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1 leukemia)-2H3 mast cells (a tumor analog of mucosal mast cells).
2 pus of mice appear to be bone marrow-derived mucosal mast cells.
3 philic esophagitis have increased numbers of mucosal mast cells.
4 antibody dissociation from its receptors on mucosal mast cells.
5 are not essential for exocytosis in RBL-2H3 mucosal mast cells.
6 blood eosinophils, and increased intestinal mucosal mast cells.
7 uft cell hyperplasia along with expansion of mucosal mast cells.
8 involved in colonic goblet cell release and mucosal mast cell activation after immobilization stress
10 expulsion is correlated with high numbers of mucosal mast cells and an increase in IL-13 and IL-10 se
11 er levels of PNA-specific IgE and intestinal mucosal mast cells and eosinophils over sham treatments.
12 accompanied by accelerated degranulation of mucosal mast cells and increased Ag-specific production
15 vity reflects the permanent sensitization of mucosal mast cells by allergen-specific IgE antibodies b
20 nstrate a critical role for IL-10 in driving mucosal mast cell expansion and activation, suggesting t
22 ifferentiation of intraepithelial intestinal mucosal mast cells expressing mouse mast cell protease 1
23 r (SCF), whereas helminth-induced intestinal mucosal mast cell hyperplasia also requires T cell-deriv
25 r results reveal a potential function of gut mucosal mast cells in HIV-1 dissemination in tissues.
26 d beta(6)(-/-) mice, we show accumulation of mucosal mast cells in the lamina propria of the small in
30 finity receptor for IgE (FcepsilonRI) on the mucosal mast cell line, RBL-2H3, results in the rapid an
32 leukemia cells (RBL-2H3 m1), an immortalized mucosal mast cell line, was studied at the single-channe
35 These results provide the mechanism whereby mucosal mast cells mediate parasite expulsion from the i
36 inal nematodes is associated with pronounced mucosal mast cell (MMC) hyperplasia, differentiation, an
39 oup has previously identified IL-9-producing mucosal mast cell (MMC9) as the primary source of IL-9 t
40 tification of multifunctional IL-9-producing mucosal mast cells (MMC9s) that can secrete prodigious a
43 toplasm of rat basophilic leukemia (RBL-2H3) mucosal mast cells or anchored (via LynB protein) to the
44 lation but not with allergen-specific IgE or mucosal mast cell protease 1 levels, indicating systemic
45 ntributing factor to the observed changes in mucosal mast cell protease and epithelial cytokine expre
46 tocytosis was absent, the protease unique to mucosal mast cells, rat mast cell protease II (RMCPII),
48 usly compromised in their ability to mount a mucosal mast cell response after infection, and there is
50 APs channel food allergens to lamina propria mucosal mast cells through an IL-13-CD38-cyclic adenosin
52 th trypsin in patients with IBS-D stimulates mucosal mast cells to release PGE2, which downregulates
53 Our aim was to elucidate the contribution of mucosal mast cells to the effector phase of a secondary