions

94 iron overload (defined as midseptal T2* < 20 msec on any prior cardiac MR images).

95 tal data, ranging from approximately 150-200 msec.

96 elin, which had a measured T2* value of 0.21 msec +/- 0.04 in the volunteer study.

97 2-KI mice (wildtype = 24.25 msec, KI = 25.22 msec; p = .011).

98 and mean T2* values of 28 msec +/- 5 and 22 msec +/- 5, respectively.

99 ndard deviation, 2242 msec +/- 116), T2 (224 msec +/- 18), and T2* (33.3 msec +/- 3.6) values and ADC

100 higher T1 (mean +/- standard deviation, 2242 msec +/- 116), T2 (224 msec +/- 18), and T2* (33.3 msec

101 cantly lower than those from NPZ (mean, 2247 msec +/- 450, 169 msec +/- 61, and 1.711 x 10(-3) mm(2)/

102 6 months (</=2.0 V at a pulse width of 0.24 msec and an increase of </=1.5 V from the time of implan

103 nd controls for placenta (5.25 msec vs 11.25 msec; P < .001) and fetal brain (3.7 msec vs 7.17 msec;

104 s observed in Tph2-KI mice (wildtype = 24.25 msec, KI = 25.22 msec; p = .011).

105 n IUGR cases and controls for placenta (5.25 msec vs 11.25 msec; P < .001) and fetal brain (3.7 msec

106 values for pulse duration and amplitude, 25 msec and 1 muT, respectively.

107 e CEST parameters of saturation duration (25 msec) and amplitude (1 muT) were chosen on the basis of

108 msec +/- 59 and 44 msec +/- 9; and fat, 253 msec +/- 42 and 77 msec +/- 16, respectively.

109 combination of a gantry rotation time of 275 msec, wide volume coverage, iterative reconstruction, au

110 relaxation times of 840 msec +/- 113 and 28 msec +/- 3 (P < .0001 and P < .01) and those in hepatic

111 - 9, respectively, and mean T2* values of 28 msec +/- 5 and 22 msec +/- 5, respectively.

112 ge damage defined with a T2* threshold of 28 msec and less.

113 At ROC curve analysis, a T2* value of 28 msec was identified as the threshold for damaged cartila

114 A short echo-time (29 msec) single-voxel (1-cm(3)) proton (hydrogen 1 [(1)H])

115 re in good agreement in per-patient (T2: 0.3 msec +/- 2.9; ECV: -0.3% +/- 1.7), per-slice (T2: 0.1 ms

116 ec +/- 106; T2, 31.0 msec +/- 2.4; T2*, 11.3 msec +/- 1.7; and ADC, 1.40 x 10(-3) mm(2)/sec +/- 0.03)

117 myocarditis than in control subjects (1185.3 msec +/- 49.3 vs 1089.1 msec +/- 44.9, respectively; P <

118 /- 116), T2 (224 msec +/- 18), and T2* (33.3 msec +/- 3.6) values and ADCs (1.53 x 10(-3) mm(2)/sec +

119 mes for normal cartilage (Beck score 1, 35.3 msec +/- 7.0) were significantly higher than those for c

120 lower in canines (23.0 msec +/- 4.0 vs 39.3 msec +/- 2.5, P < .001).

121 ) was associated with longer QTc (beta = 4.3 msec, P = .031).

122 - 2.9 [standard deviation]; range, 33.9-46.3 msec) were significantly lower than those of patients wh

123 a maximum exit velocity of 60 m/sec in a 30-msec burst.

124 T2-weighted VIP was equal to 0 msec and 303 msec (range, 39-1012 msec), respectively (P < .001).

125 t (466 msec +/- 14, 406 msec +/- 59, and 303 msec +/- 53, respectively; P < .001).

126 n healthy volunteers (745 msec +/- 65 and 31 msec +/- 6, P < .0001 and P = .021, respectively).

