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1 activated immune cells (including NK, T, and myeloid dendritic cells).
2 acrophages and subsets of neurons but not in myeloid dendritic cells.
3 body myositis and polymyositis are primarily myeloid dendritic cells.
4 accumulation of T helper 17 lymphocytes and myeloid dendritic cells.
5 activated macrophages, NK cells, and mature myeloid dendritic cells.
6 ctivator of several cell types, particularly myeloid dendritic cells.
7 lopment of mature myeloid progeny, including myeloid dendritic cells.
8 ified hemozoin on the in vitro activation of myeloid dendritic cells.
9 t for the recruitment and differentiation of myeloid dendritic cells.
10 induced by cytosolic double-stranded DNA in myeloid dendritic cells.
11 lls, NK-cells, B-cells, monocyte subsets and myeloid dendritic cells.
12 try restriction factor to HIV-1 infection in myeloid dendritic cells.
13 ext of caspase-1 activation in monocytes and myeloid dendritic cells.
14 es HagB-induced chemokine responses in human myeloid dendritic cells.
15 Therefore, we isolated dsRNA complex in myeloid dendritic cells.
16 eriod to 9 mo of age in monocytes but not in myeloid dendritic cells.
17 ndritic cells clustered together with CD11c+ myeloid dendritic cells, a distinct dendritic cell subse
18 dendritic cells, but not functionally mature myeloid dendritic cells, accumulated in tumor microenvir
19 d cells B cells, neutrophils, monocytes, and myeloid dendritic cells all efficiently bound Spike prot
20 in plasmacytoid dendritic cells, whereas in myeloid dendritic cells, all three family members were e
23 on of infection, eosinophils expand IL-10(+) myeloid dendritic cells and CD4(+) IL-10(+) T lymphocyte
25 e TSLP (29-130 + 131-159) strongly activated myeloid dendritic cells and group 2 innate lymphoid cell
26 as associated with a significant increase in myeloid dendritic cells and in vivo induction of CD4+/CD
27 sting that the antiviral cytokine sensitized myeloid dendritic cells and macrophages toward TLR4 liga
30 CCR7 deficiency led to an accumulation of myeloid dendritic cells and T cells in the lung in respo
31 a2 induced the recruitment and activation of myeloid dendritic cells and Th2 cells in the lung, causi
32 ET favors neutrophil proteins uploading into myeloid dendritic cells and the induction of ANCAs and a
33 gulated kinase 1/2-positive monocyte-derived myeloid dendritic cells, and increased vimentin expressi
34 macrophages, CD3+ T cells, monocyte-derived myeloid dendritic cells, and keratinocytes expressing in
35 elative dearth of MOG35-55-specific T cells, myeloid dendritic cells, and neutrophils, as well as an
36 hemokine MCP-1, which attracts monocytes and myeloid dendritic cells, and of the cytokine macrophage-
37 an monocyte-derived dendritic cells (MDDCs), myeloid dendritic cells, and plasmacytoid dendritic cell
38 acrophages, CD4+ T cells, and CD11b+ CD11c+ (myeloid) dendritic cells, and increased arginase1 and Ym
42 erferon (IFN-gamma), TNF-alpha, and IL-12 in myeloid dendritic cells are of importance in generating
44 tage of CD1c(+)CD19(-)CD14(-)CD303(-) type 1 myeloid dendritic cells at disease onset had a significa
46 ignificant separation between species, while myeloid dendritic cells best associated macaque populati
48 pha) in wild-type murine bone marrow-derived myeloid dendritic cells (BMDCs) but did not stimulate in
51 s gave rise to granulocyte, monocyte, and/or myeloid dendritic cells, but not MegE lineage cells in t
52 psoriasis pathogenesis, termed inflammatory myeloid dendritic cells (CD11c(+)/blood dendritic cell (
53 r stroma showed enrichment of two subsets of myeloid dendritic cells (cDC), cDC1 and cDC2, that secre
54 so seen in other subsets, including CD11c(+) myeloid dendritic cells, classical monocytes, two subset
56 in contrast to plasmacytoid dendritic cells, myeloid dendritic cells, co-cultured with Barrett's esop
57 nofluorescence staining, CD11c+, a marker of myeloid dendritic cells, colocalized with PD-L1 and PD-L
59 the study of signaling pathways involved in myeloid dendritic cell (DC) differentiation, we have dev
61 mammalian target of rapamycin (mTOR), during myeloid dendritic cell (DC) generation confers resistanc
63 oviral delivery of human TGF-beta1 to murine myeloid dendritic cell (DC) progenitors on (i) their in
64 unctional differences between the human skin myeloid dendritic cell (DC) subsets, epidermal CD207(+)
65 mice resulted in the reduced accumulation of myeloid dendritic cells (DC) and activated CD4(+) T cell
67 toid cells (PC) and CD1c(+) peripheral blood myeloid dendritic cells (DC) are two human DC precursors
68 e by modulating the functional properties of myeloid dendritic cells (DC) during the acute immune res
69 red the maturation of human monocyte-derived myeloid dendritic cells (DC) following inoculation with
71 In small animals and humans, conventional myeloid dendritic cells (DC) have been shown to select o
