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1  were ameliorated by the presence of dietary n-3 fatty acid.
2 concentrations of long-chain polyunsaturated n-3 fatty acids.
3  studies of AMD risk reduction by long-chain n-3 fatty acids.
4 script levels in the mice fed a diet of high n-3 fatty acids.
5 igh dietary AA but mitigated by high dietary n-3 fatty acids.
6 e not correlated with dietary consumption of n-3 fatty acids.
7 infarction, and stroke in subjects consuming n-3 fatty acids.
8  10% Scia in diets balanced out with n-6 and n-3 fatty acids.
9  rapid increase in erythrocyte DHA and total n-3 fatty acids.
10 ifferences based on metabolism of long-chain n-3 fatty acids.
11 uming higher quantities of n-6 or long-chain n-3 fatty acids.
12 se genes are responsible for many effects of n-3 fatty acids.
13 anic is not a guarantee of higher long chain n-3 fatty acids.
14 the origin of specific volatiles from parent n-3 fatty acids.
15 ing optimal amounts of dietary n-9, n-6, and n-3 fatty acids.
16 may be mediated by an enzymatic affinity for n-3 fatty acids.
17 n of saltwater fish and intake of long-chain n-3 fatty acids.
18 received placebo (adjusted hazard ratio with n-3 fatty acids, 0.97; 95% confidence interval, 0.88 to
19           Patients were randomly assigned to n-3 fatty acids (1 g daily) or placebo (olive oil).
20                             Both groups took n-3 fatty acids (2.4 g/d) for 4 wk with the addition of
21 after we adjusted further for marine-derived n-3 fatty acids (22 mum, 95% CI: -1 to 46 mum, p = 0.065
22 eas consumption of the vegetable oil-derived n-3 fatty acid a-linolenic acid is not as effective.
23  were similar for estimated intake of marine n-3 fatty acids: a 1 g/day higher intake was associated
24  domains) in relation to baseline intakes of n-3 fatty acids (absolute and relative to n-6 fatty acid
25           Recent studies have suggested that n-3 fatty acids, abundant in fish oil, protect against h
26                             However, whether N-3 fatty acids, active ingredients of fish oil, have di
27 in Barnes maze performance compared with the n-3 fatty acid-adequate rats during the initial training
28 3 fatty acid-deficient rats in comparison to n-3 fatty acid-adequate rats.
29                       The mechanisms whereby n-3 fatty acids affect gene expression are complex and i
30   However, the molecular mechanisms by which n-3 fatty acids affect tumor growth remain unknown.
31 re studied in chicken patties, enriched with n-3 fatty acids, after 8days of storage at 4 degrees C,
32 s have evaluated the association between the n-3 fatty acid alpha-linolenic acid (ALA) and the incide
33      Because conversion of the plant-derived n-3 fatty acid alpha-linolenic acid (ALA) to EPA and DHA
34         Intake of the plant-derived omega-3 (n-3) fatty acid alpha-linolenic acid (ALA, 18:3; n-3) ma
35                                   The use of n-3 fatty acids also had no significant effect on the ra
36                          Oxidation decreases n-3 fatty acid and other bioactive compounds contents, w
37                                  The % total n-3 fatty acids and % EPA directly correlated with pro-i
38 nts enrolled, 6244 were randomly assigned to n-3 fatty acids and 6269 to placebo.
39 %), of whom 733 of 6239 (11.7%) had received n-3 fatty acids and 745 of 6266 (11.9%) had received pla
40 d control subjects in response to intakes of n-3 fatty acids and aspirin.
41                 Shrimp oil, a rich source of n-3 fatty acids and astaxanthin, was encapsulated in nan
42 ociation between the ratio of dietary n-6 to n-3 fatty acids and bone mineral density (BMD) in 1532 c
43 d the relation between the dietary intake of n-3 fatty acids and chronic obstructive pulmonary diseas
44              Modulation of specific genes by n-3 fatty acids and cross-talk between these genes are r
45              However, the role of long-chain n-3 fatty acids and fish intake in the development of T2
46   We further analyzed the effects of n-6 and n-3 fatty acids and found that the PUFA-APOA5 interactio
47 ineered fat-1 mice, which can convert n-6 to n-3 fatty acids and have a balanced ratio of n-6 to n-3
48 showed no evidence that supplementation with n-3 fatty acids and instructions to reduce arachidonic a
49 evidenced by an increase in the ratio of n-6/n-3 fatty acids and levels of 4-hydroxynonenal, a marker
50        The results of the comparison between n-3 fatty acids and placebo are reported here.
51 sible to make firm recommendations regarding n-3 fatty acids and the prevention of dementia and AMD.
