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1 h muscle, colonic smooth muscle, and colonic mucosa).
2 and function at barrier sites (oral and lung mucosa).
3 in EAEC colonization of the gastrointestinal mucosa.
4 ral mucosa measured in the tongue and buccal mucosa.
5 bacterial species and strains on the colonic mucosa.
6 PGE2, histamine, and tryptase in the colonic mucosa.
7 and progenitor cells (ESPCs) of the skin and mucosa.
8 f DC-based vaccination to the female genital mucosa.
9 from reaching immune target cells within the mucosa.
10 ial products translocated across the damaged mucosa.
11 ased the levels of ATG16L1 in the intestinal mucosa.
12 way epithelial cells and MROH3P in esophagus mucosa.
13  between EAEC and the human gastrointestinal mucosa.
14 respiratory tract, and oral/gastrointestinal mucosa.
15 diosensitive tissues, such as the intestinal mucosa.
16 tion of type I and -III interferons in nasal mucosa.
17 id cells surrounding the crypts in the colon mucosa.
18 nitors and the formation of OEC in the nasal mucosa.
19 er recruitment of neutrophils to the colonic mucosa.
20 tly from connective tissue mast cells in pig mucosa.
21 acity to inflamed tissues, including the gut mucosa.
22 aptive immune responses at the oropharyngeal mucosa.
23  Env-specific humoral immunity in the rectal mucosa.
24 i.) in the oral cavity and upper respiratory mucosa.
25 ral infection of the denture-bearing palatal mucosa.
26 alpha4beta7 and were not enriched at the gut mucosa.
27 t present DQ2.5-glia-alpha1a in the inflamed mucosa.
28  targets epithelial cells lining the genital mucosa.
29 taken of both expanded and non-expanded oral mucosa.
30 ing the critical function of A20 in the oral mucosa.
31 al immunity by both antibody isotypes in the mucosa.
32  and iNOS were significantly increased in BE mucosa.
33 ited Th1 and Th17 responses in their gastric mucosa.
34 ncreatic secretions to the distal intestinal mucosa.
35 nch biopsies were taken of the expanded oral mucosa.
36 lated with CCL20 and CCL25 expression in the mucosa.
37  and Ki-67 staining was unchanged in jejunal mucosa.
38 fine-scissor dissection without touching the mucosa.
39 e potential of infected Stat3(SA/SA) gastric mucosa.
40 vels of specific T cells only in the vaginal mucosa.
41 estinal tract, on the skin or on the vaginal mucosa.
42 rotective phospholipid layers of the gastric mucosa.
43 mmon malignant tumor that occurs in the oral mucosa.
44  No eosinophils were found in the esophageal mucosa.
45 ocyte population in early pregnancy decidual mucosa.
46 ergic inflammatory disease of the esophageal mucosa.
47 oorganisms between body parts or surfaces to mucosa.
48 gnificant c-Met overexpression in dysplastic mucosa.
49 tly upregulated in cancer relative to benign mucosa.
50 obiota as well as transcription in the colon mucosa.
51                                 Peri-implant mucosa (1 x 2 x 2 mm) was harvested around the healing a
52  from patients with treated but unhealed CeD mucosa (15.1 +/- 7.5) than in patients with treated and
53  a mean peak of 5.97x10 2 ng/ml in the nasal mucosa 2-3 days after infusion.
54 pplied to 1) all visible esophageal columnar mucosa; 2) 5-10 mm proximal to the squamocolumnar juncti
55 idering the pH gradient found in the gastric mucosa (3 < pH < 7.4).
56 than in patients with treated and healed CeD mucosa (5.5 +/- 3.4) (P < .001).
57 om matched inflamed and non-inflamed colonic mucosa [50 Crohn's disease (CD); 80 ulcerative colitis (
58 t 1 year of function placed in thin vertical mucosa achieved similar clinical parameters and radiogra
59 activation, mucosal Th17 and Th22 cells, and mucosa-adherent microbiota signatures in gut mucosal spe
60 stal colon microbiota and a reduction in the mucosa-adjacent community, concomitant with depletion of
61 ta-glucan or chitosan/chitin in the serum or mucosa after challenge.
