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1 often develops in HLVH patients and involves multiple abnormalities.
2 nd Sox2(COND) hypomorphic alleles, result in multiple abnormalities.
3                       However, the cells had multiple abnormalities.
4  normal in LEA rats, whereas LEC rats showed multiple abnormalities.
5 with 0 to 1 abnormality versus patients with multiple abnormalities.
6 nexpected layers of genetic complexity, with multiple abnormalities associated with disease progressi
7      The PPARdelta agonist GW501516 reverses multiple abnormalities associated with the metabolic syn
8                              The presence of multiple abnormalities common to all thyroid cancer vari
9                                          The multiple abnormalities described here might be responsib
10                                              Multiple abnormalities have been described, including pe
11 megaly, lymphadenopathy, thymic atrophy, and multiple abnormalities in blood cell populations.
12        Significantly, all of the mice showed multiple abnormalities in cartilage differentiation, inc
13 phenotype in mice versus Turner syndrome and multiple abnormalities in humans.
14 d HFpEF subjects were more likely to display multiple abnormalities in reserve.
15 lts indicate that it is possible to identify multiple abnormalities in some offspring of type II diab
16 ice is lethal in the embryonic period due to multiple abnormalities in the liver, heart, and lung, th
17 e is lethal in the embryonic period, causing multiple abnormalities in the liver, heart, lung, and bl
18          However, Alk3 CKO embryos displayed multiple abnormalities in vascular development, includin
19 HR) model of genetic hypertension, there are multiple abnormalities in VSMC function, including incre
20  (HOpic) increases Akt activity and improves multiple abnormalities in Zmpste24-deficient mice.
21 embryos die at embryonic day 9.5, displaying multiple abnormalities including lack of somites, forebr
22 ed by generating Ace -/- mice, which exhibit multiple abnormalities including renal structural defect
23 otic entry, RINT-1-deficient cells exhibited multiple abnormalities, including aberrant Golgi dynamic
24 s are present on most tumor vessels but have multiple abnormalities, including altered expression of
25            Magnetic resonance imaging showed multiple abnormalities, including diffuse polymicrogyria
26 hat Bmal1(lox/lox)/Ren1(d)Cre mice exhibited multiple abnormalities, including increased urine volume
27 r targeted disruption of the Alx-4 gene have multiple abnormalities, including preaxial polydactyly.
28 e structures in these adult mutants unmasked multiple abnormalities, including supernumerary spermath
29                                              Multiple abnormalities occur in the homeostasis of essen
30 lonic carcinoma tissue mRNA and confirm that multiple abnormalities of CD44 mRNA processing occur in
31 ositive chronic myelogenous leukemia exhibit multiple abnormalities of cytoskeletal function.
32      Sixteen of 18 patients showed single or multiple abnormalities of FMZ-V(d).
33 lls isolated from ERbeta-deficient mice show multiple abnormalities of ion channel function.
34                                     We found multiple abnormalities of mitochondrial function, sugges
35 th multitask learning to classify and detect multiple abnormalities on images from high-volume modali
36 (n = 13; 95% CI, 4-12 months) for those with multiple abnormalities (P <.01).
37                                              Multiple abnormalities were observed in glomeruli of mut
38                                              Multiple abnormalities were present in 13 subjects.
39    The CR rates for patients with 0 to 1 and multiple abnormalities were similar (69% and 54%, respec