コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ing into intraluminal vesicles (ILVs) of the multivesicular body.
2 ng, including defects in sorting through the multivesicular body.
3 ired for subsequent sorting of Arn1 into the multivesicular body.
4 radation of Smf1p by trafficking through the multivesicular body.
5 ker of exosomes and intralumenal vesicles of multivesicular bodies.
6 the accumulation of the uroplakin-degrading multivesicular bodies.
7 such as microdomain-dependent biogenesis of multivesicular bodies.
8 port (ESCRT) complexes 0, I, II and III into multivesicular bodies.
9 both retrogradely transported to the soma in multivesicular bodies.
10 Golgi phosphoprotein of 130 kDa (GPP130) to multivesicular bodies.
11 xosomes and could originate from cytoplasmic multivesicular bodies.
12 posed of 50- to 80-nm exosomes released from multivesicular bodies.
13 AuNPs were present but no longer paired in multivesicular bodies.
14 g and scission of intralumenal vesicles into multivesicular bodies.
15 d, sciatic nerve and brain, but no excess of multivesicular bodies.
16 omes, including late endosomes/lysosomes and multivesicular bodies.
17 rnae, accumulation of abnormal lysosomes and multivesicular bodies.
18 ation of cargo into intraluminal vesicles of multivesicular bodies.
19 uced within and secreted from late-endocytic multivesicular bodies.
20 te and help generate the luminal vesicles of multivesicular bodies.
21 xtracellular vesicles derived from endosomal multivesicular bodies.
22 e phagosome through the endocytic network to multivesicular bodies.
23 uman immunodeficiency virus (HIV) virions in multivesicular bodies.
24 of host proteins into endosomal vesicles and multivesicular bodies.
25 olving endosomal budding of HAV capsids into multivesicular bodies.
26 helial cells, infected AmEpCs made dispersed multivesicular bodies.
27 ittle internalized PAM-1/OSX was detected in multivesicular bodies.
28 ease of endosomal intraluminal vesicles into multivesicular bodies.
29 IL-1beta was detected within LAMP-1-positive multivesicular bodies.
30 ne at the ciliary base and not via fusion of multivesicular bodies.
31 ed into internal vesicles of late endosomes (multivesicular bodies), a ubiquitin-dependent event that
34 delayed sequestering of p75NTR-bound NGF in multivesicular bodies and delayed degradation in lysosom
35 tor-interacting protein LIP5 targets AQP2 to multivesicular bodies and facilitates lysosomal degradat
37 uces a marked accumulation of cholesterol in multivesicular bodies and late endosomes, which inhibits
38 res, likely to be endosomes, and with sparse multivesicular bodies and lysosomes found in our reconst
39 tion is required for the trafficking of both multivesicular bodies and lysosomes to the LD surface du
41 NGF accumulates with a significant delay in multivesicular bodies and organelles of the degradation
42 V) are cell membranous sacs originating from multivesicular bodies and plasma membranes that facilita
43 ACA-containing vesicles, likely secreted as multivesicular bodies and presumably involved in the for
45 at the limiting membranes of late endosomes/multivesicular bodies and that Anx2 depletion is associa
46 his complex is required for the formation of multivesicular bodies and the degradation of internalize
47 ed a novel interaction between CD63-positive multivesicular bodies and the intracellular chlamydiae,
48 e vesicles produced through the formation of multivesicular bodies and their subsequent fusion with t
49 2 is involved in sorting at both the TGN and multivesicular body and that the first step can occur wi
50 es formed through fusion of early endosomes, multivesicular bodies, and early autophagosomes induced
51 sicles in late endosomal compartments called multivesicular bodies, and for the sorting of ubiquitina
52 res endosome trafficking, is associated with multivesicular bodies, and is regulated by Wnt5a through
53 arly endosomes, Rab7-positive late endosomes/multivesicular bodies, and LAMP1-positive lysosomes and
54 signaling endosomes, which are distinct from multivesicular bodies, and provide mechanistic insight i
55 er of plasma membranes, recycling endosomes, multivesicular bodies, and released exosomes; 2) a commo
56 oscopy and found to be present in endosomes, multivesicular bodies, and vacuoles, all known to be end