127 745 msec +/- 65 (standard deviation) and 31 msec +/- 6; renal medulla, 1702 msec +/- 205 and 60 msec

128 ared with HC participants (PF, 33 msec vs 31 msec; P < .001; KE, 32 msec vs 31 msec; P = .002) and in

129 msec vs 31 msec; P < .001; KE, 32 msec vs 31 msec; P = .002) and in the lower leg when compared with

130 times of lymphatic fluid at 3.0 T were 3100 msec +/- 160 (range, 2930-3210 msec; median, 3200 msec)

131 ts (PF, 33 msec vs 31 msec; P < .001; KE, 32 msec vs 31 msec; P = .002) and in the lower leg when com

132 participants without DPN (PF, 33 msec vs 32 msec; P = .03).

133 +/- 160 (range, 2930-3210 msec; median, 3200 msec) and 610 msec +/- 12 (range, 598-618 msec; median,

134 0 T were 3100 msec +/- 160 (range, 2930-3210 msec; median, 3200 msec) and 610 msec +/- 12 (range, 598

135 N when compared with HC participants (PF, 33 msec vs 31 msec; P < .001; KE, 32 msec vs 31 msec; P = .

136 mpared with participants without DPN (PF, 33 msec vs 32 msec; P = .03).

137 c images (intermediate-weighted sequence, 34-msec echo time; T2-weighted sequence, 80-msec echo time)

138 oint-resolved spectroscopy (PRESS) with a 35-msec echo time.

139 than those in cancers (1450 msec +/- 110, 36 msec +/- 11, and [0.57 +/- 0.13] x 10(-3) mm(2)/sec, res

140 volved contralateral brain were the same (36 msec+/-4 [standard deviation] vs 36 msec+/-5, respective

141 same (36 msec+/-4 [standard deviation] vs 36 msec+/-5, respectively), which might suggest similar oxy

142 acceptable pacing threshold (</=2.0 V at 0.4 msec) and an acceptable sensing amplitude (R wave >/=5.0

143 eled control cells (T2 in vivo, 15.4 vs 24.4 msec; P < .05) and could be tracked in osteochondral def

144 .7 msec +/- 2.5 [standard deviation] vs 45.4 msec +/- 3.3, respectively; MBR, 1.19 +/- 0.08; both, P

145 test attainable echo time of approximately 4 msec (T2* mapping).

146 3.0 T identified vendor (beta at 1.5 T = -4 msec [with Philips as reference], P < .001; beta at 3.0

147 th a temporal resolution of approximately 40 msec.

148 t cardiac MRI with temporal resolution at 40 msec or less.

149 y increased (high-frequency power: 54 vs. 40 msec(2), p = 0.005; high-frequency normalized power: 23.

150 , TS+ tones evoked a positive shift (100-400 msec) at right frontotemporal electrodes.

151 gadolinium enhancement (466 msec +/- 14, 406 msec +/- 59, and 303 msec +/- 53, respectively; P < .001

152 adenocarcinoma were 1673 msec +/- 331 and 43 msec +/- 13, respectively, significantly different from

153 with hypertension, with mean T2 values of 43 msec +/- 5 (standard deviation) and 46 msec +/- 9, respe

154 19; skeletal muscle, 1100 msec +/- 59 and 44 msec +/- 9; and fat, 253 msec +/- 42 and 77 msec +/- 16,

155 c, 53 msec) and 46 msec at 3.0 T (95% CI: 44 msec, 48 msec) (P <= .001).

156 s lower with ShMOLLI (62 msec) and MOLLI (44 msec) than with SASHA (13 msec; P < .05) and SAPPHIRE (1

157 11.55 msec for male rat stem cells vs 15.45 msec for sex-matched rat stem cells; P = .02 and P = .04

158 mm(3); temporal resolution, approximately 45 msec) in 20 patients with hepatic cirrhosis, 20 healthy

159 r QTc prolongation, defined as a QTc >/= 450 msec in men and >/= 460 msec in women, was 1.17 (95% CI:

160 ssues at 3.0 T were 1256 msec +/- 171 and 46 msec +/- 7, respectively.

