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1 al myocarditis, in particular of that due to mumps virus.
2  least two doses of a vaccine containing the mumps virus.
3 measuring serological responses to wild-type mumps virus.
4 d have reduced efficacy against heterologous mumps viruses.
5 copy of the genome of a Jeryl Lynn strain of mumps virus (15,384 nucleotides) was assembled from cDNA
6  proteins of Nipah virus, measles virus, and mumps virus also abolishes MDA5 interaction.
7                          We demonstrate that mumps virus also eliminates cellular STAT3, a protein th
8 nfluenza virus type 2 (PIV2), PIV3, PIV5, or mumps virus and determined the abundances of individual
9                                     Like MV, mumps virus and dsRNA failed to induce I kappa B alpha d
10 nist activity exhibited by the V proteins of mumps virus and human parainfluenza virus type 2.
11 mber of cellular genes compared to wild-type mumps virus and increases cell death in infected cells,
12 metric mean titers of antibody to measles or mumps viruses and low seroconversion rates.
13 including respiratory syncytial virus (RSV), mumps virus, and human metapneumovirus (HMPV).
14 ished human pathogens such as measles virus, mumps virus, and the human parainfluenza viruses; highly
15  to affect attenuation were detected in OPV, mumps virus, and varicella-zoster virus.
16 , characterize different aspects of the anti-mumps virus antibody response after vaccination.
17                                    Wild type mumps viruses are highly neurotropic and a frequent caus
18  also infected with a related paramyxovirus, mumps virus, as a specificity control.
19 nfluenza virus (PIV) type 2, PIV3, PIV5, and mumps virus, as well as on the relative abundance of the
20                                              Mumps virus-associated CNS complications in vaccinees co
21 luate the antigenic relationship between bat mumps virus (BMV) and the JL5 vaccine strain of mumps vi
22 l culture, or by detection of the RNA of the mumps virus by reverse transcription (RT)-PCR.
23                                A recombinant mumps virus carrying the E95D mutation in its P and V pr
24 er paramyxoviruses in the genus Rubulavirus, mumps virus catalyzes the proteasomal degradation of cel
25 nesis of the disease, with enteroviruses and mumps virus considered potential causes.
26  associated with the presence of antibody to mumps virus, data from the 1999-2004 National Health and
27 e-adjusted seroprevalence of IgG antibody to mumps virus during 1999-2004 was 90.0% (95% CI, 88.8%-91
28                                              Mumps virus, enteroviruses (including human parechovirus
29  of the L protein, did not prevent rescue of mumps virus, even though an amino acid alignment for the
30 pressing the canine distemper virus (CDV) or mumps virus F protein.
31 on of three animal models with an isolate of mumps virus from a recent outbreak (MuV-IA).
32 me cDNA was demonstrated by amplification of mumps virus from transfected-cell cultures and by subseq
33                             We sequenced 166 mumps virus genomes collected in Washington and other US
34 d assay for whole-genome sequencing (WGS) of mumps virus genotype G.
35                          Vaccination against mumps virus has been effective in reducing mumps cases.
36 , parainfluenza virus type 5, measles virus, mumps virus, Hendra virus, and Nipah virus.
37 n enteroviruses/rhinoviruses, measles virus, mumps virus, Hepatitis A-E Virus, Chikungunya virus, den
38 k provides insight into a previously unknown mumps virus-host interaction.
39 ramyxovirus pathogens include measles virus, mumps virus, human respiratory syncytial virus, and the
40                                         Anti-mumps virus immunoglobulin M (IgM) antibodies were detec
41 n of immunoglobulin M-specific antibodies to mumps virus in acute-phase serum samples, the isolation
42  no evidence of resistance to infection with mumps virus in any cell line.
43  acute-phase serum samples, the isolation of mumps virus in cell culture, or by detection of the RNA
44 developed to allow rapid characterization of mumps virus in clinical samples.
45  primers, indicating that the persistence of mumps virus in the myocardium may be related to the sele
46 tly assesses the neurovirulence potential of mumps viruses in humans and is robust and reproducible.
47 indered analysis of the neuropathogenesis of mumps virus infection and the identification of molecula
48 al fibroelastosis associated with persistent mumps virus infection by vaccination supports the notion
49 sed antibody levels that may protect against mumps virus infection for longer than previously assumed
50 r neuropathological and clinical outcomes of mumps virus infection of the neonatal rat brain demonstr
51 his report we show, for the first time, that mumps virus infection of the neonatal rat leads to devel
52 tly more participants were protected against mumps virus infection up to 1 year after vaccination (ie
53 croRNAs playing role in the host response to mumps virus infection.
