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1 dent growth of the M. smegmatis tam deletion mutant strain.
2 e domain, is sufficient to complement a rocA mutant strain.
3 uted to an altered membrane potential in the mutant strain.
4  the DeltawcaM DeltacsgA DeltayihO DeltabcsE mutant strain.
5 ated iron acquisition is impaired in an sbnI mutant strain.
6 gnificantly upregulated in the fabT deletion mutant strain.
7 h a newly characterized curved-rod Delta1228 mutant strain.
8 sistant to oxidants than a catalase-deletion mutant strain.
9 type M59 GAS, we constructed an isogenic mga mutant strain.
10  failed to support growth of an E. coli acpP mutant strain.
11 x II failed to support growth of the E. coli mutant strain.
12 pts of intron-spliced isoforms in the MoHMT1 mutant strain.
13 perature-sensitive phenotype of an spo7Delta mutant strain.
14 but at a substantially lower level in a CMD1 mutant strain.
15 ut were undetectable in the pigmentless snf2 mutant strain.
16 enes were significantly reduced in the dkppx mutant strain.
17 se activity was altered in the yjbH and yjbI mutant strains.
18 f Deltainv, DeltayadA, and DeltainvDeltayadA mutant strains.
19 xposed to H pylori, including cagPAI and cgt mutant strains.
20  sequences in both wild type and degradosome mutant strains.
21 ainst the wild type virus and drug-resistant mutant strains.
22 did not lead to the development of resistant mutant strains.
23 lated RNA ends in ribonuclease wild-type and mutant strains.
24 and cellular phenotypes of the wild-type and mutant strains.
25 in both the E. coli parent and aceE deletion mutant strains.
26 d A isolated from the corresponding deletion mutant strains.
27 eading to the recovery of covR, covS or rocA mutant strains.
28  Hsp70 biology has emerged from studying ssa mutant strains.
29 eron, by developing knock-out and functional mutant strains.
30 specific antibodies via the emergence of new mutant strains.
31  compared to single Deltagra12 or Deltarop18 mutant strains.
32 tical to those of the corresponding deletion mutant strains.
33 iter, with little effect on nearly identical mutant strains.
34  microcolonies between the EmaA positive and mutant strains.
35 species: a collection of genetically related mutant strains.
36  genetic interactions in single and multiple mutant strains, (2) can identify drug targets, (3) detec
37                             Using an E. coli mutant (strain 536DeltafyuA) unable to acquire yersiniab
38                                Notably, most mutant strains accumulate wild-type cellular levels of t
39                                         This mutant strain accumulates abnormally high levels of puri
40                                  A DeltaeseJ mutant strain adheres to epithelioma papillosum of carp
41               Chemical analyses of generated mutant strains allowed for the identification of a trike
42 terized straight-rod Deltapgp1 and Deltapgp2 mutant strains, along with a newly characterized curved-
43                                          The mutant strains also produced very reduced amounts of eth
44 t (Saccharomyces cerevisiae) SEIPIN deletion mutant strain and a plant (Nicotiana benthamiana) transi
45 ry aspergillosis since a sulfate transporter mutant strain and a sulfite reductase mutant strain are
46              We then constructed these novel mutant strains and compared their observed phenotypes to
47 e are increased in both fast and slow Pol II mutant strains and the magnitude of half-life changes co
48 urement of the relative fitness of bacterial mutants, strains and species in mixed inocula in the hos
49 orphisms (n = 48), an isogenic rocA deletion mutant strain, and five isogenic rocA polymorphism mutan
50  different inoculation techniques, bacterial mutant strains, and assays for the hypersensitive respon
51 ences were examined in the single and double mutant strains, and the virulence of select strains was
52 oot architectures, including wildtype and 10 mutant strains, and we uncovered a design principle that
53 troso-guanidine mutagenesis and selection, a mutant strain Apmu4 was derived, in which the rate of lo
54                              Akinetes in the mutant strain appeared normal, but these cultures were l
55 porter mutant strain and a sulfite reductase mutant strain are fully virulent.
56 The guide sequences, editing constructs, and mutant strains are freely available to researchers to in
57 oli communities can be established where the mutant strains are metabolically coupled.
58 sulfate was about 3-4x as great in the A666G mutant strain as in the wild type.
