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1 almonella enterica serovar Typhi relies on a muramidase.
2 id II, we report that both MpgA and MpgB are muramidases.
3 lytic transglycosylases actually function as muramidases.
4 with differences in the cellular contents of muramidase-1 or muramidase-2, with the levels of PBP 5 p
5  in the cellular contents of muramidase-1 or muramidase-2, with the levels of PBP 5 produced, or with
6 idoglycan O-acetylation modulates endogenous muramidase activity affecting the cell-surface propertie
7                                 In contrast, muramidase activity alone does not support rod assembly.
8 ectly involved in rod formation and that the muramidase activity of FlgJ, though needed for formation
9              These results indicate that the muramidase activity of lysozyme is not required for bact
10  the hypothesis that the catalytic activity (muramidase activity) of lysozyme is not required for bac
11 orresponding amino acid from MpgA results in muramidase activity, allowing us to predict from the pre
12 ein in the airspaces without any increase in muramidase activity.
13 D53S) in mouse lysozyme M completely ablated muramidase activity.
14 wall hydrolase with two catalytic domains, a muramidase and a peptidase.
15 d sensitivity of mutant cell walls to the M1 muramidase and decreased sensitivity to lysostaphin, whi
16  MS ring-rod junction protein (FliE), or the muramidase and putative rod-capping protein (FlgJ).
17 in a region of the protein distinct from the muramidase catalytic site.
18 l-L-alanine amidase) but were cleaved by the muramidases CPL and cellosyl.
19                                              Muramidase-deficient recombinant lysozyme (LysM(D53S)) k
20 t not by lipoteichoic acid, was abolished by muramidase digestion.
21               In Pseudomonas aeruginosa, the muramidase effector Tse3 (Type VI secretion exported 3)
22 s: (i) an N-terminal domain with homology to muramidases from several gram-positive bacterial species
23 s, generating the only glycosyl hydrolase 25 muramidases in plants and archaea.
24 the two catalytic domains and found that the muramidase is essential, whereas the peptidase is partia
25 cally associated proteins Class A aPBP1a and muramidase MpgA.
26 ed accumulation of SecA2 substrate, N-acetyl muramidase (NamA) in the cell wall, providing evidence f
27    Surprisingly, FlgJ functions as neither a muramidase nor a lytic transglycosylases but rather as a
28 recedented view of the mechanisms by which a muramidase recognizes its peptidoglycan substrate to fac
29                               The N-terminal muramidase-released-protein-like domain mediated BapA1-B
30                               Digestion with muramidase resulted in cleavage of the glycosidic bonds
31 tants were more sensitive than the parent to muramidases such as hen egg white lysozyme and to the Cw
32                               The lipase and muramidase T6SS effectors identified in this study under
33 ycan to the hydrolytic action of lysozyme, a muramidase that cleaves in the glycan backbone.
34 anism strictly dependent on TtsA, a specific muramidase that facilitates toxin transport through the
35 s present on mucosal surfaces is lysozyme, a muramidase that hydrolyzes the peptidoglycan backbone of
36 ted with bacterial resistance to lysozyme, a muramidase that serves as a central component of innate
37 l vent archaeon with a bacterial competitor, muramidase transcription is upregulated.
38                                  Pesticin, a muramidase, was characterized by active site mutations d
39 en suggested that it is a flagellum-specific muramidase which locally digests the peptidoglycan layer