161 of 43 msec +/- 5 (standard deviation) and 46 msec +/- 9, respectively, and mean T2* values of 28 msec

162 ence interval [CI]: 51 msec, 53 msec) and 46 msec at 3.0 T (95% CI: 44 msec, 48 msec) (P <= .001).

163 ned as a QTc >/= 450 msec in men and >/= 460 msec in women, was 1.17 (95% CI: 1.01, 1.35) for a 1-SD

164 ith evident late gadolinium enhancement (466 msec +/- 14, 406 msec +/- 59, and 303 msec +/- 53, respe

165 n those in noncancers (1620 msec +/- 120, 47 msec +/- 16, and [0.82 +/- 0.13] x 10(-3) mm(2)/sec, res

166 on were 5.9%, 19.6%, 306.79 msec, and 162.47 msec, respectively.

167 /- 21; renal cortex, 1314 msec +/- 77 and 47 msec +/- 10; spleen, 1232 msec +/- 92 and 60 msec +/- 19

168 c) and 46 msec at 3.0 T (95% CI: 44 msec, 48 msec) (P <= .001).

169 ve was stimulated hourly (30 pulses/min, 1.5 msec duration, 17.0 +/- 4.4 mA) during the surgery.

170 gh-frequency normalized power: 23.5 vs. 20.5 msec, p = 0.001; root mean square successive differences

171 transfer (T1, 137.2 msec +/- 39.3 and 239.5 msec +/- 17.6, respectively; P < .001).

172 yocardial T2 at stress (rest vs stress, 44.5 msec +/- 2.6 vs 49.0 msec +/- 5.5, respectively; P = .00

173 .02) and pulse sequence (beta at 1.5 T = -5 msec [with GRASE as reference], P < .001; beta at 3.0 T

174 as reference], P < .001; beta at 3.0 T = -5 msec, P = .02) and pulse sequence (beta at 1.5 T = -5 ms

175 es with six widely spaced echo times (in 3.5-msec increments) were acquired to correlate R2* and musc

176 for native T1 mapping ( approximately 25-50 msec; P > .05) and ECV quantification ( approximately 0.

177 bjects to both long (100 msec) and short (50 msec) duration deviant sounds.

178 CT at a temporal resolution of less than 50 msec.

179 .001) and T2 (72 msec [95% CI: 69, 75] vs 50 msec [95% CI: 49, 51]; P < .001), the magnitude of the d

180 to stimulate the brain noninvasively with 50-msec bursts at a 5% duty cycle, repetition frequency of

181 calculated with Fridericia's formula) to 500 msec occurred in 11.0% of participants in the short-regi

182 c at 1.5 T (95% confidence interval [CI]: 51 msec, 53 msec) and 46 msec at 3.0 T (95% CI: 44 msec, 48

183 homotor speed (scores range from 100 to 5100 msec, with faster times representing better performance)

184 The pooled mean of T2 across studies was 52 msec at 1.5 T (95% confidence interval [CI]: 51 msec, 53

185 rmore, FA (b = -0.007), T2(water) (b = -0.53 msec), and FF (b = -4.0%) were associated with nerve con

186 T (95% confidence interval [CI]: 51 msec, 53 msec) and 46 msec at 3.0 T (95% CI: 44 msec, 48 msec) (P

187 n, 10.72 msec for human stem cells and 11.55 msec for male rat stem cells vs 15.45 msec for sex-match

188 /- 14; P < .001) and T2 relaxation times (56 msec +/- 4 vs 59 msec +/- 3 vs 62 msec +/- 8; P = .04) a

189 ereas T2 decreased from 84 msec +/- 10 to 58 msec +/- 4 (P < .0001).

190 and T2 relaxation times (56 msec +/- 4 vs 59 msec +/- 3 vs 62 msec +/- 8; P = .04) and ECV (30% +/- 5

191 with ferumoxytol-labeled viable MASIs (26.6 msec +/- 4.9 vs 20.8 msec +/- 5.3; P = .001).

