ライフサイエンスコーパス: muscle tone

1 26-hDMPK indicated deficient arterial smooth muscle tone.

2 activity is critically dependent upon smooth muscle tone.

3 n important component in modulating vascular muscle tone.

4 the ACE gene have increased vascular smooth muscle tone.

5 l contractility and decrease vascular smooth muscle tone.

6 yl LT alter vascular permeability and smooth muscle tone.

7 role for ADM as a local modulator of smooth muscle tone.

8 e to alterations in the regulation of airway muscle tone.

9 tain the blood supply, and to support smooth muscle tone.

10 is a critical determinant of vascular smooth muscle tone.

11 tive compounds that regulate vascular smooth muscle tone.

12 sts into the same or nearby sites suppresses muscle tone.

13 ponsiveness relates in part to airway smooth muscle tone.

14 embrane potential and thereby control smooth muscle tone.

15 to cardiac function, metabolism, and smooth muscle tone.

16 inc over a wide range of pressure and smooth muscle tone.

17 taplexy without significantly reducing basal muscle tone.

18 n of pulmonary arterial and bronchial smooth muscle tone.

19 bility, seizures, microcephaly, and abnormal muscle tone.

20 ted channels in the control of airway smooth muscle tone.

21 in the cochlea and adjusting vascular smooth muscle tone.

22 c muscle activation and in maintaining basal muscle tone.

23 ay smooth muscle and regulates airway smooth muscle tone.

24 nt neuromotor development, in particular low muscle tone.

25 that alter vascular permeability and smooth muscle tone.

26 activation, PS with muscle atonia, or W with muscle tone.

27 The results showed marked effects of DCS on muscle tone.

28 ctive during PS in negative correlation with muscle tone.

29 to sleep-wake states, cortical activity, and muscle tone.

30 r neurons and interneurons, hence increasing muscle tone.

31 ctive during PS in positive correlation with muscle tone.

32 bution of K(+) and CaV1.2 channels to smooth muscle tone.

33 es and drug targets for regulation of smooth muscle tone.

34 riability and with modulation of respiratory muscle tone.

35 air cell tuning, insulin release, and smooth muscle tone.

36 al fluid secretion, gut motility, and smooth muscle tone.

37 particularly important in regulating smooth muscle tone.

38 ave a structural role in maintaining ciliary muscle tone.

39 tage-dependent Ca(2+) channels and of smooth muscle tone.

40 ines and that it increases myometrial smooth muscle tone.

41 gram (EEG), rapid eye movements, and loss of muscle tone.

42 C-associated behaviors including anxiety and muscle tone.

43 e head control, gross motor development, and muscle tone.

44 d 3-NPA exhibited only torpidity and loss of muscle tone 1-3 h after dosing.

45 ); (c) neurological impairment, with altered muscle tone (14/22); (d) dysmorphisms (13/20).

46 Muscle tone abnormalities are associated with many CNS p

47 tilage anomalies, dysmorphic facial feature, muscles tone abnormalities, skin changes and breathing d

48 in responses: vocalization, social behavior, muscle tone and activity level included opposite respons

49 enoic acids, which modulate bronchial smooth muscle tone and airway transepithelial ion transport.

50 lexy was marked by brief losses of mylohyoid muscle tone and by the observation of episodes of facial

51 We investigated the involvement of muscle tone and circular muscle (CM) contraction in peri

52 However, local muscle tone and contraction are important for the initia

53 ium (BK) channel negatively regulates smooth muscle tone and contraction in ASM.

54 (S1P), in regulating rabbit detrusor smooth muscle tone and contraction.

55 de release functionally as changes in smooth-muscle tone and direct measurement of neuropeptide relea

56 rdiovascular system where sGC governs smooth muscle tone and growth, vascular permeability, leukocyte

57 ts into how orexins regulate wakefulness and muscle tone and how OX2R agonists improve the symptoms o

58 upV BPNs in tonically modulating jaw-closing muscle tone and in mediating bilateral jaw closing.

59 al regulation of environmental awareness and muscle tone and in the pathophysiology of narcolepsy.

60 hysical examination revealed decreased axial muscle tone and increased muscle tone in her extremities

61 /- 3.5 s, n = 11) decrease of rigid hindlimb muscle tone and inhibition of all tested RMC (n = 7) and

62 d to participate in the regulation of smooth muscle tone and intestinal motility.

63 nt regulator of short-term changes in smooth muscle tone and longer-term responses to chronic drug tr

64 LC dendrites with a concurrent reduction in muscle tone and marble burying behavior, an increase in

65 nin receptor homolog on motor axons, setting muscle tone and movement vigor.

66 Additional features included increased muscle tone and muscle cramps.

67 as motor coordination, respiratory control, muscle tone and pain processing.

