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1  as a key mediator of RSPO-LGR4 signaling in myogenic differentiation.
2 requirement of LGR4 in RSPO signaling during myogenic differentiation.
3 and FGF signaling, thus resulting in reduced myogenic differentiation.
4 ta-catenin and TGF-beta signaling pivotal in myogenic differentiation.
5  a number of miRNAs, thereby contributing to myogenic differentiation.
6 bes under conditions that normally stimulate myogenic differentiation.
7 uration of myogenic microRNAs, thus favoring myogenic differentiation.
8 coma (RMS) and leiomyosarcoma (LMS), feature myogenic differentiation.
9  cilium and cessation of Hh signaling during myogenic differentiation.
10 ore identify miR-186 as a novel regulator of myogenic differentiation.
11 tion and proliferation and promoted terminal myogenic differentiation.
12 ession, functions as a positive regulator of myogenic differentiation.
13 feration, with the cells undergoing enhanced myogenic differentiation.
14 rum-derived factors, Mesp1 promotes skeletal myogenic differentiation.
15 ession rescues Map4k4-mediated inhibition of myogenic differentiation.
16 ion and Dock3 KO myoblasts are defective for myogenic differentiation.
17 t as a transcriptional repressor and inhibit myogenic differentiation.
18  which nonetheless did not prematurely enter myogenic differentiation.
19     On the other hand, Vps34 is required for myogenic differentiation.
20 roteins has not extensively been explored in myogenic differentiation.
21 oundly impairs MEF2 protein accumulation and myogenic differentiation.
22 2 myoblasts, they have opposing functions in myogenic differentiation.
23 otubes resulted in reduced ATP synthesis and myogenic differentiation.
24  in muscle precursor cells before and during myogenic differentiation.
25 gene expression consistently associated with myogenic differentiation.
26 ons that are yet unknown may be required for myogenic differentiation.
27 ls, but increased during the early stages of myogenic differentiation.
28 re, GPR39 siRNA reduced obestatin action and myogenic differentiation.
29 oss of Notch1 signaling leads to spontaneous myogenic differentiation.
30 miRNA with a previously demonstrated role in myogenic differentiation.
31 ll proliferation and the prevention of early myogenic differentiation.
32 rves as a regulator for preventing premature myogenic differentiation.
33 x is expressed in skeletal muscle throughout myogenic differentiation.
34 scle progenitor cell proliferation and delay myogenic differentiation.
35 rs and epigenetic enzymes cooperate to guide myogenic differentiation.
36  actin, indicative of malignant sarcoma with myogenic differentiation.
37  potential roles of the RSPO proteins during myogenic differentiation.
38 r its function in muscle gene expression and myogenic differentiation.
39  myogenic transcription factors that promote myogenic differentiation.
40 gesting that they are integral components of myogenic differentiation.
41 suggesting a major role for AS regulation in myogenic differentiation.
42 oxylase activity, led to repression of C2C12 myogenic differentiation.
43 blasts, and activating this pathway promoted myogenic differentiation.
44 ins that are involved in distinct aspects of myogenic differentiation.
45 s catalytically inactive mutant, potentiated myogenic differentiation.
46 recently reported to be downregulated during myogenic differentiation.
47 sary for full activation of p38 MAPK, during myogenic differentiation.
48 r205 persists on Pax3-FOXO1 throughout early myogenic differentiation.
49 n of the fusion protein changes during early myogenic differentiation.
50 associated kinase (ROCK1) that occurs during myogenic differentiation.
51 me that both NET25 and MAN1 are required for myogenic differentiation.
52 ecific role for mTORC2 signaling in terminal myogenic differentiation.
53 unction of NET37 appears to be important for myogenic differentiation.
54  and nucleosome positioning as a function of myogenic differentiation.
55 ing with the rat myocytes that induced their myogenic differentiation.
56 romoter of a key regulatory gene involved in myogenic differentiation.
57 tory factors, which plays a critical role in myogenic differentiation.
58 ls (NCSCs) in vivo and is required for their myogenic differentiation.
59 e for SUMO modification in the regulation of myogenic differentiation.
60 to reduce myoblast proliferation and promote myogenic differentiation.
61 ogenic precursor cell expansion but inhibits myogenic differentiation.
