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1 centrations in advanced heart failure impair myocardial contractility.
2 al inducible nitric oxide synthase decreases myocardial contractility.
3 t-mediated glutathione efflux and maintained myocardial contractility.
4 lase activity and an increased inhibition of myocardial contractility.
5  significant changes in oxygen extraction or myocardial contractility.
6 now understood to be a critical repressor of myocardial contractility.
7 meric proteins are important for fine-tuning myocardial contractility.
8 s, including blood pressure, heart rate, and myocardial contractility.
9 ating behavior, amplifying Ca(2+) influx and myocardial contractility.
10 tion is inherently independent of underlying myocardial contractility.
11 expression in the human RV and its impact on myocardial contractility.
12 he RV and the impact of this upregulation on myocardial contractility.
13 artner phospholamban (PLN) are essential for myocardial contractility.
14 ulating intracellular Ca(2+) homeostasis and myocardial contractility.
15 yroid hormones play a key role in modulating myocardial contractility.
16  concentration, plasma oncotic pressure, and myocardial contractility.
17 s by which activated p38alpha MAPK depresses myocardial contractility.
18  whether Arg inhibition would increase basal myocardial contractility.
19 n analysis allows quantitative assessment of myocardial contractility.
20 d is therefore of interest as a regulator of myocardial contractility.
21 n interactions that could lead to diminished myocardial contractility.
22 uppression of dobutamine-induced increase in myocardial contractility.
23 neovascularization and improve perfusion and myocardial contractility.
24 g the cardiac myosin heavy chain to increase myocardial contractility.
25 Ca2+ release, leading to variable effects on myocardial contractility.
26 eart development and is important for normal myocardial contractility.
27 nificant role in the autocrine regulation of myocardial contractility.
28 O in oxidative phosphorylation and, in turn, myocardial contractility.
29 ding effects on the systemic circulation and myocardial contractility.
30 CSAR activation in the normal state enhances myocardial contractility.
31 ements of endocardial voltage potentials and myocardial contractility.
32  and volume, which defines a single state of myocardial contractility.
33 coplasmic reticulum Ca2+-ATPase activity and myocardial contractility.
34 estation, with no significant alterations in myocardial contractility.
35 pigs showed significantly better recovery of myocardial contractility 3 months after infarction injur
36 inine (L-NNA) on free radical generation and myocardial contractility after ischemia-reperfusion.
37                                              Myocardial contractility and blood flow provide essentia
38 ction due to the effect of V1a activation on myocardial contractility and cell growth.
39                                We found that myocardial contractility and CRC activity were decreased
40 at TLR2 signaling contributes to the loss of myocardial contractility and cytokine production in the
41 ibitors are cardiotonic agents that increase myocardial contractility and decrease vascular smooth mu
42 metabolic abnormalities, including decreased myocardial contractility and decreased plasma ionized ca
43    -Cytokine-induced NO production depresses myocardial contractility and has been shown to be cytoto
44 t ventricular cardiac myocytes as a model of myocardial contractility and in whole blood from childre
45 osis, ventricular arrhythmias, and decreased myocardial contractility and left ventricular pressure.
46  HF is characterized by a marked decrease in myocardial contractility and loss of pump function.
47 importance of acidic N' region in regulating myocardial contractility and mediating the response of t
48  troponin I (cTnI) phosphorylation modulates myocardial contractility and relaxation during beta-adre
49                                              Myocardial contractility and relaxation were continuousl
50       To study the effects of tachycardia on myocardial contractility and relaxation, we evaluated th
51 ic receptors is pivotal in the regulation of myocardial contractility and remodeling.
52 n of Sca-1 causes primary cardiac defects in myocardial contractility and repair consistent with impa
53 espite their importance in the regulation of myocardial contractility and rhythm.
54 ures of dilated cardiomyopathy (DCM) are low myocardial contractility and risk of thromboembolism.
55                                 Decreases in myocardial contractility and shortening of ventricular s
56 ic oxide (NO) modulates autonomic effects on myocardial contractility and sinus and atrioventricular
57 ed for an ESHP platform capable of assessing myocardial contractility and suggests that metabolic par
58 re overload-induced hypertrophy and improved myocardial contractility and systolic function in PTP1B
59 study the rate related effects of sotalol on myocardial contractility and to test the hypothesis that
60                                     Regional myocardial contractility and wall thickness were used in
61 s associated with significant improvement in myocardial contractility, and a fall in ATP delivery, su
62 lves significantly improved cardiac preload, myocardial contractility, and cardiac output, without ch
63 ure associated with lower ejection fraction, myocardial contractility, and greater force developed by
64 I alters biventricular systolic function, RV myocardial contractility, and LV diastolic performance.
65  and results in enhanced calcium transients, myocardial contractility, and relaxation that may have f
66                               and diminished myocardial contractility are important factors leading t
67 LV strain/strain rate, surrogate measures of myocardial contractility, are reduced in pediatric PH an
68 lic stresses and heart rate increased, while myocardial contractility, as reflected by LV dP/dt and m
69               We find that OM only increases myocardial contractility at submaximal Ca(2+) activation
70 red hPSC-CMs as powerful models for studying myocardial contractility at the cellular level.
