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1 uninfected mice and influenced responses to mycoplasma infection.
2 pulations capable of mediating resistance to mycoplasma infection.
3 requirements for generating immunity against mycoplasma infection.
4 hanges in lipid content of cell lines due to mycoplasma infection.
5 of a chronic erosive arthritis subsequent to mycoplasma infection.
6 al contact for the possibility of hemotropic mycoplasma infection.
7 nized and potentially severe complication of mycoplasma infections.
9 piratory tract immunization protects against mycoplasma infection and compared preferential immunizat
11 cell populations and their activation after mycoplasma infection and determines their association wi
12 ort the need to constantly monitor cells for mycoplasma infection and keep stored samples of all cell
13 ies into venules in the airways of mice with Mycoplasma infection and that TNFalpha signaling is nece
14 IFN-gamma and IL-4 production in controlling mycoplasma infection and the pathogenesis of disease in
16 making Cypl a possible target for combating mycoplasma infections and tumorigenic transformation.
17 act immunity plays a major role in resisting mycoplasma infection, and it should be considered during
18 either protection or immunopathology against mycoplasma infection, and optimal vaccination appears to
19 a model of the vertebrate immune response to mycoplasma infections, and use it to identify which path
20 on, the multiorgan protean manifestations of mycoplasma infection are discussed in terms of the role
21 of upper respiratory tract immunity reduced mycoplasma infection at this site, but CT was needed to
24 asma pneumoniae P1 gene was used to diagnose mycoplasma infection in two cohort patients with severe
27 rrent available methods for the diagnosis of Mycoplasma infection, including cultivation, serological
36 monary Th and CD8(+) T cells increased after mycoplasma infection, there was a preferential expansion
37 hanism of Lcn2 gene regulation by MALP-2 and mycoplasma infection was investigated in mouse mammary e