ライフサイエンスコーパス: myocardial perfusion

1 There was no change in myocardial perfusion.

2 reference standard for the quantification of myocardial perfusion.

3 [N]NH3 imaging was performed to evaluate myocardial perfusion.

4 ng SPECT to precisely quantify segment-level myocardial perfusion.

5 content, and increased vessel densities and myocardial perfusion.

6 e injection did not impair coronary flow and myocardial perfusion.

7 ta can lead to misinterpretation of regional myocardial perfusion.

8 D than in those without PTSD, denoting worse myocardial perfusion.

9 aine challenge evokes a sizeable decrease in myocardial perfusion.

10 multi-echo Dixon technique for quantitative myocardial perfusion.

11 integration of data on coronary anatomy and myocardial perfusion.

12 with low event rates in patients with normal myocardial perfusion.

13 onary arteries is the prerequisite of normal myocardial perfusion.

14 increased with the extent of abnormality of myocardial perfusion.

15 yocardial strain), coronary artery flow, and myocardial perfusion.

16 essure (DBP) to levels that could compromise myocardial perfusion.

17 opathy (HCM) and is associated with abnormal myocardial perfusion.

18 y showed visually concordant DE and regional myocardial perfusion abnormalities in 31 patients and ab

19 dynamic obstruction, presence and extent of myocardial perfusion abnormalities, and fibrosis.

20 3 DE-negative patients had (apical) regional myocardial perfusion abnormalities.

21 ents (17 men; 69+/-9 years) who had improved myocardial perfusion after the first injection but had r

22 n) stress, including 15 subjects with normal myocardial perfusion and 27 patients referred for corona

23 The imaging data of patients with normal myocardial perfusion and 30 patients with mid-sized to l

24 allowing routine, noninvasive assessment of myocardial perfusion and anatomy.

25 injection is associated with improvements in myocardial perfusion and anginal symptoms in patients wi

26 not been systemically validated for absolute myocardial perfusion and coronary flow reserve (CFR) by

27 Quantitative assessment in myocardial perfusion and determination of absolute myoca

28 ood cardiometabolic control, women had worse myocardial perfusion and diastolic function compared wit

29 provided incremental prognostic value beyond myocardial perfusion and ejection fraction data.

30 rteriolar density and significantly improved myocardial perfusion and endothelium-dependent vasorelax

31 tional and structural MVD had similar stress myocardial perfusion and exercise perfusion efficiency v

32 conducted vasodilation for a more efficient myocardial perfusion and improved left ventricle (LV) di

33 (STEMI) is common and results in suboptimal myocardial perfusion and increased infarct size.

34 o, emerging evidence indicates that impaired myocardial perfusion and inflammation secondary to multi

35 gadobutrol for detection of CAD by assessing myocardial perfusion and late gadolinium enhancement (LG

36 d MPS underwent rest and adenosine stress 3D myocardial perfusion and late gadolinium enhancement CMR

37 Myocardial perfusion and left ventricular ejection fract

38 Myocardial perfusion and left ventricular function were

39 obutrol-enhanced CMR (0.1 mmol/kg) to assess myocardial perfusion and LGE in adult patients with know

40 Absolute quantification of myocardial perfusion and MPR with PET have proven diagno

41 (CMR) and positron emission tomography (PET) myocardial perfusion and myocardial perfusion reserve (M

42 ess-induced and adenosine-induced changes in myocardial perfusion and neurohormonal activation in CHF

43 armacokinetic studies in mice by quantifying myocardial perfusion and oxygen consumption with (11)C-a

44 Left ventricular myocardial perfusion and sympathetic innervation were as

45 This study sought to assess myocardial perfusion and tissue oxygenation during vasod

46 sitive patients demonstrated normal regional myocardial perfusion, and 3 DE-negative patients had (ap

47 levels, left ventricular ejection fraction, myocardial perfusion, and adverse events.

48 ise tolerance, and antianginal medications), myocardial perfusion, and clinical end points.

49 t of myocardial edema, myocardial siderosis, myocardial perfusion, and diffuse myocardial fibrosis.

50 scar, halting adverse remodeling, increasing myocardial perfusion, and improving hemodynamic status a

51 ance (cine, T2* iron, vasodilator first pass myocardial perfusion, and late gadolinium enhancement im

52 s is a cause of cardiac dysfunction, reduced myocardial perfusion, and ultimately heart failure.