52 ions between fish, shellfish, and long-chain n-3 fatty acids and the risk of T2D in a middle-aged Chi
53                                 The level of n-3 fatty acids and their metabolite prostaglandin E(3)
54 ever, the relation between dietary intake of n-3 fatty acids and ventricular arrhythmias has not been
55  and spring proved to have a high content of n-3 fatty acids and vitamin D, and hence they serve as a
56 tive was to determine whether walnuts (plant n-3 fatty acid) and fatty fish (marine n-3 fatty acid) h
57 34)), levels of polyunsaturated fatty acids, n-3 fatty acids, and docosahexaoenic acid relative to to
58 ific food components, such as soluble fiber, n-3 fatty acids, and fructose, are discussed.
59 nolenic acid (ALA), a plant-derived omega-3 (n-3) fatty acid, and age-related macular degeneration (A
60 nolenic acid (ALA), a plant-derived omega-3 (n-3) fatty acid, and cardiovascular disease (CVD) risk h
61 uding saturated fatty acids (SFAs), omega-3 (n-3) fatty acids, and refined sugar, with hippocampal-de
62 rticularly lutein and zeaxanthin, as well as n-3 fatty acids, appears beneficial for AMD and possibly
63  alkenyl chain and that of a sn-2-esterified n-3 fatty acid are additive.
64  We sought to examine whether marine-derived n-3 fatty acids are associated with less atherosclerosis
65                                              n-3 Fatty acids are known to reduce many risk factors as
66  n-6 fatty acids and concomitant decrease in n-3 fatty acids are thought to be a major driver of the
67                                     Omega-3 (n-3) fatty acids are essential for proper neuronal funct
68 ioperative immunonutrition (IMN) enriched in n-3 fatty acids, arginine, and nucleotides may improve p
69 ificantly decreased among patients receiving n-3 fatty acids, as compared with those receiving placeb
70 ple, dietary intake of fish and fish-derived n-3 fatty acids, as well as plasma biomarkers of fish in
71                               Marine-derived n-3 fatty acids at low levels are cardioprotective throu
72 3) (at a dose of 2000 IU per day) and marine n-3 fatty acids (at a dose of 1 g per day) in the primar
73 involved in lipid oxidation, with long-chain n-3 fatty acids being potent activators of PPARA.
74  indicate that syndecan 1 is up-regulated by n-3 fatty acids by a transcriptional pathway involving P
75 ed fatty acids in optimizing the benefits of n-3 fatty acids (C18-C22) on cardiovascular health.
76 genous fatty acids, with a preference toward n-3 fatty acids (C18:3 and C22:6).
77 iet that is high in omega-3 polyunsaturated (n-3) fatty acids can slow disease progression in patient
78 ga-3 fatty acids (omega-3-FAs; also known as n-3 fatty acids) can exert potent anti-inflammatory effe
79 th health-related biomarkers [e.g., omega-3 (n-3) fatty acids, citrate, alpha1-acid glycoprotein, HDL
80   In contrast to their agonistic properties, n-3 fatty acids competitively inhibit the responses of v
81 ffects of varying n-9, n-6, and longer-chain n-3 fatty acid composition on markers of coronary heart
82 ctive was to examine the association between n-3 fatty acid consumption and ventricular ectopy among
83 tigate whether a diet enriched with fish and n-3 fatty acid consumption are associated with changes i
84                    Critical time periods for n-3 fatty acid consumption may differ by sex.
85 udies have evaluated the effects of fish and n-3 fatty acid consumption on fatal coronary heart disea
86 se findings extend existing evidence linking n-3 fatty acid consumption to a reduced risk of ventricu
87 ariable adjustment, neither fish nor dietary n-3 fatty acid consumption was significantly associated
88 im was to study the associations of fish and n-3 fatty acid consumption with risk of total major chro
89 problems in evaluating the health effects of n-3 fatty acid consumption, highlighted challenges to re
90                           Increased omega-3 (n-3) fatty acid consumption is reported to benefit patie
91 sions based on plant oils differing in their n-3 fatty acid content were compared.