54                                              Mumps virus is a common infectious agent of humans, caus
55                                          The mumps virus is a negative-strand RNA virus in the family
56 MuV replication and transcription.IMPORTANCE Mumps virus is an important human pathogen.
57                                              Mumps virus is highly neurotropic and, prior to widespre
58 pinal fluid (CSF) samples and in extracts of mumps virus isolates from patients with various clinical
59        Furthermore, we generated recombinant mumps viruses lacking expression of both the V protein a
60 hown that clinical isolate-based recombinant mumps viruses lacking expression of either the V protein
61                                              Mumps virus, like other paramyxoviruses in the Rubulavir
62       Despite the presence of these multiple mumps virus lineages, the genomic data show that one lin
63 uently, PIV5 NP protein is incompatible with mumps virus M protein.
64 myxoviruses parainfluenza virus 5 (PIV5) and mumps virus, M-NP interaction also contributes to effici
65  virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyssavirus, herpes simplex v
66                However, results with several mumps virus MNVTs have raised questions as to whether th
67 ic and neurovirulent properties of wild-type mumps viruses, most national regulatory organizations re
68                     Here we demonstrate that mumps virus (MuV) and vesicular stomatitis virus (VSV) a
69    Here we report that while PIV2, PIV5, and mumps virus (MuV) are sensitive to IFIT1, nonrubulavirus
70                                              Mumps virus (MuV) caused the most viral meningitis befor
71                                              Mumps virus (MuV) causes a highly contagious human disea
72                                              Mumps virus (MuV) causes an acute infection in humans ch
73                                          The mumps virus (MuV) fusion protein (F) plays a crucial rol
74                                          The mumps virus (MuV) genome encodes a phosphoprotein (P) th
75 otype F has been the predominant genotype of mumps virus (MuV) in the last 20 years in mainland China
76                                              Mumps virus (MuV) infection may cause serious diseases i
77 unctional CD8+ T-cell response after natural mumps virus (MuV) infection that was not present after v
78                                              Mumps virus (MuV) is a highly contagious paramyxovirus t
79                                              Mumps virus (MuV) is a highly contagious pathogen, and d
80                                              Mumps virus (MuV) is a reemerging paramyxovirus that cau
81                                              Mumps virus (MuV) is highly neurotropic and was the lead
82  circulating and emerging strains.IMPORTANCE Mumps virus (MuV) is the causative agent of the highly i
83 nts of the MMR vaccine: measles virus (MeV), mumps virus (MuV) Jeryl Lynn (JL1) strain, and MuV JL2 s
84 ) of Omicron XBB.1.5 based on the attenuated mumps virus (MuV) Jeryl Lynn (JL1) vaccine strain.
85      We systematically mapped the domains in mumps virus (MuV) P and investigated their interactions
86 iral proteins play in the phosphorylation of mumps virus (MuV) P.
87                                    To define mumps virus (MuV) proteins important for this process, v
88        Formerly a common childhood pathogen, mumps virus (MuV) remains active worldwide, despite rela
89               To investigate the role of the mumps virus (MuV) SH protein in virulence, multiple stop
90                           Three hundred nine mumps virus (MuV) strains detected in the United Kingdom
91  with high phylogenetic relatedness to human mumps virus (MuV) was identified recently at the nucleic
92                                              Mumps virus (MuV), a paramyxovirus containing a negative
93             The nucleocapsid protein (NP) of mumps virus (MuV), a paramyxovirus, was coexpressed with
94                                              Mumps virus (MuV), a rubulavirus of the paramyxovirus fa
95 luenza virus 3 (HPIV3), measles virus (MeV), mumps virus (MuV), and respiratory syncytial virus (RSV)
96 esults were obtained for the closely related mumps virus (MuV), except that MuV particles derived fro
97 RNA-mediated signaling; these are encoded by mumps virus (MuV), human parainfluenza virus 2 (hPIV2),
98  failure.We identified for the first time 41 mumps virus (MuV)-specific HLA-A*02 epitopes.
99 to detect the small hydrophobic (SH) gene of mumps virus (MuV).
100 s a common childhood infection caused by the mumps virus (MuV).
101 breaks and to understand the pathogenesis of mumps virus (MuV).
102 ps virus (BMV) and the JL5 vaccine strain of mumps virus (MuVJL5), we rescued a chimeric virus bearin
103                                          For mumps virus N, a binding site for the P protein maps to
104                         For both measles and mumps virus N, truncated proteins encompassing amino aci
105            Nonetheless, the genetic basis of mumps virus neurotropism and neurovirulence was until re
106  effective safety test with which to measure mumps virus neurovirulence has also hindered analysis of
107 the United States, to test a novel rat-based mumps virus neurovirulence safety test.
108  value in elucidating the molecular basis of mumps virus neurovirulence.
109                                              Mumps virus-neutralizing antibodies are believed to be t
110 mmunoglobulin G (IgG) antibody responses and mumps virus-neutralizing antibody responses (based on th
111 oteins of the paramyxoviruses measles virus, mumps virus, Newcastle disease virus, human parainfluenz
112  shown systemic efficacy: vaccinia, measles, mumps, viruses, Newcastle disease virus, and reovirus.
