1 dule and MphR ligand-binding module by using
mutant analysis.
2 acquired thermotolerance were identified by
mutant analysis.
3 ry cell wall synthesis, were investigated by
mutant analysis.
4 sequence motif identified here by a deletion
mutant analysis.
5 ther PLL2 or PLL3 based on single and double
mutant analysis.
6 ification, and differentiation has come from
mutant analysis.
7 in the germination process were confirmed by
mutant analysis.
8 meristem determinacy was revealed by double
mutant analysis.
9 cassettes for subsequent transformation and
mutant analysis.
10 ose synthase (SS) by immunoprecipitation and
mutant analysis.
11 protection, oligonucleotide competition, and
mutant analysis.
12 een shown to be critical for its function by
mutant analysis.
13 ription were identified through an extensive
mutant analysis.
14 ctivity by LEAFY, as was deduced from double
mutant analysis.
15 at amino acid position 291, as indicated by
mutant analysis.
16 the entire gene family in somatic tissues by
mutant analysis.
17 chypodium distachyon) using gene editing and
mutant analysis.
18 time points during development and through a
mutant analysis.
19 n) through substrate feeding experiments and
mutant analysis.
20 gen (HBsAg), HBeAg levels, HBV genotype, and
mutant analysis.
21 ng, is becoming a powerful tool for en-masse
mutant analysis.
22 racing, electrophysiology, pharmacology, and
mutant analysis.
23 wer than half have roles established through
mutant analysis.
24 saul1 phenotypes, as demonstrated by double
mutant analysis.
25 We used designer arrays for defined
mutant analysis,
a high-throughput subtractive competiti
26 The
mutant analysis also confirms the proposed role of Frh i
27 Mutant analysis also revealed interactions of ABA and LR
28 A loss-of-function
mutant analysis also revealed that single mutants of bHL
29 s an approximately 85% reduction in omega-7s
Mutant analysis also showed that FATTY ACID ELONGASE1 is
30 Crystal structures followed by
mutant analysis and affinity pull-downs have revealed th
31 Mutant analysis and asymmetric reconstructions show that
32 Using a combination of double
mutant analysis and biochemistry, we found that in maize
33 Strikingly, through null
mutant analysis and cell-specific rescue experiments, we
34 Furthermore, by promoter
mutant analysis and Chromatin immune precipitation assay
35 Double
mutant analysis and comparison of AP-3(-) and BLOC-1(-)
36 Molecular epistasis tests, double
mutant analysis and dosage-sensitive interactions demons
37 We tested a small set of these genes through
mutant analysis and found that one significantly increas
38 Reciprocal grafting, double
mutant analysis and gene cloning suggest that all MAX ge
39 ng a combination of in vitro explant assays,
mutant analysis and gene delivery into mouse embryos cul
40 Both the
mutant analysis and gene regulation studies suggest that
41 Using gene expression studies, genetic
mutant analysis and genetic mosaics, we show that egl-38
42 e CER2 homologs CER2-LIKE1 and CER2-LIKE2 by
mutant analysis and heterologous expression in yeast.
43 Quadruple
mutant analysis and in situ localization of A, B, C and
44 Using a combination of
mutant analysis and in vitro explant assays, we demonstr
45 studies, in vitro migration assays, genetic
mutant analysis and in vivo fate mapping in mice, we fou
46 Through
mutant analysis and in vivo imaging, we show that gjd4/C
47 Mutant analysis and live cell imaging indicate that PDS5
48 Mutant analysis and optogenetic studies reveal that GABA
49 igands at the midline, and we show by double
mutant analysis and physical interaction tests that WRK-
50 A combination of loss-of-function
mutant analysis and protein interaction data indicates t
51 al gene 20 product (gp20) were determined by
mutant analysis and sequence localization within the str
52 By means of deletion
mutant analysis and site-directed mutagenesis, we have i
53 teraction as previously determined by escape
mutant analysis and site-directed mutation, is located i
54 Mutant analysis and subsequent molecular studies have re
55 To confirm the results of the
mutant analysis and to determine the relative contributi
56 e used a combination of multiplexed imaging,
mutant analysis,
and gene network modelling to resolve t
57 Using global expression studies, double
mutant analysis,
and protein interaction assays, we find
58 nt systems have been addressed by a targeted
mutant analysis approach and almost all are shown to be
59 pylori stress resistance was evaluated by a
mutant analysis approach.