92                        An increase in plasma n-3 fatty acid content, particularly eicosapentaenoic ac
93      Cooking treatments had little effect on n-3 fatty acid content; however, fried treatments genera
94 tudy, the authors demonstrate that rats with n-3 fatty acid deficiency display spatial learning defic
95                  Results showed that dietary n-3 fatty acid deficiency elevates the vulnerability to
96                      The effect of long-term n-3 fatty acid deficiency on rod and cone phototransduct
97 upplementation, combined with the effects of n-3 fatty acid deficiency, on acute blue-light photochem
98                                          The n-3 fatty acid-deficient rats had reduced tissue levels
99 s of brain docosahexaenoic acid (DHA) in the n-3 fatty acid-deficient rats in comparison to n-3 fatty
100           In the reversal learning task, the n-3 fatty acid-deficient rats showed a profound deficit
101                      The long-chain omega-3 (n-3) fatty acids derived from fish, eicosapentaenoic aci
102 achieved through overexpressing a C. elegans n-3 fatty acid desaturase gene, mfat-1.
103 norhabditis elegans gene, fat-1, encoding an n-3 fatty acid desaturase.
104 onics have higher levels of glycolipids with n-3 fatty acids (DGDG 36:3; SQDG 36:3; MGDG 36:6) compar
105  antiinflammatory, and vasodilatory omega-3 (n-3) fatty acids (DHA and EPA) are significantly reduced
106                   The addition of aspirin to n-3 fatty acids did not alter any SPMs in either group.
107 ovascular risk factors, daily treatment with n-3 fatty acids did not reduce cardiovascular mortality
108            Daily supplementation with 1 g of n-3 fatty acids did not reduce the rate of cardiovascula
109                         Supplementation with n-3 fatty acids did not result in a lower incidence of m
110 2(-/-)/Cx3cr1(-/-) mice that ingested a high n-3 fatty acid diet showed a slower progression of retin
111                                        Thus, n-3 fatty acids differentially regulate TRPV1 and this f
112 enter addresses is whether botanical n-6 and n-3 fatty acids directly block recognized biochemical pa
113         Together, these results suggest that N-3 fatty acids directly inhibit vascular calcification,
114 esult in altered brain concentrations of the n-3 fatty acid docosahexaenoic acid (DHA) during the per
115 of the transcription factor PPARgamma by the n-3 fatty acid docosahexaenoic acid (DHA) is implicated
116                  Our studies showed that the n-3 fatty acid docosahexaenoic acid (DHA) up-regulated s
117 ation of TLR2 with TLR1 was inhibited by the n-3 fatty acid docosahexaenoic acid.
118 ns unresolved, possibly due to low levels of n-3 fatty acids docosahexaenoic acid (DHA) and eicosapen
119                                 The omega-3 (n-3) fatty acids docosahexaenoic acid and eicosapentaeno
120                                          One n-3 fatty acid, docosahexaenoic acid, is an important co
121                        Dams were deprived of n-3 fatty acids during pregnancy and lactation, and thei
122 r diets with different levels of the various n-3 fatty acids during pregnancy and lactation, and thei
123 d interventional, assessing the influence of n-3 fatty acids during pregnancy or the early postpartum
124 itro experiments showed that addition of the n-3 fatty acid eicosapentaenoic acid or PGE(3) inhibited
125 at consuming fish or fish oil containing the n-3 fatty acids eicosapentaenoic acid (EPA) and docosahe
126 rvings/wk), especially species higher in the n-3 fatty acids eicosapentaenoic acid (EPA) and docosahe
127  attributed to the marine-derived long-chain n-3 fatty acids eicosapentaenoic acid (EPA) and docosahe
128  correlated with the RBC long-chain omega-3 (n-3) fatty acids eicosapentaenoic acid (EPA) and docosah
129                          A greater intake of n-3 fatty acids (eicosapentaenoic acid + docosahexaenoic
130 flaxseed addition in the chicken feed (i.e., n-3 fatty acid enrichment), and for the different packag
131             Supplemental long-chain omega-3 (n-3) fatty acids (EPA and DHA) raise erythrocyte EPA + D
132 ns, a significant increase in serum omega-3 (n-3) fatty acids (EPA and DHA), and a decrease in serum
133                                        Total n-3 fatty acids, EPA, and the SI for palmitic to palmito
134 iew current evidence on the relation between n-3 fatty acids, especially docosahexaenoic acid (DHA),
135                                              n-3 Fatty acids exert important effects on eicosanoid me
136     We questioned if acute administration of n-3 fatty acids (FA) carried in n-3 rich triglyceride (T
137 molecular studies relating to the effects of n-3 fatty acids (FA) on inhibiting atherogenesis.