192 P = .021) in patients and by 8% +/- 5 (44.6 msec +/- 4.8, P = .012) in canines.

193 imilar precision to ShMOLLI (4.0 msec vs 5.6 msec; P = .07) but higher precision than SAPPHIRE (6.8 m

194 bleaching light energy at a pulse width of 6 msec and a duty cycle of 50%.

195 1.5 T = -1 msec, P = .88; beta at 3.0 T = 6 msec, P = .42).

196 as reference], P < .001; beta at 3.0 T = -6 msec, P = .002) as significant covariates, but it did no

197 msec +/- 10; spleen, 1232 msec +/- 92 and 60 msec +/- 19; skeletal muscle, 1100 msec +/- 59 and 44 ms

198 - 6; renal medulla, 1702 msec +/- 205 and 60 msec +/- 21; renal cortex, 1314 msec +/- 77 and 47 msec

199 ion time, 10-12 min; temporal resolution, 60 msec) or one cardiac cycle and time-of-flight (TOF) MR a

200 , 2930-3210 msec; median, 3200 msec) and 610 msec +/- 12 (range, 598-618 msec; median, 610 msec), res

201 sec +/- 12 (range, 598-618 msec; median, 610 msec), respectively.

202 al SSFSE imaging decreasing from 1358 to 613 msec for sagittal acquisitions and from 1494 to 621 msec

203 00 msec) and 610 msec +/- 12 (range, 598-618 msec; median, 610 msec), respectively.

204 uracy in phantoms was lower with ShMOLLI (62 msec) and MOLLI (44 msec) than with SASHA (13 msec; P <

205 times (56 msec +/- 4 vs 59 msec +/- 3 vs 62 msec +/- 8; P = .04) and ECV (30% +/- 5 vs 33% +/- 5 vs

206 r sagittal acquisitions and from 1494 to 621 msec for coronal oblique acquisitions.

207 1, T2, and ADC of NTZ (1800 msec +/- 150, 65 msec +/- 22, and [1.13 +/- 0.19] x 10(-3) mm(2)/sec, res

208 reast tissues, higher T2 relaxation time (68 msec +/- 13) was observed in invasive ductal carcinoma (

209 Adding T2 information (98 msec+/-7 vs 68 msec+/-2, respectively) alone yields results that sugges

210 sequence, with slight T2 overestimation (2.7 msec).

211 ilage with early changes (Beck score 2, 20.7 msec +/- 6.0) and cartilage with more advanced degenerat

212 varial defects of recipient mice (mean, 21.7 msec vs 27.1 msec, respectively; P = .0444).

213 s 11.25 msec; P < .001) and fetal brain (3.7 msec vs 7.17 msec; P = .02), whereas there was no signif

214 to adjacent intact cartilage (mean T2 = 32.7 msec +/- 4.2).

215 ater than at rest (mean rest vs stress, 38.7 msec +/- 2.5 [standard deviation] vs 45.4 msec +/- 3.3,

216 ly 19% shorter with the DEPC method (TR, 5.7 msec) than with the SEPC method (TR, 2.8 msec +/- 4.2) (

217 (62.0 msec +/- 4.9) and nonhemorrhagic (71.7 msec +/- 7.3) infarctions in canines was elevated relati

218 SAPPHIRE (6.8 msec; P = .002) and SASHA (8.7 msec; P < .001).

219 chondral defects of female rats (mean, 10.72 msec for human stem cells and 11.55 msec for male rat st

220 nificant difference in the fetal liver (2.72 msec vs 3.18 msec; P = .47).

221 c [95% CI: 1034, 1056]; P < .001) and T2 (72 msec [95% CI: 69, 75] vs 50 msec [95% CI: 49, 51]; P < .

222 ADC from cancer (mean, 1628 msec +/- 344, 73 msec +/- 27, and 0.773 x 10(-3) mm(2)/sec +/- 0.331, res

223 ve values included the following: liver, 745 msec +/- 65 (standard deviation) and 31 msec +/- 6; rena

224 epatic parenchyma in healthy volunteers (745 msec +/- 65 and 31 msec +/- 6, P < .0001 and P = .021, r

225 ing a GRE sequence (repetition time, 600-750 msec; echo time, 10 msec; in-plane resolution, 196 mm).