60 gene targets in CGNs using inhibitor and Nfi
mutant analysis as well as chromatin immunoprecipitation
61 A dimerization motif, an alanine scan double
mutant analysis at the helix-helix interface was carried
62 Based on double
mutant analysis,
AXL1 in a MATalpha strain acted genetic
63 Double-
mutant analysis between abi3-3 and wri1-1 suggested that
64 Double-
mutant analysis between mnp-1(RNAi), ina-1, and vab-1 mu
65 Double-
mutant analysis between scraps, a mutation in anillin th
66 ion observed in mig-10 mutants, while double
mutant analysis between unc-53 and mig-10 showed no incr
67 , we previously used the multistep method of
mutant analysis by PCR and enzyme cleavage (MAPREC).
68 g of oral poliovirus vaccine with the use of
mutant analysis by PCR and restriction enzyme cleavage (
69 Mutant analysis by PCR and restriction enzyme cleavage (
70 Direct RNA extraction and
mutant analysis by polymerase chain reaction and restric
71 Inr consensus sequence based on an extensive
mutant analysis carried out in HeLa cell extracts.
72 Mutant analysis confirms that affinity proteomics is a v
73 Mutant analysis confirms that Asp-97 and Glu-108 are pro
74 However,
mutant analysis correlated specific HMW2 domains with co
75 The
mutant analysis,
coupled with physiological data, indica
76 ular localization in response to Shh, double
mutant analysis demonstrates that Rab23 does not work th
77 Mutant analysis demonstrates that Vac8p functions separa
78 Deletion
mutant analysis determined that the N- and C-termini are
79 Deletion
mutant analysis determines that there is a difference in
80 -mapping studies using neutralization escape
mutant analysis,
deuterium exchange mass spectrometry, a
81 ed subtly elevated levels of lesions, double
mutant analysis disagreed with a simple epistatic model
82 Mutant analysis established residues that were involved
83 tro findings to in vivo biological function,
mutants analysis establishes the role of Shr in GAS grow
84 The
mutant analysis further allows us to propose that a subs
85 To do this we used a global multi-
mutant analysis (
GMMA) approach, which can identify subs
86 Mutant analysis has defined two parallel genetic pathway
87 Mutant analysis has identified mycoplasma proteins assoc
88 Although deletion
mutant analysis has suggested that the region of the gen
89 The
mutant analysis highlights some important motifs for sub
90 chloroplast biogenesis have been obtained by
mutant analysis;
however, in C(4) plants a relevant muta
91 enzymes in H. pylori have been studied using
mutant analysis;
however, the gene encoding adenosine de
92 r downstream of ETR1 and ERS based on double
mutant analysis;
however, the signaling mechanisms leadi
93 ading to the epidermis in L. japonicus While
mutant analysis identified redundancy in several biosynt
94 ter activity in lymphoid cells, and deletion
mutant analysis identified three distinct domains of TEL
95 Mutant analysis in Arabidopsis has indicated that absenc
96 By combining physical perturbations and
mutant analysis in both species, we show that difference
97 Mutant analysis in Caenorhabditis elegans reveals that c
98 eterochrony was first established by genetic
mutant analysis in the nematode C. elegans, revealing a
99 Recent
mutant analysis in zebrafish points to an important role
100 e-cell transcriptomics, lineage tracing, and
mutant analysis in zebrafish, we uncover key development
101 Results from deletion and
mutant analysis indicate that Pak1 regulates cyclin D1 t
102 Double-
mutant analysis indicated that blocking the salicylic ac
103 Double
mutant analysis indicated that ERS2, similar to ETR1, ET
104 In addition, double-
mutant analysis indicated that gamma-tubulin-dependent n
105 ATR
mutant analysis indicated that it is required for checkp
106 A
mutant analysis indicated that the Ino2p/Ino4p/Opi1p reg