138 h low dietary omega-6 (n-6) or high omega-3 (n-3) fatty acid (FA) content resulted in reduced body fa
139                The omega-6 (n-6) to omega-3 (n-3) fatty acid (FA) ratio (n-6:n-3 ratio) was previousl
140 ma triglyceride (TG) response to an omega-3 (n-3) fatty acid (FA) supplementation.
141 and Treatment of Coronary Heart Disease with n-3 Fatty Acids," faculty who presented at the conferenc
142      Studies on the relation between dietary n-3 fatty acids (FAs) and cardiovascular disease vary in
143                                              n-3 Fatty acids (FAs) have been shown to be beneficial f
144                                              n-3 Fatty acids (FAs) when used in doses of 3-4 g/d eico
145                         The role of omega-3 (n-3) fatty acids (FAs) in the development of type 2 diab
146  the relation of plasma and dietary omega-3 (n-3) fatty acids (FAs) with heart failure (HF) risk have
147  Nevertheless, the most consistent effect of n-3 fatty acids feeding in rats is to decrease lipogenes
148 then recommendations for specific intakes of n-3 fatty acids for different conditions relating to car
149  Alpha-linolenic acid, an intermediate-chain n-3 fatty acid found primarily in plants, may decrease t
150                         Intake of long-chain n-3 fatty acids found in fish is low in many countries w
151 ps: n-6 fatty acids found in plant seeds and n-3 fatty acids found in marine vertebrates.
152                         Intake of long-chain n-3 fatty acids from fish was not associated with depres
153 fat-1 transgenic pigs produce high levels of n-3 fatty acids from n-6 analogs, and their tissues have
154       Twice-daily enteral supplementation of n-3 fatty acids, gamma-linolenic acid, and antioxidants
155       Twice-daily enteral supplementation of n-3 fatty acids, gamma-linolenic acid, and antioxidants
156 ion of retinal lesions compared with the low n-3 fatty acids group.
157                                   Long chain n-3 fatty acids had considerably lower concentrations in
158     Some mice that were given high levels of n-3 fatty acids had lesion reversion.
159         Whereas dietary intake of long-chain n-3 fatty acids has been associated with risk of nonfata
160                 Whether supplementation with n-3 fatty acids has such effects in general populations
161           Very high levels of marine-derived n-3 fatty acids have antiatherogenic properties that are
162     Most of the health benefits observed for n-3 fatty acids have been attributed to the marine-deriv
163                                              n-3 Fatty acids have important visual, mental, and cardi
164                                     Omega-3 (n-3) fatty acids have antiinflammatory and antiprolifera
165 plant n-3 fatty acid) and fatty fish (marine n-3 fatty acid) have similar effects on serum lipid mark
166 t Greenland Eskimos, who have a diet high in n-3 fatty acids, have a lower mortality from coronary he
167 W n-6) or a diet with a relative increase in n-3 fatty acid (HIGH n-3) compared with the CTR from pos
168 al cord blood samples (n = 101), we measured n-3 fatty acids, IgE concentrations, and immunologic res
169 ammation in animal models, we tested whether n-3 fatty acids impact SPM profiles in patients with CAD
170 ctable way to increase a specific long-chain n-3 fatty acid in plasma, tissues, or human milk is to s
171  Docosahexaenoic acid (DHA) is the principal n-3 fatty acid in tissues and is particularly abundant i
172                       Limited storage of the n-3 fatty acids in adipose tissue suggests that a contin
173 imed to investigate the effect of long-chain n-3 fatty acids in blood on the risk of nonfatal MI.