62  AMT-101 was taken up by inflamed intestinal mucosa and activated pSTAT3 in the lamina propria with l
63     TL1A expression is increased in inflamed mucosa and associated with fibrostenosing Crohn's diseas
64 e and compare immune cell populations in the mucosa and blood from patients with IBD and without IBD
65 identify immune cell populations specific to mucosa and blood samples from patients with active or in
66 entiated active UC from active CD in colonic mucosa and blood samples; top discriminating features in
67 tween microenvironmental niches, the luminal mucosa and crypts.
68  quantifying gene expression in the cervical mucosa and cytokine levels in cervicovaginal lavage flui
69 at were consistently upregulated in the oral mucosa and demonstrated that induction of one of the ide
70  investigated DCs and the connection between mucosa and joints in a murine model of Yersinia enteroco
71 ty in directly sampling the small intestinal mucosa and microbial community (microbiota).
72 eas A. lumbricoides larvae penetrate the gut mucosa and migrate through the liver and lungs before re
73 ion of islet-reactive T cells within the gut mucosa and onset of T1D.
74        H. pylori colonizes the human gastric mucosa and persists for decades.
75 ufficient to pull CD8 T cells to the vaginal mucosa and provide protection against genital herpes inf
76 virus to invade epithelial cells of the oral mucosa and salivary gland ducts via ACE2 receptors.
77    Helicobacter pylori colonizes the gastric mucosa and secretes a pore-forming toxin (VacA).
78 on the transcriptome of normal human palatal mucosa and seems to target genes important for innate im
79  create and validate the ex vivo function of mucosa and smooth muscle (n = 25).
80 ltration of yet uncategorized cells into the mucosa and submucosa of the jejunum.
81 stoscopic biopsies of bladder wall including mucosa and submucosa were obtained from all patients.
82 is critical for OEC development in the nasal mucosa and subsequent GnRH-1 neuronal migration.
83 licates in gut-associated tissues, eliciting mucosa and systemic immunity.
84         Yet, the function of A20 in the oral mucosa and the biological pathways in which A20 mitigate
85 lete endoscopic eradication, the neosquamous mucosa and the gastric cardia are sampled by 4-quadrant
86 ltration of gut microbes into the intestinal mucosa and the resulting inflammation are causal factors
87 obiome communicates with both the intestinal mucosa and the systemic immune system, given that these
88 islet-specific T cells within the intestinal mucosa and to autoimmune diabetes and provide a strong r
89 rotective phospholipid layers of the gastric mucosa and to describe the interactions with diclofenac,
90 d gene expression changes in healthy palatal mucosa and to identify potentially implicated immunologi
91 d T cells in various contexts in the thymus, mucosa and tumours.
92 l/glandular epithelium, stroma, vascularized mucosa and two-layered myometrium.
93 lammation, massive ulceration of the colonic mucosa, and bloody diarrhea.
94 ompetence for sustained ILC2 activity at the mucosa, and contributes to allergic pathogenesis.
95  of olfactory ensheathing cells in the nasal mucosa, and impairs GnRH-1 neuronal migration to the bra
96  expression) and FoxP3 in the cervicovaginal mucosa, and increased infiltration of CD4+ T-cells.
97 f three oral habitats: tongue dorsum, buccal mucosa, and supragingival plaque.
98 ssful treatment is a healed small intestinal mucosa, and therefore, the outcome measures in proof-of-
99 l pathogens.IMPORTANCE Antibodies within the mucosa are part of the first line of defense against muc
100         Therefore, the vaginal and anorectal mucosa are relevant sites for ZIKV infection.
101  HIV transmission via genital and colorectal mucosa are the most common routes of dissemination.
102 eration and migration stimulator of the oral mucosa as a potential therapy to prevent radiation induc
103 lecular changes within PICF and peri-implant mucosa as a response to PM and PS appear negligible.
104             These studies establish the oral mucosa as an important model system to study epithelial
105 e oral cavity, intestinal, and genitourinary mucosa as part of the normal microbiota.
106                    Finally, with respiratory mucosa as the initial coronavirus infection site, our fi
107 d by the molecular barrier properties of the mucosa, as well as environmental factors.
108  This study aimed to determine the fecal and mucosa-associated bacterial composition along the gastro
109             While breath tests reflected the mucosa-associated bacterial composition, there was no ev
110 ile differed between IBS subtypes, while the mucosa-associated bacterial profile was associated with
111                            In addition, many mucosa-associated disorders possess a specific gut micro
112 etween the gut microbiome and the intestinal mucosa-associated immune system.
