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1 l adhesin ApiA, was not affected in the same mutant background.
2 during heterocyst differentiation in a sigE mutant background.
3 ng cells is strongly enhanced in a DeltatipN mutant background.
4 ficient to mediate the Hth function in the L mutant background.
5 io protein is greatly diminished in the seh1 mutant background.
6 d localization of MRP observed in an ECA3265 mutant background.
7 genes either in the wild-type or in the rsp mutant background.
8 umulation of hydrogen peroxide in the dgs1-1 mutant background.
9 onto the LAT knock-in Bam32 knockout double-mutant background.
10 r body formation in the porB-1 porC-1 double mutant background.
11 hitecture, and motility inhibition in a sinR mutant background.
12 protein (IsdA) were most abundant in the agr mutant background.
13 from tissue-specific promoters in an Atsuc2 mutant background.
14 cent proteins of three colors in the quartet mutant background.
15 hs on the adaxial side of the leaf in an as1 mutant background.
16 a-4, rescued the rat phenotype in the impa-4 mutant background.
17 ible RNAi silencing of AtLCB2b in an Atlcb2a mutant background.
18 ent species and in the SF3B1 K700E oncogenic mutant background.
19 inhibit rhabdomeral degeneration in the tadr mutant background.
20 induce aneuploidy and tumorigenesis in a p53 mutant background.
21 ent, are largely lethal in a PINK1 or parkin mutant background.
22 ll-death defects in a genetically sensitized mutant background.
23 ere required to accumulate NifB-co in a nifN mutant background.
24 positive regulator in the tga5-1 tga6-1 null mutant background.
25 cry1 subcellular localization in a cry1-null mutant background.
26 oteins of three different colors in the qrt1 mutant background.
27 oxyR plasmid was absent in a type I fimbrial mutant background.
28 1 during S phase, even in a sensitized ddb-1 mutant background.
29 per facial nerve formation even in the Hoxb1 mutant background.
30 ation during early development in the agb1-2 mutant background.
31 a signal that is not present in the spoIIIE mutant background.
32 enhance the late-flowering phenotype in a co mutant background.
33 and sensitivity to cytotoxic agents in a dam mutant background.
34 g of 35S-NPTII but not RD29A-LUC in the ros1 mutant background.
35 as observed in E7 mice on the Rb(DeltaLXCXE) mutant background.
36 s not able to rescue AR activity in the TIF2 mutant background.
37 mal interaction differently depending on the mutant background.
38 lt in retinoblastoma formation even in a p53-mutant background.
39 ppresses Trp gene dysregulation in a cyp83B1 mutant background.
40 FRI to increase FLC mRNA levels in the abh1 mutant background.
41 that flk-1 signaling is up-regulated in the mutant background.
42 rly embryonic lethal defects in a Disp1-null mutant background.
43 length SMN are also stabilized in the degron mutant background.
44 escue of male viability in a roX1(-) roX2(-) mutant background.
45 rabidopsis carrying 35S::VP1 in an abi3 null mutant background.
46 ives totally glabrous plants only in the gl3 mutant background.
47 a2p, and Pea2p) are essential in a cla4delta mutant background.
48 anscription appears relatively robust in the mutant background.
49 onsistent with a cell division defect in the mutant background.
50 ene could be methylated de novo in the suvh4 mutant background.
51 nes expressing pri-miR163 gene in the mir163 mutant background.
52 Hoxd11 are present in the Hoxa11 homozygous mutant background.
53 hanced defence that is suppressed in a myb30 mutant background.
54 strongly reduced in the jasmonate resistant1 mutant background.
55 nterferes with granule initiation in the ss4 mutant background.
56 stitutive activation of the response in this mutant background.
57 screen was carried out in the bir1-1 pad4-1 mutant background.
58 ession of a COI1-YFP transgene in the coi1-1 mutant background.
59 n bud outgrowth in a strigolactone-deficient mutant background.
60 s further impaired in the C-terminal Hay/XPB mutant background.
61 lism modulated rhabdomere growth in a crumbs mutant background.
62 domains of DEK1 using gene targeting in null mutant background.
63 channel-dependent behaviors in a slo-1-null mutant background.
64 n ABA signaling was alleviated in the ios1-1 mutant background.
65 localization appears near normal in a spe-29 mutant background.