174                               The long-chain n-3 fatty acids in fish have been demonstrated to have a
175 ypothesized to reduce the health benefits of n-3 fatty acids in fish.
176 d, and because of limited interconversion of n-3 fatty acids in humans, ALA supplementation does not
177                        What are the roles of n-3 fatty acids in hypertriglyceridemia, in the metaboli
178 rized databases were searched for studies of n-3 fatty acids in immune-mediated diseases from 1966 to
179                       The adverse effects of n-3 fatty acids in men merit confirmation.
180 lt in appreciable accumulation of long-chain n-3 fatty acids in plasma.
181                        What are the roles of n-3 fatty acids in primary versus secondary prevention o
182 tribute to the chemopreventive properties of n-3 fatty acids in prostate cancer.
183 s well as higher percentages of both n-6 and n-3 fatty acids in relation to caprine milk.
184 odents caused by an inadequate amount of the n-3 fatty acids in the diet.
185 vitro studies indicates a beneficial role of n-3 fatty acids in the prevention and management of card
186 ty acids and have a balanced ratio of n-6 to n-3 fatty acids in their tissues and organs independent
187   Triglyceride synthesis could be reduced by n-3 fatty acids in three general ways: reduced substrate
188 d activation and provides specificity toward n-3 fatty acids in which the corresponding n-3 acyl-CoAs
189  acid (DHA, 22:6n-3), the principal omega-3 (n-3) fatty acid in brain gray matter, positively regulat
190  moderators of clinical response to omega-3 (n-3) fatty acids in subjects with major depressive disor
191 sociation of fish, shellfish, and long-chain n-3 fatty acid (in g/d) with risk of T2D.
192   Dietary recommendations have been made for n-3 fatty acids, including alpha-linolenic acid (ALA), e
193                                      Dietary n-3 fatty acids, independent of the reciprocal changes i
194 wide diversity of dietary intakes of n-6 and n-3 fatty acids influences tissue compositions of n-3 lo
195                                              n-3 fatty acids inhibit neuronal excitability and reduce
196                          Results showed that N-3 fatty acids inhibited alkaline phosphatase (ALP) act
197 fatty acid intake (in whites) and long-chain n-3 fatty acid intake (in African Americans) such that p
198                       Of these, four had low n-3 fatty acid intake and four had adequate intakes.
199 imilar association was found between seafood n-3 fatty acid intake and prostate cancer mortality (RR(
200  Our results support a protective effects of n-3 fatty acid intake and the n-7 saturation index again
201 d 0.025) remained negatively associated with n-3 fatty acid intake in women and men, and large HDLs (
202 in both) remained positively associated with n-3 fatty acid intake in women and men.
203           Significant genotype-by-long-chain n-3 fatty acid intake interactions were observed only in
204       Control subjects had typical L, Z, and n-3 fatty acid intake.
205 e was to determine whether habitual omega-3 (n-3) fatty acid intake is associated with the rate of in
206                                              n-3 Fatty acid intakes (both in terms of absolute amount
207 t is reasonable to hypothesize that maternal n-3 fatty acid intakes might have significant effects on
208              Fish, shellfish, and long-chain n-3 fatty acid intakes were inversely associated with T2
209 We examined the relation of fish and seafood n-3 fatty acid intakes with prostate cancer incidence an
210 PUFA intake, specifically n-6 and long-chain n-3 fatty acid intakes, and multiple lipid measures in t
211 ch as carbohydrates and protein, leucine and n-3 fatty acid intakes, such as of EPA, may be important
212                            Here we show that n-3 fatty acids interact directly with TRPV1, an ion cha
213 e trafficking of stable isotopically labeled n-3 fatty acids into phosphatidylcholine and phosphatidy
214               Docosahexaenoic acid (DHA), an n-3 fatty acid, is the major polyunsaturate in rod outer
215              The role of long-chain omega-3 (n-3) fatty acids (LCFAs) in the development of T2DM rema
216  blue light-induced damage, whereas adequate n-3 fatty acid levels reduced the damage in the parafove
217  the human situation in which 1.2% energy as n-3 fatty acids lowers serum triglyceride levels.
