1 identification, genetic-lineage tracing, and
mutant analyses.
2 These results were confirmed by
mutant analyses.
3 interspecies rescue experiments and further
mutant analyses.
4 med by single-mutant and compensatory double-
mutant analyses.
5 ich is apparent from reporter expression and
mutant analyses.
6 onsistent with previous predictions based on
mutant analyses.
7 ld disrupt size homeostasis, as confirmed by
mutant analyses.
8 Double
mutant analyses also demonstrate that ETT functions inde
9 Double
mutant analyses also reveal that IDN2 and three uncharac
10 Double-
mutant analyses also reveal that SIN2 shares functional
11 Mutant analyses and (13)C NMR studies also confirmed tha
12 Finally, the combination of
mutant analyses and characterization of P-body movement
13 with 3-hydroxypropionate, as demonstrated by
mutant analyses and enzyme activity measurements; (ii) t
14 Double
mutant analyses and expression studies show that althoug
15 NA binding and overexpression studies versus
mutant analyses and in vivo DNA binding, providing a cri
16 Systematic
mutant analyses and recent nervous system-wide expressio
17 Through transcriptome profiling,
mutant analyses and transgenic experiments, we aim to es
18 Through
mutant analyses and transgenic studies, we showed that t
19 A combination of promoter deletion
mutant analyses,
and the response of promoter mutants to
20 Double
mutant analyses between Ya and gnu suggest that YA plays
21 ediated, we used fluorescently tagged actin,
mutant analyses,
Ca(2+) imaging and controlled Ca(2+) re
22 Pharmacological and
mutant analyses confirmed the functional involvement of
23 Double-
mutant analyses demonstrate that Bif1 exhibits an epista
24 Double-
mutant analyses demonstrated that pif1phyA, pif1phyB, pi
25 Through a combination of loss-of-function
mutant analyses,
genetic mapping, and transgenic rescue
26 Mutant analyses have identified more than 10 positive co
27 lation of plant cell development, and double-
mutant analyses have revealed a complex genetic network
28 New
mutant analyses have revealed separable roles for Dap160
29 We report tissue-specific mouse
mutant analyses identifying the bone morphogenetic prote
30 Mutant analyses implicate ARP2 as an important subunit f
31 Point
mutant analyses in the two-hybrid system and in vitro sh
32 Genetic
mutant analyses indicate that alx3 functions to regulate
33 Interestingly, double, triple, and quadruple
mutant analyses indicate that individual SCAR genes vary
34 Double
mutant analyses indicate that tcl-2 can act synergistica
35 Mutant analyses indicate that the cardia bifida locus na
36 Double-
mutant analyses indicate that these pleiotropic cop/det/
37 Double
mutant analyses indicate that VosA and VelB play an inte
38 Mutant analyses indicated a correlation between dsRNA-bi
39 Additional
mutant analyses indicated that acs regulation was accomp
40 Mutant analyses indicated that endogenous ABA levels rep
41 Mutant analyses indicated that SSD-mediated mallostery o
42 Moreover, double
mutant analyses involving prp39a or smd1b and RNA qualit
43 ogenesis provides new insights for strategic
mutant analyses necessary to integrate the roles of tran
44 Here, we performed comprehensive
mutant analyses of angiopoietins and Tie receptors in ze
45 this study, comparative transcriptional and
mutant analyses of Desulfovibrio alaskensis strain G20 a
46 Double-
mutant analyses of era1 with the ABA-insensitive mutants
47 We present double
mutant analyses of eta2 axr1 plants indicating that libe
48 Expression and
mutant analyses of PIN1 members in the closely related c
49 Double-
mutant analyses of the ABA-hypersensitive signaling muta
50 ve binding ELISA and by alanine substitution
mutant analyses of the hIFNAR1-IgG.