66 ient to suppress its splicing defects in the mutant background.
67 ll caused cortex proliferation in the erecta mutant background.
68 ory action of CLE3 was abrogated in the clv1 mutant background.
69 ized and show increased activities in a upl3 mutant background.
70 e photorespiratory genes was observed in the mutant background.
71 steady-state photosynthesis and in the pgr5 mutant background.
72 se from 16 to 32 mug/ml in an embB codon 306 mutant background.
73 e Dictyostelium protein and expressed in the mutant background.
74 modulation of the RRP is blocked in the rim mutant background.
75 tomatal opening mediated by PHOT1 in a phot2 mutant background.
76 lerate the depletion of MvaT in an DeltamvaU mutant background.
77 1 expression and nodule formation in an ern1 mutant background.
78 ctional 35S::NRT2.1 transgene in an atnrt2.1 mutant background.
79 e the wake-promoting effects of DA in a DopR mutant background.
80 be used to image Pax2 expressing cells in a mutant background.
81 ressor of pathogenesis-related genes1 (npr1) mutant backgrounds.
82 in phytochrome (phy) and cryptochrome (cry) mutant backgrounds.
83 stablishment in a series of dot1 and histone mutant backgrounds.
84 expression and reduced proliferation in both mutant backgrounds.
85 effect on genome stability in wild-type and mutant backgrounds.
86 o promote epidermal differentiation in these mutant backgrounds.
87 ent developmental time points and in various mutant backgrounds.
88 ed maintenance of RPS methylation in various mutant backgrounds.
89 examined their expression in each of the QS mutant backgrounds.
90 he ROS1 down-regulation that occurs in these mutant backgrounds.
91 found in mice with different Cited2 and Wt1 mutant backgrounds.
92 r complex, becomes essential in mcs6 or pmh1 mutant backgrounds.
93 OSM-9 motility is disrupted in certain IFT mutant backgrounds.
94 ct formation in leafy, apetala1 and apetala2 mutant backgrounds.
95 ly flowering in wild-type and late-flowering-mutant backgrounds.
96 mice were crossed onto Ink4a/Arf and/or p53 mutant backgrounds.
97 by introducing a loss of her7 function into mutant backgrounds.
98 and carotenoid accumulation in wild-type and mutant backgrounds.
99 gregation in both the wild-type and the pilA mutant backgrounds.
100 cistronic transcripts in enhanced RNAi (Eri) mutant backgrounds.
101 both chd1Delta and, as has been shown, K123A mutant backgrounds.
102 n both large T (63 mutants) and middle T (51 mutants) backgrounds.
104 stil elongation and stigma exsertion in this mutant background, a process that requires SRK catalytic
105 r, Scnn1a knockout in a degeneration-causing mutant background accelerated motoneuron loss and diseas
106 -Activated Protein Kinase 6 (MPK6) in a mpk3 mutant background all have abscission-defective phenotyp
108 ns of gene expression in different zebrafish mutant backgrounds allow further quantitative evaluation
110 g wve-1 levels in either unc-34/ena or wsp-1 mutant backgrounds also leads to a significant enhanceme
111 A levels of Skn7-dependent genes in the fcp1 mutant background and also increased Skn7 protein levels
112 (upstream activating sequence)-ben in a ben mutant background and identified a well defined critical
113 introduced the HG transgene onto a Gbx2-null mutant background and recreated a new Otx2/Gbx2 border i
115 porter-gene activity increased in a u-shaped mutant background and that forced expression of SerpentN
116 Despite of this striking effect in the spi mutant background and the expression of EcR signaling co
117 alize the anatomy of specific neurons in Lhx mutant backgrounds and find that the development of the
118 expression in a subset of autonomous-pathway-mutant backgrounds and functions both to promote activat
119 ly different activities in the sinI and expR mutant backgrounds and in response to added SinI AHLs.