68 They regulate vascular smooth muscle tone and participate in cardiac action potential

69 count for the inhibition of colonic circular muscle tone and phasic contractions observed during infl

70 Muscle tone and power as well as functional ambulatory c

71 (loss of motor function and abnormalities of muscle tone and power assessed by a pediatrician) at 2 y

72 on of pancreatic islet mass, vascular smooth muscle tone and proliferation, and materno-fetal calcium

73 and WHO clinical stage and oedema, abnormal muscle tone and respiratory distress on examination as c

74 ental delay commonly accompanied by abnormal muscle tone and seizures identified de novo missense mut

75 In addition, we measured strength, muscle tone and sensation using standard clinical instru

76 s-busp, tSCS, and the busp plus tSCS-reduced muscle tone and spasm frequency.

77 tion revealed mild bilateral ptosis, reduced muscle tone and strength that worsened in proximal leg m

78 A physical examination revealed her muscle tone and strength were normal on both sides; howe

79 a result of dysregulation of vascular smooth muscle tone and structural remodeling.

80 strate that S1P may regulate detrusor smooth muscle tone and suggest that dysregulation of complex S1

81 ect evidence that trans-spinal DCS can alter muscle tone and suggest that this approach could be used

82 e to alterations in peripheral airway smooth muscle tone and surfactant function.

83 atory activity and is a primary regulator of muscle tone and thermogenesis, augmenting both of these

84 freezing that were associated with increased muscle tone and thus motor outflow.

85 may underlie differences in bronchial smooth muscle tone and thus pulmonary function, possibly in a s

86 These cells control muscle tone and thus regulate the opening of the airway

87 n addition to known effects on airway smooth muscle tone and transepithelial electrolyte transport, t

88 heral nervous systems, resulting in skeletal muscle tone and various cognitive effects in the brain.

89 rns of activity that differentially regulate muscle tone and voluntary motor activity via distinct ou

90 ularis (NMC) of the medial medulla increases muscle tone and/or produces locomotion, while injection

91 ption, on the vasculature to regulate smooth muscle tone, and as a central neurotransmitter, modulati

92 Muscarinic receptors regulate airway smooth muscle tone, and asthmatics exhibit increased AHR to mus

93 cidate the mechanisms responsible for smooth muscle tone, and may offer cues to pathological situatio

94 rents, these siblings also displayed reduced muscle tone, and one of them developed recurrent seizure

95 rents, these siblings also displayed reduced muscle tone, and one of them developed recurrent seizure

96 FAC, muscle tone, and power showed no association with HKP.

97 rt, control of bronchial and vascular smooth muscle tone, and stimulation of peptide hormone secretio

98 rergic signals to indirectly regulate smooth muscle tone, and thereby regulate the motor function of

99 tor neurons and interneurons, hence reducing muscle tone; and (2) direct current flowing in the oppos

100 lium-dependent regulation of vascular smooth muscle tone are unresolved.

101 ced significant increases in tracheal smooth muscle tone as assessed by pressure changes in a saline

102 s, autonomic activation and loss of postural muscle tone (atonia).

103 The motor examination revealed normal muscle tone, bulk, and power in the upper and lower extr

104 lial mediator that regulates vascular smooth muscle tone, but it may exert its cardiovascular action

105 livery through its effect on vascular smooth muscle tone, but the regulation of these processes is no

106 psaicin and resiniferatoxin can alter smooth muscle tone, but this response does not appear to involv

107 endogenous nitrovasodilators regulate smooth muscle tone by elevation of cGMP and activation of cycli

108 rate Ca2+ sparks to regulate arterial smooth muscle tone by examining the function of RyRs during ont

109 It is well known that vascular smooth muscle tone can be modulated by signals arising in the e

110 g and because, in their absence, waking with muscle tone cannot be maintained and narcolepsy with cat

111 kinases (PKGs) are key regulators of smooth muscle tone, cardiac hypertrophy, and other physiologica

112 ess for long periods and by sudden losses of muscle tone (cataplexy).

113 arcolepsy, including the involuntary loss of muscle tone (cataplexy).(1) Here, we show that the South

114 hways may be possibly correlated with smooth muscle tone changes, increased collagen content, and inf

115 Joint laxity and low muscle tone contributed to musculoskeletal problems comp

116 In addition, muscle tone decreased gradually during transitions from

117 Muscle tone depends on the level of excitability of spin

118 Loss of muscle tone during active (rapid-eye-movement, REM) slee

119 nsible for motoneuron inhibition and loss of muscle tone during active (REM) sleep can be activated b

120 tical activation with behavioral arousal and muscle tone during adaptive waking behaviors.

121 sleep and slow-wave sleep and an increase in muscle tone during REM and non-REM sleep episodes and in

122 obstruction caused by a loss of upper airway muscle tone during sleep.

123 in association with cortical activation and muscle tone during waking and because, in their absence,

124 Cataplexy, which is a sudden loss of muscle tone during waking, is an important diagnostic sy

125 ongoing demands and thus better orchestrate muscle tone, energy balance, and arousal.

126 midbrain locomotor region neurons related to muscle tone facilitation.

127 nglion neurons, which regulate airway smooth-muscle tone, glandular secretion and blood-vessel diamet

128 delayed developmental trajectories on active muscle tone (group x age, P < .001) and total neurologic

129 ctor capable of increasing myometrial smooth muscle tone, has been shown to be up-regulated in the se

130 hythm is tightly coupled with eye movements, muscle tone, heart and breathing rate in lizards, with s

131 the medulla produces changes in locomotion, muscle tone, heart rate, and blood pressure.

