ライフサイエンスコーパス: mumol

1 .7 +/- 8.4 mo; n = 94) were administered 1.0 mumol [14,15]-13C2-retinyl acetate to estimate total liv

2 ltammetry was linear in the range of 0.1-1.0 mumol L(-1) with limits of detection and quantification

3 it (U) of LE catalyzed the hydrolysis of 1.0 mumol of TAPTA per minute in a pH 7.40 phosphate buffer

4 efined as total liver retinol reserves >=1.0 mumol/g liver, which increased to 71.6% after supplement

5 OM incubation (0.5 and 1.0 mumol/L) enhanced force generation at submaximal Ca(2+)

6 se disposal, from 30.5+/-15.9 to 61.6+/-13.0 mumol per kilogram of fat-free mass per minute in the di

7 s baseline (visit 1: 38.0, 95% CI 0.00-132.0 mumol/L; P<0.001).

8 ration of Ses in the range from 3.0 to 140.0 mumol L(-1) with a limit of detection of 0.71 mumol L(-1

9 7.2 +/- 2.0 mumol( ) L(-1) ; P < 0.0001) and suppressed by antibioti

10 ith the highest H(2) generation rate of 25.0 mumol h(-1) , which is nearly 20 times above that using

11 (IQR, 79.1 to 117.8 mumol/h/kg) versus 81.0 mumol/h/kg (IQR, 75.9 to 88.6 mumol/h/kg) (P = 0.165) by

12 (IQR, 84.4 to 129.3 mumol/h/kg) versus 88.0 mumol/h/kg (IQR, 73.0 to 102.2 mumol/h/kg) (P = 0.247) b

13 and in the CP + CHD [serum: 1.8 (1.4 to 2.0) mumol/L; salivary 1.5 (1.2 to 1.7) mumol/g protein, P <

14 a large range of light intensities (0-1,000 mumol photons m(-2) s(-1); beta (PSII) ) are negatively

15 io with a H(2) evolution rate of over 11,000 mumol g(-1) h(-1) without any sacrificial reagents at 27

16 mounts of nitrous oxide (0.00012 +/- 0.00004 mumol N(2)O gDW(-1) hr(-1)) and carbon dioxide (3.556 +/

17 .017 +/- 0.004 compared with 0.041 +/- 0.004 mumol/g creatinine, respectively; P = 0.0009) and had 52

18 with a resultant mean +/- SEM 0.03 +/- 0.01 mumol . kg LBM-1 . min-1 lower net balance (P < 0.05), w

19 reased after the control diet (0.02 +/- 0.01 mumol/L and -0.03 +/- 0.02 mumol/L, respectively; P = 0.

20 30 +/- 0.05, 0.09 +/- 0.05 and 0.08 +/- 0.01 mumol/L.h for phytofluene, phytoene, lycopene and beta-c

21 day average PPFD) leads to a 0.031 +/- 0.013 mumol C m(-2) s(-1) increase in the maximum photosynthet

22 lose carbonate at a rate of - 0.17 +/- 0.02 mumol kg(-1) ppm(-1).

23 C (0.04 +/- 0.02 compared with 0.13 +/- 0.02 mumol/g creatinine, respectively; P = 0.002), and 58% le

24 dels after a single dose of the ADC-46 (0.02 mumol/kg).

25 adient of oxygenated to anoxic waters (<0.02 mumol/l O(2) ) in the Eastern Tropical South Pacific (ET

26 et (0.02 +/- 0.01 mumol/L and -0.03 +/- 0.02 mumol/L, respectively; P = 0.01).

27 4.74 to 9.33) in the diet group and by 7.02 mumol per kilogram of fat-free mass per minute (95% CI,

28 9 mumol/mg in GRs and HVs, but 0.07 +/- 0.03 mumol/mg in PRs (Kruskal-Wallis: P <0.01).

29 ltammetry with a limit of detection of 0.032 mumol L(-1).

30 of glucose production from baseline, by 7.04 mumol per kilogram of fat-free mass per minute (95% conf

31 micrograms per deciliter; 1 mug/dL = 0.0483 mumol/L) in U.S. women 15-49 years of age between 1976 a

32 (IQR) concentration of plasma TMAO was 3.05 mumol/L (2.10-4.60 mumol/L).

33 ce the sum of the activity of Pt/PC-50 (1.07 mumol h(-1) ) and Cu/PC-50 (1.9 mumol h(-1) ), it might

34 oducibility and low limit of detection (0.09 mumol L(-1)).

