1 limb weakness, and in some cases, attacks of myoglobinuria.

2 ting with acute rhabdomyolysis and recurrent myoglobinuria.

3 cause of exercise-induced rhabdomyolysis and myoglobinuria.

4 severe isolated COX deficiency and recurrent myoglobinuria.

5 l tide cycle, and the relative lack of renal myoglobinuria (26/26) suggested MSE onset on a rising ti

6 usly in a patient with recurrent episodes of myoglobinuria and an isolated COX deficiency.

7 dered in patients with recurrent episodes of myoglobinuria and peripheral neuropathy presenting in la

8 ction of D3 on in vivo ARF (glycerol-induced myoglobinuria) and isolated proximal tubule injury respo

9 e presenting clinical features are myalgias, myoglobinuria, and an elevated serum creatine kinase.

10 utations in these genes cause lipodystrophy, myoglobinuria, and inflammatory disorders.

11 terized by exercise intolerance, cramps, and myoglobinuria, and the other dominated by fixed, often p

12 ng with progressive exercise intolerance and myoglobinuria associated with complex III deficiency in

13 , especially of the exercise intolerance and myoglobinuria; biochemical and molecular features--molec

14 yopathy with severe exercise intolerance and myoglobinuria has been described in patients from northe

15 pin-2 deficiency in humans, leading to acute myoglobinuria in childhood or the inflammatory disorder

16 rse forms of acute renal tubular injury (eg, myoglobinuria, ischemia/reperfusion, urinary tract obstr

17 sing exercise intolerance, cramps, recurrent myoglobinuria, or fixed weakness, which often affects ex

18 mptomatic individuals to rhabdomyolysis with myoglobinuria, renal failure, and death.

19                             We conclude that myoglobinuria with myoglobin cast formation can occur fo