57 oteins that are deposited into the lumens of multivesicular bodies are either sorted for lysosomal-me
59 oth ECE-1 and -2 activities, suggesting that multivesicular bodies are intracellular sites of Abeta d
60 eceptors to intra-lumenal vesicles (ILVs) of multivesicular bodies, are thought to terminate signalin
61 5, known to be required for the formation of multivesicular bodies, as a key sensor of thresholds for
62 pivotal role in receptor downregulation and multivesicular body biogenesis and is conserved from yea
63 AAA+ ATPase VPS4 plays an essential role in multivesicular body biogenesis and is thought to act by
65 udding is a key step in vesicular transport, multivesicular body biogenesis, and enveloped virus rele
66 ery functions in HIV-1 budding, cytokinesis, multivesicular body biogenesis, and other pathways, in t
67 emodeling events that accompany cytokinesis, multivesicular body biogenesis, and retrovirus budding,
68 n a number of biological processes including multivesicular body biogenesis, cytokinesis, and envelop
69 ion in HIV-1 release, autophagosome closure, multivesicular body biogenesis, cytokinesis, and other c
70 or transport (ESCRT) system is essential for multivesicular body biogenesis, in which cargo sorting i
71 ed for transport (ESCRT) are responsible for multivesicular body biogenesis, membrane abscission duri
72 le cellular pathways, including cytokinesis, multivesicular body biogenesis, repair of the plasma mem
73 rt (ESCRT) pathway remodels membranes during multivesicular body biogenesis, the abscission stage of
74 MITF stabilization caused an increase in multivesicular body biosynthesis, which in turn increase
75 ndependent of GRASP proteins, autophagy, and multivesicular bodies but involves enclosure within endo
76 calized with markers for early endosomes and multivesicular bodies but not the trans-Golgi network.
77 re initially thought to be late endosomes or multivesicular bodies, but it has since been shown that
79 the cell, leading to particle assembly in a multivesicular body compartment and defective release of
82 anous vesicles inside inclusions, as well as multivesicular bodies docked on the inclusion surface, b
83 ting of ubiquitinated membrane proteins into multivesicular bodies en route to lysosomes for degradat
84 ly colocalized with endosomes positive for a multivesicular bodies/exosomes marker CD63 in regenerati
86 is transported via the ESCRT pathway through multivesicular bodies for degradation, can also target t
87 omes, where it facilitates the generation of multivesicular bodies for TCR degradation and signal ter
88 Y trafficking, whereas VipD interferes with multivesicular body formation at the late endosome and e
89 icating that the Wnt pathway is dependent on multivesicular body formation, a process called microaut
94 s that originate as the internal vesicles in multivesicular bodies from every renal epithelial cell t
95 in a robust inhibition of APP transport into multivesicular bodies, further demonstrating that Gas1 n
96 hat are secreted by cells when intracellular multivesicular bodies fuse with the plasma membrane.
98 e their discovery in 2001: the biogenesis of multivesicular bodies in endolysosomal sorting; the budd
99 f the protein degradation pathway, including multivesicular bodies in the axons and lysosomes within
100 n treatment also led to increased numbers of multivesicular bodies in the cytoplasm, some of which co
101 drive membrane scission for trafficking into multivesicular bodies in the endocytic pathway and for t
102 re elucidated, including transport involving multivesicular body inner vesicles/tubules and exocytosi
103 ed Wnt signaling by increasing the number of multivesicular bodies into which the Wnt signalosome/des
104 rade signaling through Pincher-generated Trk-multivesicular bodies is distinctively refractory to sig
105 d membrane proteins into lumenal vesicles of multivesicular bodies is mediated by the Endosomal Sorti
106 it: 1) storage of exosomal FasL and TRAIL in multivesicular bodies is protected from proteolytic clea
108 rafficking of internalized Ag to specialized multivesicular bodies known as MHC class II compartments
109 ough the early endosome to the late endosome/multivesicular body (LE/MVB) does not change, but exitin
110 eration of peptide-MHC class II complexes in multivesicular body-like MIIC compartments of B cells.