35 m lysine was 0.19 +/- 0.10 and 0.20 +/- 0.09 mumol/mg in GRs and HVs, but 0.07 +/- 0.03 mumol/mg in P

36 = 0.4%, N = 10) and limit of detection (0.1 mumol L(-1)) were obtained with the all complete AM elec

37 SEMs: 0.6 +/- 0.1 compared with 1.0 +/- 0.1 mumol/g creatinine, respectively; P = 0.002), 63% less h

38 ol/L) (normal range, 0.3-1.0 mg/dL [5.1-17.1 mumol/L]).

39 , with a nonclinically relevant bias of -2.1 mumol/L (95% CI, -3.7 to -0.5 mumol/L).

40 ized, with CO(2) production of 106.3 +/- 5.1 mumol g(-1) biochar.

41 resulted in a 50% receptor occupancy of 8.1 mumol/kg.

42 On average, a 1 mumol m(-2) s(-1) increase in [Formula: see text] (ten-d

43 defective in restoring resistance after a 1 mumol/L S1P challenge as compared with the DU145 alpha6W

44 mumol/L) or supplementing with dofetilide (1 mumol/L) failed to convert PsAF to sinus rhythm.

45 ubation of the VMs with PKA inhibitor H89 (1 mumol L(-1) ).

46 EW16, which was blocked by the addition of 1 mumol/L tetrodotoxin.

47 A placebo or 1 mumol DB/kg was given intragastrically to assess its eff

48 which was eliminated by inhibition of PKA (1 mumol L(-1) ).

49 ied to detect glucose in the range of 1.0-10 mumol L(-1) and LOD of 0.21 mumol L(-1).

50 ncreased from 0.26 +/- 0.19 to 0.52 +/- 0.10 mumol 100 g(-1) f.w.

51 ) (R(2) = 0.9992), limit of detection = 0.10 mumol L(-1) (S/N = 3), sensitivity = 35.6 +/- 0.8 mA-L m

52 ammetry was linear in the range of 0.08-2.10 mumol L(-1) with limits of detection and quantification

53 A rats, which was blocked with SB-334867 (10 mumol/L).

54 either sodium nitrite (30-min infusion of 10 mumol/min) or vehicle [0.9% (wt:vol) saline] 24 h before

55 In PxAF ranolazine (10 mumol/L) reduced dominant frequency from 8.3+/-0.4 to 6.

56 and low (250 p.p.m.) pCO(2) amended with 10 mumol L(-1) dissolved organic carbon from Emiliana huxle

57 nder 1 Sun illumination at a rate of ca. 100 mumol cm(-2) h(-1) at 0 V vs RHE, highlighting the excel

58 her in patients with serum creatinine >= 100 mumol/L at surgery (33% versus 0%, P < 0.001).

59 tory of ascites, and serum creatinine >= 100 mumol/L: 5% of the patients with none of these features

60 rkers of severe cholestasis (bilirubin >=100 mumol or alkaline phosphatase >3-fold the upper limit of

61 .05-5.37) of 524 cases for bile acids of 100 mumol/L or more (HR 30.50 [8.83-105.30]; p<0.0001).

62 n serum bile acids concentrations are of 100 mumol/L or more.

63 apable to analyze pure CO(2) samples of ~100 mumol.

64 physiologically relevant range = 0.10 to 100 mumol L(-1) (R(2) = 0.9992), limit of detection = 0.10 m

65 dopamine and a linear range from 6.0 to 100 mumol L(-1), with a LOD of 0.39 mumol L(-1), by differen

66 saccharide for 6 hours and perfused with 100 mumol/L CORM-401 (or inactive compound iCORM-401)-pretre

67 linear and quadratic contrast 10, 100, 1000 mumol/L) and DLM (0 vs. [100, 300 mumol/L] and 100 vs. 3

68 h before treatment with CC (0, 10, 100, 1000 mumol/L) and DLM (0, 100, 300 mumol/L) in a factorial de

69 , and contrasts for CC (0 vs. [10, 100, 1000 mumol/L] and linear and quadratic contrast 10, 100, 1000

70 y contrast, high-intensity white light (1000 mumol photons m(-2) s(-1)), darkness, and light preferen

71 physiologically relevant range = 100 to 1000 mumol L(-1) (R(2) = 0.9997), sensitivity = 2.83 +/- 0.01

72 Serum bilirubin higher than 115 mumol/L 2-5 days after the procedure has a significant n

73 [IQR] 72-99) in the tolerant cohort and 118 mumol/l (IQR 99-143) in the control group.

74 on samples with a mean concentration of 0.12 mumol/m(3).