72 d vigorous primary T-cell immune response by myeloid dendritic cells (DC) in blood and tissues could
73 sulted in the accumulation of iNKT cells and myeloid dendritic cells (DC) in pancreatic lymph nodes (
74 c and functional differences between classic myeloid dendritic cells (DC), and DC that reportedly dev
77 Crosslinking CD28 by agonistic antibodies or myeloid dendritic cells (DC; these express the CD28 liga
78 ough there is evidence for distinct roles of myeloid dendritic cells (DCs [mDCs]) and plasmacytoid pr
79 ial, whether expansion of inflammatory blood myeloid dendritic cells (DCs) and conversion of Tregs to
80 lope 2 proteins (E2), activate monocytes and myeloid dendritic cells (DCs) and partially reproduce ab
81 virtually all blood immune cells, including myeloid dendritic cells (DCs) and plasmacytoid DCs (pDCs
84 tural killer (NK) cells and plasmacytoid and myeloid dendritic cells (DCs) are depleted, and their fu
90 To determine the phenotype and function of myeloid dendritic cells (DCs) from human cutaneous squam
93 nocytes, nonclassical monocytes, and CD1c(+) myeloid dendritic cells (DCs) in the blood, and mutation
94 In response to locally produced calcitriol, myeloid dendritic cells (DCs) migrated from cutaneous va
96 CD207+ Langerhans cells (LCs) and CD11c+ myeloid dendritic cells (DCs) were found in both the epi
97 ells are activated by lipid Ags presented by myeloid dendritic cells (DCs), after which they generate
98 ating B lymphocytes, monocytes, neutrophils, myeloid dendritic cells (DCs), and at very low levels on
99 ession of the costimulatory molecule CD86 on myeloid dendritic cells (DCs), and reduced the number of
100 s and a mixed population of plasmacytoid and myeloid dendritic cells (DCs), including cells expressin
101 s-derived eVLPs elicited maturation of human myeloid dendritic cells (DCs), indicating their immunoge
111 inflamed tissue, and supports the concept of myeloid dendritic cell differentiation from trafficking
113 igen-presenting cells (mostly Langerhans and myeloid dendritic cells) exhibit a tolerogenic phenotype
114 -55)), microglial cells and CNS-infiltrating myeloid dendritic cells expressed CMKLR1, as determined
115 Additionally, a large frequency of colonic myeloid dendritic cells expressed IL-15Ra, implicating m
117 migration, activation, and Ag-processing of myeloid dendritic cells from the lung to the draining ly
118 tal autoimmune encephalomyelitis but, unlike myeloid dendritic cells, have a minor role in T cell act
121 lic surface ligand-coated AuNP that targeted myeloid dendritic cells in lymph nodes as a peptide anti
123 ndritic cells expressed IL-15Ra, implicating myeloid dendritic cells in trans presentation of IL-15 t
124 t IL-4 induces recruitment and maturation of myeloid dendritic cells in vivo and increases T cell rec
127 urthermore, Ly49Q expression on pDC, but not myeloid dendritic cells, is necessary for optimal IL-12
129 od mononuclear cells identified migration of myeloid/dendritic cell lineage cells one day after vacci
132 ndritic cell release of IFN-B, and assessing myeloid dendritic cell (mDC) contribution to hydroxychlo
133 Current immunological opinion holds that myeloid dendritic cell (mDC) precursors migrate from the
134 LACV infection indicated that differences in myeloid dendritic cell (mDC) responses between weanling
137 When individual populations were isolated, myeloid dendritic cells (mDC) and macrophages but not pl
138 receptor (TLR) responses of peripheral blood myeloid dendritic cells (mDC) and monocytes were tempora
143 n iNKT cells and CD1d-expressing circulating myeloid dendritic cells (mDC), as mDC of patients with a
146 of the activating FcgammaRIIa on circulating myeloid dendritic cells (mDCs) after high-dose, but not
147 fect and induce maturation of human CD11c(+) myeloid dendritic cells (MDCs) and CD123(+) plasmacytoid
148 he main activities of TSLP are activation of myeloid dendritic cells (mDCs) and modulation of cytokin
149 d the cytokine responses of peripheral blood myeloid dendritic cells (mDCs) and monocytes to in vitro
150 -induced changes in gene expression in human myeloid dendritic cells (mDCs) and monocytes, identify s
151 ly TLR7/8-L and TLR9-L induced activation of myeloid dendritic cells (mDCs) and plasmacytoid DCs (pDC
152 rus and RSV mobilize immune cells, including myeloid dendritic cells (mDCs) and plasmacytoid dendriti
154 that is characterized by reduced numbers of myeloid dendritic cells (mDCs) and Th17 cells in the CNS
157 improves the functional capacity of classic myeloid dendritic cells (mDCs) by altering expression of
158 VEE) exhibited enhanced infection of primary myeloid dendritic cells (mDCs) compared to mammalian-cel
162 ow that a fraction of blood monocyte-derived myeloid dendritic cells (MDCs) express B7-H1, a member o