113                                              Mucosa-associated invariant T (MAIT) cell loss in chroni
114                                              Mucosa-associated invariant T (MAIT) cells are an innate
115                                              Mucosa-associated invariant T (MAIT) cells are innate-li
116  donor-unrestricted T cells (DURTs), such as mucosa-associated invariant T cells (MAITs), CD1-restric
117 ablish an important T cell-intrinsic role of mucosa-associated lymphoid tissue lymphoma translocation
118 (P < .0001); these follicles had features of mucosa-associated lymphoid tissue lymphoma.
119 controlling dendritic cell (DC) functions in mucosa-associated lymphoid tissue.
120 n a subpopulation of human memory B cells of mucosa-associated lymphoid tissue.
121        There is evidence from both fecal and mucosa-associated microbial studies that patients with P
122                                              Mucosa-associated microbiota transferred from villin-TLR
123 ed after exposure to in vitro models of oral mucosa, at equilibrium by Gas-Chromatography-Flame Ioniz
124     Leukocyte subsets were assessed in nasal mucosa biopsies at baseline and after treatment.
125 FF graded biopsies of corresponding skin and mucosa biopsies.
126       Concordance between allograft skin and mucosa biopsy grades increased with an increasing skin-B
127 obing depth, recession, width of keratinized mucosa, bleeding on probing, suppuration, implant mobili
128 ctable in pharyngeal, bronchial, and colonic mucosa but not bile.
129  has a beneficial effect on the peri-implant mucosa, but the effect of grafting the buccal mucosa on
130 estinal fluid secretion, is expressed in the mucosa, but the exact cellular origin remains controvers
131 ctivating innate immunity in the respiratory mucosa, but there is no reliable and convenient method b
132 rleukin 18 (IL-18) production in the colonic mucosa by deubiquitinating NLRP6.
133    Novel uptake routes through (altered) gut mucosa by myeloid leukocyte subsets promote PGN transpor
134 of colonizing microorganisms in the gingival mucosa can shift from homeostasis to dysbiosis or a dise
135 genera earlier shown to be enriched on HNSCC mucosa, Capnocytophaga, Fusobacterium, and Porphyromonas
136 C) is an opportunistic infection of the oral mucosa caused by the commensal fungus Candida albicans I
137                              Human olfactory mucosa cells (hOMCs) have been transplanted to the damag
138  cells are activated and expand in blood and mucosa coincident with peak HIV-1 viremia, in a manner a
139 ences following ZIKV infection of the rectal mucosa compared to a subcutaneous route of infection.
140 al loads in bronchoalveolar lavage and nasal mucosa compared with after the primary infection.
141 rrett's esophagus (BE), metaplastic columnar mucosa containing epithelial cells with gastric and inte
142                                         Oral mucosa contains a unique transcriptional network that pr
143 gical differences between antral and oxyntic mucosa contribute to spatial partitioning of H. pylori p
144 ompared to those isolated from control nasal mucosa (control-NM), in order to identify which key mRNA
145 th HGD or EAC and normal esophageal squamous mucosa (controls).
146 s 18 subjects with confirmed noninflamed gut mucosa (controls, 12 presumed healthy, 5 undergoing panc
147 stinal features replaces esophageal squamous mucosa damaged by gastroesophageal reflux disease.
148                        Peri-implant marginal mucosa defects (PMMDs) are alterations of the peri-impla
149 on in response to carbachol ex vivo and that mucosa demonstrated squamous cell differentiation.
150 tions occur between aroma compounds and oral mucosa depending on aroma chemical structure.
151                                        Nasal mucosa-derived exosomes (NMDEs) harbor immunodefensive p
152  showed that HIV can persist within the lung mucosa despite long-term ART.
153  pneumonia, wherein infection of respiratory mucosa drives a robust influx of neutrophils.
154 eted from the periphery and gastrointestinal mucosa during acute HIV-1 infection.
155 ute number of CXCR3+ T cells in the duodenal mucosa during ART.
156 pression was markedly decreased in the colon mucosa during colitis.
157 adenomas against the backdrop of an inflamed mucosa (e.g. in ulcerative colitis) remains exceedingly
158   We previously reported increased bronchial mucosa eosinophil and neutrophil inflammation in patient
159  tissue-specific microRNAs for skin and oral mucosa epithelium were identified.