75 highest nitrate production rate of 130+/-12 mumol h(-1) g(MO) (-1) at an applied potential of 1.6 V

76 cells expressing KCNQ1+KCNE1; IC50=184+/-12 mumol/L), whereas azithromycin suppressed L-type Ca(++)

77 A 12 mumol h(-1) sustained rate of H2 production was maintain

78 level of 93 mumol/L (reference range, 79-125 mumol/L), a serum total protein level of 82 g/L (referen

79 L(-1) and a limit of detection (LOD) of 0.13 mumol L(-1), by square wave voltammetry for dopamine and

80 mal inhibitory concentration values, 1.4-132 mumol/L) in a concentration-dependent manner.

81 ight <=60 kg, or creatinine >=1.5 mg/dl [133 mumol/l]) were randomized to receive apixaban 2.5 mg twi

82 y of frustules were about 400 m(2)/g and 140 mumol/mL for AQ1, 390 m(2)/g and 130 umol/mL for NP.

83 reater oxygen storage (OSC) capacity of 1434 mumol O/g.

84 highest rate of methane production was 0.15 mumol CH(4) g(-1) oil d(-1) , orders of magnitude lower

85 58.44 mg GAE/g, 250.20 mumol TE/g and 720.15 mumol TE/g, respectively.

86 60.96 mg GAE/g, 380.53 mumol TE/g and 990.15 mumol TE/g, respectively.

87 otosynthesis increased by 10%-50% with a 150 mumol mol(-1) increase in atmospheric CO2 across seasons

88 Low-intensity white light (150 mumol photons m(-2) s(-1)) behaved similarly to light pr

89 r the intravenous route is approximately 160 mumol/hour in control mice.

90 lant, and the serum creatinine level was 160 mumol/L (1.8 mg/dL) at 2 months posttransplant.

91 inusoidal LD ('sinLD') cycle peaking at 1600 mumol photons m(-2) sec(-1) .

92 ponding to the seafloor O(2) fugacity of 162 mumol/L to 297 mumol/L.

93 an impressive hydrogen evolution rate >1700 mumol g(-1) h(-1) generated by the synergistic activity

94 boxylic acid group density from 0.03 to 0.18 mumol cm(-2).

95 h ferrihydrite, whereas magnetite removed 18 mumol g(-1) of aqueous vanadate after 48 h and uptake by

96 ydrite reduction increased from 0.07 to 0.19 mumol d(-1) with a 10-fold increase in the AQDS concentr

97 ditions resulted in a detection limit of 0.2 mumol L(-1) for DXC, but in a 10 times higher sensitivit

98 5 mumol min(-1) 100 g(-1) ; CON: 2.9 +/- 0.2 mumol min(-1) 100 g(-1) ; P < 0.05).

99 sed by antibiotic treatment (OA: 1.2 +/- 0.2 mumol( ) L(-1) ; P < 0.0001 vs.

100 ) versus 88.0 mumol/h/kg (IQR, 73.0 to 102.2 mumol/h/kg) (P = 0.247) by the two mass spectrometric me

101 ol/L) and ABOc recipients (creatinine, 140.2 mumol/L) (P = 0.99), with no significant change over the

102 al inhibitory concentration values, 0.2-16.2 mumol/L) and, as a result, calcium-induced mitochondrial

103 with KNO3 (visit 2: 199.5, 95% CI 98.7-300.2 mumol/L; visit 3: 471.8, 95% CI 377.8-565.8 mumol/L) ver

104 respective mass spectrometric methods: 93.2 mumol/h/kg of body weight (IQR, 84.4 to 129.3 mumol/h/kg

105 utamic acid concentrations (74.4 versus 98.2 mumol/L) at day 6 compared to non-CRRT patients (n = 24)

106 7% of reservoirs were N(2)O sinks (-12 to -2 mumol N(2)O.m(-2).d(-1)) in Canada's largest agricultura

107 PP and FRAP values of 58.44 mg GAE/g, 250.20 mumol TE/g and 720.15 mumol TE/g, respectively.

108 Doubling the ranolazine concentration (20 mumol/L) or supplementing with dofetilide (1 mumol/L) fa

109 transmittance aggregometry in response to 20 mumol/L adenosine diphosphate.

110 in patients with bilirubin levels above 200 mumol/L.

111 tosynthetically active radiation (100 or 200 mumol quanta m(-2) s(-1)) and flow velocity (5 or 15 cm

112 tial rates of ~330 mumol L(-1).h(-1) and 205 mumol L(-1).h(-1) were found for low and high NaCl conce

113 range of 1.0-10 mumol L(-1) and LOD of 0.21 mumol L(-1).

114 ables found by CHPM are in the range of 1-21 mumol-eq/g.