112 Rab7 leads to gross morphological changes of multivesicular bodies, lysosomes, and autophagosomes, co
113 ial urothelial cells, including increases in multivesicular bodies, lysosomes, and expression of the
116 tein Shrub has a central role in endosome-to-multivesicular body membrane trafficking, with synaptic
117 mice revealed a reduction in mature type-II multivesicular bodies (MVB II) and an accumulation of la
118 AA-ATPase, Vps4 is important for function of multivesicular bodies (MVB) sorting pathway, which invol
119 rk, plasma membrane, apoplast, late endosome/multivesicular bodies (MVB), transitory late endosome/ t
122 s involving membrane invagination, including multivesicular body (MVB) biogenesis, viral budding, and
125 te membrane exvagination processes including multivesicular body (MVB) formation, enveloped virus bud
127 rocess appeared to be blocked at the step of multivesicular body (MVB) fusion with the vacuolar membr
129 e sorting of transmembrane proteins into the multivesicular body (MVB) internal vesicles requires the
132 additional function of targeting CD4 to the multivesicular body (MVB) pathway for eventual delivery
134 of ubiquitinated cargo transport through the multivesicular body (MVB) pathway using a dominant negat
135 ns is a signal for active inclusion into the Multivesicular Body (MVB) pathway, resulting in lysosoma
142 27 is a component of ESCRT-0 involved in the multivesicular body (MVB) sorting pathway during endocyt
147 lization of the exosome lifecycle, including multivesicular body (MVB) trafficking, MVB fusion, exoso
149 mplex, which is required for function of the multivesicular body (MVB), an endosomal structure that f
150 HIV budding and in vesicle formation at the multivesicular body (MVB), where they interact with othe
151 um (ER) after Rab5B depletion but not in the multivesicular body (MVB), which is thought to be an org
152 hologous endosomal NHE Nhx1 is important for multivesicular body (MVB)-vacuolar lysosome fusion, the
157 unication, are formed intracellularly within multivesicular bodies (MVBs) and are released upon fusio
158 romotes the formation of ferritin-containing multivesicular bodies (MVBs) and exosomes that transport
159 resides within the intraluminal vesicles of multivesicular bodies (MVBs) and inside exosomes, which
160 T-2 co-localized with HBV surface protein at multivesicular bodies (MVBs) and physically interacted w
161 es and is required for both the formation of multivesicular bodies (MVBs) and the endocytic host cell
164 terruption of cholesterol trafficking within multivesicular bodies (MVBs) by chemical inhibitors or g
165 radation are sorted into lumenal vesicles of multivesicular bodies (MVBs) by the endosomal sorting co
167 cargo proteins into the lumenal vesicles of multivesicular bodies (MVBs) depends on the recruitment
168 mulates in a subset of LBPA-rich perinuclear multivesicular bodies (MVBs) distinct from those carryin
169 rich regions near/at the basolateral LIS and multivesicular bodies (MVBs) expressing early endosomal
170 pathway and targeting of internalized CD4 to multivesicular bodies (MVBs) for eventual degradation in
171 estration of the ubiquitinated receptor into multivesicular bodies (MVBs) for subsequent degradation.
172 sembly to intracellular compartments such as multivesicular bodies (MVBs) generally leads to a signif
175 idase S (Cps1p) into the luminal vesicles of multivesicular bodies (MVBs) in Saccharomyces cerevisiae
176 found that overexpressed PARK9 localized to multivesicular bodies (MVBs) in the human H4 cell line.
177 mbrane proteins into the lumenal vesicles of multivesicular bodies (MVBs) is dependent on the attachm
178 ta-catenin destruction complex components in multivesicular bodies (MVBs) is required for sustained c
180 acuolar lumen, sorting endosomes mature into multivesicular bodies (MVBs) through the action of ENDOS
181 the endocytic delivery of cell surface Hh to multivesicular bodies (MVBs) via an endosomal sorting co
182 vesicles released from cells after fusion of multivesicular bodies (MVBs) with the plasma membrane (P
183 secreted vesicles arising from the fusion of multivesicular bodies (MVBs) with the plasma membrane.