132 ponses, such as hemostasis and regulation of muscle tone; however, the roles of PARs in the functions

133 ors are physiological antagonists for smooth muscle tone in airways and bladder.

134 e have shown that GSNOR regulates the smooth muscle tone in airways and the function of beta-adrenerg

135 ects of repeated tsDCS to attenuate abnormal muscle tone in awake female mice with spinal cord injuri

136 In 30 patients with PD, the muscle tone in both arms was evaluated.

137 ed decreased axial muscle tone and increased muscle tone in her extremities; the latter was more seve

138 nhibitors have been found to regulate smooth muscle tone in human prostate.

139 of inhaled NO gas would relax airway smooth muscle tone in patients with mild asthma subjected to me

140 A complete suppression of muscle tone in the postural muscles and a reduction of m

141 e in the postural muscles and a reduction of muscle tone in the respiratory related musculature occur

142 tivation with urocortin III on airway smooth muscle tone in vitro and in an acute model of airway inf

143 s more tightly coupled to the maintenance of muscle tone in waking and its loss in REM sleep and cata

144 Simultaneous measures of behavior and neck muscle tone indicated that bouts of theta occurred predo

145 t rats cycle rapidly between periods of high muscle tone (indicative of wakefulness) and periods of a

146 s of spasticity (hyperreflexia, increases in muscle tone, involuntary muscle activity), but the long-

147 environment is maintained, as in waking, but muscle tone is lost, as in REM sleep.

148 with narcolepsy, is a waking state in which muscle tone is lost, as it is in REM sleep, while enviro

149 ections in maintaining balance, posture, and muscle tone, Materials and Methods All subjects provided

150 r mechanisms in addition to increased smooth muscle tone may contribute to the development of repetit

151 + waves, which are likely to underlie smooth muscle tone, mixing and propulsion, depend upon neural a

152 The primary endpoint was the change in muscle tone (Modified Ashworth Scale [MAS]) in the PTMG

153 They play an important role in muscle tone, motor coordination, respiration, and pain p

154 receptors has a pronounced effect on smooth muscle tone, mucosal electrolyte secretion, and the peri

155 ction, including regulation of airway smooth-muscle tone, mucus secretion from submucosal glands and

156 inate the different biomechanical sources of muscle tone (neural or visco-elastic components), and fi

157 hyperkinesia disorders and in motor control, muscle tone, posture, and cognitive processes.

158 e-angle instrument was developed to quantify muscle tone, providing two parameters: bias difference a

159 associated loci were also involved in smooth muscle tone regulation.

160 (CS), lung inflammation persists and smooth muscle tone remains elevated, despite ample amounts of n

161 oxinA injection in the upper limb muscles on muscle tone, spasticity, active movement, and function.

162 avily innervate neurons that maintain waking muscle tone such as those in the ventrolateral periaqued

163 in mediating motor activity and controlling muscle tone suggests that alterations in these structure

164 ucleus to determine whether the mechanism of muscle tone suppression differs in these nuclei as has b

165 be detected at the motoneuronal level during muscle tone suppression elicited by brainstem stimulatio

166 Spasticity is a disorder of muscle tone that is associated with lesions of the motor

167 tivation may therefore drive the increase in muscle tone that underlies expression of freezing behavi

168 aid to the agents that altered airway smooth muscle tone, their receptors, the signal transduction pa

169 on, including the support of enhanced smooth muscle tone, thrombosis, and smooth muscle proliferation

170 receptor interactions regulate airway smooth muscle tone through activation of guanine nucleotide bin

171 r endothelial cells regulate vascular smooth muscle tone through Ca2+-dependent production and releas

172 esses while modulating cortical activity and muscle tone to promote and maintain arousal along with l

173 t the visual feedback required to adjust eye muscle tone to realign the globes is eliminated.

174 daytime sleepiness and cataplexy, a loss of muscle tone triggered by emotional stimulation.

175 nal loop provides an enhancement of vibrissa muscle tone upon contact during active touch.

176 measured over a wide range of TP and smooth muscle tone using a new noninvasive ultrasound technique

177 s are an important contributor by modulating muscle tone using optogenetic and pharmacological tools,

178 ) has also been proposed to influence smooth muscle tone via activation of sGC.

179 ts in the supraspinal control of locomotion, muscle tone, waking, and REM sleep.

180 ) target brainstem regions known to regulate muscle tone, we hypothesized that these cells promote em

181 As a proxy for muscle tone, we measured changes in animal length under

182 ographic activity, eye movement, and somatic muscle tone were also placed.

183 utamatergic input has been found to modulate muscle tone, whereas cholinergic input has been found to

184 tive neurons that function to lower masseter muscle tone, whereas unilateral optogenetic activation o

185 t baseline and after the induction of smooth muscle tone with MCh, a DI distended the airways of heal

186 taplexy experience sudden losses of postural muscle tone without a corresponding loss of conscious aw