115 Kr (CHO cells expressing hERG; IC50=219+/-21 mumol/L) and IKs (CHO cells expressing KCNQ1+KCNE1; IC50

116 1.90 mumol.L(-1), 2.85 mmol.L(-1), and 5.22 mumol.L(-1) respectively.

117 with ammonia production rates of 128 and 220 mumol h(-1) g(-1) , respectively.

118 was reduced (high-grade tumor CMRO(2), 0.23 mumol/g/min +/- 0.07; low-grade tumor CMRO(2), 0.39 mumo

119 8 mumoles/cm(2)/h (ON, -1.5 V) and 0.17-0.23 mumoles/cm(2)/h (OFF, 0 V), corresponded to the higher d

120 ) and FRAP assays gave values of 872 to 2428 mumol Trolox eq./100 g f.w.

121 , superoxide dismutase mimetic (GC4419; 0.25 mumol/L) significantly mitigated the increased sensitivi

122 ular Ca (2.7 mmol/L) and isoproterenol (0.25 mumol/L) to induce diastolic Ca waves and subthreshold D

123 ease in the vasculature even at low dose (25 mumol/kg) of the complex.

124 re exposed to PAR levels of 105, 157, or 250 mumol quanta m(-2) s(-1) over a 12-h period each day.

125 , with EC50 values ranging from 2119 to 2559 mumol kg(-1) dry weight soil based on nominal concentrat

126 er samples with a limit of detection of 0.26 mumol L(-1).

127 seafloor O(2) fugacity of 162 mumol/L to 297 mumol/L.

128 rnace heating of two identical monomers (1.3 mumol h(-1) ).

129 with a medium root mean square error of 12.3 mumol m(-2) s(-1) , which suggests that seasonal changes

130 umol/h/kg of body weight (IQR, 84.4 to 129.3 mumol/h/kg) versus 88.0 mumol/h/kg (IQR, 73.0 to 102.2 m

131 was similar between ABOi (creatinine, 140.3 mumol/L) and ABOc recipients (creatinine, 140.2 mumol/L)

132 K1 (HEK cells expressing Kir2.1, IC50=44+/-3 mumol/L).

133 ting our working hypothesis (2.28 x 10(-)(3) mumol/g tissue for quinidine and ~4.10 x 10(-)(4) mumol/

134 enzoic acid (on average from 0.67 up to 1.30 mumol/g dry matter) throughout large intestinal fermenta

135 esulted in an apparent zero-order rate of 30 mumol L(-1).h(-1), whereas rates of ~300 mumol L(-1).h(-

136 10, 100, 1000 mumol/L) and DLM (0, 100, 300 mumol/L) in a factorial design.

137 100, 1000 mumol/L) and DLM (0 vs. [100, 300 mumol/L] and 100 vs. 300 mumol/L) were evaluated.

138 ous light or at irradiance levels of 150-300 mumol m(-2) s(-1) .

139 mug of catalytic polymer (that is, >244,300 mumol h(-1) g(-1) of catalytic polymer)).

140 al photosynthetic photon flux density of 300 mumol m(-2) s(-1).

141 30 mumol L(-1).h(-1), whereas rates of ~300 mumol L(-1).h(-1) and ~3750 mumol L(-1).h(-1) were compu

142 LM (0 vs. [100, 300 mumol/L] and 100 vs. 300 mumol/L) were evaluated.

143 rature (0-25 degrees C), light levels (0-325 mumol photons m(-2) s(-1)), and cell density factor (1.0

144 0 h(-1) and a H(2) production rate of >0.327 mumol h(-1) with 1.34 mug of catalytic polymer (that is,

145 .10 mg QE/g, 3.01-5.63 mg/g, and 13.54-17.33 mumol TE/g, respectively.

146 .44 to 8.34) and 5.37 (95% CI, 2.41 to 8.33) mumol per kilogram of fat-free mass per minute in the tw

147 serum uric acid (WA: 341.4 mumol/L; CB: 330 mumol/L; P = 0.020) compared with the CB.

148 For PCD, initial rates of ~330 mumol L(-1).h(-1) and 205 mumol L(-1).h(-1) were found f

149 the highest test concentration (EC50 > 3397 mumol L(-1) and >4892 mumol kg(-1) dry weight soil).

150 mumol/g/min +/- 0.16; overall CMRO(2), 0.34 mumol/g/min +/- 0.16) compared with normal-appearing gra

151 and 28% had liver impairment (bilirubin >34 mumol/L), each of these parameters defining organ failur

152 em provides a wide dynamic range (up to 3500 mumol/L) for all three N species for both isotope abunda

153 atter in both participants with glioma (2.36 mumol/g/min +/- 0.22 vs 0.75 mumol/g/min +/- 0.10, respe

154 y, the most active compound 6b (ED50 = 28.36 mumol/kg po in mice) showed improved inhibition for coll

155 arget concentrations are as follows: 120-360 mumol/L for individuals aged 0-12 years and for maternal

156 phenylalanine concentration is less than 360 mumol/L.

