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1 it must induce a state of nonresponsiveness (mucosal tolerance).
2  cell-transfer immunotherapy to re-establish mucosal tolerance.
3 rogenic dendritic cells function to maintain mucosal tolerance.
4 Th1 cells from naive precursors and promotes mucosal tolerance.
5 een regulatory T cell subsets that maintains mucosal tolerance.
6  (PSA), which induces regulatory T cells and mucosal tolerance.
7 ue and might therefore help maintain colonic mucosal tolerance.
8 iation pathway in the gut to actively induce mucosal tolerance.
9 and systemic unresponsiveness termed oral or mucosal tolerance.
10 atch versus epithelial cells in induction of mucosal tolerance.
11 esponses, contributing to the maintenance of mucosal tolerance.
12 hat these cells might play an active role in mucosal tolerance.
13 effective therapeutic approach for promoting mucosal tolerance.
14 ty and thus contribute to the maintenance of mucosal tolerance.
15 distinct roles during the inductive phase of mucosal tolerance.
16 from either a mucosal challenge or a loss of mucosal tolerance.
17 oles of B7-1 and IL-12 in the development of mucosal tolerance.
18 dietary and microbial antigens, which shapes mucosal tolerance.
19 regulation PD-1 ligands on CMFs upon colonic mucosal tolerance.
20 +) regulatory T cells and, thereby, maintain mucosal tolerance.
21 acteria appears to be a central component of mucosal tolerance.
22 y T (Treg) cells develop during induction of mucosal tolerance and after immunization.
23              Thus, nasal plasmid FL prevents mucosal tolerance and enhances active immunity when give
24            Vitamin A promotes development of mucosal tolerance and enhances differentiation of regula
25 ) Treg cells were essential for establishing mucosal tolerance and for suppressing IL-4 production an
26  the role of adaptive Foxp3(+) Treg cells in mucosal tolerance and in chronic allergic lung inflammat
27 microbial sensing by Treg cells in enforcing mucosal tolerance and maintaining commensalism by promot
28 ICOS plays an essential and specific role in mucosal tolerance and that distinct costimulatory pathwa
29 isation of the T reg cells that mediate both mucosal tolerance and tolerance to self antigens.
30 t B7 alone may not be sufficient to abrogate mucosal tolerance, and that cytokines such as IL-12 may
31 d associated lymphoid tissues helps maintain mucosal tolerance but also contributes to the developmen
32       These results clearly demonstrate that mucosal tolerance can be induced by oral administration
33        This study aimed to determine whether mucosal tolerance can be induced when EAU is suppressed
34 6DeltaDC) resulted, unexpectedly, in loss of mucosal tolerance, characterized by spontaneous developm
35  mimic the local effects of the mediators of mucosal tolerance could have therapeutic potential for t
36                                              Mucosal tolerance has been considered a potentially impo
37                                      Induced mucosal tolerance has been shown to be beneficial in pre
38 estoring both normal anabolic metabolism and mucosal tolerance in CD.
39 dy was to investigate further the concept of mucosal tolerance in EAG by examining the effect of nasa
40 his study, we investigated the mechanisms of mucosal tolerance in EAG by examining the effects of the
41               These results demonstrate that mucosal tolerance in EAG can be induced by nasal adminis
42  clearly demonstrate for the first time that mucosal tolerance in EAG can be induced by nasal adminis
43                 Commensal microbiota promote mucosal tolerance in part by engaging regulatory T (Treg
44 ) plays a crucial role in the development of mucosal tolerance, including the airways.
45  have been implicated as being important for mucosal tolerance induction and because reovirus attachm
46                                              Mucosal tolerance induction generally requires multiple
47                                              Mucosal tolerance is believed to be partly mediated by r
48 pes of corticosteroids on the development of mucosal tolerance, mice were exposed to respiratory alle
49                                              Mucosal tolerance prevents pathological reactions agains
50                       After the induction of mucosal tolerance, proliferation of T cells was inhibite
51 tment for patients with bronchial asthma, on mucosal tolerance remain unknown.
52 been suggested to prevent the development of mucosal tolerance, the effects of local administration o
53  receptors have been demonstrated to promote mucosal tolerance, the role of activating Fc gamma recep
54 (Treg) cells (CD4+ CD25high FoxP3+) regulate mucosal tolerance; their adoptive transfer prevents or r
55                     The authors investigated mucosal tolerance therapy as a treatment for autoimmune
56 rate the essential role of Jak3 in promoting mucosal tolerance through suppressed expression and limi
57 ansgenically expressed in the liver enhances mucosal tolerance to an oral Ag.
58                       We now examine whether mucosal tolerance to E-selectin has protective effects i
59 cate that, in addition to preventing stroke, mucosal tolerance to E-selectin is cytoprotective.
60       We have demonstrated that induction of mucosal tolerance to E-selectin, a cytokine-inducible ad
61 ng epithelial barrier function and fostering mucosal tolerance to luminal bacteria.
62  TLR9 intracellular distribution, supporting mucosal tolerance to normal microbiota.
63 , molecular mechanisms that actively promote mucosal tolerance to the microbiota remain unknown.
64 re exposed to respiratory allergen to induce mucosal tolerance with or without systemic or intranasal