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1 oduction and also enhanced the production of murine leukemia virus.
2 than to those of oncoretroviral RTs, such as murine leukemia virus.
3 loaded onto unintegrated DNAs of the Moloney murine leukemia virus.
4 or with the amphotropic envelope protein of murine leukemia virus.
5 (HIV-1), simian immunodeficiency virus, and murine leukemia virus.
6 rted infection of one cell line (DEL) with a murine leukemia virus.
7 of the RNAs packaged by a model retrovirus, murine leukemia virus.
8 dimerization and packaging domain of Moloney murine leukemia virus.
9 ived growth factor (PDGF)-containing Moloney murine leukemia viruses.
11 PIM1 (Proviral Integration site for Moloney murine leukemia virus 1) has emerged as a key regulator
12 kinase proviral integration site for Moloney murine leukemia virus-1 (PIM-1), which in turn regulates
16 l protein tyrosine kinase encoded by Abelson murine leukemia virus (Ab-MLV) transforms pre-B cells.
17 ginal pipeline, can identify integrations of murine leukemia virus, adeno-associated virus, Tol2 tran
18 rupted restriction activity against N-tropic murine leukemia virus and equine infectious anemia virus
21 provide a high-resolution atlas of m(5)C in murine leukemia virus and reveal a functional role of m(
22 fective HIV-1 or Vpu(-) retroviruses such as murine leukemia virus and simian immunodeficiency virus.
24 on site pattern of XMRV with those found for murine leukemia virus and two human retroviruses, human
25 gene delivery by wild-type ecotropic Moloney murine leukemia virus and vesicular stomatitis virus (VS
26 some glycoproteins, such as those encoded by murine leukemia virus and vesicular stomatitis virus, we
27 OBEC3F (A3F), which are potent inhibitors of murine leukemia virus and Vif-deficient human immunodefi
28 ene whose overexpression blocks infection by murine leukemia viruses and human immunodeficiency virus
29 stigate to what extent virions of HERV-Kcon, murine leukemia virus, and HIV-1 have the ability to tra
30 ents derived from three retroviruses (HIV-1, murine leukemia virus, and Mason-Pfizer monkey virus), t
31 ell lines blocked cytokine induction by HIV, murine leukemia virus, and simian immunodeficiency virus
33 een XMRV and the intensively studied Moloney murine leukemia virus, architectures of the regulatory d
34 tiviruses and distinct from those of Moloney murine leukemia virus, avian sarcoma leukosis virus, and
35 mbined immunodeficiency (SCID-X1), a Moloney murine leukemia virus-based gamma-retrovirus vector expr
38 r more than 1 year after transduction with a murine leukemia virus-based vector encoding the T-cell g
40 us, MoFe2, or with the parent virus, Moloney murine leukemia virus, caused significant reduction in B
42 so suggest that the leader region of Moloney murine leukemia virus contains inhibitory/regulatory seq
43 ollowed by reverse transcription via Moloney murine leukemia virus, degradation of chromosomal DNA wi
45 se APOBEC3 does not catalyze base changes in murine leukemia virus DNA, it can be recovered from thes
49 rt of this hypothesis, infection of HIV-1 or murine leukemia virus Env (MLV-Env)-pseudotyped HIV-1 pa
50 lays a defect in proteolytic cleavage of the murine leukemia virus Env cytoplasmic tail in pseudotype
53 urface (SU) or transmembrane (TM) protein of murine leukemia virus Env, along with point mutations th
55 native viral glycoproteins, including Friend murine leukemia virus envelope (F-MLV Env) are actively
58 or-binding sequence of the ecotropic Moloney murine leukemia virus envelope glycoprotein with the pep
59 studied how the protomeric units of Moloney murine leukemia virus envelope protein (Env) are activat
60 udotyped with vesicular stomatitis virus and murine leukemia virus envelopes, indicating that defensi
61 support viral entry/infection of pseudotyped murine leukemia viruses expressing pathogenic NWA glycop
62 ammaretrovirus glycoproteins, such as Friend murine leukemia virus (F-MLV) Env, but not with the rela
63 ) in the envelope glycoprotein of the Friend murine leukemia virus (F-MLV) ISD has been reported to a
