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1 n coupling, and disrupts the coordination of myocardial contraction.
2 dium to extract a metric that reflects local myocardial contraction.
3 signal generated in the atria and ends with myocardial contraction.
4 propagates electrical impulses to coordinate myocardial contraction.
5 index (SWI) and MRI radial strain quantified myocardial contraction.
6 the regulation of basal and beta-adrenergic myocardial contraction.
7 ties in the heart that would directly affect myocardial contraction.
8 lial cells release substances that influence myocardial contraction.
9 es acutely, indicating diminished economy of myocardial contraction.
10 s in genes coding for proteins essential for myocardial contraction.
11 enes coding for proteins essential in normal myocardial contraction.
12 gnitude of Ca(2+)-induced Ca(2+) release and myocardial contraction.
13 echanism of anesthetic-induced depression of myocardial contraction.
14 ssential chemical energy source that governs myocardial contraction.
16 the cushions expanding in a wave during peak myocardial contraction and becoming undetectable during
17 vely spliced cTnT variants may desynchronize myocardial contraction and contribute to the pathogenesi
18 myocardial perfusion are related to delayed myocardial contraction and greater extent of dyssynchron
19 tive gating method, to simultaneously access myocardial contraction and intracardiac blood flow at 20
20 e found that overexpressing SERCA2a enhances myocardial contraction and relaxation in normal mouse he
23 patients who died during SE had identifiable myocardial contraction bands compared with 5 of 22 contr
26 t muscle cells associated with activation of myocardial contraction by the N-terminal region of cMyBP
30 as despite significantly improved indexes of myocardial contraction (end-systolic pressure volume rel
34 indexed to body surface area (LVmassi, BSA), myocardial contraction fraction indexed to BSA (MCF(i))
35 itivities and high specificities (among them myocardial contraction fraction ratio had the highest ar
36 e area; systolic: ejection fraction, S', and myocardial contraction fraction) was investigated using
37 associated with lower ejection fraction and myocardial contraction fraction, respectively, but not i
39 ression alters the energy cost of increasing myocardial contraction in mouse hearts with pressure-ove
41 he heart rate (chronotropy), the strength of myocardial contraction (inotropy), and the rate of myoca
43 of the myofibrillar proteins, activation of myocardial contraction is thought to involve switch-like
45 To quantify ventricular size/shape, regional myocardial contraction, lateral shortening of the IPMD,
46 However, the initial proposal that depressed myocardial contraction leads to a 'compensatory' hypertr
47 re is associated with decreased longitudinal myocardial contraction measured by pulsed tissue Doppler
48 erformance beginning shortly after the first myocardial contractions occur in developing vertebrate e
49 ene blue to examine cGMP-mediated effects on myocardial contraction-relaxation, while other experimen
52 implications for the role of this protein in myocardial contraction, with particular relevance to bet