121 lle and Twist were introduced into different mutant backgrounds and the patterning activities were vi
122 s were expressed in both secA2 wild-type and mutant backgrounds and were tested for their ability to
123 lly to hth, (ii) Hth is upregulated in the L mutant background, and (iii) MH domain of Hth is require
124 ted with chromosomal translocations in a p53 mutant background, and heterozygosity for the mutant all
125 enced the candidate gene, NCU02713.3, in the mutant background, and phenocopied the mutation by gene
126 thermore, STR2 is haplo-insufficient in str1 mutant background, and str1(-/-)/str2(+/-) embryos were
127 acnA mutations were introduced into an rpiRc mutant background, and the effects on RNAIII were determ
129 ed in bacteria, but primarily in specialized mutant backgrounds, and the factors responsible for DNA
131 that heat shock-induced puffs in JIL-1 null mutant backgrounds are strongly labeled by antibody to t
133 Etv loss-of-function alleles into the Rfwd2 mutant background attenuated the branching phenotype, su
134 ented by any of the lspA(Mx) genes in an lpp mutant background, but not in an E. coli lpp(+) backgrou
135 icacy of genotoxic cancer therapies in a p53 mutant background by eliminating one of the checkpoints
136 d, after elimination of CESA redundancy in a mutant background, by coimmunoprecipitation and mass spe
137 pression of hDAT-L368Q in the Drosophila DAT mutant background caused developmental lethality, indica
138 show that disruption of PDAT1 in the tgd1-1 mutant background causes serious growth retardation, gam
142 PR gene expression, is abolished in a sid2-1 mutant background, consistent with a requirement for sal
143 this purpose, ETR1 levels were quantified in mutant backgrounds containing receptor loss-of-function
144 levels are reduced in the dcr-1(ok247) null mutant background; conversely, a reduction of DCR-1 prot
145 55A and M377V, made in the active FAR8-S363P mutant background converted its specificity from 16:0-Co
146 specific restoration of Tbx1 expression in a mutant background corrected most of those defects in the
148 ss on rop6(DN) plants, which was retained in mutant backgrounds defective in SA biosynthesis or signa
149 iated response in rop6(DN) was suppressed in mutant backgrounds defective in SA signaling (nonexpress
150 losis was lost in DO11.10 mice bred in a rag mutant background, demonstrating that the immune respons
151 on also occurs in the rps2, ndr1, and Atrar1 mutant backgrounds, demonstrating that this activity can
153 Inactivation of the p53 gene in the Mdm2 mutant background effectively reversed the lung size phe
154 red when MET1 is re-introduced into the met1 mutant background, either via genetic cross or DNA trans
158 converted into carpelloid organs in certain mutant backgrounds even in the absence of AG activity in
159 nic males carrying this construct in a mab-9 mutant background exhibit tail abnormalities including m
160 m is preserved across Drosophila species and mutant backgrounds exhibiting a range of eggshell phenot
161 rammed DNA double-strand breaks in a spo11-1 mutant background failed to rescue the DNA fragmentation
162 d VE-cadherin(+) vascular cells in the flt-1 mutant background first occurs between day 5 and day 6.
163 Our findings question the prudence of using mutant backgrounds for genetic screens and underscore a
165 dition, disruption of ihfA in an hns or toxT mutant background had no effect on tcpA expression.
168 sed epistasis experiments in an atonal (ato) mutant background, identified 188 genes induced by ey.
169 had shown that removal of Pax6 from the Gsh2 mutant background improves the molecular identity of the
170 inbred ddm1 (decrease in DNA methylation 1) mutant background in which various genetic and epigeneti
172 DNA looping in a variety of operator and CI mutant backgrounds in vivo, and our methodology can be a
173 eased the frequency of rootless seedlings in mutant backgrounds in which auxin regulation of basal po
174 n this study, we have combined live imaging, mutant backgrounds in which fertilization can be control
175 n (Psyr_1620-1616, paoABCDE), only in a pdhQ mutant background, in addition to pyruvate, are herein a
176 roxylase 1/beta-hydroxylase 2 (b1 b2) double-mutant background, in which both Arabidopsis beta-hydrox
177 in meiotic entry, as is exhibited in certain mutant backgrounds, inappropriately juxtaposes undiffere
178 that the taa mutants in several known auxin mutant backgrounds, including pid and npy1, mimic all of
179 phyA' in the DNA methyl transferase I (met1) mutant background increased PHYA expression and restored
180 ind that Rim20-GFP foci accumulate in a vps4 mutant background independently of external pH, Rim101 p
181 protein-protein interactions in a variety of mutant backgrounds indicate that substrates are probably
