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1 e Ca(2+)-releasing intracellular messenger d-myo-inositol 1,4,5-trisphosphate [1, Ins(1,4,5)P(3)] are
2 ates, in particular via the second messenger myo-inositol 1,4,5-trisphosphate, and phosphoinositides
3 e of large cholangiocytes by activation of D-myo-inositol 1,4,5-trisphosphate/Ca(2+) /calmodulin-depe
4 locked by neomycin, and is not affected by D-myo-inositol 1,4,5-trisphosphate (D-IP3).
5      A regioisomer of the second messenger D-myo-inositol 1,4,5-trisphosphate [D-Ins(1,4,5)P(3), 1],
6 ability of human Arf1.GDP (Arf1.GDP) to bind myo-inositol (1,4,5)-trisphosphate (I(1,4,5)P(3)), the s
7 itol 1,3,4-trisphosphate (Ins(1,3,4)P3) or D-myo-inositol 1,4,5-trisphosphate (Ins(1,4,5)P3) over 80-
8 ic activity partial agonists at the platelet myo-inositol 1,4, 5-trisphosphate [Ins(1,4,5)P(3)] recep
9  Ca(2+)-mobilizing intracellular messenger d-myo-inositol 1,4,5-trisphosphate [Ins(1,4,5)P(3)].
10 brevicompactum, are potent agonists at the D-myo-inositol-1,4,5-trisphosphate [Ins(1,4,5)P3] receptor
11 platelets but was 40-fold less potent than D-myo-inositol-1,4,5-trisphosphate [Ins(1,4,5)P3].
12                                            D-myo-inositol 1,4,5-trisphosphate (InsP(3)) is a fundamen
13 ine)-mediated enhancement of intracellular D-myo-inositol 1,4,5-trisphosphate (IP(3)) and Ca(2+) accu
14 t inhibition of endoplasmic reticulum (ER) d-myo-inositol 1,4,5-trisphosphate (IP(3))-gated calcium r
15                           2'-P-cADPR and 1-D-myo-inositol 1,4,5-trisphosphate (IP3) appear to act by
16           Liposomes filled with 100 microM D-myo-inositol 1,4,5-trisphosphate (IP3) enhanced the MEPP
17                 Intracellular application of myo-inositol 1,4,5-trisphosphate (IP3, 100 microM) from
18 igh affinity to intestinal SMC; stimulated D-myo-inositol-1,4,5-trisphosphate (IP3) formation, Ca2+ r
19 llular activity of phospholipase C-gamma, by myo-inositol 1,4,5-trisphosphate radioreceptor assay, de
20 phostin A (AdA) is a potent agonist of the d-myo-inositol 1,4,5-trisphosphate receptor (Ins(1,4,5)P3R
21 siently transfected with type I and type III myo-inositol 1,4,5-trisphosphate receptor (IP(3)R) isofo
22                                              myo-Inositol 1,4,5-trisphosphate receptor (IP3R) degrada
23 ate (8), two analogues of the superpotent 1D-myo-inositol 1,4,5-trisphosphate receptor agonist adenop
24 hostin A (AdA), the most potent agonist of d-myo-inositol 1,4,5-trisphosphate receptors (IP(3)R), is
25  down-regulation of both type I and type III myo-inositol 1,4,5-trisphosphate receptors (IP3Rs).
26 henylborate, a cell-permeant antagonist of D-myo-inositol 1,4,5-trisphosphate receptors, abrogates an
27 erimental data indicate that together with d-myo-inositol 1,4,5-trisphosphate receptors, RyRs regulat
28                     The response to KCl or D-myo-inositol 1,4, 5-trisphosphate was not different betw
29 pase C products, namely diacylglycerol and d-myo-inositol 1,4,5-trisphosphate, were identified as pro
30   A variety of inositol phosphates including myo-inositol 1,4,5-trisphosphate, which is a secondary m