53 ry angina is associated with improvements in myocardial perfusion, anginal complaints, and quality of

54 Small-scale studies have shown that myocardial perfusion assessed by SonoVue-enhanced MCE is

55 Myocardial perfusion assessed using single-photon emissi

56 ys: detection of ischemia (often with stress myocardial perfusion at SPECT, PET, and cardiac MRI) and

57 (A), microvascular flow velocity (beta), and myocardial perfusion (Axbeta).

58 f the physiology of coronary circulation and myocardial perfusion; (b) describe the technical prerequ

59 I approach for reliably examining changes in myocardial perfusion between rest and adenosine stress.

60 Knowledge on sex differences in myocardial perfusion, blood volume (MBV), and extracellu

61 ascularization) and quantitative measures of myocardial perfusion by [(13)N] ammonia positron emissio

62 ocker therapy has been also shown to improve myocardial perfusion by enhancing neoangiogenesis in the

63 CAD, regional, artery-specific, quantitative myocardial perfusion by PET, coronary revascularization

64 Myocardial perfusion cardiac MR imaging during CPT can a

65 First-pass myocardial perfusion cardiovascular magnetic resonance (

66 sed to guide revascularization: one involves myocardial-perfusion cardiovascular magnetic resonance i

67 nd risk factors for coronary artery disease, myocardial-perfusion cardiovascular MRI was associated w

68 om nonischemic cardiomyopathy; evaluation of myocardial perfusion; characterization of hypertrophic c

69 3D whole heart myocardial perfusion CMR accurately detects functionally

70 Vasodilator stress and rest myocardial perfusion CMR and LGE imaging had high diagno

71 3D myocardial perfusion CMR and MPS agreed in 38 of the 45

72 ent rest and adenosine stress 3D whole heart myocardial perfusion CMR at 3-T.

73 3D myocardial perfusion CMR is an alternative to MPS for de

74 3D whole heart myocardial perfusion CMR overcomes the limited spatial c

75 In this multicenter study, 3D myocardial perfusion CMR proved highly diagnostic for th

76 s to assess the diagnostic performance of 3D myocardial perfusion CMR to detect functionally relevant

77 s study was to compare ischemic burden on 3D myocardial perfusion CMR with (99m)Tc-tetrofosmin MPS.

78 More recently developed 3-dimensional (3D) myocardial perfusion CMR, however, provides whole-heart

79 ation of abnormalities including border zone myocardial perfusion, contractile dysfunction, and LV wa

80 blished the feasibility of performing stress myocardial perfusion CT imaging in small groups of patie

81 roups of patients and have shown that stress myocardial perfusion CT in combination with CT coronary

82 se determinants of myocardial oxygen demand, myocardial perfusion decreased by 30% (103.7+/-9.8 to 75

83 Myocardial perfusion decreased significantly during HD a

84 R) is typically based on induction of either myocardial perfusion defect or wall motion abnormality.

85 tion fraction, risk area (before treatment), myocardial perfusion defect over time (infarct size), an

86 I and CTA data may facilitate correlation of myocardial perfusion defects and subtending coronary art

87 c stress in eliciting clinically significant myocardial perfusion defects in CHF patients.

88 ry artery luminal stenosis and corresponding myocardial perfusion deficits in patients with suspected

89 In contrast, regions of reduced myocardial perfusion delineated by cardiac ASL were able

90 At baseline, myocardial perfusion differed between the MI core (media

91 ents in CT technology allow CT evaluation of myocardial perfusion during vasodilator stress, thereby

92 in were determined by quantitative real-time myocardial perfusion echocardiography and speckle tracki

93 amics and infarct size obtained by real-time myocardial perfusion echocardiography and their value in

94 Diagnostic performance of quantitative myocardial perfusion estimates is not affected by the tr

95 nctional MR images and dynamic assessment of myocardial perfusion from transit of intravascular contr

96 magnetic resonance for routine assessment of myocardial perfusion, function, and late gadolinium enha

97 Radionuclide imaging of myocardial perfusion, function, and viability has been e

98 Myocardial perfusion gated SPECT performed 1 month after

99 The incremental prognostic value of myocardial perfusion-gated single photon emission comput

100 0.019), independently of CFR<=2.0, RRR<=1.7, myocardial perfusion grade<=1, and Thrombolysis in Myoca

101 as compared with coronary flow reserve, TIMI myocardial perfusion grade, and clinical variables.