107 Double
mutant analysis indicated that the SOS genes function in
108 Double
mutant analysis indicated that the thk1 mutant is epista
109 Double
mutant analysis indicated that the two auxin transport s
110 Mutant analysis indicates a progressive loss in the amph
111 Mutant analysis indicates a role for PIAL1 and 2 in salt
112 Double
mutant analysis indicates an interaction energy between
113 Double-
mutant analysis indicates that "clean" DNA ends caused b
114 Double-
mutant analysis indicates that a related helix-loop-heli
115 Double-
mutant analysis indicates that BK and Erg K(+) channels
116 Double-
mutant analysis indicates that both sag-1 and eat-16 act
117 termediate columns of the neuroectoderm, and
mutant analysis indicates that Dichaete regulates cell f
118 Double-
mutant analysis indicates that early flowering is depend
119 Double
mutant analysis indicates that ETR2 acts upstream of CTR
120 Double
mutant analysis indicates that Gli3 repressor activity i
121 Essential for viability, plx
mutant analysis indicates that larval death is attributa
122 Mutant analysis indicates that LjVPY1 and LjVPY2 are req
123 Double
mutant analysis indicates that phyB is epistatic to hrb1
124 uces increases in the abundance of PIF3, and
mutant analysis indicates that PIF3 acts, in conjunction
125 r the S/M or G2/M checkpoint, however double
mutant analysis indicates that rad31 acts in a process w
126 Genetically, sos2sos3 double
mutant analysis indicates that SOS2 and SOS3 function in
127 Mutant analysis indicates that the cytokinin receptors A
128 Deletion
mutant analysis indicates that the juxtamembrane region
129 Mutant analysis indicates that the paralogues have share
130 Double
mutant analysis indicates that the primary target of Rab
131 In a comprehensive
mutant analysis involving single and multiple mutants of
132 Genetic double-
mutant analysis is consistent with ptp-3A acting with th
133 Double
mutant analysis is consistent with these results, showin
134 Using
mutant analysis,
laser ablation, optogenetics, and Ca2+
135 depressor, we used YFP:actin to monitor, and
mutant analysis,
laser-ablation and transgenic feminizat
136 at integrates multiple approaches, including
mutant analysis,
lineage tracing, cell purification, gen
137 A refined
mutant analysis localized this element to nucleotides -8
138 Based on deletion
mutant analysis,
MdmX inhibition of Smad transactivation
139 grative strategy including enzymatic assays,
mutant analysis,
metabolic engineering, isotope labeling
140 anridins." Data from transgenic experiments,
mutant analysis,
metabolic profiling, and phylogenetic a
141 stion and have shown by microarray analysis,
mutant analysis,
metabolite measurements, and (13)C-labe
142 res, we propose how the information from the
mutant analysis might facilitate the use of a simplified
143 This work provides the first
mutant analysis of a GYF-domain protein in either C. ele
144 tructure of the AdpA-DBD-DNA complex and the
mutant analysis of AdpA-DBD revealed its unique manner o
145 Double
mutant analysis of Atrar1 in combination with the R sign
146 Genetic evidence from double-
mutant analysis of cow1-1 and other loci involved in roo
147 Double
mutant analysis of dlf1 and indeterminate1 (id1), anothe
148 ilable terminal fate markers, we undertake a
mutant analysis of five homeobox genes (unc-30/Pitx, unc
149 Double
mutant analysis of hos5-1 and the ABA-deficient aba1-1 a
150 Mutant analysis of induced plant defense pathways showed
151 Double-
mutant analysis of lls1 with two maize mutants oil-yello
152 Mutant analysis of plants deficient in any of three in-f
153 Double-
mutant analysis of polar residues in the distal beta-hai
154 Surprisingly, a detailed substitution
mutant analysis of the amino-terminal domain revealed a
155 Here, we report a detailed