160                               The intestinal mucosa exists in dynamic balance with trillions of lumin
161 e assessed by bacterial cell counts in nasal mucosa, fecal samples, cervical lymph nodes, and blood.
162  IL-4, IL-21, and IL-6 was observed in nasal mucosa following intranasal allergen challenge in the GP
163 nd other innate immune cells patrol the oral mucosa for infecting microbes.
164 transferase theta 2 (GSTT2) mRNA in squamous mucosa from African American compared with European Amer
165           Biopsy samples of inflamed colonic mucosa from patients and mice with colitis released opio
166                                   Intestinal mucosa from patients with IBD exhibited reduced levels o
167 ease of molecules from mast cells in colonic mucosa from patients with IBS with diarrhea (IBS-D; 18 w
168                                  The gastric mucosa from the double KO mice did not show phosphorylat
169 multaneous presence of lesions on the buccal mucosa, grade of lesion extension, and presence of ulcer
170     Immunization directly to the respiratory mucosa has been shown to promote greater protection from
171 ical tissue compartments like the intestinal mucosa, has not been completed.
172           The assay identified patients with mucosa healing status with 84% sensitivity and 95% speci
173          Upon engraftment into murine rectal mucosa, human RC tumoroids gave rise to invasive RC foll
174 hort-circuit current (ISC) of the intestinal mucosa, impaired cAMP generation in acutely isolated sma
175 rotypes to diseases of the upper respiratory mucosa in a site-specific manner.
176 gulated innate immune responses of the nasal mucosa in allergic individuals may be important in deter
177 ungi directly associated with the intestinal mucosa in healthy people and Crohn's disease patients an
178        Despite excellent healing of the oral mucosa in PDT, a lack of robust enabling technology for
179                      The role of keratinized mucosa in promoting peri-implant health is controversial
180                               The intestinal mucosa in rats with cirrhosis showed a proinflammatory p
181  were significantly upregulated in the nasal mucosa in response to infection.
182 ic resection, whereas flat areas of columnar mucosa in the tubular esophagus can be treated with muco
183 at better understanding the role of the oral mucosa in this phenomenon.
184 ired for colonization of the primate vaginal mucosa in vivo and 96 genes required for infection of th
185        Smooth muscle cells of the muscularis mucosa, in close proximity to proliferative crypts, are
186             Plasma cell numbers in the nasal mucosa increased during treatment (P = .02).
187 ssociated with impaired integrity of colonic mucosa, increased epithelial hyper-proliferation, reduce
188 at persistently replicates in the intestinal mucosa increases epithelial barrier permeability and rev
189 esponse, possibly triggered in the bronchial mucosa, induces systemic autoimmunity.
190 l helminths can protect against gut and lung mucosa inflammatory conditions by modulating the microbi
191 y particle features that govern nanomaterial-mucosa interactions and that are relevant in both nanome
192                                     The oral mucosa is an attractive site for mucosal vaccination, ho
193 rs2240308 p = 0.02), and leukoplakia of oral mucosa is associated with both AXIN2 (rs2240308 p = 0.03
194 obial product translocation from a permeable mucosa is demonstrated as a driver of inflammation.
195 use infection model, we show that intestinal mucosa is infected via intranasal (i.n.) or per-oral (p.
196                               The intestinal mucosa is lined by a single layer of epithelial cells th
197                            The human gastric mucosa is the most active layer of the stomach wall, inv
198 t unlike the female reproductive tract (FRT) mucosa, it halts systemic Chlamydia dissemination.
199 ) to examine the significance of keratinized mucosa (KM) and gingival tissue (KT) on peri-implant and
200                The CAS had a mean keratinzed mucosa (KM) of 1.65 +/- 1.31 mm, whereas in the CON KM w
201 that invades epithelial cells in the colonic mucosa, leading to bloody diarrhea.
202 d with immune parameters measured in the gut mucosa, lung tissue, and serum samples.
203 nib prevented vascular pathology in the oral mucosa, lungs, and liver of the BMP9/10ib mice, as well
204  external phospholipid layers of the gastric mucosa may constitute an additional toxicity mechanism o
205  lack of PTM MAIT cell enrichment at the gut mucosa may prevent depletion during chronic infection, p
206 ality and quantity of desmosomes in the oral mucosa measured in the tongue and buccal mucosa.
