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1 ompensatory mechanism between myosin IIA and myosin IIB.
2     This zone is also enriched for nonmuscle myosin IIB.
3 nd ERK, myosin light chain kinase (MLCK) and Myosin IIB.
4 as distinct from that of a nonmuscle myosin, myosin IIb.
5 kinase C, a negative regulator of non-muscle myosin IIB.
6 ched in tropomodulin 1 (Tmod1) and nonmuscle myosin IIB.
7 cle myosin II, referred to as myosin IIA and myosin IIB.
8 d states, which is in contrast to non-muscle myosin IIB.
9  the nonmuscle isoforms of alpha-actinin and myosin IIB.
10  little effect on the assembly properties of myosin-IIB.
11  However, in this study, we demonstrate that myosin IIB, a cytoplasmic myosin II particularly enriche
12                  Moreover, the motor protein myosin IIB, a mediator of mechanical force transmission
13                           Here, we show that myosin IIB, a molecular motor that binds and contracts a
14  actin assembly at the leading edge, whereas myosin IIB accumulated in the rear 15-30 min later.
15       Pharmacologic or genetic inhibition of myosin IIB alters protrusive motility of spines, destabi
16 ults in an isoform switch from myosin IIC to myosin IIB and increased phosphorylation of myosin heavy
17 ntative of skeletal muscle myosin, nonmuscle myosin IIB, and nonmuscle myosin IIA revealed three dist
18 ntained fibrils composed of actin, nonmuscle myosin IIB, and sarcomeric alpha-actinin.
19                    The restraint consists of myosin IIB- and IIA-dependent processes: retrograde acti
20            Conversely, the microinjection of myosin IIB antibody blocks microfilament-directed chromo
21 ts in the localized selective aggregation of myosin IIB but not myosin IIA at the region of parasite
22           However, posterior accumulation of myosin IIB, but not anterior distribution of myosin IIA,
23 hese observations indicate that asymmetry of myosin IIB, but not IIA, is regulated by light-chain pho
24 -specific control of the mechanosensation of myosin IIB, but not myosin IIA or IIC.
25 yosin II drives actin flow, and depletion of myosin IIB, but not myosin IIA, showed similar nondirect
26 m-specific manner, affecting the assembly of myosin-IIB, but not myosin-IIA.
27 unction of one of the isoforms of myosin II, myosin IIB, by analyzing the movement and mechanical cha
28            One of these putative substrates, myosin IIB, can be phosphorylated in vivo by Arg.
29 siRNA)-mediated suppression of myosin IIA or myosin IIB causes an increase in mitochondrial length in
30 alized MT organizing center (MTOC)/Golgi and myosin IIB cell rear enrichment.
31 shwork instability and collapse, followed by myosin IIB concentration at the rear of the cone and nec
32                      We now demonstrate that myosin IIB contributes to tension at the epithelial ZA.
33 o develop convergence forces parallel to the myosin IIB-dependent dynamics of the actin cytoskeleton.
34 able to rescue cytokinesis failure caused by myosin-IIB depletion.
35                        Suppressing nonmuscle myosin IIB disrupts directional cell rearrangements and
36 sin IIA promoted internalization of MHCI and myosin IIB drove CD59 uptake in both HeLa and polarized
37 in IIA forms de novo filaments away from the myosin IIB-enriched center and back to form regions that
38                          Interestingly, when myosin IIb expression and MyH7B expression were simultan
39                                Abrogation of myosin IIB expression in the E1 knockdown cells has no e
40  indicate that transition between myosin IIC/myosin IIB expression is a critical feature of EMT that
41 n IIC is expressed in luminal cells, whereas myosin IIB expression is up-regulated in myoepithelial c
42 d in H-K-ATPase membrane trafficking include myosin IIB, F-actin, ezrin, and Rab GTPases.
43              Furthermore, phosphorylation of myosin-IIB filaments by Aurora-B also promotes filament
44 r affinity for myosin-IIA filaments than for myosin-IIB filaments.
45              Here, we show that Aurora-B and myosin-IIB form a complex in vivo during telophase.
46 onses to Wnt5a involve recruitment of actin, myosin IIB, Frizzled 3, and melanoma cell adhesion molec
47 t did not affect protrusion, suggesting that myosin IIB functions in pulling the rear of the cell for
48                            Cells depleted of myosin IIB, however, were efficient in thrombin-induced
49 ced lamellar spreading, whereas depletion of myosin IIB impaired not only migration but also impaired
50 e polarized localization of MCAM, actin, and myosin IIB in a Wnt5a-induced manner.
51 ongate the body axis, to examine the role of myosin IIB in convergence and extension.
52  have found that MLC-dependent activation of myosin IIB in migrating cells is required to form an ext
53 correlates the recruitment of myosin IIA and myosin IIB into this spreading margin.
54                                     Although myosin IIB is also found at the zonula adherens (ZA) in
55 ion analysis reveals for the first time that myosin IIB is associated with vimentin, linking vimentin
56                                              Myosin IIB is enriched in the postsynaptic density (PSD)
57 ht on the mechanism, showing that non-muscle myosin IIb is intimately involved.