157 over 72 h during the 40% fat (2.50 +/- 0.37 mumol/g creatinine) and 0% fat (2.37 +/- 0.37 mumol/g cr

158 umol/g creatinine) and 0% fat (2.37 +/- 0.37 mumol/g creatinine) interventions were similar, but a ~5

159 as rates of ~300 mumol L(-1).h(-1) and ~3750 mumol L(-1).h(-1) were computed for low (0.03 mol.L(-1))

160 , respectively) and healthy volunteers (2.38 mumol/g/min +/- 0.15 vs 0.63 mumol/g/min +/- 0.05, respe

161 in photocatalytic activity from 2050 to 3810 mumol h(-1) g(-1) when we infuse a suspension of bulk g-

162 m 6.0 to 100 mumol L(-1), with a LOD of 0.39 mumol L(-1), by differential pulse voltammetry for serot

163 served during the 0% fat-fast (1.05 +/- 0.39 mumol/g creatinine) intervention (compared with 0% fat,

164 /min +/- 0.07; low-grade tumor CMRO(2), 0.39 mumol/g/min +/- 0.16; overall CMRO(2), 0.34 mumol/g/min

165 O was higher with ageing (young: 2.6 +/- 0.4 mumol( ) L(-1) vs.

166 ain tissues, finding that it ranges from 0.4 mumol.kg(-1) in brain to 1.4 mumol.kg(-1) in kidney.

167 ranges from 0.4 mumol.kg(-1) in brain to 1.4 mumol.kg(-1) in kidney.

168 et group and from 29.4+/-12.6 to 54.5+/-10.4 mumol per kilogram of fat-free mass per minute in the su

169 week 24 (least squares mean difference -2.4 mumol/L [SE 0.4], 95% CI -3.2 to -1.6; p<0.0001).

170 ld reach a hydrogen evolution rate of 2072.4 mumol g(-1) h(-1) .

171 8), and increased serum uric acid (WA: 341.4 mumol/L; CB: 330 mumol/L; P = 0.020) compared with the C

172 mol/L (3.0 mg/dL); serum creatinine 8.8-35.4 mumol/L (0.1-0.4 mg/dL); radiographic evidence of ricket

173 umol/L) and 37.8 mumol/L (95% CI: 36.4, 39.4 mumol/L) in NHANES 1999-2000 and 2009-2010, respectively

174 (rabbit ventricular myocytes, IC50=66.5+/-4 mumol/L) and IK1 (HEK cells expressing Kir2.1, IC50=44+/

175 ic means for the sum of the 4 TFAs were 81.4 mumol/L (95% CI: 77.3, 85.6 mumol/L) and 37.8 mumol/L (9

176 /g tissue for quinidine and ~4.10 x 10(-)(4) mumol/g tissue for the conjugate).

177 levels of 0.5 mg or more per deciliter (>=40 mumol per liter) above baseline occurred in 7.0% of pati

178 with serum total bile acids of less than 40 mumol/L versus four (0.28%; 0.08-0.72) of 1412 cases wit

179 18 degrees C) and light intensity (30 vs. 40 mumol quanta.m(2).s(-1)), which should have decreased ph

180 G6 and G30) grown under ambient (aCO(2), 400 mumol mol(-1)) and elevated CO(2) (eCO(2), 620 mumol mol

181 periment in 2014 and 2015 under ambient (400 mumol mol(-1) ) and elevated (600 mumol mol(-1) ) CO(2)

182 a single dose of gamma irradiation 50 at 42 mumol/kg eliminated detectable clonogens in some SiHa ce

183 covered the mole fraction range of (378-420) mumol mol(-1) and were prepared and/or value assigned ei

184 and 309 to 432 mumol Fe(2+) eq./100 g f.w., respectively.

185 carbon (POC) exhibited a mean value of 0.44 mumol heme b mol(-1) POC.

186 ied, partial C(2) phenotype with C (*) of 44 mumol mol(-1) Increased investment of GLDP in MS tissue

187 LL) greater than or equal to 30 mug/dL (1.45 mumol/L) with concomitant GI symptoms.

188 plementation (1.13 +/- 0.43 to 1.29 +/- 0.46 mumol/g; P < 0.001).