207 as a wound-healing response, and how cardiac mucosa might be the precursor of the intestinal metaplas
208 y relevant human bronchial and alveolar lung mucosa models cultured at air-liquid interface.
209 ties and to topical drug delivery to vaginal mucosa more generally.
210  of olfactory ensheathing cells in the nasal mucosa, moreover, we discovered that Ascl-1 is necessary
211  cells (ILC3s) that reside in the intestinal mucosa must function within a highly dynamic environment
212                                  The colonic mucosa must therefore tightly regulate fluid influx to c
213 nation, we demonstrate here that the colonic mucosa O(2) is actively depleted by S. flexneri aerobic
214 Oral mucositis refers to lesions of the oral mucosa observed in patients with cancer being treated wi
215 tissue model with ex vivo inferior turbinate mucosa obtained from patients with chronic rhinosinusiti
216  in antiviral defense responses in the nasal mucosa occur in a sex-specific manner.
217 d proliferation were observed in the colonic mucosa of 11G5-infected Apc(Min/+)/Atg16l1(DeltaIEC) mic
218 ere differentially expressed in the cervical mucosa of 18 women with versus 39 without S. haematobium
219 scherichia coli (CoPEC) colonize the colonic mucosa of a higher proportion of patients with vs withou
220 investigated whether the esophageal squamous mucosa of African American individuals has features that
221 cterial biofilms are identified on the colon mucosa of approximately 50% of colorectal cancer (CRC) p
222                                          The mucosa of facial allotransplants is one of the primary t
223 s that described changes in allotransplanted mucosa of fVCAs.
224 se proximity to S-phase cells in the gastric mucosa of gastritis patients.
225 and ETS1 significantly increased in inflamed mucosa of human IBD patients and in human colorectal ade
226 deamidated gluten peptides in the intestinal mucosa of individuals with specific HLA-DQ haplotypes.
227 unctions of innate cells in the oral and gut mucosa of infants.
228                               In the gastric mucosa of mice and patients with gastritis, pS-STAT3 was
229 T(h)2 cell differentiation were found in gut mucosa of mice nursed by mothers exposed to D pteronyssi
230 he cytokine profile observed in the duodenal mucosa of patients with NCSRWS.
231      The bacterial profiles of feces and the mucosa of sigmoid colon, but not duodenum, differed betw
232          Bacterial composition of feces, and mucosa of the duodenum and sigmoid colon was determined
233 fit from declining host defenses in the lung mucosa of the elderly.
234 ucosa, but the effect of grafting the buccal mucosa on buccal bone thickness (BBT) has not been inves
235  premalignant potential compared with normal mucosa or nonatrophic gastritis.
236 ther barrier tissues (e.g., gastrointestinal mucosa or skin) or endocrine organs, although any organ
237 results obtained from homogenates of bladder mucosa or whole bladder do not represent pure urothelial
238   Once the micromotor penetrates the gastric mucosa (pH >= 6.0), its pH-responsive cap dissolves, pro
239                           Although olfactory mucosa possesses long-lived horizontal basal stem cells
240                                Human bladder mucosa presented similarities to guinea pigs.
241                      Furthermore, we measure mucosa protein profiles of tumor and tumor nearby tissue
242 ms (eg, Staphylococcus hominis or Roseomonas mucosa) reduces AD severity, which supports an important
243 l afferents innervating the small intestinal mucosa regulate feeding, gastrointestinal (GI) digestive
244                                              Mucosa rejection grades were on average lower in the ear
245 of therapeutics to the gastrointestinal (GI) mucosa remains primarily a function of diffusion and rap
246 ther antibodies are delivered to the type II mucosa represented by the lumen of the lower female repr
247 al loads in bronchoalveolar lavage and nasal mucosa, respectively, as compared with viral loads in sh
248 ns of IFN biology, including the role of the mucosa-restricted type III IFNs (IFN-III), informing our
249  T cytotoxic (Tc) cell subsets in the rectal mucosa (RM), a major site for HIV acquisition and replic
250 r administration but a higher uptake in oral mucosa, salivary glands, and thyroid for FAPI-21.
251       We performed CyTOF analysis of colonic mucosa samples (n = 87) and peripheral blood mononuclear
252          In an analysis of fecal and colonic mucosa samples from patients receiving FMT for active UC
253              Compared with controls, colonic mucosa samples from patients with IBD had increased abun
254                                      Colonic mucosa samples from patients with UC were characterized
255  of protein profiles of 82 apparently normal mucosa samples obtained from living individuals by endos
256 population of R. mucilaginosa in many buccal mucosa samples.
