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1 he paranode and ultimately the physiology of myelinated nerve.
2 response protein 2) transcription factors in myelinated nerves.
3 in the spinal cord and peripherally in large myelinated nerves.
4 ly placed, nonmyelinated axon segments along myelinated nerves.
5 l terminals and the juxtaparanodal region of myelinated nerves.
6 lial junctions flank the nodes of Ranvier in myelinated nerves.
7 aps through the nodes of Ranvier (NRs) along myelinated nerves.
8 ed to regions before the transition zones of myelinated nerves.
9 ully studied in different types of mammalian myelinated nerves.
10 tion of action potentials (AP) at the NRs of myelinated nerves.
11  key determinants of saltatory conduction on myelinated nerves.
12   Nerve terminal arborizations, arising from myelinated nerves and exhibiting variable morphology, we
13 de new insights into saltatory conduction on myelinated nerves and may have physiological as well as
14  myelin basic protein+/protein gene product+ myelinated nerve bundles at the index finger were reduce
15 ades as well as some intraepidermal and free myelinated nerve endings.
16 tor in vivo during development and repair of myelinated nerves explaining the deficiency of myelin ob
17 tional ordering of membrane compounds in the myelinated nerve fiber by means of polarized Raman micro
18 nerve fibers and synthetic gel fibers, a non-myelinated nerve fiber carrying an impulse was treated a
19 sults further demonstrate the important role myelinated nerve fiber degeneration plays in the pathoge
20 ermal nerve fiber density, as well as dermal myelinated nerve fiber density, which persisted througho
21 inal pigment epithelium degeneration (2.5%), myelinated nerve fiber layer (1.3%), and internal limiti
22 a+-K+-ATPase, nerve conduction velocity, and myelinated nerve fiber pathology.
23  Contactin is downregulated along the entire myelinated nerve fiber.
24 rcumscribed white matter tract consisting of myelinated nerve fibers and neuroglial cells.
25                     There was loss of larger myelinated nerve fibers and profuse regenerative activit
26                                              Myelinated nerve fibers are designed in an optimal manne
27              Paranodal axoglial junctions in myelinated nerve fibers are essential for efficient acti
28                       Paranodal junctions of myelinated nerve fibers are important for saltatory cond
29 erest because the global effects of aging on myelinated nerve fibers are more complex and profound th
30 es a simple and rapid technique to visualize myelinated nerve fibers at specific locations along the
31               Action potential conduction in myelinated nerve fibers depends on a polarized axonal me
32  that in aging there is a ubiquitous loss of myelinated nerve fibers from the brain and that fiber lo
33 d the middle-aged monkeys the mean number of myelinated nerve fibers in the anterior commissure is 2.
34 correlated with a reduction in the number of myelinated nerve fibers in the anterior commissure, an i
35 ed the morphology and area number density of myelinated nerve fibers in the cingulate bundle and genu
36                                  The loss of myelinated nerve fibers is accompanied by a significant
37                       However, the number of myelinated nerve fibers is the only variable that correl
38 g results in a 20% decrease in the number of myelinated nerve fibers per unit area, while remaining n
39                  Approximately 80 percent of myelinated nerve fibers throughout the central and perip
40 which may be related to the vulnerability of myelinated nerve fibers to the normal process of aging.
41                  Although the development of myelinated nerve fibers was not impaired, Miz1DeltaPOZ m
42 sequential nodal and axonal injury of intact myelinated nerve fibers, recapitulating pathologic featu
43 ction, it had no effect on measures of large myelinated nerve fibers, specifically sural or sciatic n
44 sent a new method to quantify differences in myelinated nerve fibers.
45 e fornix, but there is a significant loss of myelinated nerve fibers.
46 the action potential propagating elements of myelinated nerve fibers.
47 rs, associated with a dramatic loss of large myelinated nerve fibers.
48 to distinct subcellular domains in mammalian myelinated nerve fibers.
49  adhesion molecules, such as MAG, present in myelinated nerve fibers.
50 14 cutaneous nerve biopsies revealed loss of myelinated nerve fibres (86%), increased regenerative cl
51 nding site and transcript analysis of dermal myelinated nerve fibres using a novel platform, revealed
52 phology and molecular architecture of dermal myelinated nerve fibres were examined using immunohistoc
53 in the evaluation of non-myelinated, but not myelinated nerve fibres, in sensory neuropathies.
54 al signal generation and transmission in CNS myelinated nerve fibres.
55 gated sodium channels at nodes of Ranvier in myelinated nerves: here, we investigate its role in AIS
56 erized by autoreactive immune cells damaging myelinated nerves, impairing brain function.
57 o myelinate developing nerve and to maintain myelinated nerve in adulthood.
58                       We therefore evaluated myelinated nerves in skin biopsies from normal controls
59 ciated glycolipids essential for maintaining myelinated nerve integrity.
60 an be regenerated during nerve conduction on myelinated nerves, ion channel mechanisms underlying the
61 es are propagated at nodes of Ranvier in the myelinated nerves of vertebrates.
62                                              Myelinated nerves, regardless of their source, have in c
63                    Rapid nerve conduction in myelinated nerves requires the clustering of voltage-gat
64                Fast, saltatory conduction in myelinated nerves requires the clustering of voltage-gat
65 n depends on specialized membrane domains in myelinated nerve, the node of Ranvier, the paranode, and
66                                 In mammalian myelinated nerves, the internodal axon that is normally
67 e alterations in ion channel localization in myelinated nerves; this provides a rationale for the aud
68 etic screens for mutants with disruptions in myelinated nerves, we identified mutations in erbb3 and