64 ith defined mixtures of the ecotropic Friend murine leukemia virus (F-MuLV) and different polytropic
67 kely the functionally equivalent sequence in murine leukemia virus Gag has been inferred by mutationa
68 main inserted at random positions throughout murine leukemia virus Gag-Pol, then selecting for varian
69 ells are shown to restrict the expression of murine leukemia virus genomes but not retroviral genomes
70 ith the parental wild-type ecotropic Moloney murine leukemia virus glycoprotein through the ecotropic
71 on by retroviruses that are derived from the murine leukemia virus, human immunodeficiency virus type
72 magnetic resonance structure of the Moloney murine leukemia virus IN (M-MLV) C-terminal domain (CTD)
73 estricts N-tropic (N-MLV), but not B-tropic, murine leukemia virus in a manner dependent upon residue
74 hronically infected with the gammaretrovirus murine leukemia virus in which receptor has been downreg
76 crossover rate similar to that of HIV-1 and murine leukemia virus, indicating that the extremely hig
78 mic lymphocytes by a mink cell focus-forming murine leukemia virus induces apoptosis during the prele
79 se, encoded by the v-Abl oncogene of Abelson murine leukemia virus induces transformation of progenit
82 -5-positive (Lgr5(+)) and B lymphoma moloney murine leukemia virus insertion region homolog-1-positiv
84 is (Arabidopsis thaliana) B lymphoma Moloney murine leukemia virus insertion region1 homolog (BMI1) P
86 e the role of Bmi-1 (B-cell-specific Moloney murine leukemia virus integration site 1) as a regulator
89 profile of raltegravir on the replication of murine leukemia virus is similar to that for HIV, and th
91 ession of proviral insertion site of Moloney murine leukemia virus kinases (Pim-1, -2, and -3) in can
94 mal recessive mutation (Lamc2(jeb)) due to a murine leukemia virus long terminal repeat insertion in
95 LTR) was found to be higher than the Moloney murine leukemia virus LTRs in both LNCaP and WPMY-1 (sim
98 that gPr80gag facilitates release of Moloney murine leukemia virus (M-MuLV) from cells along an IFN-s
99 deficiency virus, type 1 (HIV-1) and Moloney murine leukemia virus (M-MuLV), we determined the effect
100 n immunodeficiency virus type 1) and Moloney murine leukemia virus (M-MuLV), we evaluated how individ
102 ock the release of HIV-1, as well as that of murine leukemia virus (MLV) and Ebola virus (EBOV); knoc
104 tion by mouse mammary tumor virus (MMTV) and murine leukemia virus (MLV) and that there are polymorph
105 the pol gene of gammaretroviruses, including murine leukemia virus (MLV) and xenotropic murine leukem
107 The envelope protein (Env) from the CasBrE murine leukemia virus (MLV) can cause acute spongiform n
109 IM proteins previously identified to inhibit murine leukemia virus (MLV) demonstrated an ability to i
111 tionship between two recent additions to the murine leukemia virus (MLV) ecotropic subgroup: Mus cerv
114 y, we found that the cytoplasmic tail of the murine leukemia virus (MLV) Env could functionally subst
115 The infectivity of HIV-1 pseudotyped with murine leukemia virus (MLV) Env was not affected by Vpu.
116 ty, we generated chimeric constructs between murine leukemia virus (MLV) Gag and HBV Core to determin
120 and Sleeping Beauty (SB) transposons and the murine leukemia virus (MLV) in mouse embryonic stem cell
122 nificantly affects HIV-1 replication but not murine leukemia virus (MLV) infection and that miR-128 m
123 The roles of cellular proteases in Moloney murine leukemia virus (MLV) infection were investigated
125 n 3 protein under conditions whereby Moloney murine leukemia virus (MLV) integrase failed to do so, s
126 of understanding the molecular mechanisms of murine leukemia virus (MLV) integration into host chroma
127 We found that the preintegration complex of murine leukemia virus (MLV) interacts with the dynein co
128 n A3G but is sensitive to murine A3, whereas murine leukemia virus (MLV) is relatively resistant to m
130 interferon-alpha-responsive manner, captures murine leukemia virus (MLV) particles and mediates their
131 form of this glycoprotein is compatible with murine leukemia virus (MLV) particles but incompatible w