102 y artery disease, given concerns for reduced myocardial perfusion if diastolic blood pressure is too

103 Conclusion: Myocardial perfusion images from D.SPECT are enhanced fo

104 Consequently, conventional myocardial perfusion images obtained from whole cardiac

105 SPECT and PET myocardial perfusion images show greater myocardial inte

106 fusion (CTP) with cardiac magnetic resonance myocardial perfusion imaging (CMR-Perf) for detection of

107 value of positron emission tomography (PET) myocardial perfusion imaging (MPI) and the improved clas

108 CT angiography (CTA) and SPECT myocardial perfusion imaging (MPI) are complementary ima

109 ve patients undergoing adenosine stress-rest myocardial perfusion imaging (MPI) by (99m)Tc-tetrofosmi

110 ate use criteria recommend performing stress myocardial perfusion imaging (MPI) for intermediate- to

111 -photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) has changed over time

112 -photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) have shown a survival

113 Cardiovascular magnetic resonance (CMR) myocardial perfusion imaging (MPI) is a state-of-the-art

114 Myocardial perfusion imaging (MPI) is well established i

115 The performance of SPECT myocardial perfusion imaging (MPI) may deteriorate in sm

116 Positron emission tomography (PET) myocardial perfusion imaging (MPI) offers technical bene

117 Combined analysis of SPECT myocardial perfusion imaging (MPI) performed with a soli

118 Radionuclide myocardial perfusion imaging (MPI) plays a vital role in

119 consecutive patients underwent (82)Rubidium myocardial perfusion imaging (MPI) positron emission tom

120 Although SPECT myocardial perfusion imaging (MPI) provides valuable inf

121 segmentation of the left ventricle for SPECT myocardial perfusion imaging (MPI) quantification often

122 lity of a new protocol, IQ SPECT, to acquire myocardial perfusion imaging (MPI) studies in a quarter

123 photon emission computed tomography (SPECT) myocardial perfusion imaging (MPI) underwent a comprehen

124 We sought to evaluate the accuracy of myocardial perfusion imaging (MPI) using cadmium-zinc-te

125 easibility of attenuation correction (AC) of myocardial perfusion imaging (MPI) with a virtual unenha

126 Hybrid PET myocardial perfusion imaging (MPI) with CT allows the in

127 artery calcium score (CACS) as an adjunct to myocardial perfusion imaging (MPI) with SPECT for cardia

128 Myocardial perfusion imaging (MPI) with SPECT is a well-

129 impact of appropriate use criteria (AUC) for myocardial perfusion imaging (MPI) with SPECT on the est

130 mance for positron emission tomography (PET) myocardial perfusion imaging (MPI) with Tc-99m single-ph

131 The prognostic value of myocardial perfusion imaging (MPI) with the cadmium-zinc

132 -photon emission computed tomography (SPECT)-myocardial perfusion imaging (MPI), a technique that is

133 sensitivity has facilitated fast or low-dose myocardial perfusion imaging (MPI), and early dynamic im

134 tunity to lower the injected doses for SPECT myocardial perfusion imaging (MPI), but the exact limits

135 CCTA or radionuclide stress myocardial perfusion imaging (MPI).

136 Rotenone derivatives have shown promise in myocardial perfusion imaging (MPI).

137 but not in symptomatic patients referred for myocardial perfusion imaging (MPI).

138 r single-photon emission computed tomography myocardial perfusion imaging (MPI).

139 t advantage of PET over conventional nuclear myocardial perfusion imaging (MPI).

140 h single-photon emission computed tomography myocardial perfusion imaging (SPECT MPI) has improved th

141 t single-photon emission computed tomography-myocardial perfusion imaging (SPECT-MPI) has high predic

142 ween October 2004 and September 2011 who had myocardial perfusion imaging after negative troponin T t

143 own CAD underwent prospectively simultaneous myocardial perfusion imaging and CAC scoring on a hybrid

144 2051 patients who underwent exercise stress myocardial perfusion imaging and echo (5.5+/-7.9 days),

145 The diagnostic accuracy of myocardial perfusion imaging and wall motion imaging wer

146 consecutive CRT recipients with radionuclide myocardial perfusion imaging before CRT between January

147 were Veteran patients who underwent nuclear myocardial perfusion imaging between December 2010 and J

148 +/- 11.8 years), patients were referred for myocardial perfusion imaging between May 2008 and Januar

149 computed tomographic angiography and stress myocardial perfusion imaging by single photon emission c