mutant analysis of the D' element which suggests that an
156 Deletion
mutant analysis of the LTR of a PERV-NIH isolate identif
157 Although phenotypic
mutant analysis of the wssFGHI genes has previously show
158 Finally, double
mutant analysis of unc-71 with other axon guidance signa
159 Deltahns, DeltarpoS, and Deltahns DeltarpoS
mutants, analysis of RpoS reporter fusions, quantitative
160 otein 70 (Hsp70) family, in conjunction with
mutant analysis,
permitted the characterization of a mot
161 Mutant analysis,
pharmacology and patch-clamp recording
162 Through
mutant analysis,
protein depletion and rescue experiment
163 We used
mutant analysis,
protein interaction and ubiquitylation
164 In this issue, Oda et al. use
mutant analysis,
protein tagging, and cryoelectron tomog
165 Here, we used
mutant analysis,
protein-protein interaction assays and
166 high-resolution Ca(2+) imaging and stomatal
mutant analysis reveal that [Ca(2+)](cyt) increases inst
167 Transplantation experiments and
mutant analysis reveal that cephalic mesoderm is the sou
168 Deletion and site-specific
mutant analysis revealed a critical role of a potential
169 Mutant analysis revealed a new relationship between the
170 In addition, double
mutant analysis revealed epistasis between EDM2 and the
171 Mutant analysis revealed extensive ligand redundancy in
172 A comprehensive
mutant analysis revealed only one element upstream of th
173 Mutant analysis revealed that a variable residue within
174 Double
mutant analysis revealed that all edr1-associated phenot
175 Deletion
mutant analysis revealed that both the kinase domain and
176 Mutant analysis revealed that complement-protective CD55
177 Double-
mutant analysis revealed that edr2-mediated resistance i
178 Double
mutant analysis revealed that espA was epistatic to pktA
179 Mutant analysis revealed that GndA and FDH4 are crucial
180 Mutant analysis revealed that loss of ChOMT1 strongly re
181 Double
mutant analysis revealed that SmD3b is also involved in
182 The stf lfl double
mutant analysis revealed that STF and LFL act mainly ind
183 Mutant analysis revealed that the C-terminal region of C
184 Double
mutant analysis revealed that the coi1 mutation (causing
185 Deletion
mutant analysis revealed that the complement control pro
186 Double
mutant analysis revealed that the nlp7-1 phenotype depen
187 Mutant analysis revealed that the protective function of
188 Mutant analysis revealed that the single chemotaxis syst
189 Double
mutant analysis revealed that these drought-induced phen
190 Biological profiling and
mutant analysis revealed that this compound is a prodrug
191 Mutant analysis revealed that yKu70 and Sir1 act collect
192 induce transcription of a reporter, deletion
mutant analysis revealed the presence of a strong activa
193 This
mutant analysis revealed the presence of a third glycosy
194 titration of purified proteins combined with
mutant analysis revealed the roles of the residues in th
195 Furthermore, MFSD2A
mutant analysis reveals an important function of the C t
196 Double
mutant analysis reveals an unexpected, redundant negativ
197 spatially stereotyped in wild-type animals,
mutant analysis reveals that each cell has the potential
198 wun wun2 double
mutant analysis reveals that the two genes, hereafter co
199 Deletion
mutant analysis reveals that WASP residues 101-151 are n
200 Tryptic phosphopeptide and
mutant analysis reveals that, as in mitosis, stress-indu
201 Our
mutant analysis reveals the contribution of mechanical e
202 AHF-Cre genetic tracing experiments and Tbx1
mutant analysis show that nonsomitic neck muscles share
203 Deletion and point
mutant analysis show that the activity of TNRC4 on tau E
204 Mutant analysis showed that AAD2 also contributes to ome
205 formation depends on ZCF membrane curvature:
mutant analysis showed that Cdc42p localization is negat
206 Explant essays and
mutant analysis showed that cellular guidance involved r
207 Mutant analysis showed that crdA cells were delayed in d
208 Double
mutant analysis showed that dopA interacts genetically w
209 genes involved in glycogen homeostasis, and
mutant analysis showed that Gcn4p suppresses glycogen le
210 Mutant analysis showed that HDA19/HD1 mediated deacetyla
211 Double
mutant analysis showed that only re rer1 and rer5 rer6 e
212 Mutant analysis showed that the glutamate residue corres
213 Mutant analysis showed that the RsbT kinase, which is re
214 involved in this response were identified by
mutant analysis,
showing that the EARLY FLOWERING 4 gene
215 Mutant analysis shows stationary flow patterns depend on
216 Phospho-
mutant analysis shows that Aurora contributes to the mic
217 Single
mutant analysis shows that both mrpl40 and prodha mutant
218 Double- and triple-
mutant analysis shows that DRT111 controls splicing of A
219 Our
mutant analysis shows that eliminating UNC-34 function r
220 Mutant analysis shows that RAD51B is essential for the m
221 Double
mutant analysis shows that trio interacts with Rac in a
222 In addition, our homeotic
mutant analysis shows that wing transformation in T1 ori
223 Double
mutant analysis shows that zig-3 and zig-4 act together
224 Our double-
mutant analysis shows that, in contrast to Zw3, Nkd acts
225 Precursor metabolite incorporation and
mutant analysis studies support the mode-of-action, bloc
226 Mutant analysis substantiates this idea.
227 heterozygous genetic interactions and double
mutant analysis suggest that 18W affects the Rho-GTPase-
228 Deletion
mutant analysis suggested a requirement of RopGEF activi
229 Binding studies in vitro and
mutant analysis suggested that GFP-PH bound PtdIns(4,5)P
230 not been demonstrated in vitro, but previous
mutant analysis suggested that Rhizobium etli gene wreQ
231 Mutant analysis suggested the role of the cysteine prote
232 Double
mutant analysis suggests a possible inaccessibility of e
233 In addition,
mutant analysis suggests a possible novel role for phyC
234 Double
mutant analysis suggests that ETTIN interacts with TSL,
235 Double
mutant analysis suggests that HST acts in parallel to SQ
236 Double
mutant analysis suggests that pgp-2(+) functions in para
237 Double
mutant analysis suggests that suppression of protein abn
238 Double
mutant analysis suggests that sur-6 PP2A-B acts downstre
239 iated with reduced cell wall biogenesis, and
mutant analysis suggests that this downregulation facili
240 Mutant analysis suggests that VE accumulation in these f
241 ., over hundreds of micrometers), and double
mutant analysis supports that FP-netrin1 and Shh collabo
242 Mutant analysis supports the assignment of the primary i
243 Mutant analysis supports the role of a candidate encodin
244 Here, we show through systematic
mutant analysis that GA20ox1, -2, and -3 are the dominan
245 We present double
mutant analysis that indicates that daf-28(sa191) acts a
246 he aid of genetic engineering and subsequent
mutant analysis,
the functional role of conserved cystei
247 we have developed an approach termed triple-
mutant analysis (
TMA).
248 We used double
mutant analysis to determine the relative positions of t
249 e we have used quantitative live imaging and
mutant analysis to determine whether similar mechanisms
250 ns, in vitro biochemical assays, and in vivo
mutant analysis to investigate the roles of these region
251 FRUITFULL, which we show through comparative
mutant analysis to modulate fruit shape during post-fert
252 We have used double
mutant analysis to position mei-217 in the meiotic recom
253 Through single- and double-
mutant analysis to study the mitotic cohesion proteins S
254 We used mouse in utero electroporation and
mutant analysis to test whether cortical signaling sourc
255 Mutant analysis was performed to study the role of MtHEX
256 tinin (Pta), a surface-associated cytotoxin,
mutant analysis was used in conjunction with a mouse mod
257 Mutant analysis was used to identify Moraxella catarrhal
258 Mutant analysis was used to prove that the HupA protein
259 to be composed of PFs by cross-sectional and
mutant analysis,
was found to extend along the entire le
260 Using fate mapping and
mutant analysis,
we find that PAA progenitors are derive
261 Using antibody labeling and
mutant analysis,
we have localized 12 of 13 core APC/C c
262 Based on our
mutant analysis,
we revised the rec27 open reading frame
263 Through phenotypic and double
mutant analysis,
we show that AGO1 regulates stem cell f
264 By conditional Shh
mutant analysis,
we show that Shh signaling regulates hC
265 We also report an escape
mutant analysis,
which allows the mapping of heterotypic
266 ure focused physiological studies, including
mutant analysis,
which will provide further details into
267 Double-
mutant analysis with anther ear1 and tassel seed2 reveal
268 Double-
mutant analysis with atr1D, an overexpression allele of
269 Here, we combined gene deletion
mutant analysis with deep-learning protein folding using
270 Double
mutant analysis with mutations in other genes affecting
271 Mutant analysis with potential upstream genes was used t
272 Double-
mutant analysis with suppressors and enhancers of lin-12
273 We performed
mutant analysis with the null alleles of ABP1, abp1-c1 a
274 Double-
mutant analysis with the peroxisomal ATP-binding cassett
275 Double
mutant analysis with this Hoxb1(3'RARE) allele and other