62 rmal cell proliferation without induction of myogenic differentiation.
63 atory regions controlling genes regulated by myogenic differentiation.
64  weak inducer of apoptosis (TWEAK) modulates myogenic differentiation.
65 his study suggested their role in preventing myogenic differentiation.
66 y reversed the inhibitory effect of TWEAK on myogenic differentiation.
67 gh different downstream pathways, to promote myogenic differentiation.
68 oss-talk between Notch and MEF2 to influence myogenic differentiation.
69 s sufficient for robust P2 responsiveness to myogenic differentiation.
70 riptional landscape of Twist2 binding during myogenic differentiation.
71 hermore, expression of Trim32 was induced in myogenic differentiation.
72 ic gene expression and sustained or enhanced myogenic differentiation.
73 ncement of cell survival, proliferation, and myogenic differentiation.
74 critical in muscle development and regulates myogenic differentiation.
75 y the role of Wnt-beta-catenin signalling in myogenic differentiation.
76 xpressed at the sarcolemma of myotubes after myogenic differentiation.
77 as BRD3 down-regulation resulted in enhanced myogenic differentiation.
78 anscription factor myogenin, which regulates myogenic differentiation.
79 reas siRNA-mediated knockdown of Ret induced myogenic differentiation.
80 ion protein 1 (MYOD1)-mediated activation of myogenic differentiation.
81  offer a useful resource for others studying myogenic differentiation.
82 a molecular mechanism by which 4.1R promotes myogenic differentiation.
83 o enhance cellular proliferation and inhibit myogenic differentiation.
84 oft tissue cancer characterized by disturbed myogenic differentiation.
85  detrimental effects of cachectic factors on myogenic differentiation.
86 a nanoscale stiffness range known to support myogenic differentiation.
87 inately regulate muscle-related genes during myogenic differentiation.
88 al regulators of muscle-related genes during myogenic differentiation.
89 m-dependent signalling pathways that control myogenic differentiation.
90 their myoblast counterparts, so is higher in myogenic differentiation.
91 re cultured in low-serum medium to stimulate myogenic differentiation.
92 y, up-regulates Myog expression and promotes myogenic differentiation.
93 nesis, indicating that Mdm2 is necessary for myogenic differentiation.
94  propose that ZBED6 plays a critical role in myogenic differentiation.
95 ted posttranscriptional switch that controls myogenic differentiation.
96                                          The myogenic differentiation 1 (MyoD) gene is a master regul
97  that quiescent MuSCs express high levels of Myogenic Differentiation 1 (MyoD) transcript in vivo, wh
98 -specific heterodimeric DNA-binding partner, myogenic differentiation 1 (MYOD).
99 s such as Myogenin, myosin heavy chains, and myogenic differentiation 1 (MyoD).
100 omoting myogenic lineage progression through myogenic differentiation 1 (Myod1) regulation.
101 lls to be positive for desmin, myogenin, and myogenic differentiation 1 (MyoD1).
102 al are integrally involved in the process of myogenic differentiation, acting as nodal regulators of
103 f-6 show a delay of Rac1 inactivation during myogenic differentiation and abnormal myotube formation.
104 rom Deltex2 knockout mice exhibit precocious myogenic differentiation and accelerated regeneration in
105 Pax7(Lo) subpopulation is enriched, enhances myogenic differentiation and accelerates muscle regenera
106 dicate roles for A-type lamins and emerin in myogenic differentiation and also suggest that these eff
107 ion indicates that Mesp-b acts by inhibiting myogenic differentiation and by inducing the dermomyotom
108 us, DUX4-mediated activation of Ret prevents myogenic differentiation and could contribute to FSHD pa
109 um response factor-dependent genes promoting myogenic differentiation and cytoskeletal organization.
110 al lethal, we explored its potential role in myogenic differentiation and development by generating a
111 GEFT is transcriptionally upregulated during myogenic differentiation and downregulated during adipog
112 -mfa-domain-containing) proteins function in myogenic differentiation and embryonic development by pa
113  role for GATA-4 and TAL1 to affect skeletal myogenic differentiation and EPO response via cross-talk
114 ) stimulates myoblast proliferation, induces myogenic differentiation and generates myocyte hypertrop
115 bnl3 expression is required for normal C2C12 myogenic differentiation and high-throughput sequencing
116 ating that Map4k4 is a negative regulator of myogenic differentiation and hypertrophy.