132 the efficiencies of restriction of HIV-1 and murine leukemia virus (MLV) particles containing various
133 w analyzed the mRNA content of Psi- and Psi+ murine leukemia virus (MLV) particles using both microar
134 infectious anemia virus (EIAV), or N-tropic murine leukemia virus (MLV) postentry and supported late
135 rs to integrate within active genes, whereas murine leukemia virus (MLV) prefers to integrate near tr
137 binding site (PBS)-dependent restriction of murine leukemia virus (MLV) replication in embryonic ste
139 ted glycosylated Gag (glycoGag) protein of a murine leukemia virus (MLV) similarly enhance the infect
140 viruses, we engineered a fluorescent Moloney murine leukemia virus (MLV) system consisting of MLV-int
143 nterrogated by measuring the cleavage of the murine leukemia virus (MLV) transmembrane Env protein by
146 enhancement of the photonic inactivation of Murine Leukemia Virus (MLV) via 805 nm femtosecond pulse
149 also by vectors bearing the envelope of 10A1 murine leukemia virus (MLV), a murine retrovirus that ca
151 well as simian immunodeficiency virus (SIV), murine leukemia virus (MLV), and the retrotransposon Mus
152 ein for the generation of pseudotyped HIV-1, murine leukemia virus (MLV), and vesicular stomatitis vi
154 oviral vectors, including those derived from murine leukemia virus (MLV), bearing heterologous Env pr
156 eported to be contaminated with an ecotropic murine leukemia virus (MLV), raising questions about the
157 four different retroviruses: HIV-1, Moloney murine leukemia virus (MLV), Rous sarcoma virus (RSV), a
159 y the glycosylated Gag (glycoGag) protein of murine leukemia virus (MLV), the S2 protein of equine in
160 modifications present in a model retrovirus, murine leukemia virus (MLV), using mass spectrometry and
161 se, we used a shuttle vector system in which murine leukemia virus (MLV)-based proviral targets prese
162 ng primary human CD4(+) T lymphocytes with a murine leukemia virus (MLV)-based retroviral vector or x
163 ments in the long terminal repeats (LTRs) of murine leukemia virus (MLV)-based vectors and the vector
164 an gene therapy approaches utilize HIV-1- or murine leukemia virus (MLV)-based vectors, which prefere
165 t may be safer than that of standard Moloney murine leukemia virus (MLV)-derived retroviral vectors.
166 n immunodeficiency virus type 1 (HIV-1)- and murine leukemia virus (MLV)-derived viral vectors, respe
167 ing, are a newly appreciated viral target in murine leukemia virus (MLV)-induced neurodegeneration.
169 four subsequent reports failed to detect any murine leukemia virus (MLV)-related virus gene sequences
170 virus-related virus (XMRV) as well as other murine leukemia virus (MLV)-related viruses, though not
182 The generation of cytopathic effects by murine leukemia viruses (MLVs) in different cell types c
185 Members of the gammaretroviruses--such as murine leukemia viruses (MLVs), most notably XMRV [xenot
188 he 3' end processing site within the Moloney murine leukemia virus (MMLV) LTR d(TCTTTCATT), a host-gu
189 elity, we measured the error rate of Moloney murine leukemia virus (MMLV) RT in the presence of sever
190 n the template switching property of Moloney murine leukemia virus (MMLV)-type reverse transcriptases
191 markedly inhibits the replication of Moloney murine leukemia virus (Mo-MLV) and is required for the a
192 A crystallographic study of the Moloney murine leukemia virus (Mo-MLV) RNase H domain was perfor
194 eas the prototypical gammaretrovirus Moloney murine leukemia virus (MoMLV) favors strong enhancers an
196 oviral reverse transcriptase (RT) of Moloney murine leukemia virus (MoMLV) is expressed in the form o
197 o examine the role of this domain in Moloney murine leukemia virus (MoMLV) replication, we analyzed 1
199 ase, but not the RNase H function of Moloney Murine Leukemia Virus (MoMLV) RT and also inhibited Esch
200 h show that tetherin does not affect Moloney murine leukemia virus (MoMLV) spread, and only minimally
201 cle ultrastructure highly similar to Moloney murine leukemia virus (MoMLV), another gammaretrovirus.