164 late carbon black (nCB) accumulates in human myeloid dendritic cells (mDCs) from emphysematous lung a
166 t plasmacytoid predendritic cells (pDCs) and myeloid dendritic cells (mDCs) have the functional plast
167 We found that miR-22 was upregulated in lung myeloid dendritic cells (mDCs) of smokers with emphysema
168 lood dendritic cell antigen-1(+) (BDCA-1(+)) myeloid dendritic cells (mDCs) present during BKPyVAN (o
169 - and STAT6-dependent expansion of recipient myeloid dendritic cells (MDCs) that induce contact-depen
170 intracellular double-stranded DNA (dsDNA) in myeloid dendritic cells (mDCs) that triggers a type I in
173 of CD4 T, CD8 T, B, and NK cells, monocytes, myeloid dendritic cells (mDCs), and plasmacytoid dendrit
174 production of the chemoattractant CXCL16 by myeloid dendritic cells (mDCs), causing subsequent recru
177 el (ESM), and mouse bone marrow (BM)-derived myeloid dendritic cells (mDCs), T cells (TCs), B cells,
178 s of SHFV infection, macrophages (MPhis) and myeloid dendritic cells (mDCs), were differentiated from
179 (alpha/beta interferon [IFN-alpha/beta]) in myeloid dendritic cells (mDCs), where the mosquito cell-
181 eceptor is selectively expressed by BDCA3(+) myeloid dendritic cells (mDCs), which have been proposed
182 istently diminished H3K27ac in monocytes and myeloid dendritic cells (mDCs), which was associated wit
190 i.e., B cells, macrophages, monocyte-derived/myeloid dendritic cells (MDDCs/mDCs), and by plasmacytoi
191 8+ regulatory T cells significantly suppress myeloid dendritic cell-mediated tumor-associated antigen
192 cells were observed in most CD11c(+)CD103(+) myeloid dendritic cells migrating to mediastinal drainin
193 the innate response identified monocytes and myeloid dendritic cells (MoDC) as principal responders t
194 onRI on plasmacytoid dendritic cells (pDCs), myeloid dendritic cells, monocytes, and basophils was as
195 y, TLR8 agonists directly activated purified myeloid dendritic cells, monocytes, and monocyte-derived
197 define expression of CD200R on macrophages, myeloid dendritic cells, natural killer cells, and T cel
199 essed specifically on human pDCs, but not on myeloid dendritic cells or other peripheral blood leukoc
201 d activation of primary immune cells such as myeloid dendritic cells, plasmacytoid dendritic cells, a
203 memory CD4 T cells and HLA-DR expression by myeloid dendritic cells, pointing toward defective cross
208 es demonstrated that IFN-alpha modulated the myeloid dendritic cell response pattern by upregulating
209 ased CD11b expression on a subset of splenic myeloid dendritic cells, resulting in compromised recrui
210 results demonstrated that A(H3N2)vpM-induced myeloid dendritic cells secreted significantly lower lev
211 st to classical contact dermatitis, in which myeloid dendritic cells sense haptens, pDCs are primary
212 itic cells and our observed results in human myeloid dendritic cells, show that DEFB103 significantly
213 tions through interleukin-10; (c) repetitive myeloid dendritic cell stimulation can recover CD8+ regu
214 6C+ population comprised primarily of mature myeloid dendritic cells, suggesting that the underlying
215 ) cytokine response and expanded a subset of myeloid dendritic cells that expressed a CD11c(high)CD8a
216 SE significantly increased the proportion of myeloid dendritic cells that produced tumor necrosis fac
217 accumulation of bone marrow-derived immature myeloid dendritic cells that recruit activated lymphocyt
218 of cathepsin E message and protein in human myeloid dendritic cells, the preeminent APCs of the immu
219 ved dendritic cells (DCs) and CD23+ (FceRII) myeloid dendritic cells through the action of allergen-s
220 e the function of monocytes, macrophages and myeloid dendritic cells through the action of haemozoin
222 )CD8(-)CD209a(+)) and human (CD1c(+)CD19(-)) myeloid dendritic cells (TIDC), an innate immune cell ty
224 thermore, imiquimod-mediated increase in the myeloid dendritic cells, TNF/inducible nitric oxide synt
225 irpin RNA efficiently reduced the ability of myeloid dendritic cells to produce IFN-beta, IL-6, and T
229 sion of CCL21 were oppositely regulated, and myeloid dendritic cells upregulated CCR7 expression.
230 und that an early immune response cell type, myeloid dendritic cells, was critical for protection in
232 ls, plasmacytoid dendritic cells (pDCs), and myeloid dendritic cells were characterized by flow cytom
233 more, several genes associated with immature myeloid dendritic cells were overexpressed in LCH CD207(
235 uman myeloid cell frequencies, specifically, myeloid dendritic cells, were elevated in the bone marro
236 found in isolated activated macrophages and myeloid dendritic cells, were widely distributed in all
237 e expression of FcepsilonRIalpha on pDCs and myeloid dendritic cells when compared with that seen in
238 th HIV-1, elite controllers have circulating myeloid dendritic cells with significantly increased ant