218 entre randomised trial of low-dose fish oil (n-3 fatty acids <1080 mg/day) in drug resistant epilepsy
219 hythmias by showing that a greater intake of n-3 fatty acids may be associated with low ventricular e
220 ndicate that the cardioprotective effects of n-3 fatty acids may be attributed to a number of distinc
221 d and SCD supports the hypothesis that these n-3 fatty acids may have antiarrhythmic properties.
222                                   The use of n-3 fatty acids may prevent cardiovascular events in pat
223                             Fish and seafood n-3 fatty acids may prevent or delay the progression of
224                     A high dietary intake of n-3 fatty acids may protect cigarette smokers against CO
225 nthophylls lutein (L) and zeaxanthin (Z) and n-3 fatty acids may reduce this damage and lower the ris
226                    Although dietary omega-3 (n-3) fatty acids may confer some cardiovascular benefits
227 T1A, was strongly associated with markers of n-3 fatty acid metabolism, including degree of unsaturat
228        Therefore, we hypothesized that these n-3 fatty acids might prevent potentially fatal ventricu
229 ignificant increase of phospholipids bearing n-3 fatty acids (most in PC and PE) was also recorded.
230 her concentrations of both n-6 (omega-6) and n-3 fatty acids of higher elongation and desaturation le
231                                  Erythrocyte n-3 fatty acids of marine origin and trans fatty acid co
232 ar consensus exists regarding the effects of n-3 fatty acids on any of these outcomes.
233   Our results support a protective effect of n-3 fatty acids on breast cancer risk and provide additi
234 ent to draw conclusions about the effects of n-3 fatty acids on clinical, endoscopic, or histologic s
235 ntrolled trials that assessed the effects of n-3 fatty acids on clinical, sigmoidoscopic, or histolog
236 spective, double-blind trials, the effect of n-3 fatty acids on depression during pregnancy or the ea
237  low-dose supplementation with B vitamins or n-3 fatty acids on depressive symptoms in cardiovascular
238 ules of either an ethyl ester concentrate of n-3 fatty acids or olive oil daily for 12 mo.
239 east 900 mg (90% or more) of ethyl esters of n-3 fatty acids or placebo daily and to receive either i
240  per liter]) or standard care and to receive n-3 fatty acids or placebo with the use of a 2-by-2 fact
241 -long-chain polyunsaturated fatty acids, the n-3 fatty acids (or omega-3), have distinct and importan
242  DHA (600 mg) in a 2:1 ratio; B vitamins and n-3 fatty acids; or a placebo.
243  risk factors and RBC saturated and omega-3 (n-3) fatty acids, ORs for CAD associated with each SD in
244 es have a significantly reduced ratio of n-6/n-3 fatty acids (P < 0.001).
245                                              n-3 Fatty acids, particularly docosahexaenoic acid (DHA)
246                                      Dietary n-3 fatty acids, particularly eicosapentaenoic acid and
247  and gelled double emulsion, for delivery of n-3 fatty acids (perilla oil at 300g/kg) and hydroxytyro
248 ether the ratio of omega-6 (n-6) to omega-3 (n-3) fatty acids plays a role in tumorigenesis remains t
249 forcement of the antioxidant system, through n-3 fatty acids plus antioxidant vitamin supplementation
250 children had high ratios, a higher intake of n-3 fatty acids predicted a better performance on the pl
251 rse association between intake of long chain n-3 fatty acids [primarily eicosapentaenoic acid (EPA) a
252       These data support the hypothesis that n-3 fatty acids protect from high-fat diet-induced hepat
253 useful model for elucidating the role of n-6/n-3 fatty acid ratio in tumorigenesis.