59 rocessing of 16S rRNA is also delayed in the mutant strain, as indicated by increased levels of precu
60         Even fecal-recovery numbers for both mutant strains at several time points prior to the anima
61 onsistent with these observations, the H273D mutant strain attenuated GAS virulence, whereas the H273
62  at the espACD regulatory region of the phoP mutant strain because of PhoP-EspR protein-protein inter
63 expression decreased in the CN3718 codY-null mutant strain but significantly increased in the SM101 c
64  proteins was not negatively affected in the mutant strains, but still the capacity for nonphotochemi
65                                  V. cholerae mutant strains carrying inactivated AI synthase genes we
66 ed to the wild-type strain, a H201R isogenic mutant strain caused significantly larger skin lesions i
67 cytogenes infection, a reduced number of the mutant strain compared to the parental strain was observ
68 f infection with the wild-type strain or the mutant strain complemented with lipA ROS derived from th
69  ESR was partially induced in the siw14Delta mutant strain, consistent with the increased stress resi
70                                     All madC mutant strains contain loss-of-function point mutations
71  gas was detected in a hydrogenase quadruple-mutant strain containing deletions in the hya, hyb, hyd,
72 created and examined a DeltacopAZ DeltacopBL mutant strain (cop-).
73 ity level, we asked whether matrix-defective mutant strains could be coaxed to produce functional mat
74 r cell signaling, and the germination of rpf mutant strains could not be stimulated by the addition o
75              Here we generate a new deletion mutant strain, CVS-N2c(DeltaG), and examine its neuronal
76 ingae pv tomato DC3000 and the nonpathogenic mutant strain DC3000hrpA- allowed us to establish causal
77           Experiments with a T. dentrificans mutant strain defective in U(IV) oxidation supported mic
78                            Finally, the mtsR mutant strains defective in metal sensing and oligomeriz
79 riptional profiling of roots inoculated with mutant strains defective in the synthesis of Nod Factor
80                                              Mutant strains, defective in sRNAs or in ARGONAUTE3 (a k
81 activity is higher in 33277, and an isogenic mutant strain deficient for the gingipains exhibited an
82              In contrast, both wild-type and mutant strains deficient for peroxiredoxins and glutathi
83 erexpression of COY1 inhibited the growth of mutant strains deficient in fusion activity at the Golgi
84 n flux ranges and the presence or absence of mutant strains delivering flux towards pathways of inter
85                           A MakatG1 deletion mutant strain (DeltaMakatG1) showed decreased catalase a
86  Escherichia coli, or Pseudomonas aeruginosa mutant strain DeltapopB Moreover, BMDMs deficient in IRF
87 riptional profiles were compared between the mutant strain DeltarelA [a (p)ppGpp(0) strain under gluc
88 rgeted gene deletion confirmed that isogenic mutant strains DeltaspyAD and Deltaisp2 are significantl
89   A B. pseudomallei DeltappiB (BpsDeltappiB) mutant strain demonstrates impaired biofilm formation an
90 gnificantly increased in the SM101 codY-null mutant strain, demonstrating CodY-dependent regulation d
91 er, we found that a slowly constricting fzlA mutant strain develops 'pointy' poles, suggesting that F
92 uring growth in vitro However, the DeltaphtY mutant strain did not display an increased CP sensitivit
93     Despite high expression of ACT, the rseA mutant strain did not infect the murine airway as effici
94                                The DeltaohyA mutant strain did not recover from the 16:1(9Z) challeng
95                               This bacterial mutant strain did not stimulate colitis in dnKO mice.
96 as the DeltaadcA, DeltaadcAII, and DeltaphtD mutant strains displayed less CP sensitivity during grow
97  the general fitness of the amoebae with the mutant strain displaying a substantial growth defect.
98                   Importantly, the DeltaeseJ mutant strain does not replicate efficiently in EPC cell
99 etween M. tuberculosis H37Rv and a DeltanarL mutant strain during exponential growth in broth culture
100 wild-type, rocA mutant, covS mutant and covR mutant strains during invasive infection and their fitne
101 g. 1-phenyl-2-thiourea, PTU) or pigmentation mutant strains (e.g. casper mutant).
102  Individual DeltatrxA and DeltatrxC deletion mutant strains each show a greater abundance of lipid pe
103 he hypothesis that vaccination with the batA mutant strain elicits protective immunity against subseq
104        We generated a Drosophila Adar(E374A) mutant strain encoding a catalytically inactive Adar wit
105 ed defects in Mia40 oxidation, only one erv1 mutant strain (erv1-1) had significantly decreased activ
106 murine model of aspergillosis, the Deltagna1 mutant strain exhibited attenuated virulence.