184 light-sensitive rhabdomeres and localized to multivesicular bodies (MVBs) within the photoreceptor cy
186 ane, leading to the extracellular release of multivesicular bodies (MVBs), initially contained within
187 sequestration of GSK3 from the cytosol into multivesicular bodies (MVBs), so that this enzyme become
189 n packaging, stability, the relation to CD63/multivesicular bodies (MVBs), the modulation of choleste
190 bset of late-endosomal compartments known as multivesicular bodies (MVBs), whose formation is control
223 suggest that nucleotide exchange of Ypt7p on multivesicular bodies (MVBs)/late endosomes must take pl
225 with Abeta42 being prominently localized to multivesicular bodies of neurons, as shown in Alzheimer'
227 ynaptic boutons demonstrated the presence of multivesicular bodies, organelles involved in the produc
228 ith the idea that HIV uses the late endosome/multivesicular body pathway during virion budding from m
229 Ubiquitinated Fpn is trafficked through the multivesicular body pathway en route to degradation in t
230 genetic interactions with components of the multivesicular body pathway in fission yeast and budding
231 independently of the function of Vps4 in the multivesicular body pathway, as dominant-negative Vps4 p
232 etion of Hrs and Tsg101, acting early in the multivesicular body pathway, retained APP in early endos
237 -8 functions in vesicle trafficking from the multivesicular body/pre-vacuolar compartment to the lyti
238 anelles indicates that, differently from the multivesicular bodies present in dendritic cells, in mon
239 from the tubulovesicular organization of the multivesicular bodies previously reported in dendritic c
240 the degradation of the ESCRT protein-charged multivesicular body protein (CHMP2B), thus generating a
243 different human ESCRT-III subunits, charged multivesicular body protein 1B (CHMP1B) and increased so
244 nd identify strong interactions with charged multivesicular body protein 1B (CHMP1B), CHMP2A, and inc
247 and cellular analyses, we identified charged multivesicular body protein 2B (CHMP2B), which is part o
248 valosin-containing protein (n = 5), charged multivesicular body protein 2B (n = 4), and linked to ch
250 1 (TSG101) and the ESCRT-III subunit charged multivesicular body protein 4b (CHMP4B) are sequentially
251 ed for transport (ESCRT)-III subunit charged multivesicular body protein 4B (CHMP4B) colocalizes and
252 ansport (ESCRT) machinery, including charged multivesicular body protein 6 (CHMP6), or CHMP2A in comb
253 e Arabidopsis thaliana ESCRT-related CHARGED MULTIVESICULAR BODY PROTEIN/CHROMATIN MODIFYING PROTEIN1
255 that the ESCRT-III subunit paralogs CHARGED MULTIVESICULAR BODY PROTEIN1 (CHMP1A) and CHMP1B are req
258 proteins are AAA(+) ATPases required to form multivesicular bodies, release viral particles, and comp
260 r (EGFR) to the intralumenal vesicles of the multivesicular body requires the coordinated action of s
261 Rab7) are minivacuoles that are competent in multivesicular body sorting and cargo degradation but re
262 tify a new component of the ESCRT-I complex, multivesicular body sorting factor of 12 kD (Mvb12), and
263 bscission and retroviral budding, but not in multivesicular body sorting of activated epidermal growt
264 y of membrane remodeling processes including multivesicular body sorting, abscission during cytokines
265 impact multiple cellular processes including multivesicular body sorting, abscission, and viral buddi
267 mes are lipid vesicles derived from cellular multivesicular bodies that are enriched in specific miRN
268 t may involve the P2X7R-induced formation of multivesicular bodies that contain exosomes with entrapp
270 nthesizing enzymes localize to intracellular multivesicular bodies that, upon stimulation, release th
271 athway functions in vesicle formation at the multivesicular body, the budding of enveloped RNA viruse
272 required for the sequestration of MHC II in multivesicular bodies, this modification is essential fo
273 and then fuse with lysosomes, endosomes and multivesicular bodies through mechanisms that involve ac
274 nine is required for GSK3 sequestration into multivesicular bodies through microautophagy, an essenti
277 f resident protein and lipid constituents of multivesicular bodies to the intracellular chlamydiae.
278 is trafficked through either early endosome/multivesicular bodies to the late endosome-Golgi for sur
280 ese studies confirm CD63 as a constituent in multivesicular body-to-inclusion transport; however, oth
281 of Golgi cisternae, plasma membrane, select multivesicular bodies, tonoplast, dense intravacuolar bo
282 leaved form of RILP promotes the movement of multivesicular bodies toward the cell periphery and indu
284 his trafficking pathway with an inhibitor of multivesicular body transport and the delivery of exogen
288 omparison of the morphology of intracellular multivesicular bodies, we detect changes in their distri
289 or the formation of intralumenal vesicles in multivesicular bodies, were also found to be required fo
290 IL are expressed on the limiting membrane of multivesicular bodies where, by membrane invagination, i
291 herin and HBV L protein at the intracellular multivesicular body, where the budding of HBV virions ta
292 ling was absent from the Golgi apparatus and multivesicular bodies, which are associated with protein
293 r degradation are internalized and sorted to multivesicular bodies, which fuse with lysosomes, where
294 orting of TLR7 into intralumenal vesicles of multivesicular bodies, which terminates signalling.
295 tetherin colocalizes with HBV virions on the multivesicular body, which is the HBV virion budding sit
296 reen fluorescent protein-SKD1 colocalizes on multivesicular bodies with fluorescent fusion protein en
297 olgi-derived exocytic vesicles and endosomal multivesicular bodies with the bacteria-containing paras
299 sence of VPS27, there was an accumulation of multivesicular bodies with vacuolar fragmentation and mi
300 into multivesicular endosomes (also known as multivesicular bodies) with subsequent fusion of the mul