150 was to determine the diagnostic accuracy of myocardial perfusion imaging by single-photon emission c

151 Contrast material-enhanced myocardial perfusion imaging by using cardiac magnetic r

152 trocardiography, stress echocardiography, or myocardial perfusion imaging can reveal findings associa

153 Myocardial perfusion imaging had good diagnostic accurac

154 SPECT myocardial perfusion imaging has attained widespread cli

155 Heretofore, radionuclide myocardial perfusion imaging has been primarily qualitat

156 h-spatial-resolution cardiovascular MR (CMR) myocardial perfusion imaging has been shown to be clinic

157 Myocardial perfusion imaging has limited sensitivity for

158 Myocardial perfusion imaging has long been used off labe

159 single-photon emission computed tomographic myocardial perfusion imaging improved from a summed stre

160 high-resolution and standard-resolution CMR myocardial perfusion imaging in patients with suspected

161 Stress myocardial perfusion imaging is a noninvasive alternativ

162 Regadenoson (82)Rb myocardial perfusion imaging is accurate for the detecti

163 Exercise CT myocardial perfusion imaging is feasible and accurate fo

164 Quantitative myocardial perfusion imaging is increasingly used for th

165 The yield of myocardial perfusion imaging is low in contemporary pati

166 is of myocardial dynamic computed tomography myocardial perfusion imaging lacks standardization.

167 atic analysis of dynamic computed tomography myocardial perfusion imaging may permit robust discrimin

168 h BMI >/= 40 kg/m(2) should be scheduled for myocardial perfusion imaging on a conventional SPECT cam

169 ial blood flow as assessed by stress-induced myocardial perfusion imaging or a significant fall in di

170 The (13)NH(3) rest/stress myocardial perfusion imaging procedure can be compressed

171 ought to determine whether changes in stress myocardial perfusion imaging protocols and camera techno

172 (82)Rb myocardial perfusion imaging protocols were implemented

173 significantly higher in patients with normal myocardial perfusion imaging results (6.5% +/- 5.4%) tha

174 econdary outcome was a comparison of nuclear myocardial perfusion imaging results and frequency of is

175 Site scoring of (82)Rb PET myocardial perfusion imaging scans was found to be in go

176 Flurpiridaz PET myocardial perfusion imaging shows promise as a new trac

177 -photon emission computed tomography (SPECT) myocardial perfusion imaging studies among patients with

178 ce radiation exposure to patients undergoing myocardial perfusion imaging studies, especially when co

179 ests a reference range of TID for (82)Rb PET myocardial perfusion imaging that is in the range of pre

180 were followed for 6 months after their index myocardial perfusion imaging to determine subsequent rat

181 s a novel positron emission tomography (PET) myocardial perfusion imaging tracer.

182 ombined Noninvasive Coronary Angiography and Myocardial Perfusion Imaging Using 320-Detector Row Comp

183 act of increased body mass on the quality of myocardial perfusion imaging using a latest-generation g

184 n spent nuclear fuel cycle as well as toward myocardial perfusion imaging utilizing (82)Sr/(82)Rb iso

185 CT myocardial perfusion imaging was performed within 1 minu

186 single-photon emission computed tomographic myocardial perfusion imaging were included.

187 g coronary angiogram within 4 mo after SPECT myocardial perfusion imaging were reviewed.

188 adenosine-stress dynamic computed tomography myocardial perfusion imaging with a second-generation du

189 Stress myocardial perfusion imaging with MRI, computed tomograp

190 Myocardial perfusion imaging with RTMCE may improve the

191 le myocardial ischemia was adjudicated using myocardial perfusion imaging with single-photon emission

192 tery disease (CAD) is ambiguous, but nuclear myocardial perfusion imaging with single-photon emission

193 onary artery calcium (CAC) scoring on top of myocardial perfusion imaging with single-photon emission

194 This prospective randomized study assessed myocardial perfusion imaging with the high-sensitivity D

195 We hypothesized that myocardial perfusion imaging would be low yield with lim

196 CT-FFR, CT myocardial perfusion imaging, and transluminal attenuati

197 We assessed the incidence of abnormal myocardial perfusion imaging, coronary angiography, reva

198 Myocardial perfusion imaging, including positron emissio

199 For gated SPECT myocardial perfusion imaging, when relative activity dis

200 SPECT is most commonly used for clinical myocardial perfusion imaging, whereas PET is the clinica

201 nd software for positron emission tomography myocardial perfusion imaging, which has advanced it from

202 iography, stress echocardiography, or stress myocardial perfusion imaging.