203 studies with small fragments of the Moloney murine leukemia virus (MoMuLV) genome suggested that sel
205 enic temperature-sensitive mutant of Moloney murine leukemia virus (MoMuLV-ts1), results in motor neu
206 odies against clade C HIV-1 gp140, gp120, or murine leukemia virus (MuLV) gp70-scaffolded V1/V2 and t
211 from scarce, femtomole quantities of Moloney murine leukemia virus (MuLV) RNA inside authentic virion
215 on with mouse mammary tumor virus (MMTV) and murine leukemia virus (MuLV) via an adaptive immune mech
217 n the gammaretroviruses, typified by Moloney murine leukemia virus (MuLV), gag and pol are in the sam
218 opic virus 1 (Bxv1), a xenotropic endogenous murine leukemia virus (MuLV), is present in these 2 rece
221 wever, the mechanism of APOBEC inhibition of murine leukemia viruses (MuLVs) does not appear to be G-
224 nfected with mixtures of mouse retroviruses (murine leukemia viruses [MuLVs]) exhibit dramatically al
226 rs TRIM5alpha(hu) and Fv-1 restrict N-tropic murine leukemia virus (N-MLV) infection at an early post
227 capsid, we had previously selected N-tropic murine leukemia virus (N-MLV) mutants escaping from rhes
228 iction of the TRIM5alpha-sensitive, N-tropic murine leukemia virus (N-MLV) rendered HIV-1 transductio
230 The temperature-sensitive form of Abelson murine leukemia virus offers a reversible model to study
232 The infectivity of HIV-1 virions bearing murine leukemia virus or vesicular stomatitis virus glyc
233 The provirus integration site for Moloney murine leukemia virus (Pim) 1 kinase is an oncogenic ser
234 genic (proviral integration site for Moloney murine leukemia virus (PIM) 1, 2, and 3 kinases in a NF-
236 ecific proviral integration site for Moloney murine leukemia virus (PIM) kinases PIM1 and PIM2 have b
238 cture of the double hairpin from the Moloney murine leukemia virus ([Psi(CD)](2), 132 nt, 42.8 kDa) u
239 neonatal infection of rats with the PVC-211 murine leukemia virus (PVC-211 MuLV) and its underlying
241 We investigated the prevalence of xenotropic murine leukemia virus-related virus (XMRV) among 293 par
242 tly, CFS has been associated with xenotropic murine leukemia virus-related virus (XMRV) as well as ot
247 tions regarding the prevalence of xenotropic murine leukemia virus-related virus (XMRV) in patients w
248 ecent study identified DNA from a xenotropic murine leukemia virus-related virus (XMRV) in peripheral
260 iency virus type-1 (HIV-1) and of xenotropic murine leukemia virus-related virus (XMRV), a gammaretro
262 (MLV)-based retroviral vector or xenotropic murine leukemia virus-related virus (XMRV), and isolated
264 DNA from a human gammaretrovirus, xenotropic murine leukemia virus-related virus (XMRV), in 68 of 101
265 described gammaretrovirus genome, xenotropic murine leukemia virus-related virus (XMRV), in prostate
266 e newly identified retrovirus-the xenotropic murine leukemia virus-related virus (XMRV)-has recently
268 d with the human retrovirus XMRV (xenotropic murine leukemia virus-related virus) can induce rare foc
270 a gammaretrovirus, termed "XMRV" (xenotropic murine leukemia virus-related virus) in prostate cancers
273 ow that a retrovirus called XMRV (xenotropic murine leukemia virus-related virus) was present in the
275 inding to Fc; the same hot-spots control HIV/murine leukemia virus restriction by TRIM5alpha and medi
276 eonatal infection of these mice with Moloney murine leukemia virus resulted in accelerated tumor onse
277 tor resistance of several commercial Moloney murine leukemia virus reverse transcriptase (MMLV RT) en
278 t to increase the thermostability of Moloney Murine Leukemia Virus reverse transcriptase (MMLV RT), w
281 ied unexpectedly high levels of m(5)C in the murine leukemia virus RNA, precisely mapped its location
283 Tan-1 RT enzymatically mimics oncoretroviral murine leukemia virus RT which is characterized by its l
285 l replication in vivo, I constructed a novel murine leukemia virus strain (FMLV-IL-1beta) that encode
286 , feline leukemia virus subgroup T, and 10A1 murine leukemia virus, this receptor is the human type I
287 1 with a replaced envelope gene from Moloney murine leukemia virus to allow HTLV-1 to fuse with murin
290 neurovirulent viruses FrCas(NC) and Moloney murine leukemia virus ts1 indicate that the nascent enve
291 be increased by 3- to 5-fold by placing the murine leukemia virus UAG read-through element upstream
292 icrobe, Stavrou et al. (2015) reveal how the murine leukemia virus uses a sugar-protein shield to pro
295 ffect was specific to HIV in that release of murine leukemia virus was minimally affected by the pres
296 stingly, our fusion protein did not restrict murine leukemia virus, which does not incorporate Vpr.
297 r, mouse APOBEC3 protein blocks infection by murine leukemia viruses without catalyzing this base cha
298 variant nonpermissive to XMRV and xenotropic murine leukemia virus (X-MLV) infection, suggesting that
299 iruses (MLVs), most notably XMRV [xenotropic murine leukemia virus (X-MLV)-related virus--have been r
300 g murine leukemia virus (MLV) and xenotropic murine leukemia virus (XMRV), named the CAE (cytoplasmic