107        Despite gaining phage resistance, epa mutant strains exhibited a loss of resistance to cell wa
108 t H273 and H327 relieved inhibition, and the mutant strains exhibited a wild-type phenotype.
109 In contrast to cen9Delta mutants, cen10Delta mutant strains exhibited growth defects and were aneuplo
110                       We showed that the rRV mutant strains exhibited impaired viral replication and
111                       Each of the Walker box mutant strains exhibited properties identical to those o
112 n original strategy that combined the use of mutant strains expressing catalytically inactive variant
113 strain expressing both HMW1 and HMW2 and the mutant strains expressing either HMW1 or HMW2 were able
114              We analyse 230 signature-tagged mutant strains for 114 putative phosphatases under 30 di
115  A screen with S. pombe transcription factor mutant strains for growth sensitivity to the AEO fractio
116    To further elucidate regulatory controls, mutant strains for histidine kinases PhoR and AioS were
117                   We consistently found that mutant strains for RNAi (dcr1Delta, ago1Delta, rdp1Delta
118 ld-type strain and an isogenic fabT deletion mutant strain found that between 3.7 and 28.5% of the S.
119 1;3 and OsPIP2;6 were displayed in yeast HD9 mutant strain (fps1acr3ycf1) as a result of increased B
120 ysis shows that the transmission rate of the mutant strain from mosquitoes to human is more influenti
121  representative isolates and "leaky melanin" mutant strains from each species complex to examine the
122              An exogenous EL mimic protected mutant strains from hNP-1 and hBD-2 but not RP-1, indica
123 on techniques can be used to distinguish FKS mutant strains from wild type, but testing C. glabrata w
124 ith the increased stress resistance, and the mutant strain further induced the ESR in response to oxi
125 g mutant mice, we characterized Rhbdf1 mouse mutant strains generated by three commonly used strategi
126                                          The mutant strain grew normally under iron-limiting conditio
127 nt, hopper(Bd-we), isolated from a white eye mutant strain had an intact transposase reading frame an
128 orescent reporter proteins revealed that the mutant strain had greatly enlarged peroxisomes up to 10
129 virulence characteristics, and the quadruple-mutant strain had the same (greatly attenuated) phenotyp
130                                The DeltacsrA mutant strains had a greatly reduced ability to kill Gal
131                                The DeltacsrA mutant strains had a reduced ability to survive drying a
132                                              Mutant strains harbouring transposon insertions in two s
133  release, and an ato5Delta ATO1(G53D) double mutant strain has additive alkalinization and ammonia re
134  Y181C, Y188L, IRLL98, and K103N+Y181C HIV-1 mutant strains, highlighting the importance of the alpha
135 V. cholerae and a locked low-cell-density QS-mutant strain identified 7,240 transcriptional start sit
136 Whole genome sequencing of the wild-type and mutant strains identified a point mutation that creates
137 oci analysis, machine learning, and isogenic mutant strains identified and confirmed a one-nucleotide
138 ollowed by genome sequencing of the isolated mutant strains identified mutations that disrupt DNA dam
139                                       In all mutant strains, impaired maturation of white matter astr
140                         The virulence of the mutant strain in a murine model of melioidosis demonstra
141 w that Cu strongly inhibits growth of a copA mutant strain in acidic cultures.
142 experiments with a PEP carboxylase-deficient mutant strain in blood and cerebrospinal fluid.
143  CMV infection resolution and emergence of a mutant strain in high-risk recipients of kidney transpla
144 t mortality between wild-type and DSB repair mutant strains in any model of infection.
145 re limited data on disease manifestations of mutant strains in children.
146 were necessary for fitness in lasR+ and lasR mutant strains in colony biofilms grown in microoxic con
147 forming growth curves with WT, alsT and gltS mutant strains in defined medium supplemented with ammon
148 roteome changes of wild type UA159 and lrgAB mutant strains in response to these same stresses.
149                           Surprisingly, when mutant strains in the genes involved in virulence (JDelt
150                        Indeed, evolving 7 PF mutant strains in the presence of drug revealed 3 adapta
151 lker-B) in each of the ATPases and generated mutant strains in which these motifs were altered.