182 ESL and dnaKJ operons transformed into these mutant backgrounds indicated that sig1 is required for t
183 ffects of ethylene and auxin in a variety of mutant backgrounds indicates that auxin biosynthesis, tr
184 to restore Syt1 function in a syt1(-/-) null mutant background, indicating both C2A and C2B are speci
185 ational repression were impaired in the AGO3 mutant background, indicating that AGO3 can mediate both
186 b-1 mutant lethality was enhanced in a pyr-1 mutant background, indicating that HSPG synthesis is ver
187 background, whereas it persisted in the sspA mutant background, indicating that SspA/Lpa mediate the
188 anced and blue shifted in the Cys154 --> Gly mutant background, indicating that the latter mutation c
191 of S motility in the mglA-8 masK-815 double mutant background is different than that resulting from
193 n that the upregulation of FLC in FRI- or AP-mutant backgrounds is correlated to an increase in histo
194 oxylase observed in both the daf-2 and unc-2 mutant backgrounds is suppressible either genetically by
197 is abrogated in the mrt-2, hus-1, and clk-2 mutant backgrounds, lack of the apoptotic branch of the
198 s observed only for attractant addition in a mutant background lacking the coupling proteins, CheW an
199 ts and, importantly, deleting Yap in the Nf2 mutant background largely restores hippocampal developme
200 th and ethylene responsiveness in the double-mutant background, leading to the conclusion that canoni
201 in PHB development, loss of egl-5 in a ham-1 mutant background leads to PHB differentiation defects.
202 into a 'sensitised' zygotic headless (tcf3) mutant background leads to severe forebrain and eye defe
203 n by RNAi of ats1-1 RNA levels in the ats1-1 mutant background led to a more severe growth phenotype
204 t of these cells (the leaf margin) in a dwf4 mutant background led to both restoration of differentia
207 ion of this RGS1 protein variant in the rgs1 mutant background makes plants hypersensitive to a subse
209 oss of KANADI activity in a Class III HD-Zip mutant background mitigates the defects in bilateral sym
210 l function in ovule development: in the MPK6 mutant background, MPK3 is haplo-insufficient, giving fe
212 which can be reversed upon transfer into the mutant backgrounds of decrease in DNA methylation 1 (ddm
213 auer arrest: it forms dauers in the deletion mutant backgrounds of ncr-1 or daf-28/insulin; as a sing
216 l types using the GAL4/UAS system in an amon mutant background partially rescues larval molting and g
217 f a single mutant allele of Nodal in the Tet mutant background partially restored patterning, suggest
218 he pathway was reduced by RNAi in the ats1-1 mutant background, phosphatidylglycerol amounts decrease
221 application and the ssi2 mutation in various mutant backgrounds produce similar effects and that rest
222 CeTRAP240/let-19(RNAi) in a CeTRAP230/dpy-22 mutant background produced a strong synthetic lethal phe
223 e important for cell elongation, in the ugtP mutant background produced cells with severe shape defec
227 ic ftz cis-regulatory element (CRE) in a ftz mutant background rescued for segmentation defects.
229 ion of AtBI1 transgene in the two homozygous mutant backgrounds rescued the accelerated cell death ph
230 c restoration of Gata3 function in the Gata3 mutant background rescues the expression phenotypes of t
232 pression of Shaw(OX) and NKCC(RNAi) in a qsm mutant background restored LL-induced behavioral arrhyth
234 Overexpression of this gene in the rcp1 mutant background restores carotenoid production and, un
235 Deletion of sloR or sdhB in the DeltaserR mutant background restores growth to wild-type levels, s
237 ld-type, an equivalent loss in the fshr-1(0) mutant background resulted in a highly penetrant germlin
238 Mutation of this gene in the triple-FDH mutant background resulted in a methanol-negative phenot
240 Deletion of flhA in a wild-type or luxS mutant background resulted in identical loss of motility
241 C1, using artificial microRNA in the exo70C2 mutant background, resulted in a complete pollen-specifi
242 gene, Bmpr1b, in the retina-specific Bmpr1a mutant background, results in abnormal retinal dorsovent
243 or 35S::AtSCS-B-c-myc in the scs-1 knockout mutant background revealed that, in planta, both forms a
244 GPA1(S52C) or AtGPA1(Q222L) in the gpa1-null mutant background revealed various mutant phenotypes tha
245 e (Pol) II distribution in single and double mutant backgrounds revealed that Swi6 and Chp2 proteins
246 alysis between the tumors in the various E2F-mutant backgrounds revealed that there was extensive com
247 mal promoter was placed in a scarecrow (scr) mutant background, revealing a possible role for SCR in
248 ity and female-specific lethality in a piwi2 mutant background, revealing the zygotic function of piw
249 ngly, ruvC mutants, in both recA(+) and recA mutant backgrounds, scored as hyperrecombinational.
