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1 ent states of cortical excitability (rest vs muscular contraction).
2 expressed, with functions not restricted to muscular contraction.
3 ck of a hydrostatic column and an absence of muscular contraction.
4 res that store energy from a single cycle of muscular contraction.
5 a step increase in workload at the onset of muscular contraction.
6 nerated by the actomyosin interaction during muscular contraction.
7 ncies of 25 to 30 Hz without neural input or muscular contraction.
8 nd transmits tension along myofibrils during muscular contraction.
9 s, playing a major role in the regulation of muscular contraction.
10 d by electrically silent, percussion-induced muscular contractions.
11 essential elements in neuronal signaling and muscular contraction and in humans have been causally re
12 ge, resulting in alterations in pelvic floor muscular contraction and low urethral pressure, resembli
14 otes to load the web over multiple cycles of muscular contraction and thus release more stored energy
15 t patients are learning effects, pain during muscular contraction, and alteration of consciousness.Th
25 activated in a redox-sensitive manner during muscular contraction, presumably due to increased oxidan
26 nventional NMES protocols to elicit repeated muscular contractions resulted in a significant decrease
27 or because any type of brief, yet frequent, muscular contraction throughout the day may be necessary
28 eons, fluorescence ratio changes accompanied muscular contraction, verifying detection of calcium tra
31 imb movements are generally driven by active muscular contractions working with and against passive f