117                          RSPOs also promoted myogenic differentiation and induced hypertrophic myotub
118  and Sirt1 cross-talk each other to regulate myogenic differentiation and mediate EPO activity during
119  at the cell cortex is a crucial step during myogenic differentiation and might be a general mechanis
120 ndicate a key role of Phospho1 in regulating myogenic differentiation and mitochondrial function.
121 tant increase in Pax3-mediated inhibition of myogenic differentiation and myoblast migration.
122 important extracellular cue in regulation of myogenic differentiation and myofiber size.
123 ts identify Stac3 as a new gene required for myogenic differentiation and myofibrillar protein assemb
124 early in myogenesis, and its loss attenuated myogenic differentiation and potently reduced the levels
125  as MSulf double mutant mice exhibit delayed myogenic differentiation and prolonged Pax7 expression a
126  pathway may contribute to the inhibition of myogenic differentiation and resistance to apoptosis in
127 etion of the LGR4 receptor severely disrupts myogenic differentiation and significantly diminishes th
128                            TNFalpha inhibits myogenic differentiation and skeletal muscle regeneratio
129 G translocates to the nucleus in response to myogenic differentiation and sublethal dose of cisplatin
130 ediating the repressive effect of hypoxia on myogenic differentiation and suggests that inhibition of
131 mely paired box 7 (satellite cell) and early myogenic differentiation and terminal differentiation (m
132 ified that LSD1 is the only KDM required for myogenic differentiation and that KDM3B, KDM6A, and KDM8
133 hat EB3 [6] is specifically upregulated upon myogenic differentiation and that knockdown of EB3, but
134 el can help better understand the process of myogenic differentiation and the effects of mechanical c
135 22 was physiologically induced during normal myogenic differentiation and was transcriptionally regul
136 ricular remodeling; NUP210 (expressed during myogenic differentiation) and ANK1 (cytoskeletal protein
137 s not interfere with the program of skeletal myogenic differentiation, and does not affect myoblast m
138 FR1 gene expression is down-regulated during myogenic differentiation, and FGFR1 promoter activity is
139 s of dermomyotomal markers and activation of myogenic differentiation, and higher levels induce loss
140 ative form of TCF4 reversed MYF5 expression, myogenic differentiation, and hypertrophic myotube forma
141 e significantly compromised MYF5 expression, myogenic differentiation, and myotube formation.
142 or other E2F family members, is required for myogenic differentiation, and that this requirement for
143 1 expression, impaired EPO effect on delayed myogenic differentiation, and the Sirt1 knockdown effect
144 leavage of the alpha7 chain is elevated upon myogenic differentiation, and this cleavage may be media
145                                We used human myogenic differentiation as a model system to statistica
146                                        Using myogenic differentiation as a model, we found that reduc
147 hat although mTOR knockdown severely impairs myogenic differentiation as expected, the knockdown of r
148 ses and nuclear factor kappa B and decreased myogenic differentiation, as reflected by reduced expres
149 wering Xhes6 expression levels blocks normal myogenic differentiation at tail bud stage.
150 cularly given the lack of drugs that promote myogenic differentiation available for potential clinica
151  that these myoblasts are unable to complete myogenic differentiation because of an inability to up-r
152 R-210 was induced in normoxic myoblasts upon myogenic differentiation both in vitro and in vivo.
153 a are assembled during the initial stages of myogenic differentiation but disappear as cells progress
154 rct, which did not express Nkx2.5 or undergo myogenic differentiation, but adopted a vascular fate wi
155  cAMP signaling can both promote and inhibit myogenic differentiation, but little is known about the
156                mTOR is a master regulator of myogenic differentiation, but the pathways mediating ami
157 on of skeletal muscle gene expression during myogenic differentiation by facilitating the transition
158 lecular mechanisms underlying osteogenic and myogenic differentiation by FN in C2C12 progenitor cells
159                       FN treatment activates myogenic differentiation by increasing p38MAPK and decre
160 ta suggest that inhibition of MSC suppresses myogenic differentiation by inhibiting the caspase-3 act
161 factor receptor in RMS cells, which promotes myogenic differentiation by inhibiting the Notch and the
162 heb-mTOR/raptor pathway negatively regulates myogenic differentiation by suppressing IRS1-PI3K-Akt si
163   These data demonstrate that Ascl2 inhibits myogenic differentiation by targeting MRFs and facilitat
164 tions of BCL9/9-2 inhibited the promotion of myogenic differentiation by Wnt and the normal regenerat
165  inhibitory mechanisms can be suppressed and myogenic differentiation can be induced in the RD rhabdo