189 ackinawite reached near complete removal (46 mumol g(-1)) of aqueous vanadate after 3 h and rates wer

190 hr(-1)) and carbon dioxide (3.556 +/- 0.471 mumol CO(2) gDW(-1) hr(-1)).

191 rts (change in eGFR level per 1-mg/dl [59.48 mumol/l] increase in SU: -1.99 ml/min/1.73 m2; 95% CI -2

192 entration (EC50 > 3397 mumol L(-1) and >4892 mumol kg(-1) dry weight soil).

193 uptake was lower in IUGR (IUGR: 1.3 +/- 0.5 mumol min(-1) 100 g(-1) ; CON: 2.9 +/- 0.2 mumol min(-1)

194 from baseline at 12 months (mean change -0.5 mumol/L [SD 3.0]; p=0.021).

195 t bias of -2.1 mumol/L (95% CI, -3.7 to -0.5 mumol/L).

196 he use of low amounts of lead compounds (1-5 mumol).

197 ivity, with HER rates reaching 33.2 and 17.5 mumol h(-1) from pure water under simulated solar light

198 increase of 72% in the rETRmax value (190.5 mumol electrons m(-2) s(-1)), compared with the conventi

199 cessfully separated and detected down to 2.5 mumol/L and demonstrated only 50% to 60% signal suppress

200 er detection limit in our experiment was 2.5 mumol/L.

201 creatinine rose by 50% or by 0.3 mg/dL (26.5 mumol/L) from preoperative baseline.

202 PPH reduction and ORAC stabilization at 52.5 mumol muL(-1) of Trolox Equivalent.

203 pecific CO(2) sorption rates (in excess of 5 mumol s(-1) g(-1) bar(-1)) and high selectivity due to t

204 ol functionalities (Mem-RS(tot) = 380 +/- 50 mumol g(-1) C) are situated closely enough to be involve

205 Limits of detection (LODs) of 20, 40, and 50 mumol L(-1) were obtained for ethanol, n-propanol, and m

206 diabetic DIOS patients with hepatic iron >50 mumol/g at magnetic resonance imaging to compare the met

207 .4 vs. 39.9 muM and FRAP value = 598 vs. 514 mumol/L, respectively), exerted an antioxidant activity

208 nzothiazoline-6-sulfonic acid) (ABTS) (16.52 mumol TEAC/g) assay.

209 DPPH (IC(50) = 0.89 mug/mL) and FRAP (225.53 mumol equivalent ferrous sulphate/g).

210 e (1/6-1/6-2/3), with 60.96 mg GAE/g, 380.53 mumol TE/g and 990.15 mumol TE/g, respectively.

211 eposition, with a mean concentration of 0.54 mumol/m(3).

212 ccumulation of superoxide radicals (WT, 4.54 mumol/mg tissue/min; CatA-TG, 8.62 mumol/mg tissue/min),

213 169 +/- 0.340 compared with -0.036 +/- 0.544 mumol/L in vitamin B-12 and placebo groups, respectively

214 +/- 0.21, 10.04 +/- 0.30, and 13.49 +/- 0.55 mumol phenylalanine . kg-1 . h-1, respectively; P < 0.00

215 esentation were 462 U/L (IQR 266-790) and 56 mumol/L (IQR 25-100) respectively.

216 (191 mg/dl), creatinine at admission >132.6 mumol/l (1.5 mg/dl), Thrombolysis In Myocardial Infarcti

217 ity, with LC50 values between 78.3 and 161.6 mumol L(-1) and deformation of the protective cuticle.

218 (95% CI 4.14 to 11.65) and homocysteine 4.6 mumol/l p < 0.05 (95% CI 0.32 to 8.87) CONCLUSION: The l

219 either group (Deltacreatinine, -0.83 vs 6.6 mumol/L) (P = 0.59).

220 yields 14.9 mumol g(-1) of methane and 77.6 mumol g(-1) of CO, representing a much more effective ca

221 4 TFAs were 81.4 mumol/L (95% CI: 77.3, 85.6 mumol/L) and 37.8 mumol/L (95% CI: 36.4, 39.4 mumol/L) i

222 g) versus 81.0 mumol/h/kg (IQR, 75.9 to 88.6 mumol/h/kg) (P = 0.165) by the two mass spectrometric me

223 n of plasma TMAO was 3.05 mumol/L (2.10-4.60 mumol/L).

224 0-12 years and for maternal PKU, and 120-600 mumol/L for non-pregnant individuals older than 12 years

225 bient (400 mumol mol(-1) ) and elevated (600 mumol mol(-1) ) CO(2) concentrations, and under ambient

226 rachninae species with C (*) values of 56-61 mumol mol(-1), whereas two-thirds of leaf GLDP was in th

227 (WT, 4.54 mumol/mg tissue/min; CatA-TG, 8.62 mumol/mg tissue/min), increased inflammation, myocyte hy

228 mol mol(-1)) and elevated CO(2) (eCO(2), 620 mumol mol(-1)) were assessed.