58                     Our results suggest that myosin IIB is involved not in propelling but in directin
59                                 We find that myosin IIB is localized in the cortex of intercalating c
60                                   Therefore, myosin IIB is necessary for normal growth cone spreading
61                    In the superficial layer, myosin IIB is needed for apical actin accumulation, whic
62                                 We show that myosin IIB is required for actin-cytoskeletal organizati
63                            We also show that myosin IIB is required for resistance to deformation ("s
64  that a short serine-rich motif in nonmuscle myosin IIB is required to establish the cell's rear.
65                              To test whether myosin IIB is responsible for the force generation, we q
66                              We propose that myosin-IIB is a substrate of Aurora-B and reveal a new m
67 ng that Aurora-B-mediated phosphorylation of myosin-IIB is important for abscission.
68                 We show that nonmuscle MIIB (myosin-IIB) is unpolarized in cells on soft matrix in 2D
69 mediated traction force in growth cones from myosin IIB knock-out (KO) mice and compared them with ne
70 y with blebbistatin or by using neurons from myosin IIB knockouts inhibits retraction.
71 wed, but not eliminated, in neurons from the myosin IIB KO mice.
72  with emerin, and emerin depletion prevented myosin IIB localization near nuclei.
73 alization from the basolateral domain, while myosin IIB localized at the basal cortex and apical cell
74                                    Activated myosin IIB localizes prominently at the cell rear and pr
75 l family kinase substrates and suggests that myosin IIB may be regulated by tyrosine phosphorylation.
76    Atomic force microscopy demonstrated that myosin IIB mediated apical epithelial tension in Caco2 c
77  that the individual nonprocessive nonmuscle myosin IIB molecules form a highly processive unit when
78 ne 1847 (T(1847)), abrogating the ability of myosin-IIB monomers to form filaments.
79 issue-targeting techniques, we show that the myosin IIB motor protein complex is essential for both t
80  power stroke, specifically activating human myosin IIB (MYH10) and human myosin IIC (MYH14), but not
81        Their less responsive sister paralogs-myosin IIB (MYH10), alpha-actinin 1, and filamin A-had l
82 2, beta-catenin (CTNNB1), N-cadherin (CDH2), myosin IIB (MYOIIB), aPKCzeta, LGL, PAR3, pericentrin, P
83                                    Nonmuscle myosin IIB (NM IIB)-mediated actomyosin contractility re
84 in 4.2 (TPM4), vinculin (VCL), and nonmuscle myosin IIB (NM-2B, MYH10).
85                                   Non-muscle myosin IIB (NMIIB) generates tension along actin filamen
86                                    Nonmuscle myosin IIB (NMIIB) is a cytoplasmic myosin, which plays
87 ry of a viable therapeutic target, nonmuscle myosin IIB (NMIIB), a molecular motor that supports memo
88  chain IIB (NMHCIIB), a subunit of nonmuscle myosin IIB (NMIIB), as an ER stress-dependent interactin
89 of myo1b and the motor domain from nonmuscle myosin-IIb (nmMIIb) concentrates on actin filaments in r
90                        Similar to non-muscle myosin IIB, non-muscle myosin IIA shows high ADP affinit
91                                              Myosin IIB null cells displayed multiple unstable and di
92 te bending, whereas in the deep neural cells myosin IIB organizes a cortical actin cytoskeleton, whic
93                                 We show that myosin-IIB possessing a phosphomimetic mutation at T(184
94 ic stem and progenitor cells, with polarized myosin-IIB promoting asymmetric self-renewal and constit
95                                              Myosin IIB redistributes from a broad distribution to th
96 te of Aurora-B and reveal a new mechanism of myosin-IIB regulation by Aurora-B in the late stages of
97                  Aurora-B phosphorylates the myosin-IIB rod domain at threonine 1847 (T(1847)), abrog
98 al for the mechanics of cell migration, with myosin IIB seeming to have a preferential role in the me
99                    These features imply that myosin IIB serves a set of physiologic needs different f
100             Transient kinetics of non-muscle myosin IIB showed that this motor has a very high actomy
101 atin or selective RNA-mediated repression of myosin IIB significantly inhibits (P < 0.05) C. parvum c
102                                              Myosin IIB specifically coimmunoprecipitated with emerin
103 tatin on the kinetic properties of nonmuscle myosin IIB subfragment 1 (NMIIB S1).
104 netic characterization of a human non-muscle myosin IIB subfragment-1 construct produced in the bacul
105                             Thus, non-muscle myosin IIB subfragment-1 spends a significantly higher p
106  in the offspring and increased the ratio of myosin IIb to other isoforms by 17.6 +/- 4.9% (P < 0.05)
107 hese results show that emerin functions with myosin IIB to polarize actin flow and nuclear movement i
108 e-Rap1 pathway as responsible for recruiting myosin IIB to the ZA and supporting contractile tension.
109 hese cadherin contractions require nonmuscle myosin IIB, tropomyosin 2.1, alpha-catenin, and binding
110  In contrast, genetic ablation of non-muscle myosin IIB was associated with a 60% decrease in mitocho
111   Immunofluorescence staining indicated that myosin IIB was localized preferentially along stress fib
112                    Posterior accumulation of myosin IIB was unaffected.
113 tinin 2], TTN [titin], and MYH10 [non-muscle myosin IIB]) were misspliced.
114 tinin 2], TTN [titin], and MYH10 [non-muscle myosin IIB]) were misspliced.
115 s of CARMIL2 also caused decreased levels of myosin-IIB, which may contribute to the polarity phenoty
116  in COS7 cells disrupted the localization of myosin IIB without obviously affecting actin filaments.

 
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