113 that interactions between this region of the mumps virus NP and its polymerase leads to exposure of t
114                                              Mumps virus NP protein harbors DWD in place of the DLD s
115 together with support plasmids which express mumps virus NP, P, and L proteins under control of the T
116 vCSP (VK210, VK247 and P. vivax-like) to the mumps virus nucleocapsid protein to enhance immune respo
117                    The structure of an empty mumps virus nucleocapsid-like particle is determined to
118 f the patient had laboratory confirmation of mumps virus or probable if they had clinical symptoms an
119 samples were seropositive for measles virus, mumps virus, or rubella virus antibodies, and there were
120 ast 50 amino acids of both measles virus and mumps virus P (measles virus P, 457 to 507; mumps virus
121  mumps virus P (measles virus P, 457 to 507; mumps virus P, 343 to 391) by themselves constitute the
122  a lesser degree of tertiary organization in mumps virus P.
123 ative responses to measles virus (P=.01) and mumps virus (P=.006).
124  important viruses, including measles virus, mumps virus, parainfluenza viruses, respiratory syncytia
125 esent the best animal model for the study of mumps virus pathogenesis.
126 STAT3 are independently targeted by a single mumps virus protein, called V, that assembles STAT-direc
127 mon respiratory viruses (HPIV-2, -3, and -4, mumps virus, respiratory syncytial virus, and influenza
128 scribe the generation of a novel recombinant mumps virus (rMuV) expressing HIV-1 Gag (rMuVgag) and me
129 re, we developed live attenuated recombinant mumps virus (rMuV)-based SARS-CoV-2 vaccine candidates u
130                              The duration of mumps virus RNA detection was studied during a mumps out
131                                              Mumps virus RNA was characterized directly from cerebros
132 predict genes for 12 viruses: measles virus, mumps virus, rubella virus, respiratory syncytial virus,
133 ype 6, varicellazoster virus, measles virus, mumps virus, rubella virus, the picornavirus group, infl
134 ble EIA kits were used to evaluate wild-type mumps virus serological responses in human serum samples
135 of sequence relationships, with examples for mumps virus SH gene cDNA and prion protein sequences.
136                              Moreover, since mumps virus-specific antibody titers are generally low i
137                                            A mumps virus-specific enzyme immunoassay was used to meas
138     A*26-Cw*12-B*38 was associated with both mumps virus-specific humoral (P=.007) and cell-mediated
139                                              Mumps virus-specific immunoglobulin G (IgG) antibody res
140 ccine, a measles virus Edmonston and the two mumps virus strains [Jeryl Lynn 1 (JL1) and JL2] that ar
141   The study also allowed characterization of mumps virus strains from Argentina as part of a new subg
142 accine viruses, raising concern that certain mumps virus strains may escape vaccine-induced immunity.
143                                Here, various mumps virus strains representing a wide range of neuropa
144                    Neurovirulence of several mumps virus strains was assessed in a prototype rat neur
145 and highly neurovirulent (88-1961 wild type) mumps virus strains were passaged in human neural cells
146 riminate neurovirulent from nonneurovirulent mumps virus strains.
147  (Kilham) from nonneurovirulent (Jeryl Lynn) mumps virus strains.
148 efully selected group of genetically diverse mumps virus strains.
149 te among the relative neurovirulent risks of mumps virus strains.
150 man parainfluenza virus 2 targets STAT2, and mumps virus targets both STAT1 and STAT3.
151 ue cytokine and oncogene evasion property of mumps virus that provides a molecular basis for its obse
152                   To test whether attenuated mumps viruses used in the manufacture of mumps vaccines
153 interferon-activated STAT1 or STAT2 protein, mumps virus V protein is unique in its ability to also t
154                                Consequently, mumps virus V protein prevents responses to interleukin-
155 that a single amino acid substitution in the mumps virus V protein, E95D, results in defective STAT3
156 stigate genetic variation in live attenuated mumps virus vaccine by using both MAPREC and a platform
157 t can reliably predict the neurovirulence of mumps virus vaccine candidates in humans.
158 utralizing test) against both the Jeryl Lynn mumps virus vaccine strain (hereafter, the "vaccine stra
159 ples to neutralize the genotype A Jeryl Lynn mumps virus vaccine strain and a genotype G wild-type vi
160  potentially neurovirulent, live, attenuated mumps virus vaccines stems largely from the lack of an a
161 bserved in monkeys inoculated with wild type mumps virus versus vaccine strains, although differences
162                                 In contrast, mumps virus was not inhibited by the expression of flavi
163 adoption of widespread vaccination programs, mumps virus was the leading cause of virus-induced centr
164                               Two regions of mumps virus were amplified: the nucleocapsid gene and th
165 erologous, nonubiquitous viruses such as the mumps virus were low or absent in the HIV+ group.
166 ublic health investigations with analysis of mumps virus whole genome sequences from 201 infected ind
167 iated N protein (N-RNA) for both measles and mumps viruses with proteins produced in a bacterial expr
168 lent cellular immune responses to measles or mumps viruses, with or without passive antibodies when i

 
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