152  length maintenance by utilizing yku70-R456E mutant strains, in which Ku has reduced DEB and telomere
153         Using a novel P. gingivalis W50 PPAD mutant strain, incapable of protein citrullination, and
154 vironments, to characterization of different mutant strains, including those harbouring synthetic cir
155 ested by the hypersensitivity of a Deltasod1 mutant strain, increased resistance afforded by the supe
156 n and metabolism were all accelerated in the mutant strain indicative of dysregulated oxidative phosp
157                            We show that this mutant strain is able to metabolise xylose to acetate on
158  in mouse lung 24 h post infection while the mutant strain is cleared by host defense mechanisms.
159     Finally, we show that an E. faecalis epa mutant strain is deficient in intestinal colonization, c
160       Congruous with ex vivo findings, a nfu mutant strain is more susceptible to oxidative killing b
161 munity afforded by vaccination with the batA mutant strain is predominantly mediated by IgG antibodie
162                             Two Helicobacter mutant strains (katA(H56A) and katA(Y339A)) containing c
163 abelled LA did not differ between the WT and mutant strains, LA induced more cell membrane damage in
164                                          The mutant strain lacking both SPN and SLO production is sev
165  to colonize more frequently than the double mutant strain lacking HMW1 and HMW2.
166 tion model and that HA levels increased in a mutant strain lacking hyaluronidase (HysA).
167 by Pseudomonas aeruginosa wild-type versus a mutant strain lacking multiple porins.
168 ia and soft tissue infection models, and the mutant strain lacking production of both toxins is furth
169 in several nitrogen sources, we found that a mutant strain lacking the Mycobacterium proteasomal acti
170                            An S. Typhimurium mutant strain lacking these two effectors exhibits marke
171                                              Mutant strains lacking Cagalpha, Cagbeta, or CagE (putat
172                   Furthermore, 33277 and 381 mutant strains lacking CTD cell surface proteins were mo
173 iffusion in wild-type V. cholerae to that in mutant strains lacking either toxR or the toxT promoter,
174                               Here, isogenic mutant strains lacking EmaA structures, but still expres
175                                 We show that mutant strains lacking gigC have impaired growth in the
176 s lysozyme resistance of L. monocytogenes as mutant strains lacking gpsB showed an increased lysozyme
177                             Experiments with mutant strains lacking mitochondrial superoxide dismutas
178                                              Mutant strains lacking mneP are Mn(II) sensitive and acc
179                                              Mutant strains lacking the spermine synthase-encoding ge
180                          Similarly to a carR mutant, strains lacking almE, almF, and almG exhibited e
181                                  Like a ume6 mutant, strains lacking SAP6 exhibit enhanced colonizati
182  Mongolian gerbils with an H. pylori pgdA(-) mutant strain led to significantly decreased levels of i
183 on of LipA from Xac306 and verified that the mutant strain lost most of its lipase and esterase activ
184                              We found that a mutant strain (mac1Delta) lacking Mac1, a copper-sensing
185                                    Two other mutant strains made truncated versions of the protein.
186 quire a live, stable, lipid-rich E. gracilis mutant strain, named B1ZFeL, with 40% more lipid content
187 clude the generation of large collections of mutant strains, now available for forward- and reverse-g
188                             An ibeA deletion mutant strain (NRG857cDeltaibeA) was constructed, and th
189                                            A mutant strain of C. neoformans that cannot transport xyl
190 the reference strain and homozygous deletion mutant strain of CCC2, which encodes a Cu(2+)-transporti
191  compared to a DeltarelADeltaspoT (ppGpp(0)) mutant strain of E. coli, deficient in the stringent res
192    We also determine that the high-persister mutant strain of Escherichia coli, HipQ, is associated w
193                                            A mutant strain of L. monocytogenes expressing the cystein
194  ROS inhibitor) or LGGOmegaSpaC (an adhesion-mutant strain of LGG) or FPR1-knockout mice.
195 tient 103 (PA103) supernatant] or defective [mutant strain of P. aeruginosa lacking a functional type
196 e, where symptoms are reduced in a Deltaexl1 mutant strain of P. atrosepticum.
197 easure the mechanical properties of T4P of a mutant strain of Pseudomonas aeruginosa PAO1 unable to r
198 tpatient LRTI because of a newly circulating mutant strain of RSV B.
199 1) from S. atroolivacues SB3033, a DeltalnmE mutant strain of S. atroolivaceus S-140.
200  because killed bacteria and a protease-null mutant strain of S. aureus were unable to penetrate.