71 gh beta-adrenergic stimuli are essential for myocardial contractility, beta-blockers have a proven be
72 les (blood pressure, stroke volume [SV], and myocardial contractility), but the relative strength and
73 mprovements in functional parameters such as myocardial contractility by echocardiography, perfusion
74                 Novel myotrope TA1 increased myocardial contractility by sensitizing the sarcomere to
75 95% CI: 0.46-0.70]) and local differences in myocardial contractility, compliance, and activation del
76 acterized by chamber enlargement and reduced myocardial contractility, decreases in beta-adrenergic r
77                        Epinephrine increases myocardial contractility, decreases the duration of syst
78 ecreasing CCI was also associated with lower myocardial contractility defined using global radial and
79                                              Myocardial contractility depends on Ca2+ release from an
80  LVDP: 1767.3 +/- 929.5 vs. 492.7 +/- 308.1; myocardial contractility, dP/dt(max) : 2748.9 +/- 1514.9
81 tions were used to track changes in regional myocardial contractility, geometry, and perfusion.
82 he effects of veno-venous ultrafiltration on myocardial contractility in children undergoing cardiopu
83                          It had no effect on myocardial contractility in isolated mouse cardiac myocy
84      Microtubule depolymerization normalized myocardial contractility in papillary muscles of PAB cat
85 nk coronary vascular dysfunction to abnormal myocardial contractility in patients with BD.
86        Nitric oxide inhibits beta-adrenergic myocardial contractility in patients with heart failure.
87 ed acidic N' region results in a decrease in myocardial contractility in the cTnI(Delta2-11) mice dem
88        We conclude that SMT is beneficial to myocardial contractility in this model of endotoxemia, w
89                                              Myocardial contractility in transgenic mice, as assessed
90 amban is necessary for optimal regulation of myocardial contractility in vivo.
91  mediating its optimal regulatory effects on myocardial contractility in vivo.
92                            Insulin can alter myocardial contractility, in part through an effect on t
93 wditch 139 years ago as the observation that myocardial contractility increases proportionally with i
94                                         Poor myocardial contractility is a very important issue in vi
95                                              Myocardial contractility is constantly changing from bea
96                                      Whether myocardial contractility is enhanced to contribute to th
97                                              Myocardial contractility is generally believed to be inc
98 t a volatile anaesthetic-induced decrease in myocardial contractility is mediated by a reduction in i
99 annel (CRC) activity is a mechanism by which myocardial contractility is reduced in endotoxemia; b) t
100 C) has been recognized as a key regulator of myocardial contractility, little is known about its mech
101                                  We assessed myocardial contractility, myocardial nitric oxide conten
102                     PPA had little effect on myocardial contractility of normal hearts until the high
103 e systolic dysfunction results from impaired myocardial contractility or altered loading conditions i
104                Hypothermia does not decrease myocardial contractility or induce hypotension if hypovo
105 not develop hypoxia, acidosis, alteration in myocardial contractility, or arrhythmias.
106 formation imaging might more closely reflect myocardial contractility than traditional measures of sy
107 serve), diastolic function (compliance), and myocardial contractility (the slope of the relationship
108 and regulate cardiac performance, from acute myocardial contractility to chronic gene expression and
109         The aim of this work was to evaluate myocardial contractility using the end-systolic wall str
110 mited, diastolic function was preserved, and myocardial contractility was altered (Emax=2.6+/-0.3 mm
111                                              Myocardial contractility was followed up by cardiac magn
112                                              Myocardial contractility was inversely determined from p
113 mited, ventricular compliance decreased, and myocardial contractility was preserved.
114 mice, echocardiographic analysis showed that myocardial contractility was reduced to 14 +/- 1% of con
115            Because NOS1 positively modulates myocardial contractility, we determined whether Arg inhi
116 y increased glutathione efflux and decreased myocardial contractility when compared with control anim
117 sses unique inotropic properties, increasing myocardial contractility while simultaneously reducing c
118 l evaluate the potential impact of increased myocardial contractility with omecamtiv mecarbil on mult
119 n femoral resistance arteries, and increased myocardial contractility with sympathetic dominance.
120 mendan is a calcium sensitizer that enhances myocardial contractility without increasing myocardial o
121 al application of inotropic compounds drives myocardial contractility without systemic side effects.

 
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