226 msec +/- 9; and fat, 253 msec +/- 42 and 77 msec +/- 16, respectively.

227 in a typical three-echo protocol (with 0.78-msec increments).

228 ecruited population were 5.9%, 19.6%, 306.79 msec, and 162.47 msec, respectively.

229 sec +/- 88; T2, 32.0 msec +/- 4.3; T2*, 10.8 msec +/- 0.8; ADC, 1.39 x 10(-3) mm(2)/sec +/- 0.02 (ref

230 square successive differences: 16.7 vs. 14.8 msec, p = 0.007).

231 anced degeneration (Beck scores 3-6, </=19.8 msec +/- 5.6) (P < .001).

232 5.7 msec) than with the SEPC method (TR, 2.8 msec +/- 4.2) (P < .05).

233 eled viable MASIs (26.6 msec +/- 4.9 vs 20.8 msec +/- 5.3; P = .001).

234 ured at an orientation of 54.7 degrees (21.8 msec +/- 2.8 [+/- standard error of the mean]) than at 0

235 was marginally elevated by 6% +/- 2.5 (37.8 msec +/- 2.5, P = .021) in patients and by 8% +/- 5 (44.

236 .07) but higher precision than SAPPHIRE (6.8 msec; P = .002) and SASHA (8.7 msec; P < .001).

237 34-msec echo time; T2-weighted sequence, 80-msec echo time) were compared in vivo with corresponding

238 hs (P < .0001), whereas T2 decreased from 84 msec +/- 10 to 58 msec +/- 4 (P < .0001).

239 ing liver parenchyma relaxation times of 840 msec +/- 113 and 28 msec +/- 3 (P < .0001 and P < .01) a

240 speed (change in raw score, 5.2 msec and 0.9 msec, respectively).

241 (standard deviation) lower in patients (15.9 msec +/- 4.5 vs 35.2 msec +/- 2.1, P < .001) and 40% +/-

242 ant T2 shortening (22.2 msec +/- 3.2 vs 27.9 msec +/- 1.8; P < .001) and no difference in cartilage r

243 Mean T1rho values of volunteers (mean, 40.9 msec +/- 2.9 [standard deviation]; range, 33.9-46.3 msec

244 0 msec +/- 0.7, P < .001) or 90 degrees (9.9 msec +/- 0.4, P < .001).

245 -parietal electrode sites were evident at 92 msec (C1) and at 146 msec (N1b).

246 +/- 28 [standard deviation] in diastole, 959 msec +/- 21 in systole) and all segmental T1 values betw

247 976 msec (95% confidence interval [CI]: 969 msec, 983 msec) at 1.5 T and 1159 msec (95% CI: 1143 mse

248 The pooled mean of native T1 was 976 msec (95% confidence interval [CI]: 969 msec, 983 msec)

249 Myocardial T1 (978 msec +/- 23 vs 1006 msec +/- 29 vs 1044 msec +/- 14; P <

250 Adding T2 information (98 msec+/-7 vs 68 msec+/-2, respectively) alone yields resu

251 (95% confidence interval [CI]: 969 msec, 983 msec) at 1.5 T and 1159 msec (95% CI: 1143 msec, 1175 ms

252 Mean myocardial T1 (984 msec +/- 28 [standard deviation] in diastole, 959 msec +

253 density-weighted SE (repetition time msec/TE msec, 2000/15) and ultrashort TE (300/0.008, 6.6, echo-s

254 netic field with rate of rise (dB/dt) in the msec range to cultured tumor cells to assess whether thi

255 ing sequence (repetition time msec/echo time msec, 10 123/40; b=1200 sec/mm2).