203 on rest/stress positron emission tomography myocardial perfusion imaging.

204 1 (Tl-201) as an alternative radiotracer for myocardial perfusion imaging.

205 ry angiography and single-photon emission CT myocardial perfusion imaging.

206 tive to pharmacologic stress or exercise for myocardial perfusion imaging.

207 dial infarction showed high concordance with myocardial perfusion in matched territories as revealed

208 s of angina, relevant clinical outcomes, and myocardial perfusion in patients with refractory angina.

209 on, resulting in significant improvements in myocardial perfusion in the setting of chronic ischemia.

210 Infarction occurs when myocardial perfusion is interrupted for prolonged period

211 xygen level-dependent (BOLD) cardiac MRI for myocardial perfusion is limited by inadequate spatial co

212 he early postinfarction period when regional myocardial perfusion is often severely compromised.

213 Myocardial perfusion, MBV, and ECV are higher in female

214 y late gadolinium enhancement (LGE) MRI, and myocardial perfusion/metabolism was evaluated by (99m)Tc

215 onance (CMR) with conventional 2-dimensional myocardial perfusion methods is limited by incomplete ca

216 ], wall motion abnormalities [WMA], abnormal myocardial perfusion, microvascular obstruction, late ga

217 The addition of myocardial perfusion (MP) imaging during dipyridamole re

218 one arterial input function plane and three myocardial perfusion (MP) planes per heartbeat in patien

219 was to determine the clinical presentation, myocardial perfusion on provocative stress testing, and

220 No differences in myocardial perfusion or adverse events were observed bet

221 criteria for identifying areas of decreased myocardial perfusion or for assessing tissue viability f

222 or significantly altering quantification of myocardial perfusion or LV function.

223 ary efficacy end point was change in resting myocardial perfusion over 6 months.

224 ardial flow reserve (MFR) with (13)N-ammonia myocardial perfusion PET have been implemented for clini

225 diagnostic performance of regadenoson (82)Rb myocardial perfusion PET imaging to detect obstructive c

226 ) underwent clinically indicated rest-stress myocardial perfusion PET with (82)Rb.

227 action (>=40%) at a clinical rest and stress myocardial perfusion PET/CT.

228 ronary angiography after stress testing with myocardial perfusion positron emission tomography and wi

229 years, 50.5% women) referred for rest/stress myocardial perfusion positron emission tomography scans

230 ronary angiography after stress testing with myocardial perfusion positron emission tomography were f

231 or suspected CAD with troponin before stress myocardial perfusion positron emission tomography were f

232 ield in sucrose (SUC) versus EtOH; P=0.004), myocardial perfusion (ratio of blood flow to the at-risk

233 Stress myocardial perfusion Rb-82 PET is a diagnostic alternati

234 l blood-derived mononuclear cells (PBMCs) on myocardial perfusion recovery by using cardiac magnetic

235 Myocardial perfusion reflects the macro- and microvascul

236 <2.0, 3.18+/-1.42 mm Hg/cm per second versus myocardial perfusion reserve >/=2.0, 2.24+/-1.19 mm Hg/c

237 with decreased myocardial blood flow on PET (myocardial perfusion reserve <2.0, 3.18+/-1.42 mm Hg/cm

238 Myocardial perfusion reserve (MPR) index was calculated

239 Quantification of myocardial perfusion reserve (MPR) is an emerging topic

240 on tomography (PET) myocardial perfusion and myocardial perfusion reserve (MPR) measurements in patie

241 nalysis for each method, with stress MBF and myocardial perfusion reserve (MPR) serving as continuous

242 mL/mg/min +/- 0.82, respectively (P < .001); myocardial perfusion reserve (MPR) was 2.4 +/- 0.82 (P <

243 MBF and myocardial perfusion reserve (MPR) were calculated for e

244 ce of stress myocardial blood flow (MBF) and myocardial perfusion reserve (MPR, the ratio of stress t

245 magnitude of change was proportional to the myocardial perfusion reserve (rho = 0.53; p < 0.01).

246 A myocardial perfusion reserve index (MPRI) (stress/rest s

247 Signal intensity change (SIDelta) and myocardial perfusion reserve index (MPRI) were measured

248 perfusion images was completed to determine myocardial perfusion reserve index (MPRI).