51 Double-
mutant analyses of tsh4 and several highly branched muta
52 Together with transcriptomic and double-
mutant analyses,
our data revealed that the HUB1 interac
53 zed ApcI in Synechocystis sp. PCC 6803 using
mutant analyses,
PBS binding experiments, and protein in
54 Sox4
mutant analyses reveal a requirement for Sox4 in IPC spe
55 Double-
mutant analyses reveal that vem-1 genetically interacts
56 The 64
mutant analyses revealed all the genetic determinants re
57 Mutant analyses revealed that apical Baz accumulations c
58 Tissue-specific knockdowns and genetic
mutant analyses revealed that both Tachykinin and Tachyk
59 Mutant analyses revealed that cornichon genes regulate d
60 Double
mutant analyses revealed that I9H functioned redundantly
61 In the present study, double
mutant analyses revealed that only rhd1-4 hypersusceptib
62 Systematic double-
mutant analyses revealed the genetic network context in
63 Biochemical and
mutant analyses show that an active process drives micro
64 in the conserved Asp51 phosphorylation site,
mutant analyses show that cell-cycle-controlled CtrA pro
65 Our
mutant analyses show that Lis1/dynactin have at least tw
66 Double
mutant analyses show that the hypersensitive phenotype o
67 Deletion
mutant analyses show that the most proximal 852 kb of th
68 P factors in regulating these promoters, and
mutant analyses showed that ABI4 and these bZIPs share s
69 Double
mutant analyses showed that PHYTOALEXIN DEFICIENT4 (PAD4
70 Instead, double
mutant analyses showed that the phyB mutation suppressed
71 Mutant analyses showed the inhibitory function of DC-HIL
72 ogether with results from abh1/abi1-1 double-
mutant analyses suggest that abh1 shows enhanced sensiti
73 Furthermore, results of double-
mutant analyses suggest that Ark1p and Prk1p function in
74 Target gene expression and double-
mutant analyses suggest that C2cd3 is an essential regul
75 Lastly, double
mutant analyses suggest that ETT control of floral organ
76 Gene-expression profiles and
mutant analyses suggest that methanol is the dominant ca
77 In addition, double-
mutant analyses suggest that the gene products of claret
78 Mutant analyses suggest that the primary motors for mito
79 Genetic double
mutant analyses suggest that the SUR-7-mediated effect i
80 Double
mutant analyses suggest that tube-size control by septat
81 Mutant analyses suggested a requirement of the p-coumaro
82 Double-
mutant analyses suggested that both wild-type Smo and Sm
83 In zebrafish, previous lineage-tracing and
mutant analyses suggested that SHF ventricular and OFT p
84 Gene expression and
mutant analyses support a model in which DnaA and Rok co
85 Double
mutant analyses support a model where FLAIL-mediated spl
86 Double-
mutant analyses support a specific functional interactio
87 Mutant analyses supported this proposed model at a signi
88 model, including in silico and experimental
mutant analyses,
supports the importance of a reciprocal
89 This review summarizes the
mutant analyses that give insight into these patterning
90 As result of the
mutant analyses,
the antagonistic cytokinin and auxin si
91 e been implicated in xylan synthesis through
mutant analyses,
the biochemical mechanisms responsible
92 f ABA on sugar import, and transcriptome and
mutant analyses to identify genes associated with Glc av
93 oinformatics-driven hypothesis building with
mutant analyses to identify potential epigenetic mechani
94 equired for cell-to-cell trafficking through
mutant analyses using a zucchini yellow mosaic virus exp
95 suppressor gene functions that eluded single
mutant analyses,
using a Caenorhabditis elegans genome-w
96 single site-directed mutagenesis and double
mutant analyses,
we conduct a detailed analysis on the r
97 Through prospective fate mapping and
mutant analyses,
we define the derivatives of each subgr
98 n the activity of the recombinant enzyme and
mutant analyses,
we demonstrate its prominent role in th
99 attenuated SWP duration in Arabidopsis Using
mutant analyses,
we identified Arabidopsis H(+)-ATPase 1
100 in situ hybridization and double and triple
mutant analyses,
we showed that this enhanced defect was
101 Mutant analyses were consistent with predictions that Cy
102 Thus,
mutant analyses will require multi-generational studies.
103 Double
mutant analyses with dcl2-1, dcl3-1, and dcl4-2 alleles
104 ethylation (H3K9me2) levels at AtMu1c Double
mutant analyses with epigenetic silencing mutants sugges
105 Combining
mutant analyses with gain- and loss-of-function approach
106 By combining
mutant analyses with in vivo electrophysiological record
107 Double
mutant analyses with M. truncatula single leaflet1 (sgl1
108 Double-
mutant analyses with mutations in MUCILAGE MODIFIED2 and