254 Fish were fed diets containing different n-6/n-3 fatty acid ratios (V0.5 or V1) and sesamin contents
255                                     Oxidized n-3 fatty acids reacted directly with the negative regul
256                                          The n-3 fatty acid recommendation to achieve nutritional ade
257 istent across 3 studies was the finding that n-3 fatty acids reduce corticosteroid requirements, alth
258                            Fish oils rich in n-3 fatty acids reduce serum triglyceride levels.
259 idered tentative, and the mechanism by which n-3 fatty acids reduce triglyceride levels in humans rem
260                                 As examples, n-3 fatty acids regulate 2 groups of transcription facto
261 ogram produced 11 reports on a wide range of n-3 fatty acid-related topics.
262 olic and cardiovascular benefits of omega-3 (n-3) fatty acids, several studies have suggested an adde
263             In the parafovea, animals low in n-3 fatty acids showed greater sensitivity to damage tha
264 es support a direct association between poor n-3 fatty acid status and increased risk of maternal dep
265 upplementation successfully increased plasma n-3 fatty acid status.
266                                     Omega-3 (n-3) fatty acids stimulate protein anabolism in animals
267 on trials, that examine the relation between n-3 fatty acids such as DHA and depression and child men
268  both groups, and the addition of aspirin to n-3 fatty acid supplementation did not alter any of the
269                                     Diet and n-3 fatty acid supplementation dramatically reduced seru
270 ins were increased to a similar extent after n-3 fatty acid supplementation in both groups, and the a
271 ecursors 18-HEPE, 17-HDHA, and 14-HDHA after n-3 fatty acid supplementation that was significantly at
272 ased to a similar extent in the groups after n-3 fatty acid supplementation, and the D-series resolvi
273 solvins as well as of 14-HDHA in response to n-3 fatty acid supplementation.
274 individual effects of vitamin D and omega-3 (n-3) fatty acid supplementation on endometriosis-associa
275  change of serum metabolomics in response to n-3 fatty acid supplements in Chinese patients with type
276 mol per liter) more among patients receiving n-3 fatty acids than among those receiving placebo (P<0.
277 e had 2-fold higher levels of marine-derived n-3 fatty acids than whites and Japanese Americans in th
278     SDA-enriched soybean oil is a land-based n-3 fatty acid that is a sustainable approach to increas
279 te that DHA, but not EPA, is the most potent n-3 fatty acid that suppresses glomerulonephritis and ex
280 ional studies utilizing tissue biomarkers of n-3 fatty acids that more accurately measure dietary con
281   Omega-3 polyunsaturated fatty acids (PUFA, n-3 fatty acids), the key components of fish and flaxsee
282                                  Amongst the n-3 fatty acids, the concentrations of alpha-linolenic a
283  and other fatty acids, including long-chain n-3 fatty acids, the intake of alpha-linolenic acid was
284  studies fed unphysiologically high doses of n-3 fatty acids, these findings in rats must be consider
285 trate an anticancer (antimelanoma) effect of n-3 fatty acids through, at least in part, activation of
286 ake variations of omega-6 (n-6) and omega-3 (n-3) fatty acids ultimately determine cell membrane inco
287 r prevention, including lycopene, long-chain n-3 fatty acids, vitamin D, vitamin E, and selenium, wer
288 or DRI review, by which sodium, the omega-3 (n-3) fatty acids, vitamin E, and magnesium were identifi
289           Dietary consumption of seafood and n-3 fatty acids was annually assessed by a food frequenc
290           However, the allocation to receive n-3 fatty acids was positively associated with depressiv
291 percentage of energy from fat, the intake of n-3 fatty acids was significantly associated with fewer
292 tored hen eggs enriched with very long-chain n-3 fatty acids, was investigated.
293 ted seafood intake and estimated or measured n-3 fatty acids, whereas associations with self-reported
294  Meat products are generally low in omega-3 (n-3) fatty acids, which are beneficial to human health.
295 icant inverse associations of marine-derived n-3 fatty acids with IMT and CAC prevalence, respectivel
296 cle reports the results of the comparison of n-3 fatty acids with placebo.
297  is a lack of research on the association of n-3 fatty acids with risk of benign fibrocystic breast c
298  that dietary supplementation with selective n-3 fatty acids would be most beneficial for the treatme
299 ed that children with lower ratios of n-6 to n-3 fatty acids would perform better on tests of plannin
300 phospholipid measures of long-chain omega-3 (n-3) fatty acids would be positively associated with lar

 
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