257 t on each taxon in stool and small-intestine mucosa samples.
258                               The intestinal mucosa serves both as a conduit for the uptake of food-d
259  the interaction of the allergen with airway mucosa, shedding light into its potential role in the im
260 ium (e.g. cultured urothelial cells, bladder mucosa sheets mounted in Ussing chambers or isolated bla
261                                       The GI mucosa showed eosinophil infiltration of more than 40/hi
262 IF pattern was composed of liver and gastric mucosa staining on rat kidney/liver/stomach sections.
263  decellularized porcine small intestine (SI) mucosa/submucosa enable formation and growth of endoderm
264                      He first developed oral mucosa symptoms and vomiting at 4 years and 10 months of
265  protective immune responses at the cervical mucosa that could limit chlamydial infection to the cerv
266 ocated broadly within the basal layer of the mucosa throughout the oral cavity.
267 se-key urothelial sensory process, from live mucosa tissue, full-thickness bladder but not smooth mus
268  human oral mucosa tissues, including labial mucosa tissue, gingival tissue, and tongue dorsum tissue
269 y the tissue parameters of normal human oral mucosa tissues, including labial mucosa tissue, gingival
270 dging innate and adaptive immunity from lung mucosa to lymph nodes to program DCs to direct effective
271 25(+) FOXP3(+) cells in the gastrointestinal mucosa to support immunoregulation and immunological tol
272 pro-inflammatory immune responses of the gut mucosa to systemic autoimmunity in lupus.
273 ovides a reference dataset of the intestinal mucosa transcriptional responses to chronic EtOH exposur
274 ctor T cells from circulation to the genital mucosa via topical vaginal application of chemokines in
275 s of hypermethylated genes in murine colonic mucosa (vs.
276 uman in vitro co-culture model of the airway mucosa was evaluated herein.
277 ges on the diversity of viruses in the nasal mucosa was investigated in three Colombian villages with
278 nuously; RBC velocity of the ileal serosa or mucosa was recorded by intravital videomicroscopy.
279                     A congestive and friable mucosa was seen, and the resection specimen showed enlar
280  which is found at depressed levels on HNSCC mucosa, was high.
281 marily associated with the mammalian gastric mucosa, we conclude that loss of Tff2 in the developing
282 d their distribution in the small intestinal mucosa were examined by quantitatively comparing tdTomat
283 peroxidasin gene expressions in peri-implant mucosa were noted within both groups (P < 0.05) without
284  Homogenates of human biofilm-positive colon mucosa were prepared from tumor patients (tumor and pair
285                       Biopsies of esophageal mucosa were taken from the (1) proximal esophagus, (2) m
286 r substitute for tissue biopsy of esophageal mucosa when evaluating microflora patterns.
287 lance of the epidermis, intestinal, and oral mucosa, whereas the presence of pathogenic microorganism
288 ion and signaling pathways in the colorectal mucosa, which promotes viral establishment by creating a
289 cal ingredients directly through the gastric mucosa while avoiding perforation.
290  phenotype (PSP) encompasses the keratinized mucosa width (KMW), mucosal thickness (MT), and supracre
291 prorepair mechanisms in the gastrointestinal mucosa will aid in the development of novel therapeutics
292     We then treated ex vivo human esophageal mucosa with a cytokine cocktail to closely mimic the Th2
293 re, immunological priming through the rectal mucosa with an attenuated ZIKV strain resulted in signif
294 4a-3, miR-129-2, and miR-137, in the gastric mucosa with and without GC, i.e., in atrophic mucosal gl
295                              Patient colonic mucosa with CoPEC colonization had higher levels of mRNA
296 ally integral elements in the fabric of oral mucosa with significant functional roles.
297                                 Treatment of mucosa with Th2 and fibrotic cytokines recapitulated the
298 d across the cribriform plate into the nasal mucosa, with a small fraction of NPs localizing with ner
299  proteins involved in autophagy than colonic mucosa without these bacteria.
300 tia (43.1%; 47/109), whereas 7.3% was in the mucosa (ypT1a), 16.5% in the submucosa (ypT1b) and 6.4%

 
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