229 olunteers (2.38 mumol/g/min +/- 0.15 vs 0.63 mumol/g/min +/- 0.05, respectively) (P < .001 and P = .0

230 e detected with the detection limits of 0.65 mumol.L(-1), 1.90 mumol.L(-1), 2.85 mmol.L(-1), and 5.22

231 reported, with a H(2) production rate of 655 mumol g(-1) h(-1) , which could rival excellent photocat

232 ogether with adequate detection limit of 4.7 mumol L(-1).

233 wo mass spectrometric methods in SM and 93.7 mumol/h/kg (IQR, 79.1 to 117.8 mumol/h/kg) versus 81.0 m

234 4 to 2.0) mumol/L; salivary 1.5 (1.2 to 1.7) mumol/g protein, P < 0.001] group compared to CP patient

235 1.2 to 1.8) mumol/L; salivary 1.3 (1 to 1.7) mumol/g protein, P < 0.01] and in the CP + CHD [serum: 1

236 retinol-binding protein <14.7 mug/mL or 0.70 mumol/L) and inflammation status (C-reactive protein >3.

237 umol L(-1) with a limit of detection of 0.71 mumol L(-1).

238 ving a maximum U(VI) membrane uptake of 6.73 mumol g(-1) and an affinity constant of 9.85 . 10(6) L m

239 C (8.90-14.72 mg GAE/g) and TAA (11.37-12.74 mumol TE/g) decreased significantly with increase in roa

240 th glioma (2.36 mumol/g/min +/- 0.22 vs 0.75 mumol/g/min +/- 0.10, respectively) and healthy voluntee

241 t 13 mM and specific activity of 1470 +/- 75 mumol/min/g.

242 e amount of AA from 990 +/- 16 to 830 +/- 76 mumol 100 g(-1) f.w.

243 her (TEAC assay for C(alc): 848.11 +/- 60.78 mumol TE/g protein).

244 st, and TEOA donor, H2 evolution rate of 782 mumol h(-1) g(-1) and TON of 54.4 has been obtained in a

245 o a maximum methane chlorination rate of 0.8 mumol(CH(4,conv)) (g(catalyst) s)(-1).

246 ol/L [SD 4.1]; p=0.0042) and 48 months (-0.8 mumol/L [3.8]; p=0.016).

247 n SM and 93.7 mumol/h/kg (IQR, 79.1 to 117.8 mumol/h/kg) versus 81.0 mumol/h/kg (IQR, 75.9 to 88.6 mu

248 0 +/- 0.2%) and sugar content (101.3 +/- 3.8 mumol g(-1) fw) and very low (0.007-0.009) Na/K ratio we

249 umol/L (95% CI: 77.3, 85.6 mumol/L) and 37.8 mumol/L (95% CI: 36.4, 39.4 mumol/L) in NHANES 1999-2000

250 ffusive sea-air methane efflux (17.3 +/- 4.8 mumol m(-2)d(-1)).

251 mumol/L; visit 3: 471.8, 95% CI 377.8-565.8 mumol/L) versus baseline (visit 1: 38.0, 95% CI 0.00-132

252 howed a highest yield of C(2) product of 6.8 mumol h(-1) at a space velocity of 2400 h(-1) , more tha

253 peutically useful nicotine fluxes of 1.3-1.8 mumoles/cm(2)/h (ON, -1.5 V) and 0.17-0.23 mumoles/cm(2)

254 er in the CHD group [serum: 1.5 (1.2 to 1.8) mumol/L; salivary 1.3 (1 to 1.7) mumol/g protein, P < 0.

255 chieved a linear response between 30 and 800 mumol L(-1) and a limit of detection (LOD) of 0.13 mumol

256 Phosphate was added at 0, 80, 400, and 800 mumoles g(-1) (i.e., zero, low, medium, and high loading

257 was 62 (10) years and for GlycA was 375 (82) mumol/L; 53% women.

258 ion of CO2 with high CO generation rate (825 mumol h(-1) g(-1)) and outstanding stability under visib

259 -group difference (95% CI): 4266 (261, 8270) mumol.min-1.kg-1.180 min; P = 0.04; eta2p = 0.31] and br

260 was associated with grade 3-4 HE (116 vs. 83 mumol/L) and mortality at day 21 attributed to neurologi

261 1 vs. 90 mumol/L) and all causes (114 vs. 83 mumol/L; P < 0.001 for all).