256 weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) of the upper abd

257 weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) of the upper abd

258 weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) of the upper abd

259 weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) of the upper abd

260 weighted MRI (repetition time msec/echo time msec, 1000/89; section thickness, 4 mm) of the upper abd

261 ion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo spin-echo T2-w

262 ion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo spin-echo T2-w

263 ion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo spin-echo T2-w

264 ion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo spin-echo T2-w

265 (23)Na (repetition time msec/echo time msec, 160/0.35) and (35)Cl (40/0.6) MR imaging of both l

266 re generated (repetition time msec/echo time msec, 2000/67; section thickness, 4 mm; in-plane resolut

267 st spin-echo (repetition time msec/echo time msec, 2220/57; section thickness, 4 mm), (b) axial unenh

268 st spin-echo (repetition time msec/echo time msec, 2220/57; section thickness, 4 mm), (b) axial unenh

269 st spin-echo (repetition time msec/echo time msec, 2220/57; section thickness, 4 mm), (b) axial unenh

270 cho sequence (repetition time msec/echo time msec, 3000/20-320).

271 T2-weighted (repetition time msec/echo time msec, 4574/86.5) MR image of the pelvis.

272 T2-weighted (repetition time msec/echo time msec, 4574/86.5) MR image of the pelvis.

273 5-T MR image (repetition time msec/echo time msec, 541/15).

274 al MR images (repetition time msec/echo time msec, 8.86/4.51; flip angle, 25 degrees ) acquired with

275 cho sequence (repetition time msec/echo time msec, 800/1.8-49.8) was performed at embryonic day 19.

276 cho-planar imaging sequence (repetition time msec/echo time msec, 10 123/40; b=1200 sec/mm2).

277 -suppressed T2-weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) o

278 -suppressed T2-weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) o

279 -suppressed T2-weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) o

280 -suppressed T2-weighted MRI (repetition time msec/echo time msec, 1000/87; section thickness, 6 mm) o

281 e 2:Coronal T2-weighted MRI (repetition time msec/echo time msec, 1000/89; section thickness, 4 mm) o

282 tenuated inversion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo

283 tenuated inversion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo

284 tenuated inversion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo

285 tenuated inversion recovery (repetition time msec/echo time msec, 11 000/125) MRI and (b) axial turbo

286 (23)Na (repetition time msec/echo time msec, 160/0.35) and (35)Cl (40/0.6) MR im

287 and ADC maps were generated (repetition time msec/echo time msec, 2000/67; section thickness, 4 mm; i

288 at-saturated fast spin-echo (repetition time msec/echo time msec, 2220/57; section thickness, 4 mm),

289 at-saturated fast spin-echo (repetition time msec/echo time msec, 2220/57; section thickness, 4 mm),

290 at-saturated fast spin-echo (repetition time msec/echo time msec, 2220/57; section thickness, 4 mm),

291 d from a spin-echo sequence (repetition time msec/echo time msec, 3000/20-320).

292 2a: (a) Coronal T2-weighted (repetition time msec/echo time msec, 4574/86.5) MR image of the pelvis.

293 2b: (a) Coronal T2-weighted (repetition time msec/echo time msec, 4574/86.5) MR image of the pelvis.

294 T1-weighted 1.5-T MR image (repetition time msec/echo time msec, 541/15).

295 three-dimensional MR images (repetition time msec/echo time msec, 8.86/4.51; flip angle, 25 degrees )

296 iple gradient-echo sequence (repetition time msec/echo time msec, 800/1.8-49.8) was performed at embr

297 ultiecho spin-echo sequence (repetition time msec/echo times msec, 1500/24, 36, 48, 60, 72, 84, 96, 1

298 ry short echo time sequence (repetition time msec/echo times msec, 30/0.075, 2, 5, 12, 18).

299 proton density-weighted SE (repetition time msec/TE msec, 2000/15) and ultrashort TE (300/0.008, 6.6

300 ho sequence (repetition time msec/echo times msec, 1500/24, 36, 48, 60, 72, 84, 96, 108, 120, 132, 14

301 me sequence (repetition time msec/echo times msec, 30/0.075, 2, 5, 12, 18).