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1 hotspot driver mutations specifically in the mucinous tumor.
2 e a distinct subset of genes was specific to mucinous tumors.
3 ociation of NKX2-1 with the genes induced in mucinous tumors.
4 between ductal type of intraductal papillary mucinous tumors.
5 ely related to nonmucinous tumors but not to mucinous tumors.
6 l/Met variant of COMT decreases the risk for mucinous tumors.
7 y mucinous ascites and multifocal peritoneal mucinous tumors.
8 cer of all principal histologic types except mucinous tumors.
9 d fewer DNA copy-number alterations than non-mucinous tumors.
10 erapy, whereas no association was found with mucinous tumors.
11                          Nine neoplasms were mucinous tumors (64.3%), including all neoplasms associa
12 ations for serous tumors, KRAS mutations for mucinous tumors, and beta-catenin and PTEN mutations and
13 tones, localization of intraductal papillary mucinous tumors, and localization and treatment of hemob
14 ant of adenocarcinoma, intraductal papillary mucinous tumors are observed in patients with McCune-Alb
15 us tumors is associated with size, and small mucinous tumors are very unlikely to be malignant.
16                                              Mucinous tumors, both borderline and invasive, were more
17 ollowing characteristics: signet ring cells, mucinous tumor, budding, lymphovascular invasion, perine
18                        Intraductal papillary mucinous tumors continue to be commonly encountered and
19 d with an increased risk in endometrioid and mucinous tumors [e.g., testosterone, endometrioid tumors
20 inous tumors and 1.03 (95% CI 0.88-1.21) for mucinous tumors; for each year of oral contraceptive use
21 erefore, we present the first mouse model of mucinous tumor formation from ovarian cells and supporti
22                                        While mucinous tumors from different organs are histologically
23                                              Mucinous tumors had a much higher SI on the T2-weighted
24 s 67%, 72%, and 67%, respectively, with most mucinous tumors having predominantly high SI and a perip
25 s and some included a heterogeneous group of mucinous tumors, including both benign and malignant app
26                    Women with PMP often have mucinous tumors involving both the appendix and the ovar
27 indings strongly support the conclusion that mucinous tumors involving the appendix and ovaries in wo
28                        Intraductal papillary-mucinous tumor (IPMT) of the pancreatic ducts is increas
29 creas of patients with intraductal papillary mucinous tumors (IPMT) and to analyze their relation to
30 rofiles in a series of intraductal papillary-mucinous tumors (IPMTs) of the pancreas, which represent
31 ce interval: 1.0, 2.4) but decreased risk of mucinous tumors (IRR = 0.3, 95% confidence interval: 0.1
32  The encompessing term intraductal papillary-mucinous tumors is appropriate.
33                            Malignancy within mucinous tumors is associated with size, and small mucin
34                                              Mucinous tumors (n = 18) were the most common malignant
35        Compared to population control cases, mucinous tumors occurred less frequently in the familial
36 endiceal mucinous adenomas (MAs) and ovarian mucinous tumors of low malignant potential (MLMPs) unass
37                                      Because mucinous tumors of the appendix similarly express MUC2,
38 iant was not significant overall, it was for mucinous tumors of the ovary, with an adjusted RR of 0.2
39                        Intraductal papillary mucinous tumors of the pancreas have a biologic behavior
40 inous tumors and 0.98 (95% CI 0.93-1.04) for mucinous tumors (p = 0.00051 and p = 0.0040, respectivel
41             The molecular drivers underlying mucinous tumor pathogenicity are poorly understood.
42 ic variants, including GNAS, associated with mucinous tumor phenotype.
43 only 50% and 0% of clear cell carcinomas and mucinous tumors, respectively, were positive.
44 AS mutations in granulosa cell tumors and in mucinous tumors, respectively.
45                                              Mucinous tumor samples exhibited the most distinct chang
46 an be differentiated with MR imaging because mucinous tumors show high SI on T2-weighted fast SE imag
47                             None of the five mucinous tumors showed allelic deletion at any of the 13
48  molecular and clinical phenotype defined by mucinous tumor status, increased peritoneal metastasis,
49 ordinate variants between paired Brenner and mucinous tumors supports a shared origin or progression.
50 convey greater increased risk for women with mucinous tumors than for women with neoplasms of other h
51      DNA from six Brenner tumors with paired mucinous tumors, two Brenner tumors not associated with
52  demonstrated that a set of genes induced in mucinous tumors was shared with genes induced in a nontu
53      The risk of nonmucinous tumors, but not mucinous tumors, was positively associated with a family
54                                              Mucinous tumors were enriched for oncogenic GNAS variant
55              Two of 11 intraductal papillary mucinous tumors were positive for telomerase activity, b
56                          Nonmucinous but not mucinous tumors were significantly associated with menst
57           DCC is retained in the majority of mucinous tumors, which produce high levels of mucins, an
58 ily involving the ovary from primary ovarian mucinous tumors with peritoneal implants.