249 Patients with SCD also had a 21% lower myocardial perfusion reserve index than control subjects

250 nt) were evaluated for perfusion upslope and myocardial perfusion reserve index with Student t test a

251 The CMR myocardial perfusion reserve significantly outperformed

252 ention index to describe global and regional myocardial perfusion reserve using a dedicated solid-sta

253 pared with 22% of controls (P<0.001); global myocardial perfusion reserve was 2.01+/-0.41 and 2.68+/-

254 The CMR myocardial perfusion reserve was the only independent pr

255 inal pro-brain-type natriuretic peptide) and myocardial perfusion reserve were associated with the pr

256 Quantification of myocardial perfusion reserve with PET can assist in the

257 fusion cardiac magnetic resonance to measure myocardial perfusion reserve.

258 stress (peak) for the assessment of regional myocardial perfusion (rMP), left ventricular ejection fr

259 erences in the prognostic accuracy of stress myocardial perfusion rubidum-82 (Rb-82) positron emissio

260 derwent a comprehensive echocardiogram and a myocardial perfusion scintigraphy (MPS) at inclusion.

261 The extent and severity of ischemia on myocardial perfusion scintigraphy (MPS) is commonly used

262 e is progressive and recurring; thus, stress myocardial perfusion scintigraphy (MPS) is widely used t

263 Whether abnormal myocardial perfusion scintigraphy (MPS), dobutamine stre

264 yed to determine appropriateness ratings for myocardial perfusion scintigraphy (MPS), stress echocard

265 mean stenosis diameter 55%+/-11%), underwent myocardial perfusion scintigraphy for documentation of r

266 criminative value for myocardial ischemia on myocardial perfusion scintigraphy of all parameters was

267 There was no reduction in ischemic burden on myocardial perfusion scintigraphy or in the safety endpo

268 ying segments of greater perfusion defect on myocardial perfusion scintigraphy.

269 or mortality, the threshold of quantitative myocardial perfusion severity was analyzed for associati

270 atients with available rest and stress gated myocardial perfusion single-photon emission computed tom

271 probability with the prevalence of abnormal myocardial perfusion single-photon emission computed tom

272 Myocardial perfusion single-photon emission computed tom

273 ardiovascular magnetic resonance while using myocardial perfusion single-photon emission computed tom

274 Compared with myocardial perfusion single-photon emission computed tom

275 ween CE-SSFP and T2-STIR from this study and myocardial perfusion single-photon emission computed tom

276 improve the diagnostic accuracy of automatic myocardial perfusion SPECT (MPS) interpretation analysis

277 nuation-corrected (AC) and noncorrected (NC) myocardial perfusion SPECT (MPS) with the corresponding

278 easing concern about radiation exposure from myocardial perfusion SPECT (MPS).

279 myocardial wall motion and thickening during myocardial perfusion SPECT are typically assessed separa

280 % underwent both adenosine and mental stress myocardial perfusion SPECT on consecutive days.

281 In myocardial perfusion SPECT, transient ischemic dilation

282 f this study was to determine whether stress myocardial perfusion (SPECT) optimized with stress-only

283 rdized MD, 0.331; 95% CI, 0.08 to 0.55), and myocardial perfusion (standardized MD, -0.49; 95% CI, -0

284 DG uptake on torso PET scans is unrelated to myocardial perfusion status.

285 this incongruence when they interpret DE CT myocardial perfusion studies.

286 photon emission computed tomography (SPECT) myocardial perfusion study underwent coronary CT angiogr

287 alignment of coronary arterial segments and myocardial perfusion territories, designed for the CORE3

288 ariability in coronary anatomy and potential myocardial perfusion territory overlap.

289 Myocardial perfusion under vasodilator stress is impaire

290 , whereas CMR offers detailed assessments of myocardial perfusion, viability, and function.

291 The summed difference score for regional myocardial perfusion was also assessed.

292 Myocardial perfusion was assessed during rest, peak CPT,

293 Myocardial perfusion was assessed using stress imaging.

294 Myocardial perfusion was assessed with stress perfusion

295 dioxide (PetCO2) increased by 10 mm Hg, and myocardial perfusion was monitored with myocardial blood

296 Assessment of myocardial perfusion was performed using stress imaging.

297 Myocardial perfusion was quantified automatically for le

298 Myocardial perfusion was quantified using H2 (15)O PET.

299 CT scans of 10 patients with normal regional myocardial perfusion were analyzed.

300 mL/min/g; p = 0.03), whereas differences in myocardial perfusion were not statistically significant