166       We first show that cell cycle exit and myogenic differentiation can be uncoupled.
167  considered to play an important role during myogenic differentiation, chronological alterations in D
168 ion of genes involved in neuroectodermal and myogenic differentiation, closely simulating the develop
169 ur data suggest that the role of Fn14 during myogenic differentiation could be independent of TWEAK c
170 strated that cells lacking c6orf32 exhibit a myogenic differentiation defect, characterized by a decr
171  in other inflammatory myopathies, display a myogenic differentiation defect.
172 aling pathway, and exogenous FGF rescues the myogenic differentiation defects upon loss of MyHC-emb f
173 on of NKX2-5 or mutant DMPK 3'UTR results in myogenic differentiation defects, which can be rescued b
174 n of the master regulator MyoD, resulting in myogenic differentiation defects.
175 tained expression of Pax3 proteins inhibited myogenic differentiation, demonstrating that Pax3 degrad
176 he dynamic protein changes that occur during myogenic differentiation, demonstrating the feasibility
177 Accordingly, MyoD or Myog expression rescues myogenic differentiation despite Ascl2 overexpression.
178 satellite cells impairs their activation and myogenic differentiation during muscle regeneration.
179 longed activation of MAPK/ERK pathway during myogenic differentiation, expression of myogenin does no
180                           Here, we show that myogenic differentiation factors regulate miR-182 levels
181 aired gene expression and protein content of myogenic differentiation factors were preceded by decrea
182        FSHD patient myoblasts have defective myogenic differentiation, forming smaller myotubes with
183 we identified HDAC3 as a major suppressor of myogenic differentiation from a high-efficiency Clustere
184 c fibroblasts failed to undergo MyoD-induced myogenic differentiation, further suggesting that Maml1
185 12 myoblasts markedly enhances expression of myogenic differentiation genes, myoblast fusion, and myo
186 diated H3-K9 methylation on the promoters of myogenic differentiation genes.
187 in embryonic development, bone formation and myogenic differentiation; however, its role in human can
188 ported that the tyrosine kinase Abl promotes myogenic differentiation in a manner dependent on its cy
189 e show that MyoD up-regulates miR-378 during myogenic differentiation in C2C12 cells.
190 e identified three compounds which inhibited myogenic differentiation in C2C12 myoblasts; (+)-JQ1, PF
191 w that myomiR release accompanies periods of myogenic differentiation in cell culture and in vivo.
192                                 Furthermore, myogenic differentiation in Mef2a-deficient myoblasts is
193 show that cell migration, proliferation, and myogenic differentiation in pre-culture SBB-treated grou
194 but not constitutive active Akt restored the myogenic differentiation in TAK1-deficient mouse embryon
195 signaling rescues CNC cell proliferation and myogenic differentiation in these mutant mice.
196 ons impairs MyoD function, thereby arresting myogenic differentiation in these tumor cells.
197 ppaBalphaDeltaN) significantly increased the myogenic differentiation in TWEAK-treated C2C12 cultures
198 he mdx mice (paired with GFP mice) underwent myogenic differentiation in vitro and expressed markers
199 levated canonical Wnt signaling resulting in myogenic differentiation in vitro and in mouse xenograft
200 superfamily member that positively regulates myogenic differentiation in vitro and in vivo and signal
201 regulated in hypertrophic muscles and during myogenic differentiation in vitro and in vivo.
202               We found that Deltex2 inhibits myogenic differentiation in vitro, and that skeletal mus
203                                       During myogenic differentiation in vitro, Has2 was the most hig
204           Although various protocols achieve myogenic differentiation in vitro, resulting myotubes ty
205 ive-CD45 negative epCSCs and increased their myogenic differentiation in vitro.
206 he requirement of most of these microRNAs in myogenic differentiation in vivo.