250 cessive ppd mutants on a spring-flowering hr mutant background show early, photoperiod-insensitive fl
251 scopy of the P(nifHD)-gfp reporter in a sigE mutant background showed delayed development and undetec
252 -of-function 14-3-3 lambda mutant in a phot1 mutant background showed that the 14-3-3 lambda protein
253 ression of regulatory genes in wild-type and mutant backgrounds showed that RspR controls transcripti
254 Mating these mutant mice to various gene mutant backgrounds showed that the mouse disease phenoty
255 Assay of the FUS1-lacZ reporter (in a hog1 mutant background) showed that sho1 and msb2 mutations b
256 n a feedback-insensitive threonine deaminase mutant background, shows that these two enzymes have ove
257 of one allele of Pax6 on the homozygous Tlx mutant background significantly worsens the phenotype.
259 dev promoter activity in wild-type and devS mutant backgrounds strongly suggest that upstream and do
260 ts is abolished in the ssi2 act1 coi1 triple-mutant background, suggesting that both JA- and act1-gen
261 phenotype of dao is suppressed in a seizure mutant background, suggesting that Dao acts by an effect
262 ly restores tripartite SC structure in a hal mutant background, suggesting that the defect in pairwis
263 ependent and rates are not altered in a CUL1 mutant background, suggesting that this ARF is targeted
264 ffects were lost or altered in patS and hetN mutant backgrounds, supporting a role of SepJ in the int
266 r screen in a gastrulation-sensitized double-mutant background, targeting genes likely to be expresse
268 sis and are hyperactive PTI suppressors in a mutant background that lacks the cell death response.
269 erwise isogenic background, as well as a nod mutant background that primarily undergoes loss of chrom
271 al of 72 tested, we identify 6 miRNA allelic mutant backgrounds that modulate the survival response t
278 d overexpression in a desiccation-intolerant mutant background to play an important role in seed DT.
279 nts were generated in either of the permease mutant backgrounds to identify the ATPase(s) interacting
280 s9 activity in heterozygous loss-of-function mutant backgrounds, to rapidly evaluate the phenotypic i
281 nd phyC deficiency had no effect in the phyA mutant background under FRc, suggesting that phyC does n
282 evels of HFR1 protein accumulate in the spa1 mutant background under various light conditions, includ
283 d score a double interval in a wild-type and mutant background using this protocol will take 22-27 we
284 reen for synthetic lethals in an unc-34 null mutant background utilizing an RNAi feeding approach.
285 h L, while its homeodomain is not, (iv) in L mutant background ventral eye suppression function of Ht
286 uction of SPCH is compromised in a GATA gene mutant background, we hypothesize that PIF- and light-re
288 holino-mediated knockdown of cdx1a in a cdx4 mutant background, we show that a deficiency in both cdx
289 crossing a Bmp4 mutation into the RBPJ/Axin2 mutant background, we show that Wnt and Bmp4 signaling a
290 HO-1:GFP) in wild-type and kinesin (unc-104) mutant backgrounds, we establish in the living nematode
291 d DeltasspAB mutants in both the WT and fakA mutant backgrounds, we found that the absence of these p
293 sations of ABCB19 and PIN1 in the reciprocal mutant backgrounds were like those in wild type, PIN1 pl
294 tations did not affect virulence in the msbB mutant background when administered orally to BALB/c mic
295 ion and trichome placement occur normally in mutant backgrounds where asymmetry of polarity protein d
297 uorescent kinesin-5, KLP61F-GFP, in a klp61f mutant background, where it rescues mitosis and viabilit
298 that callose levels are reduced in the qsk1 mutant background with a root phenotype resembling ectop
299 ion, which was broken in the pmr4 disruption mutant background, with callose deposits at the site of
300 nal fusion was placed in 11 of the 12 Tda(-) mutant backgrounds, with cysI being the sole exception.