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1 ing a CPT1 blocker or by using a Cpt1a P479L mutant mouse strain.
2  generated a cleavage-resistant Ripk1(D325A) mutant mouse strain.
3 e role of E2F3 in vivo, we generated an E2f3 mutant mouse strain.
4 ked compositional differences between WT and mutant mouse strains.
5 ele of Dscam to use alongside existing Dscam mutant mouse strains.
6 ating glucose metabolism and fat mass in two mutant mouse strains.
7 e scientific community for the generation of mutant mouse strains.
8 ossed Myo15(sh2/sh2) mice to the three other mutant mouse strains.
9 nous imprinted genes in inbred wild-type and mutant mouse strains.
10 n of (59)Fe absorption in both wild-type and mutant mouse strains.
11 els were quantitatively similar in the three mutant mouse strains and normal mice.
12  complementation system (LCS) makes use of a mutant mouse strain, aphakia (ak), homozygotes of which
13           Here we show that odors of two MHC mutant mouse strains (bm1 and bm3) can be distinguished,
14  in individual hippocampi from the mast cell mutant mouse strain, C57BL/6 Kit(W-sh/W-sh).
15                               We generated a mutant mouse strain carrying an F832A mutation in Rb1 th
16 ed a forward genetics approach to identify a mutant mouse strain characterized by the absence of CNS
17                                              Mutant mouse strains deficient in RGS7 or -11 were chara
18         Compared with wild-type mice, 2 FHM1 mutant mouse strains developed earlier onset of anoxic d
19  which are defective in the pallid and muted mutant mouse strains, encode small, coiled-coil-forming
20                                              Mutant mouse strains expressing either p31 or p41 Ii cha
21 y important yet simple approach to establish mutant mouse strains for functional study at defined sta
22                   The recent construction of mutant mouse strains for several of these diseases shoul
23                     Here we describe a novel mutant mouse strain, Goldenticket (Gt), that fails to pr
24                                  At least 15 mutant mouse strains have been classified as models of H
25                                   Studies of mutant mouse strains have revealed that each family memb
26                              We identified a mutant mouse strain, HcB-19/Dem (HcB-19), that shares fe
27                                Using several mutant mouse strains, immunoblotting, and microcomputed
28                                              Mutant mouse strains in 60 different GPCRs were generate
29                     The fast accumulation of mutant mouse strains in recent years has provided an inv
30                                A conditional mutant mouse strain, in which Pitx2 function is inactiva
31 we allografted carotid artery loops from six mutant mouse strains into immunocompetent CBA/CaJ recipi
32                                              Mutant mouse strains lacking Ii chain expression have be
33          The class II molecules expressed by mutant mouse strains lacking Ii chain or DM activities d
34 e previously showed that a hypomorphic Mre11 mutant mouse strain (Mre11 (ATLD1/ATLD1) ) was highly su
35                              The spontaneous mutant mouse strain, plt/plt, lacks the secondary lympho
36                                              Mutant mouse strains producing only p31 or p41 under con
37                                         This mutant mouse strain provides a unique model to further e
38                        We show that a CRD-BP mutant mouse strain retains expression of the shorter tr
39 er (EOC) in vivo We used the Pten/Kras(G12D)-mutant mouse strain that develops serous EOC with 100% p
40      Surprisingly, our studies of a knock-in mutant mouse strain that expresses a stabilized and tran
41 ological conditions was investigated using a mutant mouse strain that expresses a truncated Cabin1 la
42 red conditional compound heterozygous Dicer1 mutant mouse strain that fully recapitulates the biallel
43                               We generated a mutant mouse strain that lacks exons 13-20 of L3mbtl1.
44 V-enriched, AMPH-regulated genes in an Mecp2 mutant mouse strain that shows heightened behavioral sen
45  strains, ACAID cannot be induced in several mutant mouse strains that are coincidentally deficient i
46           Here we show, by analyzing several mutant mouse strains, that selective activation of hepat
47 small intestinal phenotype found in most Apc-mutant mouse strains, this strain has been designated th
48 tic defects of GATA-2-/- cells, we interbred mutant mouse strains to assess the effects of p53 loss o
49                                  Here we use mutant mouse strains to investigate E2F3's role in vivo.
50                                            A mutant mouse strain was generated by replacing the IL-2
51                              Different Adar1-mutant mouse strains were employed for gene deletion or
52 he lungs of infected mice revealed that both mutant mouse strains were only transiently impaired in t
53                                          The mutant mouse strains were subjected to lethal doses of b
54 ole of ADAR1 in the heart, we used different mutant mouse strains, which allows to distinguish immuno
55                                              Mutant mouse strains with "autistic-like" phenotypes (Fm
56 d wild-type mice and several newly developed mutant mouse strains with clenbuterol, a selective beta(
57 e-driven breast tumorigenesis in a series of mutant mouse strains with specific DDR deficiencies to r
58 he DFI of frozen-thawed sperm from 83 unique mutant mouse strains with sperm count, motility and morp