262 gm at 25 degrees C ranged from 2.90 to 7.85 mumol m(-2) s(-1) Pa(-1) .

263 Median creatinine levels were 85 mumol/l (interquartile range [IQR] 72-99) in the toleran

264 rsus baseline at 12 months (mean change -0.9 mumol/L [SD 4.1]; p=0.0042) and 48 months (-0.8 mumol/L

265 /PC-50 (1.07 mumol h(-1) ) and Cu/PC-50 (1.9 mumol h(-1) ), it might also be the highest among photoc

266 s-solid interface with formation yields 14.9 mumol g(-1) of methane and 77.6 mumol g(-1) of CO, repre

267 e detection limits of 0.65 mumol.L(-1), 1.90 mumol.L(-1), 2.85 mmol.L(-1), and 5.22 mumol.L(-1) respe

268 e 1,1-diphenyl-2-picrylhydrazyl (DPPH) (8.90 mumol TEAC/g) and 2,2'-Azino-bis (3-ethylbenzothiazoline

269 the AA content from 766 +/- 89 to 849 +/- 90 mumol 100 g(-1) f.w.

270 ay 21 attributed to neurological (181 vs. 90 mumol/L) and all causes (114 vs. 83 mumol/L; P < 0.001 f

271 ospheric CO2 uptake rates (-33,300 +/- 7,900 mumol m(-2)d(-1)) twice that of surrounding waters and a

272 37 mukat/L]), a serum creatinine level of 93 mumol/L (reference range, 79-125 mumol/L), a serum total

273 duction over 70 h with a maximum rate of 941 mumol h(-1) g(-1), turnover number TON(Ni) > 103, and to

274 of 1412 cases with total bile acids of 40-99 mumol/L (hazard ratio [HR] 2.35 [95% CI 0.52-10.50]; p=0

275 9 +/- 0.6), and the short-chain fatty acids (mumol/g) acetate (decrease 27.4 +/- 22.6 vs 21.6 +/- 20.

276 bilization of paraoxonase 1 (PON1) activity (mumol/min/mg) when compared with WT apoA-I and comparabl

277 ivity (666 +/- 51.8 GBq/mumol; 18 +/- 1.4 Ci/mumol).

278 nd was 853 +/- 29.6 GBq/mumol (23 +/- 0.8 Ci/mumol).

279 est antioxidant (0.013-1.432mumol Trolox eq./mumol peptide) and ACE inhibitory activities (IC50=44-12

280 d their dual activity (10-14mumol Trolox eq./mumol peptide; IC50=11-21muM).

281 average yield of 69.7% and more than 100 GBq/mumol specific activity.

282 and molar activities between 33 and 114 GBq/mumol.

283 r radioactivities ranging from 20 to 250 GBq/mumol.

284 of >98% and molar activity of 98 +/- 30 GBq/mumol EOS.

285 nd molar activities of 25-40 and 200-300 GBq/mumol, respectively.

286 urity and molar activities of 192 +/- 33 GBq/mumol.

287 Molar activity ranged from 40 to 336 GBq/mumol, and an excellent radiochemical purity of greater

288 ield = 31.6%, specific activity >/= 48.5 GBq/mumol).

289 tivity of this compound was 853 +/- 29.6 GBq/mumol (23 +/- 0.8 Ci/mumol).

290 ty, >97%) with molar activities of 12-60 GBq/mumol.

291 d molar activities ranging from 52 to 65 GBq/mumol (radiochemical purity > 99%).

292 , with a specific activity of 279 +/- 75 GBq/mumol.

293 ell as high molar activity (666 +/- 51.8 GBq/mumol; 18 +/- 1.4 Ci/mumol).

294 1)C-Me-NB1 was synthesized at 290 +/- 90 GBq/mumol molar activity, 7.4 +/- 1.9 GBq total activity at

295 formula: (creatinine plasma concentration in mumol/L) = (creatinine concentration in DBS in mumol/L)/

296 c regression revealed baseline creatinine in mumol/L [OR 0.99 (95% CI 0.99 - 1.00), p = 0.019] and du

297 mol/L) = (creatinine concentration in DBS in mumol/L)/0.73, with a nonclinically relevant bias of -2.

298 methacholine (adjusted beta-coefficient log-mumol, -0.80 [95% CI, -1.55 to -0.06]; P = 0.0348), and

299 The DME productivity (mumol g(cat) (-1) h(-1) ) was comparable to oxygenate pr

300 tide concentrations ranged from high nmol to mumol g(dry)(-1) with slightly higher cell quotas in the