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1  delivery of internalised viral particles to multivesicular endosomes.
2  budding of the endosomal membrane generates multivesicular endosomes.
3 0-nm extracellular vesicles that derive from multivesicular endosomes.
4 ciated with the intralumenal vesicles of the multivesicular endosomes.
5  by the localization of MR1 molecules in the multivesicular endosomes.
6 e cancer cells were principally derived from multivesicular endosomes.
7 cs with the formation of luminal vesicles in multivesicular endosomes.
8 ana, which localizes to the cytoplasm and to multivesicular endosomes.
9 containing late endosomes/lysosomes, but not multivesicular endosomes.
10 utant cells, resulting in the restoration of multivesicular endosomes.
11 e-to-lysosome transport intermediates called multivesicular endosomes.
12  originate from maturation of endosomes into multivesicular endosomes (also known as multivesicular b
13  antisense mRNA, resulted in accumulation of multivesicular endosomes and immature rhoptries.
14 chain of T. gondii AP-1 (a) was localized to multivesicular endosomes and the limiting and luminal me
15                             We conclude that multivesicular endosomes are essential components of the
16 omet tails had the morphological features of multivesicular endosomes as revealed by electron microsc
17 during the ostensibly disparate processes of multivesicular endosome biogenesis, cytokinesis, and ret
18  vitro strategies to study the mechanisms of multivesicular endosome biogenesis.
19    Membrane contact sites between the ER and multivesicular endosomes/bodies (MVBs) play important ro
20 R) are trafficked through a subpopulation of multivesicular endosomes/bodies (MVBs) that are distinct
21 ed, and sorted into intraluminal vesicles of multivesicular endosomes by endosomal sorting complexes
22  extracellular DNA through an autophagy- and multivesicular-endosome-dependent but exosome-independen
23 and-receptor complexes through the early and multivesicular endosomes followed by segregation of the
24 in, for early-to-late-endosome transport and multivesicular endosome formation.
25                             AnxA2-containing multivesicular endosomes fuse directly with the plasma m
26           The factors that determine whether multivesicular endosomes fuse with the plasma membrane o
27                         We show that loss of multivesicular endosome-generating endosomal sorting com
28                     eHAV is released through multivesicular endosomes in a process dependent on endos
29 educed intralumenal vesicle concatenation in multivesicular endosomes in both tapetal cells and devel
30                       An alternative fate of multivesicular endosomes is fusion with lysosomes, resul
31 cling fate, routing the receptor to modified multivesicular endosomes (MVBs) and lysosomal compartmen
32                                              Multivesicular endosomes (MVBs) are major sorting platfo
33 p is sorted from the trans-Golgi through the multivesicular endosome (MVE) and to the vacuole.
34 ns that participate in endosomal sorting and multivesicular endosome (MVE) biogenesis, many of them w
35 tein sorting into vesicles that bud into the multivesicular endosome (MVE) en route to the vacuole.
36 plex, which is required for formation of the multivesicular endosome (MVE).
37 y been identified as important components of multivesicular endosomes (MVEs) and are involved in the
38 re secreted to the extracellular milieu when multivesicular endosomes (MVEs) dock and fuse with the p
39                             The formation of multivesicular endosomes (MVEs) mediates the turnover of
40                                              Multivesicular endosomes (MVEs) sequester membrane prote
41 ollectively participate in the biogenesis of multivesicular endosomes (MVEs).
42 oes into intralumenal vesicles (ILVs) within multivesicular endosomes (MVEs).
43 ity control via sorting into the degradative multivesicular endosome pathway.
44                     These class II MHC-rich, multivesicular endosomes receive incoming antigen and ca
45  of a large enveloped DNA virus engaging the multivesicular endosome sorting machinery to enable infe
46 gion and in vesicles and tubules surrounding multivesicular endosomes, suggesting impaired recycling
47 as accompanied by protein sorting defects at multivesicular endosomes that divert the exosomal marker
48  of exosomes is intraluminal vesicles within multivesicular endosomes that undergo exocytic fusion wi
49 ation of intralumenal vesicles that bud into multivesicular endosomes, the ESCRT-II complex initiates
50  the budding of newly assembled capsids into multivesicular endosomes, the first step in nonlytic rel
51 tivation, thereby disrupting the delivery of multivesicular endosomes to lysosomes.
52               This enhances the targeting of multivesicular endosomes to the cell periphery, their fu
53 tioning into intralumenal vesicles (ILVs) of multivesicular endosomes underlies such cellular process
54 e vesicles whose biogenesis by exocytosis of multivesicular endosomes was discovered in 1983.
55 ial for the formation of luminal vesicles in multivesicular endosomes, we now show that Vps4 function
56 )-dependent release from hepatocytes through multivesicular endosomes, what is known about how they e
57 ses RAB27A and RAB27B regulate exocytosis of multivesicular endosomes, which lead to exosome secretio
58 ion of sEV, and triggered co-localization of multivesicular endosomes with lysosomes for degradation.
59 lar vesicles that originate by the fusion of multivesicular endosomes with the plasma membrane [1-8].
60 icular bodies) with subsequent fusion of the multivesicular endosomes with the plasma membrane, it re
61 les that include functional mitochondria and multivesicular endosomes, yet lack a definable nucleus.