201 c-psbA4 gene from Cyanothece 51142 in a 4E-3 mutant strain of the model non-nitrogen-fixing cyanobact
202                            We use a dynactin mutant strain of the multinucleate fungus Ashbya gossypi
203                     A colonization-deficient mutant strain of this intracellular bacterium is able to
204 Bs2 gene since no effect was observed when a mutant strain of Xcc with a disruption in avrBs2 gene wa
205 much faster "colony surfing" still occurs in mutant strains of Bacillus subtilis lacking flagella.
206 -producing and a noninvasive hypha-producing mutant strains of C. albicans.
207 xperimental data for different wild type and mutant strains of C. botulinum Group I type A1.
208  of photoautotrophically grown wild-type and mutant strains of Chlamydomonas reinhardtii to determine
209 option of choice to check both wild-type and mutant strains of different origins.
210 ease progression using Caenorhaditis elegans mutant strains of dnj-14, the worm orthologue of DNAJC5.
211 irus (IAV) infection including wild-type and mutant strains of H1N1 and H3N2 subtypes.
212 mic has been exacerbated by the emergence of mutant strains of Mtb that are resistant to frontline an
213 oughput sequencing (Hi-C) with wild-type and mutant strains of Neurospora crassa to gain insight into
214 al lesions, both capsular-defective knockout mutant strains of P. gingivalis induced less alveolar bo
215                                     Isogenic mutant strains of S. aureus with varying degree (Deltare
216                                Surprisingly, mutant strains of S. mutans with impairment in RGP side
217                            Here we show that mutant strains of S. praecaptivus that lack genetic comp
218                                 Furthermore, mutant strains of T. forsythia, devoid of either mirolys
219                                        Using mutant strains of these bacteria that are defective in t
220  to a toxic concentration of toluene, a tolR mutant strain or a strain overexpressing a diguanylate c
221                                 Using a pde2 mutant strain, pApA was detected for the first time in S
222                     On plants, the Deltanmo2 mutant strain penetrated host cuticles like wild type, b
223 tii nopA::Tn or a Dot/Icm-defective dotA::Tn mutant strain present a functional innate immune respons
224 transformants of a somatic regenerator (Reg) mutant strain receiving sgRNA plasmid with glsA protospa
225  during the growth of the wild-type and sigL mutant strains reduced expression of the toxin genes, in
226 that the majority of cells in a Deltasll1130 mutant strain remained unicellular and viable after prol
227  produce an mif(-/-) strain of L. major This mutant strain replicated normally in vitro but had a 2-f
228                      SMG-free, quadruple vic mutant strains representing both allelic backgrounds of
229 n situ reconstitution of fnm in the deletion mutant strain restored adherence.
230                Our analyses of wild-type and mutant strains reveal key elements of chromosome archite
231 ic Cu(+) Analysis of DeltacopR and DeltacueR mutant strains revealed a CopR regulon composed of genes
232 scriptome analysis of the wild-type and dcl3 mutant strains revealed a further difference from higher
233                   Phenotypic analysis of the mutant strains revealed that disruption of icsA abolishe
234 DeltadevR, DeltadevSDeltadosT, and DeltanarL mutant strains revealed that in response to nitrite prod
235 nal carbon; the core oscillator in the prd-1 mutant strain runs with a long period under glucose-suff
236 itch variability is heritable, and comparing mutant strains selectively bred to high and low penetran
237 ated MRSA parental and their respective sarA mutant strain sets.
238                            Unexpectedly, the mutant strain showed increased serum resistance.
239                      Goats infected with the mutant strain showed only transient fever.
240 tion in the wild type and an IsoMO-disrupted mutant strain showed that epoxyisoprene, or a subsequent
241 for correct fungal development as the AaGPx3 mutant strains showed a severe reduction in conidiation.
242                       EM of the compensatory mutant strains showed complete virus particles, but thes
243 s and biological confirmation with different mutant strains showed that this life extension is due to