207 d in reduced cell proliferation coupled with myogenic differentiation, including increased expression
208 sults predict the kinetics of the process of myogenic differentiation, including the number of cells
209                                    Thus, the myogenic differentiation-induced microRNAs miR-1 and -20
210                                  Conversely, myogenic differentiation into multinucleated myotubes wa
211 transient Myod1 induction efficiently drives myogenic differentiation into multinucleated myotubes.
212                     The process of stem cell myogenic differentiation is interpreted as the interplay
213     However, the role of those components in myogenic differentiation is not entirely clear.
214  of E2F proteins, and in particular E2f3, in myogenic differentiation is not well understood.
215 pleted from proliferating myoblasts by RNAi, myogenic differentiation is significantly impaired, and
216 molecular mechanism by which RSPO2 regulates myogenic differentiation is unknown.
217  Pak1 and Pak2 display delayed expression of myogenic differentiation markers and myotube formation.
218         Results showed that relaxin promoted myogenic differentiation, migration, and activation of m
219 tion and onset time relative to the stage of myogenic differentiation, miniagrin was found to induce
220          Of note, MTF1 formed a complex with myogenic differentiation (MYOD)1, the master transcripti
221 At genes expressed throughout the program of myogenic differentiation, Myod can bind and recruit hist
222 show that in addition to inhibiting MyoD and myogenic differentiation, NICD(OE) upregulates Pax7 and
223 O1 or PAX7-FOXO1 fusions that block terminal myogenic differentiation, no functionally comparable gen
224  transmembrane protein NET37 is required for myogenic differentiation of C2C12 cells.
225          During myogenesis, Sfmbt1 represses myogenic differentiation of cultured and primary myoblas
226          Immunohistology at 4 weeks revealed myogenic differentiation of donor cells at the site of c
227 -Fc chimera or short interfering RNA induced myogenic differentiation of IBM mesoangioblasts.
228 atin to the differentiation medium increased myogenic differentiation of L6E9 cells.
229 s repression of the Runx2 P1 promoter during myogenic differentiation of mesenchymal cells.
230 se (TERT) enhanced the survival, growth, and myogenic differentiation of mesenchymal stromal cells (M
231                    Moreover, TWEAK inhibited myogenic differentiation of mesoangioblasts.
232                                              Myogenic differentiation of MSCs in culture was induced
233 ycling that plays a nonredundant role in the myogenic differentiation of muscle precursors, limiting
234 D2 (CCND2) was shown to dramatically enhance myogenic differentiation of muscle progenitor cells and
235 ted ERK nuclear translocation induced robust myogenic differentiation of muscle progenitor/stem cells
236  Ebp1 was expressed in the dermomyotome, and myogenic differentiation of muscle progenitors was inhib
237 unostaining showed extensive engraftment and myogenic differentiation of preconditioned Sca-1+ cells.
238                                              Myogenic differentiation of retinal cells prior to their
239 ntrolled MyoD expression benefits functional myogenic differentiation of transdifferentiated myoblast
240           In vitro, CDCs showed the greatest myogenic differentiation potency, highest angiogenic pot
241 ogenic potential in myoblasts, the augmented myogenic differentiation potential observed is likely th
242 -culturing the dKO-nmMSCs with dKO-MPCs, the myogenic differentiation potential of the dKO-MPCs was r
243 g high levels of endoglin (NCSC(CD105+)) had myogenic differentiation potential.
244 pansion during regeneration, but compromised myogenic differentiation prevented the contribution of t
245 (genuine state), and CD34(Low), committed to myogenic differentiation (primed state).
246                                              Myogenic differentiation proceeds through a highly coord
247 rs for noninvasive in vivo monitoring of the myogenic differentiation process from muscle precursor c
248 rogenitor cells followed by the execution of myogenic differentiation, processes that are coordinated
249 e from which these tumors arise, restore the myogenic differentiation program and block the tumorigen
250 th IP3R and RYR, is expressed as part of the myogenic differentiation program and enhances NFAT-depen
251 ssemble a regulatory network controlling the myogenic differentiation program in mammalian cells.
252 d a critical role for eRNAs in regulation of myogenic differentiation program through increasing chro
253 ng cell cycle progression and initiating the myogenic differentiation program.
254 leads to Pax7 cleavage and initiation of the myogenic differentiation program.
