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2 ls of CWP1, resulting from the activation of myeloblastosis domain protein 2 (MYB2), a TF previously
6 ween GTP-binding elongation factor HBS1L and myeloblastosis oncogene MYB on chromosome 6q that are as
8 ow that the Drosophila homologue of the Myb (Myeloblastosis) oncoprotein family is tightly associated
9 on assays while no specific binding to avian myeloblastosis or Moloney murine leukemia RTs was detect
10 FLD (Flowering locus D), GI (GIGANTEA), Myb (Myeloblastosis), SFH3 (SEC14-like 3), bZIP (basic-leucin
11 genetic locus encoding BERRY1, an R2R3 MYB (myeloblastosis) that regulates anthocyanin pigment produ
13 tric gliomas in the related gene v-myb avian myeloblastosis viral oncogene homolog (MYB) on 6q23.3.
16 ry gland origin, has a signature v-myb avian myeloblastosis viral oncogene homolog-nuclear factor I/B
17 l stabilities of wild-type recombinant avian myeloblastosis virus (AMV) and Moloney murine leukemia v
18 construction experiments with purified avian myeloblastosis virus (AMV) IN and retrovirus-like donor
20 nuclear protein v-Myb, encoded by the avian myeloblastosis virus (AMV), can induce acute monoblastic
22 ilarly inhibits the activities of both avian myeloblastosis virus and human immunodeficiency virus ty
23 e and full-site reactions catalyzed by avian myeloblastosis virus and human immunodeficiency virus ty
24 Two acute transforming retroviruses, avian myeloblastosis virus and the E26 leukemia virus, transdu
26 ed attachment (att) site sequences and avian myeloblastosis virus IN (4 nm) were as competent for ful
27 ular insights into the organization of avian myeloblastosis virus IN at the viral DNA ends were gaine
28 specific activities of virion-derived avian myeloblastosis virus integrase and bacterial recombinant
31 tem, extension of the primer by either avian myeloblastosis virus reverse transcriptase (AMV-RT) or h
33 here the deleted cDNA was generated by avian myeloblastosis virus reverse transcriptase from a synthe
34 , DNA polymerase I Klenow fragment and avian myeloblastosis virus reverse transcriptase incorporated
35 The shortened cDNA was generated by avian myeloblastosis virus reverse transcriptase, but not by t
36 v-Myb, the transforming protein of avian myeloblastosis virus, causes acute myeloid leukemia in c
37 ologue of the transforming gene of the avian myeloblastosis virus, is preferentially expressed in all
38 Thus, the v-myb gene encoded by the Avian Myeloblastosis Virus, is truncated at both the 5' and 3'
39 t other reverse transcriptases tested (avian myeloblastosis virus, Moloney murine leukemia virus, and