244 FACT in deubiquitinating H2B in vivo, a FACT mutant strain shows elevated levels of H2B-Ub.
245 tion frequency, particularly in an ung1Delta mutant strain, suggesting that dCas9 induces mutations t
246 aexlx-gh5 rescued the movement defect of the mutant strain, suggesting that expansin and GH5 function
247  transcripts that were more abundant in dcl3 mutant strains than in wild-type cells were not due to s
248 C (11G5 strain) isolated from a patient or a mutant strain that does not produce colibactin (11G5Delt
249  we describe the genome engineering of a RF1 mutant strain that enhances suppression efficiency durin
250 enetically engineered centrin gene knock-out mutant strain that is antibiotic resistant marker free a
251                 Specifically, we generated a mutant strain that lacks all 13 PTS transporters, and fr
252                                              Mutant strains that are dsbA or dsbB null facilitate lab
253 ding increasing concentrations of sulfate to mutant strains that are unable to incorporate H2S effici
254 combine phenotypic and genotypic analyses of mutant strains that suggest discrepancies in the literat
255                                   In the two mutant strains, the flexural rigidity is not significant
256  colonized with R. gnavus wild-type (WT) and mutant strains-the fitness of the nan mutant was signifi
257 ully restored the wild-type phenotype of the mutant strain; this indicates that P55 plays no importan
258 still substantially impact the phenotypes of mutant strains through epistasis.
259 or screening mutants and establishing stable mutant strains through genetic crosses.
260  of Chlamydomonas reinhardtii and the use of mutant strains to analyze photosynthesis was conducted i
261                The varied sensitivity of the mutant strains to CP-mediated Zn limitation suggests dis
262 notation of new genes, and the generation of mutant strains to define the role of genes in complex en
263  strain, and five isogenic rocA polymorphism mutant strains to perform genome-wide transcript analysi
264               Here, we utilize P. gingivalis mutant strains to show that pathogen-differentiated mDCs
265                                              Mutant strains tolerant to levels of aromatic acids near
266 satisfies fluxomic data for wild-type and 25 mutant strains under different substrates and growth con
267                                  The capsule mutant strain was also impaired for survival in guinea p
268 tbreak data in North-Eastern Italy where the mutant strain was detected.
269  5 transporters confirmed that each isogenic mutant strain was significantly (P < 0.05) impaired in c
270                     The B. abortus bab1_1517 mutant strain was significantly attenuated in macrophage
271 ii, we observed that the growth of DeltacsrA mutant strains was inhibited in the presence of amino ac
272 xpression by epithelial cells exposed to psm mutant strains was significantly increased compared to t
273                               Using isogenic mutant strains, we demonstrate that deleting one or more
274                             Using isoallelic mutant strains, we found that 3 polymorphisms in this to
275                                Using FGDelta-mutant strains, we showed that specific combinations of
276   S(0) globules from a Chlorobaculum tepidum mutant strain were purified and used to show that the wi
277                                          The mutant strains were examined for CPS quantity, size, and
278                                          Two mutant strains were generated: one where expression of t
279                    We also observed that the mutant strains were more sensitive than WT cells to high
280                            Whereas DeltaagrB mutant strains were not able to produce biofilms, a Delt
281 differences in MICs between WT and DeltaTolC mutant strains were not reflected by equal differences i
282 ptation, exocytosis, and endocytosis in sec3 mutant strains were similarly alleviated by mutation of
283                The DeltaadcC and DeltarpsN.2 mutant strains were the most susceptible to CP, whereas
284 d Pseudomonas putida G7 Y1, a nonchemotactic mutant strain, were simultaneously introduced into the s
285 ExaF reduces methanol sensitivity in the fae mutant strain when lanthanides are present, providing ev
286 egulated by RpoE, and surprisingly, the rseA mutant strain where RpoE activity was elevated expressed
287       Capsule production is abolished in the mutant strain, which is concomitant with its inability t
288  a consistent failure of C. reinhardtii vtc1 mutant strains, which are deficient in polyphosphate syn
289                                      Using a mutant strain with a high differentiation rate and fluor
290 rbidity and mortality, particularly if a CMV mutant strain with antiviral resistance emerges.
291 nserted into the C. besciigenome, creating a mutant strain with its S-layer extensively decorated wit
292 ion with either wild-type cryptococci or the mutant strain with reduced surface xylose; although iBAL
293                       Complementation of the mutant strain with the wild-type SNF2 gene restored pigm
294 sporter mediated this increase, we generated mutant strains with a deletion of SPE1 or SPE2 combined
295  generate and phenotypically characterize 29 mutant strains with deletions of individual transporter
296                                              Mutant strains with disrupted RGP synthesis failed to pr
297 hpA The expression of uhpT was absent in the mutant strains with uhpT deletion and was not inducible
298 ing molecular mass in both the wild-type and mutant strains with various subsets of phenylalanine-gly
299                                    One (+/0) mutant strain, with multiple mutations at the mating loc
300  whereas in the csr-1 partially rescued null mutant strain (WM193), this mark is ectopically deposite

 
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