255 form essential for normal progression of the myogenic differentiation programme.
256                          Expression of early myogenic differentiation proteins Myf-5 and MyoD increas
257  multiple human pluripotent stem cell (hPSC) myogenic differentiation protocols and mapped hPSC-deriv
258 c function, the highest cell engraftment and myogenic differentiation rates, and the least-abnormal h
259 tion of immunoproteasomes (i-proteasomes) in myogenic differentiation remains unknown.
260 ion of MyoD and its target genes, diminished myogenic differentiation, repression of glutathione redo
261 HD and control myogenesis, revealing altered myogenic differentiation results in hypotrophic myotubes
262 iated E2A protein degradation depends on the myogenic differentiation state (t 1/2 approximately 2 h
263  MyoD and Id1 degradation are independent of myogenic differentiation state.
264 id not inhibit other biochemical measures of myogenic differentiation, suggesting a specific role of
265 to multiple lineages, such as neurogenic and myogenic differentiations; they also display a superior
266 of beta-catenin-recovered their capacity for myogenic differentiation through myocyte enhancer factor
267 ate the ubiquitin-proteasome system (UPS) in myogenic differentiation through regulating cell cycle p
268 2C12 cells, we postulate that 3-MPA promoted myogenic differentiation through the inhibition of PEPCK
269                Deletion of STIM2beta delayed myogenic differentiation through the MEF2C and NFAT4 pat
270 at exit the cell cycle during the process of myogenic differentiation to form myofibers.
271 apidly divide, and are capable of undergoing myogenic differentiation to form myotubes.
272       Importantly, Bhlhe40 knockdown rescues myogenic differentiation under hypoxia.
273     We have dissected the role of the UPS in myogenic differentiation using an in vitro muscle differ
274     We previously showed that RSPO2 promoted myogenic differentiation via activation of WNT/beta-cate
275 t PAX3/FOXO1A and PAX7/FOXO1A act to prevent myogenic differentiation via suppression of the transcri
276              Insulin growth factor 1-induced myogenic differentiation was also found to involve TAK1.
277                                      Induced myogenic differentiation was associated with a decrease
278             The inhibitory effect of DMOG on myogenic differentiation was markedly impaired in C2C12
279  The inhibitory effect of gadolinium ions on myogenic differentiation was reversible and independent
280                                        C2C12 myogenic differentiation was significantly induced follo
281 ur understanding on how microtubules support myogenic differentiation, we analyzed the role of EB1-re
282 um of distal regulatory elements that govern myogenic differentiation, we generated chromatin state m
283             Remarkably, during the course of myogenic differentiation, we observed retention and acqu
284       Moreover, cell mechanical features and myogenic differentiation were significantly reduced in s
285 immortalized cells retained the capacity for myogenic differentiation when treated with the steroid h
286 F-II, IGFBP-5 restores IGF-II expression and myogenic differentiation, whereas an IGF binding-deficie
287 rference revealed that BRD4 was required for myogenic differentiation, whereas BRD3 down-regulation r
288 ed that blockage of this pathway accelerated myogenic differentiation, whereas its activation diminis
289 ly, composed of miR-1 and miR-206, promoting myogenic differentiation, whereas miR-133 maintains the
290 least partially overlapping functions during myogenic differentiation, which are distinct from those
291 PGC1alpha-ERRalpha axis leading to perturbed myogenic differentiation, which can effectively be rescu
292 nd suggests that it is involved in mediating myogenic differentiation, which is HIF-independent.
293 alities in cell-cycle parameters and delayed myogenic differentiation, which were associated with per
294 y cilia strongly suppresses Hh signaling and myogenic differentiation while enhancing proliferation.
295 ntigen family protein, promotes neuronal and myogenic differentiation while inhibiting adipogenesis.
296 urthermore, the model of bexarotene-enhanced myogenic differentiation will provide an important avenu
297 ferentiation and mediate EPO activity during myogenic differentiation with Sirt1 playing a role upstr
298 entified Twist2 as a reversible inhibitor of myogenic differentiation with the remarkable ability to
299 eta superfamily signaling is an inhibitor of myogenic differentiation, with elevated activity in aged
300 myogenin and a specific Mef2 isoform induced myogenic differentiation without activating endogenous M

 
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